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Pernicious Anemia:     more books (94)

Splenectomy for pernicious anemia;: Apparent recovery; death by James Fairchild Baldwin, 1915 Pernicious Anemia; Report of the Progress of Cases presented to the As by Frank BILLINGS, 1900 Recent development in the treatment of pernicious anemia and a consideration of the etiology of the disease: Middleton Goldsmith lecture, New York Pathological Society, October 18, 1930 by Cyrus Cressey Sturgis, 1931 A diet rich in liver in the treatment of pernicious anemia: Study of one hundred and five cases by George Richards Minot, 1927 Pernicious Anemia; Report of the Progress of Cases presented to the As by Frank BILLINGS, 1900 Vitamin B12 in Pernicious Anemia: Parenteral by Charles Cady UNGLEY, 1949 Renal function in pernicious anemia, as determined by dietary renal tests by Henry A Christian, 1916 THE ANEMIAS : WITH SPECIAL REFERENCE TO PERNICIOUS ANEMIA ... by INDIANAPOLIS, IN. ELI LILLY AND COMPANY (FIRM), 1942-01-01 Heredity in pernicious anæmia;: A proband study of the heredity and the relationship to cancer of the stomach by Johannes Mosbech, 1953 Pernicious anaemia (Postgraduate medical journal. [Offprint]) by Keith B Taylor, 1961 Anemia: disease of the supra-renal capsules, 1849: On the constitutional and local effects of disease of the supra-renal capsules, 1855 (Medical classics) by Thomas Addison, 1937 Atrophy of the stomach, with the clinical features of progressive pernicious anæmia by Frederick P Henry, 1886 The treatment of anemia: With comments on food deficiency and its relation to the nervous system by George Richards Minot, 1931 The development of liver therapy in pernicious anæmia by George Richards Minot, 1935

lists with details

101. Vitamin B12 - Ask The Dietitian
     Got questions about Vitamin B 12 or pernicious anemia Ask Joanne Larsen,Registered Dietitian and nutrition counselor.  
     http://www.dietitian.com/vitaminb12.html
     
     Extractions: Pernicious anemia is caused by a deficiency of vitamin B 12. It would show up on a blood test because of the large immature red blood cells that are present. These large red blood cells are very inefficient at carrying oxygen. It can have at least 2 causes - one a lack of vitamin B 12 rich foods in the diet - two an inadequate amount of the intrinsic factor in the stomach. People at risk for pernicious anemia are veg*ans unless they have a high intake of folacin which helps red blood cells develop to the correct size and shape. However adequate folacin does not repair the nerve damage caused by a vitamin B 12 deficiency. Actually it can take quite long, up to 5 years, to develop a vitamin B 12 deficiency depending on a person's body stores. The amount of vitamin B 12 your body stores is dependent on the amount of vitamin B 12 rich foods you eat. Animal products are the best source of vitamin B 12 as there are no real good vegetable sources. Unfortunately, by the time a deficiency does occur, it can cause irreversible nerve and brain damage. In order to absorb vitamin B 12, a person needs the "intrinsic factor" in their stomach. This factor is made in the stomach, but if a person has part or all of their stomach removed, their ability to make intrinsic factor is reduced or eliminated. Basically, the intrinsic factor allows your body to absorb vitamin B 12 and without it, even if you eat foods rich in vitamin B 12, you can still develop a deficiency. In the case of someone having their stomach removed, vitamin B 12 can be administered by injection which must be continued every three weeks for the remainder of life. This method of vitamin administration ensures that it is delivered directly into the blood and bypasses the stomach where the intrinsic factor is insufficient or missing.

102. Hematology
     pernicious anemia is caused by a lack of sufficient quantities of vitamin B12.As discussed in lecture, the disorder is rare one needs only small dietary  
     http://www.pnb.sunysb.edu/hby531/Problem_Sets/clinical_hematology.htm
     
     Extractions: In Word Format Clinical Hematology Sickle-cell disease Sickle-cell disease is an inherited mutation that produces a hemoglobinopathy the glutamate at the number 6 amino acid in the 146-amino acid b chain of hemoglobin (Hb) is replaced by valine. This mutation results from a single base-pair substitution in the gene encoding for the b chain. The resulting Hb in sickle-cell patients is denoted HbS In sickle-cell disease, when the HbS encounters regions where there is a low O tension (like venous blood arising from an exercising muscle), the HbS polymerizes, forming long strands within the RBC that distorts its shape (see figure, right). The distorted RBCs lose there normal flexibility and pile up, blocking blood flow through capillaries and small vessels. One can enter a positive feedback situation whereby the local ischemia (reduced blood flow) causes further drops in the O levels, which causes still further sickling, and the patient is said to be in sickle-cell crisis Patients also suffer from severe hemolytic anemia —a reduction of the number of circulating RBCs due to hemolysis of the fragile sickle cells.

103. Arch Intern Med -- Abstract: Unsuspected Pernicious Anemia In A Patient With Sic
     Unsuspected pernicious anemia in a patient with sickle cell disease pernicious anemia with Neuropsychiatric Dysfunction in a Patient with Sickle Cell  
     http://archinte.ama-assn.org/cgi/content/abstract/147/10/1828
     
     Extractions: Vol. 147 No. 10, October 1, 1987 Featured Link E-mail Alerts ARTICLE Article Options Send to a Friend Similar articles in this journal Literature Track Add to File Drawer Download to Citation Manager PubMed citation Articles in PubMed by Sinow RM Carmel R Articles that cite this article Contact me when this article is cited R. M. Sinow, C. S. Johnson, D. S. Karnaze, M. E. Siegel and R. Carmel Department of Radiology, Los Angeles County-University of Southern California Medical Center. Although the issue of folate supplementation in sickle cell anemia remains controversial, routine supplementation has become common. The major drawback to indiscriminate folate therapy is the potential of masking findings of vitamin B12 (cobalamin) deficiency. This has been dismissed as

104. American RSDHope
     At this point, the symptoms of pernicious anemia will develop. pernicious anemia is Most patients with pernicious anemia are older, usually over 60.  
     http://www.rsdhope.org/Showpage.asp?PAGE_ID=33&PGCT_ID=2651

105. Anemia, Pernicious Definition - Medical Dictionary Definitions Of Popular Medica
     Online Medical Dictionary and glossary with medical definitions.  
     http://www.medterms.com/script/main/art.asp?articlekey=6855

106. Megaloblastic (Pernicious) Anemia
     Detailed information on megaloblastic (pernicious) anemia, including causes,symptoms, diagnosis, and treatment.  
     http://www.healthsystem.virginia.edu/uvahealth/peds_hematology/megalob.cfm
     
     Extractions: [ Skip Navigation ] Topics Adolescent Medicine Allergy/Asthma/Immunology Blood Disorders Burns Cancer Common Injuries/Poison Tips Craniofacial Anomalies Dermatology Diabetes/Endo/Metabolism Eye Care Genitourinary/Kidney High-Risk Newborn High-Risk Pregnancy Infectious Diseases Medical Genetics Mental Health Nervous System Disorders Normal Newborns Oral Health Orthopaedics Pediatric Surgery Respiratory Disorders Terminal Illness in Children Transplantation MAKE AN APPOINTMENT CALENDAR OF EVENTS ... CLINICAL TRIALS Search This Site Megaloblastic anemia is a type of anemia characterized by very large red blood cells. In addition to the cells being large, the inner contents of each cell are not completely developed. This malformation causes the bone marrow to produce fewer cells, and sometimes the cells die earlier than the 120-day life expectancy. Instead of being round or disc-shaped, the red blood cells can be oval. What causes megaloblastic anemia?

107. Health Information
     Megaloblastic (pernicious) anemia. What is megaloblastic anemia? Megaloblasticanemia is a type of anemia characterized by very large red blood cells.  
     http://www.baptisthealth.net/greystone/content.jsp?pageid=P00080

108. The Methodist Hospital: Health Topic Home
     Megaloblastic (pernicious) anemia is a rare disorder in which the body does Megaloblastic (pernicious) anemia results from a lack of intrinsic factor in  
     http://www.methodisthealth.com/cgi-bin/hmdim/home/healthTopic.do?pageTypeId=0&ch

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