Karger Publishers portalsystemic encephalopathy (PSE) results from portal-systemic shunting ofvenous blood that arises either spontaneously or due to surgical portacaval http://content.karger.com/ProdukteDB/produkte.asp?Doi=51414
Extractions: HOME HELP FEEDBACK SUBSCRIPTIONS ... TABLE OF CONTENTS This Article Full Text Full Text (PDF) Alert me when this article is cited ... Alert me if a correction is posted Services Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal Download to citation manager PubMed PubMed Citation Articles by Wang, M. Q. Articles by Gao, Y. A. Journal of Vascular and Interventional Radiology Mao Qiang Wang, MD Michael D. Dake, MD Zhi Peng Cui, MD Zhi Qiang Wang, MD and Yu Ao Gao, MD From the Division of Cardiovascular and Interventional Radiology (M.Q.W., M.D.D.), Stanford University Medical Center, Stanford, California, and Departments of Diagnostic Radiology (M.Q.W., Z.P.C., Y.A.G.) and Gastroenterology (Z.Q.W.), Chinese PLA General Hospital, Beijing, China. Received June 14, 1999; revision requested July 17; final revision received February 5, 2001; accepted February 6. Address correspondence to M.D.D., Division of Cardiovascular and Interventional Radiology, Stanford University Medical Center, Rm H-3647, 300 Pasteur Dr., Stanford, CA 94305; E-mail: mddake@ leland.stanford.edu
Product Abstract:Branch Chain Amino Acids The value of the portalsystemic encephalopathy index was chosen as main outcome,because of lack of more significant clinical outcomes. http://www.lef.org/prod_hp/abstracts/php-ab224.html
Extractions: var WebSiteBaseURL = "http://www.lef.org" var ThisPageFullURL = "http://www.lef.org/prod_hp/abstracts/php-ab224.html" translation by SYSTRAN MEMBERSHIP PRODUCTS MAGAZINE ... CHECKOUT Oral supplementation with branched-chain amino acids improves transthyretin turnover in rats with carbon tetrachloride-induced liver cirrhosis Overview of randomized clinical trials of oral branched-chain amino acid treatment in chronic hepatic encephalopathy Leucine metabolism in rats with cirrhosis. Nutrient-induced thermogenesis and protein-sparing effect by rapid infusion of a branched chain-enriched amino acid solution to cirrhotic patients. ... Alanylglutamine-enriched total parenteral nutrition improves protein metabolism more than branched chain amino acid-enriched total parenteral nutrition in protracted peritonitis
FIRSTConsult - Sdfdsf Hepatic encephalopathy Medical Condition File. portal-systemic encephalopathy *Hepatic coma. Human immunodeficiency virus (HIV) test. http://www.firstconsult.com/?action=view_article&id=1014781&type=101&bref=1
FIRSTConsult - Sdfdsf of patients with cirrhosis and chronic portalsystemic encephalopathy, and neomycin in the treatment of chronic portal-systemic encephalopathy. http://www.firstconsult.com/?action=view_article&id=1014768&type=101&bref=1
Hepatitis C (Steve Harris, M.D.; David Rind) In portalsystemic encephalopathy resulting from c hronic liver failure, astrocytesmanifest altered expression of several key proteins and enzymes http://yarchive.net/med/hepatitis_c.html
Extractions: Index Home About Search for Google's copy of this article Index Home About From: sbharris@ix.netcom.com(Steven B. Harris) Subject: Re: HELP FOR CIRRHOSIS (Hepatitis C) Date: 30 Sep 1996 Newsgroups: alt.health.oxygen-therapy,alt.health,misc.health.alternative,misc.health,own.health.herbs,sci.med On Wed, 25 Sep 1996 21:52:38, JPear@gnn.com (John Pearson) wrote: >>A close friend of mine was recently diagnosed with hepatitis and >>cirrhosis of the liver, and only has about 20% usage of his liver. >>His doctor says he is too far gone to do a liver transplant, but will >>give him interferon. He does not drink. There is no such thing as a person "too far gone" for a liver transplant. That's ridiculous. People have been saved by liver transplants when they are so far gone in hepatic coma that some of their doctors thought they were brain dead (in one famous case, a "brain-dead" man presented as a potential heart donor, ended up being a liver *recipient*, and did well). If your friend is getting interferon, he's probably got hepatitis C. A couple of antivirals also seem to work for hepatitis C, though neither is used routinely in the US. One is ribavirin (Virazole), which is used with interferon in England, and has a number of studies behind it. Another is amantidine, which is used in the US against influenza A, but also has activity against hep C. In a preliminary study done by Jill P. Smith at Hershey medical center in Pennsylvania was presented in May at the Digestive Disease Week Conferences in San Francisco, amantidine seems to be worth looking into. In this study, 8 out of 20 patients who completed the study had a partial response to 6 months of amantidine, and another 6 had complete response (liver enzymes normalized). This is as good as the results of interferon, and at about 4% of the price. Neither amantidine or ribivirin is very toxic at the doses used for hep C (1.2 grams/d for ribivirin, and 200 mg/d for amantidine). Ribivirin can be bought as the drug "Vilona" in Mexico and brought across the border quite legally. Amantidine is a standard pharmaceutical available anywhere in the US on prescription. If I had hep C and was a layman, I'd be looking for a doctor willing to follow my liver enzymes with me as I experimented with both medications, singly and in combination, PLUS interferon alfa-2b. Viral load tests for hep C are also now available, and can probably help customize treatment with antivirals, in much the same way they are doing for AIDS/HIV patients. Specialist help is needed for this, and also because interferon has side effects which can be managed with other drugs. FIND A DOCTOR WHOSE PRACTICE IS MAINLY TREATING CHRONIC INFECTIOUS HEPATITIS PATIENTS. These exist. Good luck. Your friend may also want to try the American Liver Foundation Hotline at 1-800-223-0179. They keep track of the latest research. Search for Google's copy of this article Search for Google's copy of this article Search for Google's copy of this article Search for Google's copy of this article ... About
Help! Green Plasma? Books Chronic portalsystemic encephalopathy with normal portal vein pressure 20-year history of chronic progressive portal-systemic encephalopathy. http://mailman1.u.washington.edu/pipermail/hlib-nw/2000-February/004510.html
Extractions: Tue Feb 29 14:28:24 PST 2000 PNC librarians; One of my pathologists is asking me to find information on "green plasma" or "green serum". When I searched on Medline via PubMed, I get information on green tea and effects on serum blood levels as well as green phosphorescence in plants. etc. He tells me this is a real disorder, but I can't find it in the physician's guide to rare diseases or any other reference book in my collection. I also tried the Internet and found many games have a theme about green plasma (a energy source?) in their descriptions, but nothing about a medical condition causing the serum plasma to appear green in color. Any ideas? I am uncertain where to look next. Maybe this is an early April fools joke, but I am hoping you can give me some help solving this medical mystery. Kathy Nelson Eastern Idaho Regional Medical Center Idaho Falls ID 83440 library at eirmc.org
Category Browsing Results Cerebrospinal Fluid Taurine in portalsystemic encephalopathy (J. Lavoie, G.Therrien, and RF Butterworth). Cerebral Zinc Content in Hepatic Encepthalopathy http://opamp.com/cf/browse.cfm?Main=SCIENCES&Sub1=CHEMISTRY
Gastro En La Red Translate this page Dietary management of portal-systemic encephalopathy. In Conn HO, Bircher J,editors. Hepatic encephalopathy syndromes and therapies. http://www.gastroenlared.com.ar/template.php?pagina=./Articulos/Educacion/Encefa
NEJM -- Sign In Dietary management of portalsystemic encephalopathy. In Conn HO, Bircher J, eds.Hepatic encephalopathy syndromes and therapies. http://content.nejm.org/cgi/content/full/337/26/1921-a
Extractions: SIGN IN User Name Password Forgot your Password? Click here and we'll e-mail it to you. If you do not use cookies, sign in here. Remember my User Name and Password. Log in via Athens. PURCHASE THIS ARTICLE Purchase a single article and get immediate online access for just $10. If you're a subscriber but have not yet activated your full online access ACTIVATE YOUR SUBSCRIPTION Subscribers to NEJM are entitled to full access to all online content and features, including 20 FREE online CME exams. OR Receive full access to ALL current content and online features including Personal Archives, PDF article downloads, PDA access, E-mail alerts and 20 FREE online CME exams. OR Receive FREE online access to NEJM Original and Special Articles 6 months after publication and choose to receive the Table of Contents and notification of early release articles via e-mail.
Extractions: This Article Full Text Full Text (PDF) Alert me when this article is cited ... Alert me if a correction is posted Services Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal Download to citation manager ... Cited by other online articles PubMed PubMed Citation Articles by Hazell, A. S. Articles by Butterworth, R. F. Proceedings of the Society for Experimental Biology and Medicine Hepatic encephalopathy (HE) is a neuropsychiatric disorder that occurs in both acute and chronic liver failure. Although the precise pathophysiologic mechanisms responsible for HE are not completely understood, a deficit in neurotransmission rather than a primary deficit in cerebral energy metabolism appears to be involved. The neural cell most vulnerable to liver failure
Portal Systemic Encephalopathy - General Practice Notebook portal systemic encephalopathy. Hepatic encephalopathy is a metabolic disorderof the central nervous system and neuromuscular system that may complicate http://www.gpnotebook.co.uk/cache/-1087045620.htm
Extractions: portal systemic encephalopathy Hepatic encephalopathy is a metabolic disorder of the central nervous system and neuromuscular system that may complicate liver failure from any cause. It is particularly associated with advanced cirrhosis on account of the diffuse parenchymal damage and portosystemic shunting. The features depend on the aetiology and precipitating factors, eventually developing into stupor and then coma. Some aetiologies of the hepatic failure, for example paracetamol overdose, can precipitate this condition within three or four days, with a very rapid progression through the grades of encephalopathy. Other aetiologies, for example viral hepatitis, are associated with a much more variable onset. The grade of the encephalopathy can be used to predict the prognosis - for example grades I and II have and excellent prognosis, grades III and IV have poorer prognoses with the development of complications.
Portal Systemic Encephalopathy - General Practice Notebook portal systemic encephalopathy. Medical search. Hepatic encephalopathy is ametabolic disorder of the central nervous system and neuromuscular system that http://www.gpnotebook.co.uk/medwebpage.cfm?ID=-1087045620
Portal Systemic Encephalopathy Complete online version of The Encyclopaedia of Medical Imaging including textand images from The Encyclopaedia of Medical Imaging s eight book volumes http://www.amershamhealth.com/medcyclopaedia/medical/Volume VI 1/PORTAL SYSTEMIC
Penn State Faculty Research Expertise Database (FRED) encephalopathy, Portal Systemic, Hepatic Comas. Hepatic Encephalopathies,Hepatic Stupors. Hepatocerebral Encephalopathies, Portal Systemic encephalopathy http://fred.hmc.psu.edu/ds/retrieve/fred/meshdescriptor/D006501
Extractions: Vol Page [Advanced] This Article Submit a response Alert me when this article is cited Alert me when eLetters are posted Alert me if a correction is posted Services Email this link to a friend Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal ... Download to citation manager PubMed PubMed Citation Articles by Gyr, K. Articles by Amrein, R. K Gyr, R Meier, J Haussler, P Bouletreau, WE Fleig, A Gatta, A Holstege, G Pomier-Layrargues, SW Schalm, M Groeneweg, G Scollo-Lavizzari, E Ventura, ML Zeneroli, R Williams, Y Yoo and R Amrein
HEPATIC ENCEPHALOPATHY HEPATIC encephalopathy. PORTAL SYSTEMIC encephalopathy (PSE). Background.PSE is defined as a change in mentation in a patient with cirrhosis and portal http://www.columbia.edu/~jj2085/residency/fulltext/HEPATICXENCEPHALOPATHYX3.html
Extractions: I Euphoria/Depression, Day-Night Sleep reversal, Mild asterixis (may be absent) poor concentration, mild confusion, slurred speech II Increased drowsiness, confusion, inability to sustain Significant Asterixis, Concentration Brisk reflexes III Marked confusion, arousable but sleeping continuously Asterixis, clonus Asterixis absent Risk factors:
Portal Toolkit Invalid Site URL Recently, one case of portal systemic encephalopathy was reported, INOUE E,HORI S, NARUMI Y. Portal systemic encephalopathy presence of basal ganglia http://ppv.ovid.com/pt/re/obes/fulltext.00000132-200208000-00007.htm
Log In Problems Portal systemic encephalopathy or hepatic encephalopathy is a The diagnosisof portal systemic encephalopathy is made on a clinical basis but can be http://www.medscape.com/viewarticle/501977_2
LAXETTE SOLUTION In portal systemic encephalopathy administration of large doses of LAXETTE results Portal systemic encephalopathy, hepatic coma or precoma stages where http://home.intekom.com/pharm/cipla/laxette.html
Extractions: LAXETTE is a synthetic disaccharide of fructose and galactose, which is not split into its monosaccharide constituents in the small intestine due to the lack of a specific enzyme. It reaches the colon unchanged where it is broken down by the saccharolytic flora into organic acids, such as lactic acid and acetic acid - acids formed in the colon under physiological conditions. Due to this local osmotic effect in the colon, water is retained, the faecal mass softened and normal colonic peristalsis restored. In portal systemic encephalopathy administration of large doses of LAXETTE results in a significant reduction in the pH of the colonic contents. Lowering the pH promotes conversion of non-ionised ammonia into ionised form. The latter form being non-absorbable leads to reduction of absorption of ammonia from the intestine into the portal circulation and may even promote the excretion of ammonia from the circulation into the faeces.
