81. IZOTÓP | Cikklista A Helizo-tesztkészlethez 14Curea breath analysis, a non-invasive test for campylobacter pylori in the campylobacter pylori detected noninvasively by the 13C-urea breath test; http://www.izotop.hu/radpha_h/pylori2.htm

Termékek Terméktámogatás Keresés Honlaptérkép ... IZOTÓP INTÉZET KFT. Kapcsolódó cikkek listája CÉGINFORMÁCIÓ Cégünkrõl Elérhetõségek ÜZLETÁGAK Radiogyógyszer Immunoassay Molekuláris biológia Szintézis ... Sugártechnika TERMÉKEK Teljes terméklista Új termékek Terméktámogató anyagok ÁRAJÁNLATKÉRÉS ... English A Helicobacter pylori fertõzés kimutatására szolgáló Helizo-tesztkészlethez Atherton JC, Spiller RC: The urea breath test for Helicobacter pylori; GUT 1994:35:723-725 Bell GD Weil J. In Helicobacter pylori and Gastroduodenal Disease; Ed. Rathbone BJ Heatlei RV. pp 74-87. Blackwell Scientific Publications, Oxford England Bell GD et al.: C-urea breath analysis, a non-invasive test for Campylobacter pylori in the stomach; Lancet 1987;1:1367-1368 Eggers R H et al.: A methodological analysis of the C-urea breath test for detection of Helicobacter pylori infection: high sensitivity and specificity within 30 min using 75 mg of Ferrero RL, Hazell SL, Lee A.: The urease enzymes of Campylobacter pylori and a related bacterium; J Med Microbiology 1988;27:33-40 Graham D Y et al.: Campylobacter pylori detected noninvasively by the

82. Jones Publications Optimization of detection of campylobacter pylori by urease and DNA hybridization. Gastroduodenal pathology and campylobacter pylori. (ed. http://www.medicine.uiowa.edu/microbiology/publications/jones.htm

Faculty Research Research Highlights Programs Graduate Undergrad Honors Summer Research ... Home This page was modified December 8, 2004 Webmaster, Julie Nealson Bradley D. Jones, Ph.D. bradley-jones@uiowa.edu List of Publications Peer-reviewed Journals Mobley, H.L.T., B.D. Jones and A.E. Jerse. 1986. Cloning of urease gene sequences from Providencia stuartii. Infect. Immun. 54:161 169. Jones, B.D. and H.L.T. Mobley. 1987. Genetic and biochemical diversity of ureases of Proteus, Providencia, and Morganella species isolated from urinary tract infection. Infect. Immun. 55:2198 2203. Mobley, H.L.T., B.D. Jones and J.L. Penner. 1987. Urease activity of Proteus penneri. J. Clin. Microbiol. 25:2302 2305. Mobley, H.L.T., M.J. Cortesia, L.E. Rosenthal and B.D. Jones. 1988. Characterization of urease from Campylobacter pylori. J. Clin. Microbiol. 26:831 836. Jones, B.D. and H.L.T. Mobley. 1988. Proteus mirabilis urease: genetic organization, regulation, and expression of structural genes. J. Bacteriol. 170:3342 3349. Mobley, H.L.T., L.E. Rosenthal, A.F. Trofa and

83. Tietopankki - Termi helikobakteeri helicobakteeri , helicobacter pylori , campylobacter pylori etenkin ruuansulatuskanavassa esiintyvä bakteerien ryhmä (mm. http://www.tohtori.fi/termi.php3?terID=2780&termit=campylobacter pylori

84. Publication List Antibody response to campylobacter pylori in diverse ethnic groups. Scand. J. Infect. Dis. The Prevalence of campylobacter pylori in Human Populations. http://www.microbiol.unimelb.edu.au/staff/mds/lab_pics/WeeTee2000/WTeePubs.html

Publications by Wee Tee Highest cited papers, ie. the number of times her papers have been cited by others (as at Oct 2004). This is a measure of how important/useful her studies have been found by other researchers. Citations Posthumous publications Over the 18 months to July 2001, I had the great pleasure of supervising Wee's five last manuscripts through to publication in leading medical journals. I don't consider myself to be a medical microbiologist (I work on extremely halophilic archaea!), and getting these manuscripts through the review process was challenging to say the least. Fortunately, her co-authors and colleagues (who are medical microbiologists) were extremely helpful in addressing the issues raised by reviewers and editors. Thanks. A special thanks to Dr Norbert Ryan (VIDRL). Amazingly, all five manuscripts were published (or accepted for publication) before the first anniversary of her death. 31. Tee, W., Jenney, A., McPhee, A., Mijch, A. and Dyall-Smith, M. 2001. " Helicobacter rappini " isolated from two homosexual males. Clin Inf Dis 33:e8-e11

85. IT SHOULD BE POSSIBLE TO ELIMINATE PEPTIC ULCER DISEASE AND GASTRIC CARCINOMA FR campylobacter pylori its link to gastritis and peptic ulcer disease. Eradication of campylobacter pylori and recurrence of duodenal ulcer. http://www.kfshrc.edu.sa/annals/143/ed93185.html

May 1994 IT SHOULD BE POSSIBLE TO ELIMINATE PEPTIC ULCER DISEASE AND GASTRIC CARCINOMA FROM SAUDI ARABIA Until recently, it would have been unthinkable to suggest that it might be possible to eliminate peptic ulcers and gastric carcinoma from the Kingdom of Saudi Arabia. This possibility now deserves serious consideration. This change in thinking is a direct result of recent studies linking the seemingly different diseases to an infection with a bacterium, Helicobacter pylori. H. pylori infection has been linked to gastric carcinoma, peptic ulcer disease, and gastric B-cell lymphoma.1-5 The tremendous morbidity and mortality from H. pylori infection are only now becoming appreciated. The cumulative risk of developing a peptic ulcer is 15% to 20% and 1% risk of developing gastric carcinoma. The risk of gastric lymphoma is unknown. Overall, the recognized rate of mortality or morbidity related to H. pylori infection is 20% to 30% of those infected. It has long been appreciated that chronic atrophic gastritis was a precursor lesion to gastric carcinoma and that, in areas of the world where atrophic gastritis was common in young adults, gastric cancer was frequent. Gastritis was also known to be almost universally present in patients with gastric or duodenal ulcer disease.2,6,7 Studies of the epidemiology of these gastritis-associated diseases demonstrated that unidentified environmental factor(s) were involved in development of gastritis and in progression from atrophic gastritis to gastric cancer. Despite a large number of studies and clues such as differences in prevalence of gastritis depending on socioeconomic class, ethnic group, age, implicating environmental factors, no common theme or factor emerged until approximately 10 years ago when

86. Actinobacillus Pleuropneumoniae Translate this page Campylobacter jejuni subsp. jejuni ? Campylobacter jejuni campylobacter pylori ? Helicobacter pylori Corynebacterium equi ? Rhodococcus equi http://niah.naro.affrc.go.jp/sat/joseki/Taxonomy/Taxonomy_change.htm

i —LŒø‹Û–¼Abˆã×‹ÛŠw‚ÉŠÖŒW‚·‚éŽå‚È‹ÛŽí‚Ì”²ˆj Actinobacillus pleuropneumoniae © Haemophilus pluropneumoniae Actinomyces pyogenes Corynebacterium pyogenes Actinomyces pyogenes ¨ Arcanobacterium pyogenes Actinomyces suis Eubacterium suis Arcanobacterium pyogenes © Actinomyces pyogenes Corynebacterium pyogenes Bacillus larvae ¨ Paenibacillus larvae subsp. larvae Bacteroides nodosus ¨ Dichelobacter nodosus Bergeyella zoochelcum © Weeksella zoohelcum Brachyspira hyodysenteriae © Serpulina hyodysenteriae Treponema hyodysenteriae Brachyspira innocens © Serpulina innocens Treponema innocens Brachyspira pilosicoli © Serpulina pilosicoli Brucella abortus ¨ Brucella melitensis biovar Abortus F ˆãŠwAbˆãŠw‚ł͍¬—‚ð”ð‚¯‚é‚½‚߁AŠe¶•¨Œ^‚ð]—ˆ‚Ì‹ÛŽí‚Æ‚µ‚Ĉµ‚¤ Brucella canis ¨ Brucella melitensis biovar Canis F ˆãŠwAbˆãŠw‚ł͍¬—‚ð”ð‚¯‚é‚½‚߁AŠe¶•¨Œ^‚ð]—ˆ‚Ì‹ÛŽí‚Æ‚µ‚Ĉµ‚¤ Brucella ovis ¨ Brucella melitensis biovar Ovis F ˆãŠwAbˆãŠw‚ł͍¬—‚ð”ð‚¯‚é‚½‚߁AŠe¶•¨Œ^‚ð]—ˆ‚Ì‹ÛŽí‚Æ‚µ‚Ĉµ‚¤ Brucella suis ¨ Brucella melitensis biovar Suis F ˆãŠwAbˆãŠw‚ł͍¬—‚ð”ð‚¯‚é‚½‚߁AŠe¶•¨Œ^‚ð]—ˆ‚Ì‹ÛŽí‚Æ‚µ‚Ĉµ‚¤ Burkholderia mallei © Pseudomonas malleii Burkholderia pseudomallei © Pseudomonas pseudomallei Campylobacter jejuni ¨

87. Bacteriology 330 Lecture Topics: Helicobacter PyloriBacteriology 3 C. pyloridis was renamed campylobacter pylori to fit in with the names of other enteric pathogens. In 1989, it was finally named Helicobacter pylori based http://www.bact.wisc.edu/Bact330/lecturehelico2

Bacteriology at UW-Madison Helicobacter pylori: An Emerging Pathogen by Karrie Holston, Department of Bacteriology University of Wisconsin-Madison Description of Helicobacter pylori Helicobacter pylori is a spiral-shaped, Gram-negative rod approximately 0.5 x 3.0 micrometers in size. The catalase-positive organism has 4-6 sheathed flagella attached to one pole which allow for motility. History Barry Marshall and Robin Warren of Perth, Western Australia, discovered H. pylori in 1983. Originally, the organism was named Campylobacter pyloridis because it was structurally similar to other Campylobacter species, such as C. jejuni C. jejuni is a gut pathogen which has the ability to colonize the gastric mucosa. C. pyloridis was renamed Campylobacter pylori to fit in with the names of other enteric pathogens. In 1989, it was finally named Helicobacter pylori based on functional and enzymatic properties. Linkage to Gastrointestinal Disease Studies have linked H. pylori to gastrointestinal disease in humans. Even though H. pylori

88. FORCES - SMOKING DOES NOT CAUSE ULCERS OR STOMACH CANCER Aktuelle Gastroenterologie, campylobacter pylori. campylobacter pylori, NSAIDs and smoking Risk factors for peptic ulcer disease. http://www.forces.org/evidence/carol/carol15.htm

Forces International Back to The evidence SMOKING DOES NOT CAUSE ULCERS OR STOMACH CANCER Back to main page GASTRIC ULCER: According to an editorial in the New England Journal of Medicine, "We now recognize three major causes of peptic ulcer disease: Helicobacter pylori infection, the use of nonsteroidal anti-inflammatory drugs (NSAIDs), and pathologic hypersecretory states such as the Zollinger-Ellison syndrome... H. pylori infection is the most common known cause of peptic ulcer and accounts for the majority of cases. NSAIDs are the second most common cause and are responsible for the majority of cases not caused by H. pylori infection." (DY Graham. Treatment of peptic ulcers caused by Helicobacter pylori. NEJM 1993;328(5):349-350). Out of 115 consecutive gastric ulcer patients, 61% were HP positive, including 30% who also used NSAIDs. Of 44 HP negatives, 66% used NSAIDs, 5% had malignant GUs, and 30% were of unknown cause. Only 11% of total GUs were of unknown cause. (TJ Borody, S Brandl et al. Helicobacter pylori negative gastric ulcer. Am J Gastroenterol 1992;87(10):1403-1406).

89. Revista De La Sociedad Venezolana De Microbiología - campylobacter pylori, duodenal ulcer and gastric http://www.scielo.org.ve/scielo.php?pid=S1315-25562002000100007&script=sci_artte

90. Editorial Thapa BR, Chhina RS, Ayyagri A, Malik AK, Mehta S. campylobacter pylori campylobacter pylori associated gastritis and peptic ulcer disease in children. http://www.indianpediatrics.net/march-275-283.htm

Home Past Issue About IP About IAP ... Subscription Personal Practice Indian Pediatrics 2000;37: 275-283 Helicobacter Pylori Infection in Children Ujjal Poddar B.R. Thapa Helicobacter pylori is a Gram-negative spiral bacterium found in association with the gastric epithelium. It stays on the surface of the cells and does not invade them. In 1983, Warren and Marshall(1) discovered this organism and it has fulfilled Koch's postulation as a cause of chronic active gastritis in human(2). Since its discovery it has generated much interest among medical scientists. There were 8996 publications till April 1999 (on MEDLINE search) and even whole sections of gastroenterology journals like Gut, Gastroenterology etc . are being devoted exclusively to H. pylori for quite some years now. It has made a place for itself in the mainstream of modern medical practice. Though the infection begins in childhood, the literature and research on H. pylori

91. !HELICOB campylobacter pylori in artificial contaminated milk. Clin Path 1989; 42 778 http://www.meb.uni-bonn.de/kinder/!HELICOB.html

Abteilung für allgemeine Pädiatrie und Poliklinik (Dir. Prof. Dr. Michael J. Lentze) am Zentrum für Kinderheilkunde der Universität Bonn Adenauerallee 119, D-53113 Bonn, Tel 49-228-2873213, Fax 49-228-2873314 KRANKENHAUS HYGIENE Erstellt von Dr. Arne Simon Hygienedatenblatt am Zentrum für Kinderheilkunde der Universität Bonn, Stand 03/98 erstellt von Dr. B.Utsch (Durchsicht PD Dr. K.M. Keller) Helicobacter pylori [Hp] Gramnegatives, spiralförmiges oder gebogenes Stäbchenbakterium. Ureasebildner. Meldepflicht: Keine. Krankheiten: Rezidivierende, chronische Bauchschmerzen (periumbilical oder epigastrisch), nicht-ulzeröse Dyspepsie, Gastritis, Ulcus duodeni, Ulcus ventrikuli, MALT-Lymphom (Wotherspoon 1993), Magenkarzinom (HP wurde von der WHO als karzinogen klassifiziert) Vorkommen, Infektiöses Material: ca 50% der Weltbevölkerung sind Hp-infiziert bzw. besiedelt (Graham 1991). Neuinfektionsrate pro Jahr zunehmendes Lebenalter ca. 1% (Veldhuyzen 1994). Magen, selten extragastral im Oesophagus, Meckel'schen Divertikel, Duodenum, Rektum. Außerdem: Mundschleimhaut, Zahnbelag, Speichel. Trinkwasser (Karim 1989), Nahrungsmittel (West 1990).

92. El Visick Inicial De Los Pacientes Estudiados Fue campylobacter pylori a las distintas http://www.encolombia.com/medicina/gastroenterologia/gastro16101invitado2.htm

Con respecto a la evaluación endoscópica en todos los pacientes a quienes se les había realizado cirugía resectiva, se observó un patrón compatible con gastritis de tipo alcalino acompañado con importante reflujo biliar; esto se encontró en los pacientes infectados o libres de infección y no se modificó con el tratamiento de erradicación. Aquellos pacientes que no se sometieron a cirugía resectiva (vagotomía) mostraron un patrón endoscópico normal o de gastropatía crónica, sin modificarse con la erradicación. El Visick inicial de los pacientes estudiados fue: Visick I, 24 pacientes (54,5%); Visick II, 17 pacientes (38,6%); Visick III, 3 pacientes (6,8%), y ningún paciente con Visick IV. La relación entre el Visick inicial y la infección por H. pylori se muestra en la Tabla 2. Tabla 2. Resultado de la detección de H. pylori por mediode biopsia (Bx) y prueba de ureasa (U). MÉTODO N Bx +U + Bx -U + Bx +U - Bx -U - Los pacientes con H. pylori negativo no registraron cambios en el patrón endoscópico, histológico ni clínico, durante el período de seguimiento. Comentario Actualmente, la cirugía ha sido relegada a la resolución de las complicaciones de la enfermedad ulcerosa gastroduodenal, debido al papel fundamental que juegan los inhibidores de la bomba de protones. Esto ha sido aún más manifiesto al conocerse la relación causal de la enfermedad con la infección por H. pylori, aceptándose que la erradicación del mismo es un pilar fundamental en su tratamiento.

93. Helicobacter Pylori And Peptic Ulcer Originally called campylobacter pyloridis, the name was changed to campylobacter pylori and then later to Helicobacter pylori as specific morphologic, http://www.jr2.ox.ac.uk/bandolier/bandopubs/hpyl/hpall.html

@import "../../styles/advanced.css"; Helicobacter Pylori and Peptic Ulcer A systematic review of effectiveness and an overview of the economic benefits of implementing what is known to be effective R A Moore MA DPhil Pain Research The Churchill Headington Oxford December 1994 Executive Summary Helicobacter pylori - background Introduction H pylori Evidence implicating H pylori in gastritis and peptic ulcer H pylori and ulcers How does H pylori cause gastritis and ulcers? Methods of diagnosing H pylori infection Invasive methods Culture Histology CLO test Non-invasive testing Breath testing Antibody measurement Quantitating antibody tests Antibody testing after H pylori eradication Prevalence UK prevalence Endoscopy studies Dyspepsia in seropositive and seronegative populations Incidence peptic ulcer bleeding Helicobacter pylori eradication and peptic ulcer Antibiotics and H pylori eradication H pylori eradication and relapse rates Effect of treatment on rebleeding rates Effect of eradication therapy on ulcer healing H pylori and dyspepsia Systematic review Conclusions H pylori and gastric cancer Background Gastric cancer and H pylori Epidemiological considerations Clinical guidelines on H pylori NIH Consensus Conference Endoscopy and H pylori testing Economic consequences of H pylori testing and eradication

94. Carbon-14 Urea Breath Test For The Diagnosis Of Campylobacter Pylori Associated Carbon14 urea breath test for the diagnosis of campylobacter pylori associated gastritis. BJ Marshall and I Surveyor Australian National Health and Medical http://jnm.snmjournals.org/cgi/content/abstract/29/1/11

HOME HELP FEEDBACK SUBSCRIPTIONS ... TABLE OF CONTENTS QUICK SEARCH: [advanced] Author: Keyword(s): Year: Vol: Page: This Article Alert me when this article is cited Alert me if a correction is posted Services Email this article to a friend Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal ... Cited by other online articles PubMed PubMed Citation Articles by Marshall, B. J. Articles by Surveyor, I. JOURNAL ARTICLE Carbon-14 urea breath test for the diagnosis of Campylobacter pylori associated gastritis BJ Marshall and I Surveyor Australian National Health and Medical Research Council, Royal Perth Hospital. Urease in the human gastric mucosa is a marker for infection with Campylobacter pylori (CP), an organism suspected of causing chronic gastritis and peptic ulceration. To detect gastric urease, we examined 32 patients who were being evaluated for possible peptic ulcer disease. Fasting patients were given 10 microCi (370 kBq) of 14C-labeled urea. Breath samples were collected in hyamine at intervals between 1 and 30 min.

95. Published Articles On Helicobacter Pylori( CGC) campylobacter pylori as a Major Determinant in Hypothetical Carcinogenesis of A Prospective Study on the campylobacter pylori Isolated from Patients of http://kid.gsnu.ac.kr/gastritis/helico1.html

Site for Published Articles on Helicobacter pylori (with English abstract) Prevalence of Campylobacter pylori in Normal Korean Person Campylobacter pylori ... E-mail

96. Chin Med J (Taipei) 1997;59:171-6. ( Bacteriological and pharmacological basis for treatment of campylobacter pylori infection. In campylobacter pylori and gastroduedenal disease. http://www.vghtpe.gov.tw/~cmj/5903/590305.htm

Helicobacter pylori Treatment with Clarithromycin) Next Prev Abs Chi ... Home Chin Med J (Taipei) 1997;59:171-6. Clarithromycin in the Combination Therapy for the Eradication of Helicobacter pylori in Peptic Ulcer Disease Wen-Ching Lo, Hwai-Jeng Lin, Kun Wang, Chin-Lin Perng, Shou-Dong Lee Division of Gastroenterology, Department of Medicine, Veterans General Hospital-Taipei, and National Yang-Ming University School of Medicine, Taipei, Taiwan, R.O.C. Abstract Background. Clarithromycin is a new macrolide antibiotic which is known to be highly effective in eradicating Helicobacter pylori (H. pylori). In Chinese, the role of clarithromycin for H. pylori is still unclear. Methods. Between January 1995 and February 1996, 75 patients with active H. pylori-positive duodenal ulcer were enrolled in this study. Three groups were randomized to have (1) 2x150 mg nizatidine twice daily, 2x250 mg amoxicillin four times daily, and 2x250 mg clarithromycin three times daily for two weeks (niz-amox-clar group, N=25); or (2) 20 mg omeprazole twice daily plus 2x250 mg clarithromycin three times daily for two weeks (ome-clar group, N=25); or (3) 300 mg bismuth subsalicylate four times daily, and 2x250 mg amoxicillin four times daily, 250 mg metronidazole four times daily for two weeks (triple therapy group, N=25). All the patients received H2 receptor antagonist (150 mg nizatidine or ranitidine, or 400 mg cimetidine, twice daily) for the consecutive six weeks. Results.

97. Infezione Da Helicobacter Pylori Translate this page Eaton KA, Morgan DR, Krakowka S campylobacter pylori virulence factors in Paull G, Yardley JH Gastric and esophageal campylobacter pylori in patients http://www.benessere.com/salute/arg00/helicobacter.htm

INFEZIONE DA HELICOBACTER PYLORI A cura del dott. G. Salvemini Cenni storici Nel 1892 batteri spiraliformi nello stomaco di animali furono mostrati per la prima volta da Giulio Bizzozero, patologo dell'Università di Torino. Nel 1983 Robin Warren, patologo, e Barry Marshall, giovane specializzando in Medicina interna, riuscirono a coltivare il microorganismo da campioni bioptici di mucosa gastrica. Nel corso degli anni il batterio è stato diversamente denominato, da Campylobacter pyloridis all'attuale Helicobacter pilori Hp ), e la sua presenza è stata associata a diverse patologie. Microbiologia L' Hp è un batterio Gram-negativo, spiraliforme, microaerofilo, a forma di "ali di gabbiano". E' mobile per la presenza di flagelli di superficie localizzati sull'estremità polare; infatti, è caratterizzato da movimenti "a cavaturaccioli". Il microorganismo possiede fattori di colonizzazione come l'ureasi e fattori tossici come il lipopolisaccaride, la "Heat Shock Protein" (HSP), il "CagA" ed il "VacA". Epidemiologia L'incidenza dell'infezione da Hp è andata sempre più riducendosi con lo sviluppo industriale: infatti, nei Paesi sviluppati, soggetti di elevato stato socio-economico hanno percentuali più basse di infezione e l'infezione rimane comune in quasi tutti i Paesi in via di sviluppo.

98. Campylobacter-pylori-Antikörper Translate this page campylobacter-pylori-Antikörper. Siehe. bullet, Helicobacter-pylori-Antikörper. Haftungsausschluß wichtiger Hinweis. falls Sie diese Seite über eine http://www.laborlexikon.de/Lexikon/Infoframe/c/Campylobacter-pylori-Antikoerper.

Siehe: Haftungsausschluß: wichtiger Hinweis falls Sie diese Seite über eine Suchmaschine gefunden haben: Link zur Startseite (Impressum)

99. Atatürk Üniversitesi Tip Fakültesi Ana Sayfa Yönetim Tarihçe Helicobacter pylori campylobacter jejuni - Antimikrobiyal direnç. Bilimsel Yayinlari. ULUSLARARASI DERGILERDE - AKTAS O, Calik, Z. In vitro activity of http://tip.atauni.edu.tr/ogretim_uye.asp?u_id=825

100. HON Mother & Child Glossary, Bacterial Infections In Childhood: H. Pylori Helicobacter pylori was originally classified in the genus campylobacter, but RNA sequencing, cellular fatty acid profiles, growth patterns, http://www.hon.ch/Dossier/MotherChild/child_bacteria/bacteria_pylori.html

Introduction Reproduction Pregnancy During Pregnancy ... Glossary A-Z Childhood Illness Bacteria Virus Cancer Gastrointestinal ... Mental Health Bacterial Infections in Childhood: H. Pylori Description Helicobacter pylori : A spiral bacterium active as a human gastric pathogen. It is a gram-negative, urease-positive, curved or slightly spiral organism initially isolated in 1982 from patients with lesions of gastritis or peptic ulcers in Western Australia. Helicobacter pylori was originally classified in the genus CAMPYLOBACTER, but RNA sequencing, cellular fatty acid profiles, growth patterns, and other taxonomic characteristics indicate that the micro-organism should be included in the genus HELICOBACTER. It has been officially transferred to Helicobacter gen. nov. (see Int J Syst Bacteriol 1989 Oct;39(4):297-405). [1] Peptic Ulcer : Ulcer that occurs in those portions of the alimentary tract which come into contact with gastric juice containing pepsin and acid. It occurs when the amount of acid and pepsin is sufficient to overcome the gastric mucosal barrier. [1] Other HON resources From MedHunt (websites) Helicobacter Pylori From HONselect Helicobacter pylori (www.cellsalive.com)

A  B  C  D  E  F  G  H  I  J  K  L  M  N  O  P  Q  R  S  T  U  V  W  X  Y  Z

Page 5     81-100 of 100    Back | 1  | 2  | 3  | 4  | 5