Charles Miller Fisher (www.whonamedit.com) Charles miller fisher Canadian neurologist, born 1913, Waterloo, Ontario.Associated with GuillainBarré-Strohl syndrome,miller fisher s syndrome. http://www.whonamedit.com/doctor.cfm/1466.html
Extractions: This survey of medical eponyms and the persons behind them is meant as a general interest site only. No information found here must under any circumstances be used for medical purposes, diagnostically, therapeutically or otherwise. If you, or anybody close to you, is affected, or believe to be affected, by any condition mentioned here: see a doctor. Charles Miller Fisher graduated from the University of Toronto Medical School in 1938. During the Second World War he was in a German prison camp for three and a half years. He became the doctor for the other prisoners and took the opportunity to learn German. This would later allow him, upon his return home to Canada, to access important original German literature regarding cerebrovascular disease.
GBS Pure motor syndrome (IgG vs GM1); millerfisher syndrome (IgG vs GQ1b) miller-fisher syndrome 1% of childhood AIDP; CNS signs More frequent; At onset http://www.neuro.wustl.edu/neuromuscular/antibody/gbs.htm
Miller-Fisher Syndrome - General Practice Notebook millerfisher syndrome is a rare variant of Guillain-Barre syndrome comprising.ataxia; ophthalmoplegia; areflexia. Like the Guillain-Barre, http://www.gpnotebook.co.uk/cache/-1395654646.htm
Miller-Fisher Syndrome Info? millerfisher syndrome info? This article submitted by Jack Creegan on 12/9/95.I am looking for sources of information on miller-fisher syndrome. http://neuro-www.mgh.harvard.edu/neurowebforum/GeneralFeedbackArticles/MillerFis
Extractions: written by his wife The dates below are approximate but give you a pretty accurate idea of the time span involved in such a case. At the hospital a doctor was present when David tried to eat some jelly and could not swallow it. By repeating the tests done by the GP during the day they were able to assess the progress of the still undiagnosed problem. During the night his condition worsened, and he was extremely embarrassed when he fell down while going into the toiled to deal with yet another bout of diarrhea, completely losing control of both feet and bowel functions. Monday the 13th December the decision was made to transfer him to Wellington Hospital and the doctor and a nurse accompanied him in an ambulance. Breathing had become difficult and movement of arms and legs almost ceased. David panicked (and so did the Masterton doctor) when David was left in the Emergency admissions for over an hour because the orderlies were at lunch.
Extractions: Extracted from IndMED Narula AS; Rana PVS; Narula HS Dept. of Medicine, Command Hospital (SC), Pune 400 040, (Maharashtra) Miller-fisher syndrome a variant of guillian barre syndrome : a case report Medical Journal Armed Forces India. 1986 Jul; 42(3): 231-2 ABSTRACT: A case of Miller-Fisher Syndrome has been presented. This is an unusual variant of Guillian Barre Syndrome. Relevant literature has also been reviewed. KEYWORDS: Polyradiculoneuritis/DI; Polyradiculoneuritis/DT; Prednisolone/AD; Middle Age; case Report References: 8 Record Identifier: NI209465
Guillain-Barre Syndrome millerfisher syndrome The most common subtype is miller-fisher syndrome (MFS), miller-fisher syndrome associated with Campylobacter jejuni bearing http://www.davidson.edu/academic/psychology/ramirezsite/neuroscience/psy324/jeca
Extractions: In 1859, Jean B.O. Landry, a French physician, described a disorder in which the nerves of the legs, arms, neck, and breathing muscles were paralyzed. Georges Guillain, Jean Alexander Barre, and Andre Strohl discovered the characteristic abnormality of increased protein but normal cell count in the cerebrospinal fluid in 1916. Several names have been given for the syndrome including acute idiopathic polyneuritis, acute idiopathic polyradiculoneuritis, Landrys ascending paralysis, and Guillain Barre syndrome. The causes of Guillain Barre syndrome are unknown. Many researchers theorize that the autoimmune reaction in which macrophages and T-cells attack myelin in the peripheral and cranial nerves is associated with a bacteria or virus, as many cases occur a few days to a few weeks after an infection including the common cold, sore throat, and stomach and intestinal viruses with vomiting and diarrhea. The virus might induce the demyelination via a possible mimicry between the effector virus and a human ganglioside. Salloway and colleagues (1996) discovered that the lipopolysaccharide structure of some strains of Campylobacter jejuni , specifically, the terminal structures of the core oligosaccharide, resemble the human gangliosides GM1 and GD1a. A possible mimicry also exists between the influenza A NS2 protein and a sequence region of the human P2 (myelin) protein thought to be neuritogenic in animals and mitogenic for lymphocytes from patients with GBS. This finding could provide a possible link between the large number of cases of GBS associated with the 1976 USA swine flu vaccination program (Weise and Carnegie, 1988).
Extractions: (advertisement) Home Specialties Resource Centers CME ... Patient Education Articles Images CME Patient Education Advanced Search Consumer Health Link to this site Back to: eMedicine Specialties Emergency Medicine Neurology Last Updated: August 14, 2004 Rate this Article Email to a Colleague Synonyms and related keywords: GBS, acute idiopathic polyneuritis AUTHOR INFORMATION Section 1 of 10 Author Information Introduction Clinical Differentials ... Bibliography Author: David Fanion, MD , Consulting Staff, Department of Emergency Medicine, AO Fox Hospital Coauthor(s): David Fanion, MD , Consulting Staff, Department of Emergency Medicine, AO Fox Hospital David Fanion, MD, is a member of the following medical societies: American Academy of Emergency Medicine Editor(s): Edward A Michelson, MD , Program Director, Associate Professor, Department of Emergency Medicine, University Hospital Health Systems in Cleveland; Francisco Talavera, PharmD, PhD , Senior Pharmacy Editor, eMedicine; J Stephen Huff, MD
Extractions: (advertisement) Synonyms, Key Words, and Related Terms: Guillain-Barré syndrome, GBS, acute inflammatory demyelinating polyradiculoneuropathy, AIDP, Landry-Guillain-Barré syndrome, Landry-Guillain-Barré-Strohl syndrome, acute idiopathic neuropathy, acute demyelinating neuropathy, infectious polyneuritis, acute polyradiculoneuritis, axonal Guillain-Barré syndrome, acute motor axonal neuropathy, AMAN, acute motor-sensory axonal neuropathy, AMSAN, Miller-Fisher syndrome, pharyngeal-cervical-brachial GBS Background: Guillain-Barré syndrome (GBS) is described most accurately as a collection of clinical syndromes manifested by an acute inflammatory polyradiculoneuropathy with resultant weakness and reflex changes. With poliomyelitis under control in developed countries, GBS is now the most important cause of acute flaccid paralysis. GBS remains a diagnosis made primarily by clinical history and findings. Though classically thought of as a demyelinating neuropathy with ascending weakness, many clinical variants have been well documented in the medical literature. Acute inflammatory demyelinating polyradiculoneuropathy (AIDP) is the most widely recognized form in Western countries, but the recently described variants of acute motor axonal neuropathy (AMAN) and acute motor-sensory axonal neuropathy (AMSAN) also are well recognized. Many believe that strictly defined subgroups of GBS are not distinguished easily but that the subgroups exist, based on a clinical spectrum of symptoms and findings.
Bioline International Official Site (site Up-dated Regularly) Our patient with recurrent millerfisher syndrome (MFS) had no antecedent illnessduring the second episode as mentioned in the case report.1 Antecedent http://www.bioline.org.br/request?ni03091
Fýrat Týp Dergisi A Case Of millerfisher syndrome Displaying A Fast Improvement Following Intravenous miller-fisher syndrome is a rare and benign form of Guillain-Barre http://www.firattipdergisi.com/summary_en.php3?id=125
Pathogenic Blocking Antibodies In Guillain-Barre Syndrome Pre and postsynaptic blockade of synaptic transmission by miller-fisher syndromeIgG at mouse motor nerve terminals.. European Journal of Neuroscience, http://www.zv.uni-wuerzburg.de/forschungsbericht/FOBE-akt/IN-30502000/Toyka DFG
Extractions: The Guillain-Barre Syndrome is the most common inflammatory peripheral nerve disorder with a mortality of up to 8 per cent and longstanding disability in almost half of the patients. The project deals with the recently identified blocking IgG autoantibodies which may act in concert with cell mediated immune mechanisms and complement. These antibodies have a unique dual action with presynaptic blockade of calcium current and of postsynaptic acetyl choline receptor channels. The project deals with the precise mechanisms as analyzed by electrophysiological patch clamp technology. These autoantibodies are likely directed to membrane gangliosides and can be neutralized by polyclonal immunoglobulins from healthy humans. Key words:
JW Neurology -- Sign In Sixty percent of patients with ordinary acute inflammatory demyelinatingpolyneuropathy (AIDP) have had gastrointestinal or respiratory infection within 1 http://neurology.jwatch.org/cgi/content/full/2001/1206/7
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