Neurology -- Sign In Page Acquired hepatocerebral degeneration (AHCD) is usually described in AHCD isa portalsystemic encephalopathy rather than a complication of liver disease http://www.neurology.org/cgi/content/full/63/10/1981
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Extractions: Vol. 60 No. 4, April 2003 Featured Link E-mail Alerts Original Contribution Article Options Full text PDF Send to a Friend Readers Reply Submit a reply Related articles in this issue Similar articles in this journal Literature Track Add to File Drawer Download to Citation Manager PubMed citation Articles in PubMed by Burkhard PR Spahr L Articles that cite this article ISI Web of Science (10) ... Contact me when this article is cited Topic Collections Liver/ Biliary Tract/ Pancreatic Diseases Movement Disorders Parkinson Disease/ Parkinsonian Disorders Topic Collection Alerts
Extractions: Select Journal or Resource JAMA Archives of Dermatology Facial Plastic Surgery Family Medicine (1992-2000) General Psychiatry Internal Medicine Neurology Ophthalmology Surgery Student JAMA (1998-2004) JAMA CareerNet For The Media Meetings Peer Review Congress Citations 1-7 of 7 total displayed. Most recent content (1 May 2005): Original Contributions
Bioline International Official Site (site Up-dated Regularly) Acquired (NonWilsonian) hepatocerebral degeneration degeneration (AHD),different from acute hepatic encephalopathy and classical Parkinson?s disease. http://www.bioline.org.br/request?ni04189
Revista Médica De Chile - Encefalopatía Hepática Crónica: Rol De Niveles San Translate this page Chronic hepatic encephalopathy (CHE) is a disabling complication of chronic Jog M, Lang A. Chronic Acquired hepatocerebral Degeneration Case reports http://www.scielo.cl/scielo.php?pid=S0034-98872001000900011&script=sci_arttext&t
Extractions: George Eversman, MD Alcohol . Few drugs wreak so much havoc so often and with such predictability. And to complicate matters, alcoholic patients are frequently uncooperative, have more than one underlying disease, and a compliance profile that is usually less than optimal. Critical diagnostic findings may be obscured by ethanol intoxication, and disposition decisions are complicated by the patients compromised capacity for vigilant self-care and appropriate follow-up. Add to these pitfalls the fact that chronic alcoholics tend to be poor historians and that the range of medical and surgical disorders afflicting them can fill a textbook of medicine, and it is clear why these patients are prone to falling between the diagnostic cracks. Although alcoholic patients are frequent users of emergency care, physician recognition of various alcohol-related diseases is poor. Some alcohol-related conditions require immediate lifesaving interventions, whereas others require hospital admission or outpatient referral for resolution. Whatever the diagnosis and ultimate disposition, recognition of chronic alcohol abuse as a cause for these problems is imperative so that early care can be instituted.
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World J Gastroenterol AIM Acquired hepatocerebral degeneration (AHD) is an exceptional type Movement dysfunction and hepatic encephalopathy. Metab Brain Dis 2001; 16 2735 http://www.wjgnet.com/1007-9327/11/764.asp
Research Findings Register: Summary Number 1127 The commonest complications are a diffuse encephalopathy and seizures. myelopathic presentation of the acquired hepatocerebral degeneration syndrome. http://www.refer.nhs.uk/ViewRecord.asp?ID=1127
Extractions: Anodontia Absence of teeth (complete) (congenital) (partial) Hypodontia Oligodontia Excludes : acquired absence of teeth (525.1) Supernumerary teeth Distomolar Fourth molar Mesiodens Paramolar Supplemental teeth Excludes : supernumerary roots (520.2) Abnormalities of size and form Concrescence of teeth Fusion of teeth Gemination of teeth Dens evaginatus Dens in dente Dens invaginatus Enamel pearls Macrodontia Microdontia Peg-shaped [conical] teeth Supernumerary roots Taurodontism Tuberculum paramolare Excludes : that due to congenital syphilis (090.5) tuberculum Carabelli, which is regarded as a normal variation Mottled teeth Dental fluorosis Mottling of enamel Nonfluoride enamel opacities Disturbances of tooth formation Aplasia and hypoplasia of cementum Dilaceration of tooth Enamel hypoplasia (neonatal) (postnatal) (prenatal) Horner's teeth Hypocalcification of teeth Regional odontodysplasia Turner's tooth Excludes : Hutchinson's teeth and mulberry molars in congenital syphilis (090.5) mottled teeth (520.3)
Cecil Textbook Of Medicine : /> Some alcoholics with repeated bouts of hepatic encephalopathy develop acquiredchronic hepatocerebral degeneration, with dementia, dysarthria, ataxia, http://www.merckmedicus.com/ppdocs/us/common/cecils/chapters/458_004.htm
Extractions: ALCOHOL-RELATED DISORDERS Alcohol intoxication and withdrawal are discussed in Chapter 17 WERNICKE-KORSAKOFF SYNDROME. is often dramatic. Eye movements sometimes begin to improve within a few hours, and except for residual nystagmus, may be normal within 1 or 2 weeks. Ataxia tends to improve less completely; more than half of patients are left with a broad-based, unsteady gait. More serious are persisting mental symptoms. Drowsiness, inattentiveness, and apathy tend to clear with treatment, but an amnestic disorder, termed Korsakoff's syndrome, often persists and includes both anterograde and retrograde memory loss, sometimes accompanied by confabulation, which is out of proportion to additional mental abnormalities. Once established, the memory disorder is permanent in the majority of patients. In alcoholics and others with low thiamine stores, administration of glucose can precipitate Wernicke's syndrome. Patients receiving parenteral glucose (e.g., for parenteral alimentation or in an acute setting for diagnosis and treatment of unexplained seizures or coma) should also be given parenteral thiamine (and other water-soluble vitamins).
Virus Hepatitis Liver Virology Toxicology Encephalopathy 2005 (full text) ACQUIRED hepatocerebral DEGENERATION IN HEPATITIS C INFECTION December 2004 (full text) Minimal hepatic encephalopathy Should we http://www.psic.info/HCV_Einleitung.htm
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Extractions: This Article Submit a response Alert me when this article is cited Alert me when eLetters are posted Alert me if a correction is posted ... Citation Map Services Email this article to a friend Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal ... Cited by other online articles PubMed PubMed Citation Articles by Kreis, R. Articles by Ackerman, Z. R Kreis, BD Ross, NA Farrow and Z Ackerman Magnetic Resonance Spectroscopy Laboratory, Huntington Medical Research Institute, Pasadena, CA 91105. Proton magnetic resonance (MR) spectroscopy of the brain was performed in 11 patients with chronic hepatic encephalopathy (CHE), and the results were compared with those of patients with liver disease but without CHE; clinical control subjects with diabetes, uremia, or cortical atrophy; and
Chronic Liver Injury Hepatic encephalopathy. neuropsychiatric abnormalities in patients with hepatocerebral syndromes ABCs. Acute liver failure associated with HE (HE http://members.tripod.com/d3jonline/21-Gastrointestinal/Chronic_Liver_Injury.htm
Extractions: Search: Lycos Tripod Star Wars Share This Page Report Abuse Edit your Site ... Next Chronic Liver Injury Causes of Chronic Liver Injury Three Mechanisms 80% of all cases are related to lupus; some autoimmunity possibly due to superantigens from drug metabolism high dose steroid therapy can be much better in weeks Viral due to hepatitis B, delta, or C (A and E are not chronic) often subclinical acute infection in hepatitis B, damage is due to inflammatory response, NOT direct cytotoxicity
Clinical Neuropharmacology - UserLogin Patients with chronic hepatic encephalopathy display monoaminergic Bengtsson F,Bugge M, Nobin A. hepatocerebral dysfunction and brain serotonin. http://www.clinicalneuropharm.com/pt/re/clnneupharm/fulltext.00002826-200111000-
AJNR -- Sign In Page Hepatic encephalopathy is a complication of hepatic failure or Cole M.The acquired (nonWilsonian) type of chronic hepatocerebral degeneration. http://www.ajnr.org/cgi/content/full/26/2/347
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