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Hepatic Encephalopathy:     more books (47)

Advances in Hepatic Encephalopathy and Metabolism in Liver Disease CIRRHOSIS, HYPERAMMONEMIA, AND HEPATIC ENCEPHALOPATHY (AEMB, VOL. 341) (HB) " by Grisolia/felipo, 1993 Hepatic encephalopathy (Medical Grand Rounds) by James Shorey, 1978 Hepatic encephalopathy, by Frederick Steigmann, 1971 Advances in Cirrhosis, Hyperammonemia, and Hepatic Encephalopathy (Advances in Experimental Medicine and Biology, 420) by Santiago Grisolia, Hyperammonemia, and Hepatic Ence International Symposium on Cirrh Vicente Felipo, 1997 Hepatic Encephalopathy (Digestive Diseases,) by K. Mullen, 1996-08 Brain monoamine metabolism in experimental hepatic encephalopathy (Bulletin No. 65 from the Department of Surgery, Lund University) by Mogens Bugge, 1987 Hepatic Encephalopathy and Nitrogen Metabolism by D. H ussinger, 2009 Hepatic encephalopathy: The role of blood-brain barrier derangements, ammonia, amino acids and neurotransmitters (Bulletin of the Department of Surgery, University of Lund) by Torbjörn Jonung, 1985 Hepatic encephalopathy: A new perspective for therapy by Peter B Soeters, 1979 HEPATIC ENCEPHALOPATHY PATHOPHYSIOLOGY by ROGER & G LAYRARGUES BUTTERWORTH, 1989-01-01 Hepatic Encephalopathy, Hyperammonemia and Ammonia Toxicity by Vicente Felipo, 1994 Hepatic Encephalopathy by Roger F. Butterworth, 2009 Hepatic Encephalopathy Hyperammonemia by Vicente Felipo, 1995

lists with details

41. Alcohol Research & Health: Hepatic Encephalopathy
    Full text of the article, hepatic encephalopathy from Alcohol Research Health,a publication in the field of Health Fitness, is provided free of  
    http://www.findarticles.com/p/articles/mi_m0CXH/is_3_27/ai_n6357651
    
    Extractions: The liver and the brain interact in numerous ways to ensure normal brain functioning. For example, the liver plays a key role in supplying nutrients to the brain, which cannot produce these compounds itself. The liver also removes toxic substances from the blood, including substances that have been generated in the brain and must be eliminated from the body, as well as compounds produced in other tissues that are harmful to the brain's nerve cells (i.e., are neurotoxic). Thus, liver dysfunction can cause disturbances of brain function and even contribute to brain damage. This article describes the characteristics and diagnosis of HE and the changes in brain cell structure associated with this condition. The article also reviews imaging techniques that allow researchers to study changes in brain structure and function occurring in patients with HE and describes the contributions of ammonia and manganese to the development of HE, as elucidated by these techniques. Finally, the article explores some approaches currently used or being investigated for treating patients with HE resulting from alcoholic liver disease.

42. Alcohol Research & Health: Hepatic Encephalopathy—a Serious Complication Of Alc
    Full text of the article, hepatic encephalopathy—a serious complication ofalcoholic liver disease from Alcohol Research Health, a publication in the  
    http://www.findarticles.com/p/articles/mi_m0CXH/is_2_27/ai_n6125561
    
    Extractions: Save a personal copy of this article and quickly find it again with Furl.net. It's free! Save it. Alcohol's harmful effects on liver cells not only interfere not only with the normal functioning of the liver but also impact distant organs, including the brain. Prolonged liver dysfunction resulting from excessive alcohol consumption can lead to the development of a serious and potentially fatal brain disorder known as hepatic encephalopathy (HE). Patients with HE suffer from sleep disturbances, changes of mood and personality, severe cognitive effects (e.g., a shortened attention span), psychiatric conditions such as anxiety and depression, as well as motor disturbances, including motor incoordination and a type of flapping tremor of the hands called asterixis. In the most serious cases, the patients no longer respond to external stimuli and may fall into a coma (i.e., hepatic coma), which can be fatal.

43. FIRSTConsult - Sdfdsf
    FIRSTConsult, hepatic encephalopathy (Medical Condition File). Published formedical students and primary healthcare providers by Elsevier.  
    http://www.firstconsult.com/?action=view_article&id=1014781&type=101&bref=1

44. Benzodiazepine Receptor Antagonists For Hepatic Encephalopathy (Cochrane Review)
    Abstract of a systematic review of the effects of health care prepared by theCochrane Collaboration.  
    http://www.cochrane.org/cochrane/revabstr/AB002798.htm
    
    Extractions: From The Cochrane Library, Issue 2, 2005 Benzodiazepine receptor antagonists for hepatic encephalopathy (Cochrane Review) Als-Nielsen B, Gluud LL, Gluud C ABSTRACT What's new in this issue Search abstracts Browse alphabetical list of titles Browse by Review Group A substantive amendment to this systematic review was last made on 23 February 2004. Cochrane reviews are regularly checked and updated if necessary. Background: Hepatic encephalopathy may be associated with accumulation of substances that bind to a receptor-complex in the brain resulting in neural inhibition. Benzodiazepine receptor antagonists may have a beneficial effect on patients with hepatic encephalopathy. Objectives: To evaluate the beneficial and harmful effects of benzodiazepine receptor antagonists for patients with hepatic encephalopathy. Search strategy: Eligible trials were identified through The Cochrane Hepato-Biliary Group Controlled Trials Register, The Cochrane Controlled Trials Register on The Cochrane Library, MEDLINE and EMBASE (last search: January 2004), reference lists of relevant articles, authors of trials, and pharmaceutical companies. Selection criteria: Randomised trials comparing any benzodiazepine receptor antagonist versus placebo or no intervention for hepatic encephalopathy.

45. Branched-chain Amino Acids For Hepatic Encephalopathy (Cochrane Review)
    Abstract of a systematic review of the effects of health care prepared by theCochrane Collaboration.  
    http://www.cochrane.org/cochrane/revabstr/AB001939.htm
    
    Extractions: From The Cochrane Library, Issue 2, 2005 Branched-chain amino acids for hepatic encephalopathy (Cochrane Review) Als-Nielsen B, Koretz RL, Kjaergard LL, Gluud C ABSTRACT What's new in this issue Search abstracts Browse alphabetical list of titles Browse by Review Group A substantive amendment to this systematic review was last made on 19 September 2002. Cochrane reviews are regularly checked and updated if necessary. Background: Hepatic encephalopathy may be caused by a decreased plasma ratio of branched-chain amino acids (BCAA) to aromatic amino acids. Treatment with BCAA may therefore have a beneficial effect on patients with hepatic encephalopathy. Objectives: To evaluate the beneficial and harmful effects of BCAA for patients with hepatic encephalopathy. Search strategy: We identified trials through The Cochrane Hepato-Biliary Group Controlled Trials Register (September 2002), (Issue 3, 2002), MEDLINE (1966-2002/09) and EMBASE (1980-2002/05), manual searches of bibliographies and journals, authors of trials, and pharmaceutical companies. Selection criteria: Randomised trials comparing BCAA with any kind of control therapy for hepatic encephalopathy were included, regardless of blinding, language, or publication status.

46. Hepatic Encephalopathy- Health Encyclopedia And Reference
    hepatic encephalopathy Symptoms, Treatments and Medications.  
    http://www.healthcentral.com/encyclopedia/408/368/Hepatic_Encephalopathy.html
    
    Extractions: Dr. Dean TV Specials Newsletters Help Apr 14, 2005 Home News Diseases Symptoms ... Health Tools Search Choose a Health Topic * All Health Topics * Acne Addictions AIDS/HIV Alcohol Abuse Allergies Alternative Medicine Alzheimer's Arthritis Asthma Attention Deficit Disorder (ADHD) Backache Bipolar Affective Disorder Birth Control Blood Blood Pressure Bone/Joint/Tendon Bowel Brain Breast cancer Cancer Cerebral Palsy Cholesterol Cramps Crohn's Disease Cysts Dental/Oral Depression Diabetes Diet, Fitness, Looks Disabled/Special Needs Drug Abuse Ear/Nose/Throat Eating/Appetite Eczema Encephalitis Eye/Vision Fatigue Fever Flu Food Poisoning Foot Gallbladder Gastrointestinal Genetic/Congenital GERD/Heartburn Hair Loss Hair/Scalp Headache Hearing Heart Disease, Stroke Heat/Sunstroke Hepatitis Hernia Herpes High Blood Pressure Hormonal Immune Disorders Immunizations/Vaccines Impotence Incontinence/Bladder Infections Injuries Kidney/Urinary Kids Learning Disabilities Liver Lung Cancer Men Menopause, Postmenopause Migraine Mobility/Balance Multiple Sclerosis Muscle Neural Nosebleeds Pain Parasites Pituitary PMS Pregnancy Prostate Cancer Prostate Disorders Psoriasis Psychological Raynaud's Disease Respiratory Reye Syndrome Rheumatoid Arthritis Schizophrenia Seniors Sensory Sexually Transmitted Skin Sleep Speech Disorders Spinal Thyroid Trauma Weight Loss Women Yeast Infections

47. Halfbakery: Hepatic Encephalopathy Treatment
    What to do for the folks with hepatic encephalopathy? As Nick8 s link describes,the reason for this trouble is thought to be accumulation of ammonia,  
    http://www.halfbakery.com/idea/Hepatic_20encephalopathy_20treatment
    
    Extractions: Just when everyone was having fun talking about the booze buster pills, along came buzzkill IVnick8or with the gloom and doom of hepatic encephalopathy. Those folks are in a bad way. There is no good dialysis equivalent for livers, so if you have used yours up, you can't just hook on to the machine like the renal failure folks do. Even if you quit the booze, if your liver is shot you could still get hepatic encephalopathy from time to time. The only cure is a liver transplants, but folks seem to want to save the available livers for kids and those who came by liver failure by accident, rather than prolonged effort. What to do for the folks with hepatic encephalopathy? As Nick8's link describes, the reason for this trouble is thought to be accumulation of ammonia, which normally the liver can get rid of. When this stuff builds up it stuns the brain. Zeolite is a natural mineral widely used to adsorb ammonia in applications such as wastewater treatment and aquarium maintenance. I propose that a slurry be devised for the liver impaired, consisting of zeolite slurry. This stuff will adsorb ammonia on the way thru the gut and eventually be pooped out. Possibly, if has not been exhausted, it will go on to aid with municipal wastewater treatment. The "Z-slurry" will buttress what is left of the liver, allowing the person to cloud his mind only with the chemicals of his choosing.

48. Hepatic Encephalopathy - Alegent Health Serving Eastern Nebraska And Southwest I
    hepatic encephalopathy courtesy of Alegent Health serving eastern Nebraska andsouthwest Iowa - Council Bluffs, Iowa - Corning, Iowa - Missouri Valley,  
    http://www.alegent.com/12503.cfm
    
    Extractions: @import url(default.css); Back to Health Library Print This Page Email to a Friend Digestive system organs Definition: Hepatic encephalopathy is brain and nervous system damage that occurs as a complication of liver disorders. It is characterized by various neurologic symptoms including changes in reflexes, changes in consciousness , and behavior changes that can range from mild to severe. However, when the liver cannot properly metabolize and detoxify substances in the body, toxic substances build up in the bloodstream. One substance believed to be particularly toxic to the central nervous system is ammonia , which is produced by the body when proteins are digested, but is normally detoxified by the liver. Many other substances may also accumulate in the body and contribute to damage to the nervous system.

49. Hepatic Encephalopathy - Washington DC
    hepatic encephalopathy Washington Hospital Center is located in Washington DC.  
    http://www.whcenter.org/13545.cfm
    
    Extractions: Home Back to Health Library Print This Page ... Digestive system organs Definition: Hepatic encephalopathy is brain and nervous system damage that occurs as a complication of liver disorders. It is characterized by various neurologic symptoms including changes in reflexes, changes in consciousness , and behavior changes that can range from mild to severe. However, when the liver cannot properly metabolize and detoxify substances in the body, toxic substances build up in the bloodstream. One substance believed to be particularly toxic to the central nervous system is ammonia , which is produced by the body when proteins are digested, but is normally detoxified by the liver. Many other substances may also accumulate in the body and contribute to damage to the nervous system.

50. NEJM -- Treatment Of Hepatic Encephalopathy
    Review Article from The New England Journal of Medicine Treatment of HepaticEncephalopathy.  
    http://content.nejm.org/cgi/content/short/337/7/473
    
    Extractions: Add to Personal Archive ... PubMed Citation Hepatic encephalopathy is a complex neuropsychiatric syndrome that may occur in such diverse clinical situations as inherited errors of the urea cycle, acute or chronic liver disease, and spontaneous or iatrogenic portosystemic venous shunting, including that following procedures to establish a transjugular intrahepatic portosystemic shunt. The clinical manifestations of this syndrome range from subtle abnormalities detectable only by psychometric testing to deep coma. Several grading systems have been proposed; one based on clinical and electroencephalographic abnormalities is shown in Table 1. Hepatic encephalopathy may be present in 50 to 70 percent of all patients with cirrhosis, including

51. NEJM -- Management Of Hepatic Encephalopathy
    Correspondence from The New England Journal of Medicine Management of HepaticEncephalopathy.  
    http://content.nejm.org/cgi/content/short/337/26/1921-a
    
    Extractions: Add to Personal Archive Add to Citation Manager E-mail When Cited ... Find Similar Articles To the Editor: In their review, Riordan and Williams (Aug. 14 issue) state that protein restriction is a well-documented therapy for hepatic encephalopathy and imply that a high dietary protein intake is a common cause of hepatic encephalopathy in patients with liver cirrhosis. To our knowledge, scientific proof of the benefit of protein restriction is lacking, and there are no controlled studies of protein restriction. The authors refer to a chapter in a book and an experimental study in dogs. They recommend minimal daily protein intakes of 0.8 to 1.0 g per kilogram of body weight to Full Text of this Article References

52. THE MERCK MANUAL, Sec. 4, Ch. 38, Clinical Features Of Liver Disease
    PortalSystemic Encephalopathy (hepatic encephalopathy; Hepatic Coma) Portal-systemic encephalopathy may occur in fulminant hepatitis caused by viruses,  
    http://www.merck.com/mrkshared/mmanual/section4/chapter38/38f.jsp

53. HEPATIC ENCEPHALOPATHY
    hepatic encephalopathy A condition which is used to describe the deleteriouseffects of liver failure on the central nervous system.  
    http://www.medhelp.org/glossary/new/gls_2197.htm
    
    Extractions: HEPATIC ENCEPHALOPATHY - A condition which is used to describe the deleterious effects of liver failure on the central nervous system. Features include confusion ranging to unresponsiveness ( coma ). A common cause is alcoholic cirrhosis . Treatment includes the binding and elimination of ammonia in the intestinal tract . General life support including respiratory support and cardiovascular support may also be required. See encephalopathy for additional details.

54. Hepatic Encephalopathy
    Patient medical question and answer from The Hepatitis Forum. Health topic areaand articles about liver other Topics ,.  
    http://www.medhelp.org/forums/Hepatitis/messages/37462.html

55. A Review Of Congenital Portosystemic Shunting And Hepatic Encephalopathy
    The pathogenesis of hepatic encephalopathy is multifactorial and not hepatic encephalopathy develops because of failure of the diseased liver to  
    http://www.addl.purdue.edu/newsletters/1997/winter/cps.shtml
    
    Extractions: A Review of Congenital Portosystemic Shunting and Hepatic Encephalopathy Heptic encephalopathy is a neurologic disorder which may develop in animals who have advanced liver disease and/or severe portosystemic shunting. Congenital porto-vascular anomalies, which allow portal blood to circumvent hepatic detoxification in affected dogs and cats, and chronic severe hepato-cellular disease with acquired intra- or extrahepatic portosystemic shunting (PSS) in dogs account for most of the cases of hepatic encephalopathy. Signalment and History Congenital PSS is more commonly seen in purebred (Yorkshire terriers and Miniature schnauzers) than in mix-breed dogs. Most animals are presented by 2 years of age, often by 6 months of age, and sporadically at any age. Owners' concerns are commonly related to neurologic, gastrointestinal, and/or urinary tract disorders. Furthermore, affected animals may have a history of stunted growth or failure to gain weight compared with unaffected littermates. Laboratory Evaluation Definitive diagnosis of a portosystemic shunt requires identification of the shunt by ultrasonography, contrast radiography, or exploratory laporatomy.

56. Dopaminergic Agonists For Hepatic Encephalopathy (Cochrane Review)
    Abstract of a systematic review of the effects of health care prepared by theCochrane Collaboration.  
    http://www.update-software.com/Abstracts/AB003047.htm
    
    Extractions: From The Cochrane Library, Issue 3, 2005 Dopaminergic agonists for hepatic encephalopathy (Cochrane Review) Als-Nielsen B, Gluud LL, Gluud C ABSTRACT What's new in this issue Search abstracts Browse alphabetical list of titles Browse by Review Group A substantive amendment to this systematic review was last made on 24 August 2004. Cochrane reviews are regularly checked and updated if necessary. Background: Hepatic encephalopathy may be associated with an impairment of the dopaminergic neurotransmission. Dopaminergic agonists may therefore have a beneficial effect on patients with hepatic encephalopathy. Objectives: To evaluate the beneficial and harmful effects of dopaminergic agonists for patients with hepatic encephalopathy. Search strategy: Trials were identified through The Cochrane Hepato-Biliary Group Controlled Trials Register (July 2004), The Cochrane Central Register of Controlled Trials (Issue 3, 2004), MEDLINE (1966-2004/07), EMBASE (1980-2004/07), manual searches of bibliographies and journals, authors of trials, and pharmaceutical companies. Selection criteria: All randomised trials comparing dopaminergic agonists versus placebo or no intervention for hepatic encephalopathy.

57. Icon Health Publications Hepatic Encephalopathy A Medical
    Icon Health Publications hepatic encephalopathy A Medical Dictionary, Bibliography,and Annotated Research Guide to Internet References,Conservative,Book  
    http://www.forbesbookclub.com/bookpage.asp?prod_cd=IR08D

58. UCL Eprints - The Molecular Pathogenesis Of Hepatic Encephalopathy
    hepatic encephalopathy (HE) incorporates a spectrum of neuropsychiatric abnormalitiesseen in patients with liver dysfunction with a potential for full  
    http://eprints.ucl.ac.uk/archive/00000312/
    
    Extractions: Help Jalan, R. and Shawcross, D.L. and Davies, N. The molecular pathogenesis of hepatic encephalopathy. International Journal of Biochemistry and Cell Biology . ISSN Type: Article Status: Published Refereed: Yes Publisher version: http://www.sciencedirect.com/science/journal/13572725 Keywords: Hepatic encephalopathy; ammonia; cirrhosis; acute liver failure; hypothermia UCL Eprints classification: UCL Departments and Research Centres Faculty of Clinical Sciences Department of Medicine Institute of Hepatology Deposited on: 10 February 2005 Archive Staff Only: edit this record

59. UCL Eprints - Dispelling Myths In The Treatment Of Hepatic Encephalopathy
    Context Guidelines for the treatment of hepatic encephalopathy suggest Where next Two approaches to new therapies for hepatic encephalopathy are  
    http://eprints.ucl.ac.uk/archive/00000302/
    
    Extractions: Help Shawcross, D.L. and Jalan, R. Dispelling myths in the treatment of hepatic encephalopathy. The Lancet . ISSN PDF - Requires Adobe Acrobat Reader or other PDF viewer. PDF - Requires Adobe Acrobat Reader or other PDF viewer. Other .tif Type: Article Status: Published Refereed: Yes Publisher version: Additional information: Table and figure accompany text as separate files. UCL Eprints classification: UCL Departments and Research Centres Faculty of Clinical Sciences Department of Medicine Institute of Hepatology Deposited on: 08 February 2005 Archive Staff Only: edit this record

60. Hepatic Encephalopathy
    Dr. Marcos Pedrosa s lecture given at 2004 Advanced Liver Disease Training Program.This content is intended for healthcare providers.  
    http://hepatitis.va.gov/vahep?page=cfny04-08

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