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         Campylobacter Pylori:     more detail
  1. Campylobacter Pylori & Gastroduodenal Disease by Rathbone, 1989-08
  2. Campylobacter Pylori in Gastritis and Peptic Ulcer Disease
  3. Campylobacter Pylori: Proceedings by H. Menge, M. Gregor, et all 1988-08
  4. Gastroduodenal Pathology and Campylobacter Pylori (International congress series)
  5. Helicobacter Pylori, Gastritis and Peptic Ulcer by P. Malfertheiner, Germany) European Campylobacter Pylori Study Group Meeting 1989 (Ulm, 1990-10
  6. Campylobacter pylori: Proceedings of the First International Symposium on Campylobacter Pylori, Kronberg, June 12-13th, 1987
  7. Epsilonproteobacteria: Helicobacter Pylori, Campylobacter (French Edition)
  8. Aktuelle Gastroenterologie - Campylobacter pylori (German Edition)
  9. Campylobacter pylori by Rauws and Tytgat, 1989
  10. Campylobacter Pylori in Gastritis and Peptic Ulcer Disease
  11. Proteobacteria: Thiomargarita Namibiensis, Enterobacteriaceae, Bdellovibrio, Campylobacter, Neisseria Gonorrhoeae, Helicobacter Pylori
  12. Comparison of Helicobacter pylori colonization on the tonsillar surface versus tonsillar core tissue as determined by the CLO test.(Campylobacter-like ... article from: Ear, Nose and Throat Journal by Bijan Khademi, Nika Niknejad, et all 2007-08-01
  13. Helicobacter Pylori 1990: Proceedings of the Second International Symposium on Helicobacter Pylori Bad Nauheim, August 25 26th, 1989 by H. Menge, Gregor M., et all 1991-07
  14. Molecular Mechanisms of Bacterial Infection via the Gut (Current Topics in Microbiology and Immunology)

21. Biomerica, Inc.: EZ-HP, One-Step Helicobacter Pylori Test
In 1983, Helicobacter pylori (previously known as campylobacter pylori) was found campylobacter pylori and histological gastritis in duodenal ulcer A
http://biomerica.com/html/ezhp.html
home the company financial info news/press ... contact us EZ-HP One-Step Helicobacter pylori Test
Detection of IgG Antibodies to H.pylori in Whole Blood
Overview
Summary and Explanation

Principle of the Test

Materials Provided
...
References

Overview
Simple Flexible Sensitive Quick Reliable Saves time, money
Summary and Explanation
In 1983, Helicobacter pylori (previously known as Campylobacter pylori) was found in close contact with gastric epithelium in biopsy samples showing active type B or chronic gastritis (6,7,8,9,10). Although the source of H. pylori infection is unknown, reports (3,12,14) have shown quite convincingly that H. pylori infection can be associated with chronic gastritis. A positive correlation has been found between the presence of H. pylori and gastric lesions in some cases of duodenal ulcers (15,16). Complete resolution of gastritis after eradication of the organism has also been reported (17,18). H. pylori is a gram negative, curved, spiral shaped rod bacteria (0.2 - 0.8 µm in width by 0.5 - 5.0 µm in length). H. pylori colonization is found in the deep portions of the mucous layer that coats the gastric mucosa, and between the mucous layer and apical surface of the gastric mucosal epithelial cells in some infected patients (1). H. pylori may also be located in the regions of junctions between adjacent mucosal epithelial cells. It produces three enzymes in large amounts; urease, superoxide dismutase and catalases (14,17,18). Urease splits urea to produce ammonia, which provides the conditions needed for the multiplication and sustenance of the organism in the gastric environment. Colonization may induce the host's local and systemic immune response, and may cause clinical signs and symptoms including neutrophilic infiltration and the production of specific antibodies.

22. NEJM -- Association Of Campylobacter Pylori On The Gastric Mucosa With Antral Ga
Original Article from The New England Journal of Medicine Association of campylobacter pylori on the gastric mucosa with antral gastritis in children.
http://content.nejm.org/cgi/content/short/316/25/1557
HOME SEARCH CURRENT ISSUE PAST ISSUES ... HELP Please sign in for full text and personal services Volume 316:1557-1561 June 18, 1987 Number 25 Next Association of Campylobacter pylori on the gastric mucosa with antral gastritis in children
B Drumm, P Sherman, E Cutz, and M Karmali
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This article has been cited by other articles:
  • Kato, S., Sherman, P. M. (2005). What Is New Related to Helicobacter pylori Infection in Children and Teenagers?. Arch Pediatr Adolesc Med [Abstract] [Full Text]
  • Clyne, M., Drumm, B. (2004). Helicobacter pylori Infection of Human and Murine Primary Gastric Cells. Infect. Immun. [Abstract] [Full Text]
  • Straubinger, R. K., Greiter, A., McDonough, S. P., Gerold, A., Scanziani, E., Soldati, S., Dailidiene, D., Dailide, G., Berg, D. E., Simpson, K. W. (2003). Quantitative Evaluation of Inflammatory and Immune Responses in the Early Stages of Chronic Helicobacter pylori Infection. Infect. Immun.

23. NEJM -- Sign In
Inhibitory antimicrobial concentrations aganst campylobacter pylori in gastric mucosa. J Antimicrob Chemother 1988;22729738.
http://content.nejm.org/cgi/content/full/329/1/59

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24. Medmicro Chapter 23
Helicobacter pylori (formerly known as campylobacter pylori), All Campylobacter species except H pylori are similar in structure and appearance.
http://gsbs.utmb.edu/microbook/ch023.htm
Campylobacter and Helicobacter
Guillermo I. Perez-Perez
Martin J. Blaser
General Concepts
Campylobacter Jejuni and other Enteric Campylobacters
Clinical Manifestations
Campylobacter species cause acute gastroenteritis with diarrhea, abdominal pain, fever, nausea, and vomiting. Recently, Campylobacter infections have been identified as the most common antecedent to an acute neurological disease, the Guillain-Barré syndrome.
Structure
Campylobacter species are Gram-negative, microaerophilic, non-fermenting, motile rods with a single polar flagellum; they are oxidase-positive and grow optimally at 37° or 42°C.
Classification and Antigenic Types
Campylobacter species have many serogroups, based on lipopolysaccharide (O) and protein (H) antigens. However, only a few serogroups account for most human isolates in a given geographic region. C jejuni possesses several common surface-exposed antigens, including porin protein and flagellin.
Pathogenesis
The bacteria colonize the small and large intestines, causing inflammatory diarrhea with fever. Stools contain leukocytes and blood. The role of toxins in pathogenesis is unclear. C jejuni antigens that cross-react with one or more neural structures may be responsible for triggering the Guillian-Barre syndrome.

25. Arch Intern Med -- Abstract: Gastritis And Campylobacter Pylori In Healthy, Asym
Gastritis and campylobacter pylori in healthy, asymptomatic volunteers. JS Barthel, TU Westblom, AD Havey, F. Gonzalez and ED Everett
http://archinte.ama-assn.org/cgi/content/abstract/148/5/1149
Select Journal or Resource JAMA Archives of Dermatology Facial Plastic Surgery Family Medicine (1992-2000) General Psychiatry Internal Medicine Neurology Ophthalmology Surgery Student JAMA (1998-2004) JAMA CareerNet For The Media Meetings Peer Review Congress
Vol. 148 No. 5, May 1, 1988 Featured Link E-mail Alerts ARTICLE Article Options Send to a Friend Similar articles in this journal Literature Track Add to File Drawer Download to Citation Manager PubMed citation Articles in PubMed by Barthel JS Everett ED Articles that cite this article Contact me when this article is cited
Gastritis and Campylobacter pylori in healthy, asymptomatic volunteers
J. S. Barthel, T. U. Westblom, A. D. Havey, F. Gonzalez and E. D. Everett
University of Missouri School of Medicine, Columbia. A high prevalence of histologic gastritis in asymptomatic individuals has been reported in the literature. The studies have been poorly controlled for gastritis risk factors. We evaluated 20 healthy, asymptomatic volunteers free of known risk factors for gastritis with endoscopy, mucosal biopsy, culture, and gastric pH determination. The prevalence of gastritis

26. Arch Intern Med -- Abstract: Prevalence Of Campylobacter Pylori In Esophagitis,
campylobacter pylori was detected in 14 (70%) of 20 patients with chronic gastritis, in 8 (67%) of 12 patients with endoscopically documented duodenal
http://archinte.ama-assn.org/cgi/content/abstract/149/6/1373
Select Journal or Resource JAMA Archives of Dermatology Facial Plastic Surgery Family Medicine (1992-2000) General Psychiatry Internal Medicine Neurology Ophthalmology Surgery Student JAMA (1998-2004) JAMA CareerNet For The Media Meetings Peer Review Congress
Vol. 149 No. 6, June 1, 1989 Featured Link E-mail Alerts ARTICLE Article Options Send to a Friend Similar articles in this journal Literature Track Add to File Drawer Download to Citation Manager PubMed citation Articles in PubMed by Cheng EH Kawanishi H Articles that cite this article Contact me when this article is cited
Prevalence of Campylobacter pylori in esophagitis, gastritis, and duodenal disease
E. H. Cheng, P. Bermanski, M. Silversmith, P. Valenstein and H. Kawanishi
Department of Medicine, State University of New York, Stony Brook. The relationship between the presence of Campylobacter pylori and esophagitis was studied in patients undergoing paired biopsies of distal esophagus and gastric antrum during esophagogastroduodenoscopy. Biopsy specimens were examined for urease activity and for the presence of C pylori by culture and by histologic examination of hematoxylin-eosin- and Warthin-Starry-stained sections. Sixty-two patients were entered into the

27. Entrez PubMed
Comparison of partial 16S rRNA sequences from representative Campylobacter species indicates that th
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=3

28. Entrez PubMed
Antibody response to campylobacter pylori in an ethnic group lacking peptic ulceration. Dwyer B, Sun NX, Kaldor J, Tee W, Lambert J, Luppino M, Flannery G.
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=3

29. Helicobacter Pylori J99 (Campylobacter Pylori J99) Complete Proteome Set
Complete proteome of Helicobacter pylori J99 (campylobacter pylori J99). The complete nonredundant proteome of Helicobacter pylori J99 (Campylobacter
http://www.ebi.ac.uk/proteome/pre_integr8/HELPJ/download.html
Complete proteome of Helicobacter pylori J99 (Campylobacter pylori J99) The complete non-redundant proteome of Helicobacter pylori J99 (Campylobacter pylori J99) comprises:
Genome component EMBL accession number Genome Reviews Number of proteins encoded Proteome Sets Chromosome Tables Helicobacter pylori J99 (Campylobacter pylori J99) Chromosome AE001439_GR.dat Swiss-Prot format FASTA format HTML format ... Text format Interpro Matches Click here to download the location of all InterPro matches for this proteome (tab-delineated text format) Note that for proteomes whose entries have largely not yet been promoted into UniProt from TrEMBLnew, the files of Interpro hits are liable to be very incomplete. See the proteome source page to find out the current status of this proteome Click here for further help or information concerning the complete proteome sets.
Click here for help concerning the chromosome tables.
Click here for help concerning Genome Reviews.
The UniProt non-redundant proteome sets are made using proteins taken from the UniProt Knowledgebase. Click here for more information about UniProt Last updated on Thu Jun 24 14:20:59 BST 2004 by Paul Kersey Back one page or Proteome Analysis homepage or EBI homepage

30. Disclaimer - Electronic Collection
eLetters Campylobacter vs. Helicobacter? for which therapy is indicated (eg campylobacter pylori). Did Dr. Farquhar mean Campylobacter jejuni?
http://collection.nlc-bnc.ca/100/201/300/cdn_medical_association/cmaj/vol-163/is
You are viewing a document archived by Library and Archives Canada. Please note, information may be out of date and some functionality lost. You will be redirected in 15 seconds or follow this link to continue to the document For more information see the Electronic Collection's Web site. Vous visualisez un document archivé par Bibliothèque et Archives Canada. Veuillez noter que l’information peut être périmée et que certaines fonctions peuvent être inactives. Vous serez redirigé dans 15 secondes ou suivez ce lien pour voir le document Pour plus de renseignements veuillez consulter le site Web de la Collection électronique Last Updated: 2005-04-12 Important Notices Avis importants

31. Campylobacter Pylori - Definition From Biology-Online.org
Definition and other additional information on campylobacter pylori from BiologyOnline.org dictionary.
http://www.biology-online.org/dictionary/campylobacter_pylori

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helicobacter pylori
A bacterium that has been implicated in the development of duodenal and gastric ulcers Many gastroenterologists now advocate treating with antibiotic regimens to eradicate this microorganism in the upper gastrointestinal tract of those who suffer from recurrent or intractable peptic ulcer disease Combinations of bismuth metronidazole and ampicillin (or tetracycline ) have been used successfully. Others advocate the use of omeprazole in combination with amoxicillin (or clarithromycin
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32. Role Of Helicobacter Pylori In The Genesis Of Gastric Ulcerations Among Smokers
H. pylori, formerly known as campylobacter pylori, are Gramnegative Diagnosis of campylobacter pylori infections the gold standard and the
http://www.emro.who.int/Publications/EMHJ/0302/16.htm
Eastern Mediterranean Health Journal Back to Health Journal page Health Journal back issues Home
Role of Helicobacter pylori in the genesis of gastric ulcerations among smokers and nonsmokers Volume 3, Issue 2, 1997, Page 316-321 M.A. El-Barrawy, M.I. Morad and M. Gaber ABSTRACT Helicobacter pylori infection was investigated by three different methods (serologically, microbiologically and biochemically) in 210 male patients aged 20-40 years. Gastric ulcers were diagnosed in 126 patients in which H. pylori infection showed a significant association (c = 162.8). Smokers had a higher rate of infection than nonsmokers, both among patients with gastric ulcers (99% compared to 79%) and without. The risk of H. pylori infection was computed for different categories of smoker. Heavy cigarette smokers were found to have the highest risk, followed by moderate to mild cigarette smokers and communal shisha smokers; neither private shisha nor cigar/pipe smokers showed any significant risk. Rôle d' Helicobacter pylori dans la genèse des ulcérations gastriques chez les fumeurs et les non-fumeurs.

33. Bacteriology Pages 1 2 3 4 5 6 7 8 9 10 11 12 13 14 15 16 17 18
Campylobacter jejuni, ÇáÚóØíÝóÉõ ÇáÕøóÇÆöãöíøóÉ º. campylobacter pylori, ÇáÚóØíÝóÉõ ÇáÈóæøóÇÈöíøóÉ º. Campylobacter sputorum, ÇáÚóØíÝóÉõ ÇáÈóáúÛóãöíøóÉ º
http://www.emro.who.int/umd/browsingsubj.asp?PageNo=6&subj=203

34. Histologic Characteristics Of Campylobacter Pylori (Helicobacter Pylori) Mediate
A positive specimen for campylobacter pylori (CP+), (Helicobacter pylori) was defined as one in which curved or spiral shaped microbes were identified on
http://www.annclinlabsci.org/cgi/content/abstract/20/5/329
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Histologic characteristics of Campylobacter pylori (Helicobacter pylori) mediated gastritis
E Madan, J Kemp, TU Westblom, J Chaffin, and AM Foster
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35. The Australian Institute Of Political Science
ulcer relapse after eradication of campylobacter pylori, Lancet 2 14371442 of campylobacter pylori associated gastritis, J Nucl Med 29 11-16
http://www.aips.net.au/tallpoppies/floreymedal/winner1998.htm
Mayne Florey Medal 1998 Award
For the discovery of the Helicobacter Pylori
the causative agent in Gastritis and Gastric Ulceration
The winners of the inaugural Florey Medal, awarded in 1998, were Dr Robin Warren of the Department of Pathology, Royal Perth Hospital, Western Australia, and Professor Barry Marshall of the Department of Medicine, University of Western Australia. The nominated achievement for which these scientists won the Medal was their identification of a new bacterium, Helicobacter pylori , and their discovery that this bacterium was the cause of both gastritis (inflammation of the stomach) and of gastric ulcer. These discoveries have had a profound effect on both the management of gastroduodenal disease, and the clinical practice of gastroenterology. What Warren and Marshall did, in chronological order,was to:

36. DSMZ - Helicobacter Pylori
DSMZ List of Microbial Species Helicobacter pylori (Bacteria) Synonym, campylobacter pylori. Restrictions, Risk Group 2, restricted distribution (A),
http://www.dsmz.de/species/sp200395.htm
DSMZ - List of Microbial Species: Helicobacter pylori (Bacteria) Name Helicobacter pylori (Marshall et al. 1986) Goodwin et al. 1989 VP
- see also Bacterial Nomenclature Up-to-Date Synonym Campylobacter pylori Restrictions Risk Group 2, restricted distribution (A) , see also shipping costs Strains DSMZ Microorganisms

37. CAMPYLOBACTER
Arcobacter nitrofigilis Name campylobacter pylori Authors (Marshall et al. pylori Name campylobacter pylori subsp. mustelae Authors Fox et al.
http://www.dsmz.de/bactnom/nam0665.htm
Genus CAMPYLOBACTER - Bacterial Nomenclature Up-to-Date Name: CAMPYLOBACTER Authors: Sebald and Veron 1963 emend. Vandamme et al. 1991 Status: Approved Lists Type species: C. fetus Literature: Int. J. Syst. Bacteriol. 30:270 (AL); Name: Campylobacter butzleri Authors: Kiehlbauch et al. 1991 Status: Basonym Literature: Int. J. Syst. Bacteriol. 41:580 (validation list); Type strain: ATCC 49616, D2686, DSM 8739 , LMG 10828 Synonym: Arcobacter butzleri Name: Campylobacter cinaedi Authors: Totten et al. 1988 Status: Basonym Literature: Int. J. Syst. Bacteriol. 38:328 (validation list); Type strain: 165, CCUG 18818, DSM 5359 Synonym: Helicobacter cinaedi Name: Campylobacter coli Authors: (Doyle 1948) Veron and Chatelain 1973 Status: Approved Lists Literature: Int. J. Syst. Bacteriol. 30:271 (AL); Risk group: 2 (German classification) Type strain: ATCC 33559, CIP 7080, DSM 4689 , NCTC 11366 Synonyms: Campylobacter hyoilei (heterotypic synonym) Name: Campylobacter concisus Authors: Tanner et al. 1981 Status: New Species Literature: Int. J. Syst. Bacteriol. 31:442;

38. Spiral Bacteria In The Human Stomach
Cytotoxin production by campylobacter pylori strains isolated from patients with Establishment of gastric campylobacter pylori infection in the neonatal
http://www.cdc.gov/ncidod/eid/vol1no3/dubois.htm
EID Volume 1 * Number 3 July-September 1995 Synopses
Spiral Bacteria in the Human Stomach: The Gastric Helicobacters
Andre Dubois, M.D., Ph.D. Digestive Diseases Division, Department of Medicine,
Uniformed Services University of the Health Sciences Bethesda, Maryland, USA Download Article During the past decade, Helicobacter pylori has become recognized as one of the most common human pathogens, colonizing the gastric mucosa of almost all persons exposed to poor hygienic conditions from childhood. It also is often found, albeit with a lower frequency, in groups of high socioeconomic status. H. pylori causes chronic active gastritis and is a major factor in the pathogenesis of duodenal ulcers and, to a lesser extent, gastric ulcers. In addition, the presence of this bacterium is now recognized as a risk factor for gastric adenocarcinoma and lymphoma. Nevertheless, most infections appear without clinical consequences. In this second decade of intensive research, it is important to understand why H. pylori

39. CPath::Search Results
UREB Helicobacter pylori (campylobacter pylori). 2. Interaction. Interactor, Organism, Experimental Evidence, Database Source
http://cbio.mskcc.org/cpath/webservice.do?version=1.0&cmd=get_by_keyword&q=&form

40. References: H. Pylori
campylobacter pylori and its role in peptic ulcer disease. Gastroenterolo Clin North Am. 19183, 1990. Graham, D. Helicobacter pylori Infection in the
http://www.mcphu.edu/continuing/cme/medicine/h-pylori/referenc.htm

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REFERENCES
  • Bizzozero B. Beziehunger ihres Epithel za dem Oberfachenepithel der Schleimhart. Arch f Mikr Anat 23:82-152, 1893.
    Palme r ED. Investigation of the gastric mucosa spirochetes of the human. Gastroenterology 27:218-220, 1954.
    Steer HW, Colin-Jones DG. Mucosal changes in gastric ulceration and their response to carbenoxolone sodium. Gut 16:590-597, 1975.
    Marshall BJ, Armstrong JA, McGechie DB, Glancy RJ. Attempt to fulfil Koch's postulates for pyloric Campylobacter. Med J Aust 142:436-439, 1985.
    Morris A, Nicholson G. Ingestion of Campylobacter pyloridis causes gastritis and raised fasting pH. Am J Gastroetnerol 82:191-199, 1987.
    Handt LK, Fox JG, Stalis IH, Rufo R, Lee T, Linn J, LiX, Kleanthous H. Characterization of feline Helicobacter pylori strains and associated gastritisi in a colony of domestic cats. J Clin Microbiol 33:2280-2289, 1995.
    Cave DR. How is Helicobacter pylori transmitted? Gastroenterology 113:S9-S14, 1997.
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