H Hypertension . Hyperlexia . Insulin Resistance . Hirsutism hepatocerebral encephalopathy Hepatolenticular Degeneration + and treatment.Also copper content in various + Also a brief article on Dr. Samuel Wilson. http://www.thema-gute-gesundheit.de/Health|DE-6404-6640|Conditions,and,Diseases|
Neuroimaging Aspects Of Chronic Acquired Hepatocerebral Chronic, nonWilsonian hepatocerebral degeneration was described by Victor Hepatic encephalopathy and coma can be recurrent and resolve completely with http://med.ege.edu.tr/norolbil/2001/NBD16601.html
Chapter 14 - Section 13: First Principles Of Gastroenterology Asterixis is not specific to hepatic encephalopathy and may be present in often forms part of the spectrum of acquired hepatocerebral degeneration). http://gastroresource.com/GITextbook/En/Chapter14/14-13.htm
Extractions: - Select a chapter - 1. Symptoms and Signs 2. Nutrition 3. Ethics 4. Research/Clinical Trials 5. Esophagus 6. Stomach and Duodenum 7. Small Intestine 8. Intestinal Ischemia 9. H.I.V. 10. Inflammatory Bowel 11. Colon 12. Pancreas 13. Biliary System 14. Liver 15. Paediatrics 16. Video Endoscopic Images Search 13. Hepatic Encephalopathy / L.J. Worobetz page 537 Hepatic encephalopathy (HE) is a complex, potentially reversible neuropsychiatric condition that occurs as a consequence of acute or chronic liver disease. It is characterized by changes of personality, consciousness, behavior and neuromuscular function ( Table 20 ). Early features include reversal of sleep pattern, apathy, hypersomnia, irritability and personal neglect. In later stages, delirium and coma may occur. Neurologic signs may include hyperreflexia, rigidity, myoclonus and asterixis. Asterixis is not specific to hepatic encephalopathy and may be present in other causes of metabolic encephalopathy. Seizures and lateralizing signs are uncommon and are more commonly seen in acute than chronic liver failure. Clinically, a number of encephalopathic patterns can be observed: acute, acute recurrent, chronic recurrent and chronic permanent encephalopathy (the last often forms part of the spectrum of acquired hepatocerebral degeneration).
CCHS Clinical Digital Library Chapter 268 Hepatic encephalopathy and Acquired hepatocerebral DegenerationAccess document. Ferri Ferri s Clinical Advisor Instant Diagnosis and http://cchs-dl.slis.ua.edu/clinical/gastroenterology/hepatic/hepatic/hepatic_enc
Extractions: Clinical Resources by Topic: Gastroenterology Hepatic Encephalopathy Clinical Resources Emergency Pediatrics Pathology Clinical Guidelines ... Miscellaneous Resources See also: Chapter 258. Critical Care Neurology: Table of contents Feldman: Sleisenger and Fordtran's Gastrointestinal and Liver Disease 7th Ed.-2002 (MD Consult): Table of contents Health Sciences Library subscription INFO Brenner and Rector's The Kidney 7th Ed.-2004 (MD Consult):
Extractions: Vol Page [Advanced] This Article Extract Submit a response View responses Alert me when this article is cited ... Alert me if a correction is posted Services Email this link to a friend Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal ... Cited by other online articles PubMed PubMed Citation Articles by STRACCIARI, A Articles by PISI, P Related Collections Other Neurology Other Psychiatry J Neurol Neurosurg Psychiatry 136-137 ( January ) Liver diseases may give rise to variable degrees of neurological impairment, which mostly consist of the syndrome of hepatic encephalopathy, due to the toxic effect of ammonia on the brain during episodes of liver decompensation. In a minority of patients
Extractions: Vol Page [Advanced] This Article Full Text Submit a response View responses Alert me when this article is cited ... Alert me if a correction is posted Services Email this link to a friend Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal ... Cited by other online articles PubMed PubMed Citation Articles by STRACCIARI, A Articles by PISI, P Related Collections Other Neurology Other Psychiatry J Neurol Neurosurg Psychiatry 136-137 ( January ) The first 150 words of the full text of this article appear below. Liver diseases may give rise to variable degrees of neurological impairment, which mostly consist of the syndrome of hepatic encephalopathy, due to the toxic effect of ammonia on the brain
Final Diagnosis -- Case 221 Final Diagnosis Acquired (NonWilsonian) hepatocerebral degeneration with shuntmyelopathy Fraser CL, Arieff AI (1985) Hepatic encephalopathy. http://path.upmc.edu/cases/case221/dx.html
Extractions: FINAL DIAGNOSIS: Acquired (Non-Wilsonian) hepatocerebral degeneration with "shunt myelopathy" DISCUSSION: It is well known that patients with severe liver disease may suffer from a reversible neurologic condition known as hepatic encephalopathy [1,6]. Some of these patients suffer from a more permanent neurologic disorder, particularly if they have surgically induced or spontaneous porto-systemic shunts and experience episodes of severe hepatic encephalopathy with coma. Victor, Adams, et al. reported the features of AHCD in detail in a series of 27 cases [14]. These symptoms and signs included progressive dementia, dysarthria, involuntary movements (including tremor, asterixis, and choreoathetosis), ataxia of limb and gait, and mild pyramidal tract signs, typically in a patient with severe hepatic disease and recurrent episodes of coma. Less commonly reported is a progressive spastic myelopathy in association with AHCD [4,12]. Only about 15 cases have been reported in the world literature with detailed pathological descriptions [2,3,7,9,13]. The present case report has several distinctive features. Whereas most reported cases resulted from a surgically created porto-systemic shunt as a treatment for cirrhosis, a few other cases, including this one, are thought to result from spontaneously created shunts (e.g., varices) with the same functional consequences. The published cases usually had Alzheimer II cells in the brain, but only a few had the severe spongy degeneration in the deep cortex illustrated here. Recently, hepatocerebral degeneration has occasionally been documented in vivo by MRI [8,9]. Few electron microscopic studies of human Alzheimer II cells and hepatocerebral degeneration have been published [5,11]. The present report suggests that the vacuolation results from the disruption of astrocytes.
En : On Medical Dictionary Online encephalopathy, Hepatic encephalopathy, hepatocerebral encephalopathy, HIV encephalopathy, Hyperbilirubinemic encephalopathy, Hypertensive http://www.online-medical-dictionary.org/en.asp?q=~En
Elsevier.com - Handbook Of Medical Psychiatry Uremic encephalopathy 259.Dialysis Disequilibrium Syndrome 260.Hepatic encephalopathyand Acquired hepatocerebral Degeneration 261.Hepatic Porphyrias 262. http://www.elsevier.com/wps/product/cws_home/702307
Extractions: Home Site map Regional Sites Advanced Product Search ... Handbook of Medical Psychiatry Book information Product description Author information and services Ordering information Bibliographic and ordering information Book related information Submit your book proposal Other books in same subject area About Elsevier Select your view HANDBOOK OF MEDICAL PSYCHIATRY The HANDBOOK is a comprehensive text covering nearly every psychiatric, neurologic, and general medical condition capable of affecting mental status. It is divided into 30 sections patterned after topic headings in the DSM-IV. Within each section is a series of chapters that briefly describe an overview, time of onset, clinical features, course, complications, etiology, differential diagnosis, and treatment of a given disorder. A short list of references follows every chapter, and many chapters include radiographs that show characteristics of the disease process. The final section covers psychopharmacology, emphasizing medical features such as pharmacokinetics, side effects, drug-drug interactions, and complications of overdose.
Gazi Týp Fakültesi - Gazi Medical Journal Acquired (nonwilsonian) hepatocerebral degeneration, characterised with permanent Lockwood AH Hepatic encephalopathy and other neurological disorders http://www.med.gazi.edu.tr/gmj/1998_3_136_138.html
Extractions: Table of contents CASE REPORTS ACQUIRED HEPATOCEREBRAL DEGENERATION: A CASE PRESENTATION WITH CRANIAL MAGNETIC RESONANCE IMAGING FINDINGS Levent Sinan BÝR, M.D., Reha KURUOÐLU, M.D., Ali Ýhsan BAYSAL, M.D., Ceyla ÝRKEÇ, M.D., Suna AKKUÞ ASLAN, M.D. Gazi University, Faculty of Medicine, Department of Neurology, Ankara, Turkey Gazi Medical Journal 1998; 9 : 136-138 SUMMARY : We present a 54-year-old male patient who had previously been followed with the diagnosis of postnecrotic cirrhosis for twelve years. During the last 4 years, he had developed progressive dysarthria, tremor, and ataxic gait. Neurological examination revealed a mild right hemiparesis with bilateral cerebellar and pyramidal signs. Cranial magnetic resonance imaging study showed cerebral and cerebellar atrophy, as well as bilateral high intensity signals in dentate nuclei and periventricular white matter in T2 weighted images, reminiscent of Wilson's disease. These white matter lesions can also be erroneously diagnosed as multiple sclerosis plaques. Key Words: Brain, Magnetic Resonance Imaging, Liver Diseases.
Extractions: MRI examination revealed similar brain lesions in 5 alcoholic Korsakoff patients and 5 chronic alcoholics without cognitive impairment. Not only cerebral atrophy and demyelination, but also lesions thought to be specific for the Wernicke-Korsakoff syndrome were equally prominent in both groups. The morphological abnormalities thought to be typical of Wernicke-Korsakoff syndrome are probably common features of chronic alcoholism and malnutrition. Marked atrophy of the operculae was found in all Korsakoff patients and in 3 out of 5 chronic alcoholics. Alcohol amnestic disorder may not exclusively result from diencephalic lesions, but also from temporal lesions. Butterworth, R. F., J. J. Kril, et al. (1993). Thiamine-dependent enzyme changes in the brains of alcoholics: relationship to the Wernicke-Korsakoff syndrome. Alcohol Clin Exp Res
Liver : Epilepsy.com/Professionals A variety of liver disorders may cause hepatic encephalopathy. These includehepatitis, Wilsons disease, acquired hepatocerebral degeneration, http://professionals.epilepsy.com/page/transplant_liver.html
Extractions: Search epilepsy.com/professionals: WHO IS EPILEPSY.COM DONATE PROFESSIONAL FORUM CO-EXISTING DISORDERS ... RESOURCE LIBRARY Liver Co-Existing Disorders Liver Author: E Cruz-Martinez and RL Gilmore A variety of liver disorders may cause hepatic encephalopathy. These include hepatitis, Wilsons disease, acquired hepatocerebral degeneration, alcoholic hepatitis, and cirrhosis. Hepatic encephalopathy is classified into stages: Hypoglycemia may complicate liver failure and may be responsible for some seizures. Hyperammonemia also is associated with seizures and may contribute to the encephalopathy of primary hyperammonemic disorders. Treatments that reduce ammonia also ameliorate the encephalopathy. Treatment should be directed at the underlying etiology of the hepatic failure. The cornerstone of treatment is the reduction of gastrointestinal protein and the use of lactulose, which increases ammonia elimination Treatment with chronic antiepileptic drugs (AEDs) is usually not necessary, unless there is a known predisposition to seizures (e.g., from previous cerebral injury). Because sedating AEDs may precipitate coma, they are generally avoided. Phenytoin is probably a reasonable choice when chronic AED treatment is necessary. Valproic acid should be avoided in hepatic failure. The AEDs excreted by the kidney are particularly useful in this setting.
CNSforum Chronic Parkinsonism Associated With Cirrhosis - A With Cirrhosis A Distinct Subset of Acquired hepatocerebral Degeneration and entirely different from acute hepatic encephalopathy episodes. http://www.cnsforum.com/abstractitem/926add09-13b0-4df6-8a55-ab03606062e9/defaul
Parkinsonism - The Doctors Lounge(TM) Trauma pugilistic encephalopathy. Other parathyroid abnormalities, hypothyroidism,hepatocerebral degeneration, brain tumour, normal pressure http://www.thedoctorslounge.net/clinlounge/diseases/neurology/parkinsonism.htm
