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Graves Disease: more books (100)

Woman and her diseases, from the cradle to the grave: Adapted exclusively to her instruction in the physiology of her system by Edward H Dixon, 1846

Remarks on Graves' disease by John Arvid Octerlony, 1873

Diffuse hyperplasia of the thyroid gland (Graves' disease).(PATHOLOGY CLINIC)(Clinical report): An article from: Ear, Nose and Throat Journal by Lester D.R. Thompson, 2007-11-01

Loss of motility predicts vision loss in Graves'.(Endocrinology): An article from: Internal Medicine News by Bruce Jancin, 2004-12-01

Celiac disease occurring in a patient with hypoparathyroidism and autoimmune thyroid disease.(Case Report): An article from: Southern Medical Journal by Hasnain M. Khandwala, Rajni Chibbar, et all 2006-03-01

Improved therapies available for Graves' ophthalmopathy: follow-up of adjunctive IV steroids.(Clinical Rounds): An article from: Family Practice News by Damian McNamara, 2004-04-01

Eye and Orbit in Thyroid Disease by Colum A. Gorman, Robert R. Waller, 1984-11

Graves' Ophthalmopathy: Developments in Diagnostic Methods and Therapeutical Procedures (Developments in Ophthalmology) by C. Renate Pickardt, 1989-10

The Genetics of Complex Thyroid Diseases

Basedow's disease; by Hubert Sattler, 1952

Three rickettsioses, Darnley Island, Australia.(DISPATCHES): An article from: Emerging Infectious Diseases by Nathan B. Unsworth, John Stenos, et all 2007-07-01

Pain-free exercise training for people with peripheral vascular disease?: An article from: Palaestra by Christine Stopka, Rachel Wolper, et all 1998-03-22

Foods in health and disease, by Lulu G Graves, 1932

Carotid Disease: The Role of Imaging in Diagnosis and Management

lists with details

Graves Disease
Elaine is the author of graves disease, A Practical Guide. I haven t got my personal graves disease Journal up here yet, but I am working on getting
http://valkyrie.aarg.net/

Extractions: Friends with Graves Elaine Elaine is the author of Graves Disease, A Practical Guide. Elaine also has regular articles published at Suite 101 Pam Pam is a faithful contributor to the support group at Yahoo! Groups here is another great group at the Mediboard Network. Graves Disease: A practical Guide By: Elaine Moore The Thyroid Solution By Dr. Ridha Arem Buddhism Without Beliefs: A Contemporary Guide to Awakening By: Stephen Batchelor Mary's Shomon's Thyroid site on about.com Amercian Autoimmune Related Diseases Association" Elaine's column at Suite 101 Welcome to my new site, it has a better look and I have finally been able (mentally etc.) to face the work of the overhaul. I haven't got my personal Graves Disease Journal up here yet, but I am working on getting that back as well. To read the old site and my journal go here This site is meant to be a gateway. There is SO MUCH information about Graves Disease and I can not put it all here. I am just hoping to provide a place to start. One thing is, if you have been diagnosed, your best weapon is reading, reading and more reading! Any knowledge that you can gather about how this disease works and how to treat will help you in the long run. The doctors that generally treat Graves are called Endocrinologists. Many times they specialize in Diabetes, so they may not have all the newest information about Graves. This disease is not simple and it may be up to you to help educate your Doctor so you can receive the best treatment possible. This disease (and many others I'm sure) require that you be proactive in the healing process.
Joint Program In Nuclear Medicine
Graves disease has been estimated to occur in 0.4% of the population of the United The usual surgical treatment of graves disease consists of subtotal
http://www.med.harvard.edu/JPNM/TF94_95/Sept13/WriteUpSept13.html

Extractions: A 27 year old female presented with symptoms and signs of hyperthyroidism. Thyroid function tests confirmed the diagnosis of hyperthyroidism. Thyroid scan with uptake was performed to determine the etiology of the hyperthyroidism. Thyroid scintigraphy (RAO, anterior and LAO views, 14k bytes) shows an enlarged gland with a focal region of decreased uptake in the right lower pole ( thick arrows , 14k bytes). The is visualization of a pyaramidal lobe (thin arrows). The thyroid uptake was markedly elevated at 72%. Because of the hypofunctioning right lower pole nodule, a total right thyroidectomy and a subtotal left thyroidectomy was performed. Pathology showed a follicular adenoma with atypical characteristics and thyroid hyperplasia. Caleb Hillier Parry, a private practitioner in Bath, England, saw his first patient with a diffuse goiter and hyperthyroidism in 1786. His report of eight patients, however was not published until 1825, which was 3 years after his death (1). In 1835, Robert James Graves in Dublin described the same disease in six pregnant women. The disease was named after him in both the former British empire and in the United States (2). Carl A von Basedow described the disease a third time in three women in 1840 (3) and his name rather than Graves is applied to the disease in Europe. Graves' disease has been estimated to occur in 0.4% of the population of the United States with a lifetime risk of 1%. It is most commonly manifest in the third or fourth decade of life and the female to male ratio is 7:1 to 10:1 in published series.
Graves Disease
Graves disease is a thyroidspecific autoimmune disorder in which the body makes The autoantibodies produced in Graves disease are not subject to
http://www.bio.davidson.edu/Courses/Immunology/Students/Spring2003/Breedlove/Gra

Extractions: This web page was created for an undergraduate assignment at Davidson College Graves' disease is a thyroid-specific autoimmune disorder in which the body makes antibodies to the thyroid-stimulating hormone receptor (TSHR), leading to hyperthyroidism, or an abnormally strong release of hormones from the thyroid gland. Normally, the release of thyroid hormones is mediated by thyroid-stimulating hormone (TSH), a hormone secreted by the pituitary gland that binds to TSHR to stimulate the thyroid to release thyroid hormones. This normal cycle is self-regulating: the hormones secreted by the thyroid keep more TSH from being produced (Janeway, 2001). The autoantibodies produced in Graves' disease are not subject to negative feedback, so they continue to be produced and bind to TSHR even when thyroid hormone levels rise too high. These antibodies act as agonists, stimulating more hormones to be released and thus leading to hyperthyroidism. Figure 1: Both TSH and autoantibodies can stimulate the TSHR on thymocytes
My Personal Battle With Autoimmune Thyroid Disease (Graves Disease And Hashimoto
A personal story about fighting Graves disease and Hashimoto s thyroiditis.
http://www.webmosaics.com/thyroid/

Extractions: Below is the story (a work in progress) of my journey to good health and the paths I have chosen to follow to get there. ( Want the short version? Click here. ) I'm writing this for my sanity, as writing lets me release and heal, and I'm publishing it online in the hope that it might help someone else in their journey. You may need an open mind to read this as it turns out that many of my choices are not the traditional ones. I don't know if I've been unhealthy all my life, but looking back, I suspect I have been so on and off - for much of my adult life anyway. I remember taking a photography class and realizing that my hands shook a lot. Whenever I'd asked doctors about being tired all the time or feeling swollen after a workout, they've all said everything was normal. Maybe I should slow down and not work so much were the suggestion I got from a couple of them; it was true, I did work a lot and played hard too.
Introduction: Graves Disease - WrongDiagnosis.com
Introduction to graves disease as a medical condition including symptoms, diagnosis, misdiagnosis, treatment, prevention, and prognosis.
http://www.wrongdiagnosis.com/g/graves_disease/intro.htm

Extractions: Graves Disease: Graves' disease is one of the most common autoimmune diseases, affecting 13 million people and targeting women seven times as often as men. Patients with Graves' disease produce an excessive amount of thyroid hormone. Researching symptoms of Graves Disease: Further information about the symptoms of Graves Disease is available including a list of symptoms of Graves Disease , other diseases that might have similar symptoms in differential diagnosis of Graves Disease , or alternatively return to research other symptoms in the symptom center Misdiagnosis and Graves Disease: Research more detailed information about misdiagnosis of Graves Disease failure to diagnose Graves Disease underlying causes of Graves Disease (possibly misdiagnosed), or research
Graves Disease
Extensive information about the eye problems (ophthalmopathy) associated with both Graves disease and autoimmune thyroiditis.
http://www.eyeinstitute.net/graves.html

Extractions: For more than 150 years, conditions known as Graves Disease and chronic autoimmune thyroditits have been associated eye problems (ophthalmopathy). In 1825, Caleb Parry described a condition of goitre and eye protrusion. In 1835, Robert Graves wrote about cases of thyroid disease and proptosis (protrusion) of the eye, and this form of hyper-thyroidism was named after him. In 1840, Carl von Basedow noted swelling of tissues around the eye in patients with hyperthyroidism. Although considerable research is being done, it is not yet known why the eye is involved in thyroid conditions. Grave's ophthalmopathy focuses on the muscles around the eye, which help position the eye within the bony orbit or cavity where it rests on a bed of fat. This fat and the tear gland can also be affected in Grave's Disease although to a lesser degree. More often, apical enlargement of the eye muscles stimulates other fibroblasts. Fluid is drawn into the muscle and stored, eventually leading to the development of scar tissue. In addition, the swelling of the muscles pushes the eye forward causing it to protrude. As a result patients may have trouble closing their eyes, which causes drying, irritation and increased inflammation. Increased effort to bring the eyelids together may result in a frown, or swollen muscles may compress the optic nerve leading to loss of vision if treatment is not sought early on.
Graves Disease: Definition, Symptoms, And Treatment - Kellogg Eye Center
graves disease defined, description of symptoms, and information on treatment
http://www.kellogg.umich.edu/patientcare/conditions/graves.disease.html

Extractions: Graves disease (hyperthyroidism) is a medical condition in which an overactive thyroid gland secretes an excess amount of thyroid hormone. As a result, you may have an enlarged thyroid gland, palpitations, fast pulse, profuse sweating, high blood pressure, irritability, and a number of symptoms related to your eyes. Symptoms of hyperthyroidism include fatigue, fast heartbeat, weight loss, heat intolerance, fine hair and diarrhea. Hypothyroidism may also cause fatigue, but with slow heartbeat, constipation and weight gain. Note that symptoms of thyroid eye disease may be present even when tests show a normal level of thyroid hormone in the blood (called euthyroid). However, most patients with eye symptoms have abnormal blood levels of thyroid hormone. Symptoms A "staring" appearance Dry eyes Swelling (edema) of the eyelids and tissue around the eye Double vision
Hyperthyroidism (Graves'' Disease)
Detailed information on hyperthyroidism (graves disease), including cause, symptoms, diagnosis, and treatment.
http://www.healthsystem.virginia.edu/uvahealth/peds_diabetes/graves.cfm

Extractions: [ Skip Navigation ] Topics Adolescent Medicine Allergy/Asthma/Immunology Blood Disorders Burns Cancer Common Injuries/Poison Tips Craniofacial Anomalies Dermatology Diabetes/Endo/Metabolism Eye Care Genitourinary/Kidney High-Risk Newborn High-Risk Pregnancy Infectious Diseases Medical Genetics Mental Health Nervous System Disorders Normal Newborns Oral Health Orthopaedics Pediatric Surgery Respiratory Disorders Terminal Illness in Children Transplantation MAKE AN APPOINTMENT ... CLINICAL TRIALS Search This Site Hyperthyroidism means overactivity of the thyroid gland, resulting in too much thyroid hormone in the bloodstream. The over-secretion of thyroid hormones leads to overactivity of the body's metabolism. In newborns, the most common cause of an overactive thyroid is called neonatal Graves disease, which can be life threatening. However, hyperthyroidism rarely occurs in children and adolescents. What causes hyperthyroidism?
Thyroid Conditions And Graves Disease - Q & A
A range of questions on thyroid conditions and their effects on other metabolic functions. Our experts provide the answers.
http://www.betterhealth.vic.gov.au/bhcv2/bhcarticles.nsf/pages/Thyroid_condition
Graves Disease Definition - Allergies: Allergy Symptoms, Treatment, And Medicati
Allergy information includes articles on allergies, symptoms, treatment, medications, food allergies, and allergy relief.
http://www.medterms.com/script/main/art.asp?articlekey=3634
Graves Disease / Family Village Library
Established in 1990, the National Graves Disease Foundation is the only national, This list is for people with Graves Disease and their family and
http://www.familyvillage.wisc.edu/lib_grav.htm

Extractions: Website: http://www.ngdf.org/ Established in 1990, the National Graves' Disease Foundation is the only national, lay, nonprofit, educational organization open exclusively to people with Graves' disease, their families, friends and health care professionals. The NGDF offers a multi-disciplinary approach to a complex problem. Representatives of all related disciplines are included on the Medical and Advisory board: Endocrinology, Surgery, Nursing, Opthalmology, Dermatology, Psychiatry, Psychology, Pharmacology, Internal Medicine, Family Practice and Graves' disease patients. The National Graves' Disease Foundation is dedicated to increasing the public awareness of the disease and providing support and information to people with the illness. Graves Disease Web Board This list is for people with Graves' Disease and their family and friends who are interesting in supporting them. Topics for discussion can include problems people are experiencing, their chosen course of treatment, current medical studies and possible alternative methods of treatment. This list is not intended to replace expert medical advice and treatment, but to allow folks to discuss various aspects of Graves' with others.
Graves-Basedow Disease - Wikipedia, The Free Encyclopedia
Also known in the Englishspeaking world simply as Graves disease, it occurs most Medical treatment of graves disease includes antithyroid drugs,
http://en.wikipedia.org/wiki/Graves_Disease

Extractions: You did it! Over US$240,000 was donated in the 21 day fund drive. Thank you for your generosity! You are still welcome to make a donation or purchase Wikimedia merchandise (Redirected from Graves Disease Graves-Basedow disease is a form of thyroiditis , an autoimmune disorder that stimulates the thyroid gland, being the most common cause of hyperthyroidism (overactivity of the thyroid). Also known in the English-speaking world simply as Graves' disease , it occurs most frequently in women (8:1 compared to men) of middle age. Symptoms include fatigue weight loss and rapid heart beat . Because similar antibodies to those stimulating the thyroid also affect the eye, eye symptoms are also commonly reported. Treatment is with medication that reduces the production of thyroid hormone (thyroxin), or with radioactive iodine if refractory. edit Graves-Basedow disease is a disorder characterized by a triad of hyperthyroidism goitre , and exophthalmos (bulging eyeballs).
Lifetimetv.com: Strong Medicine - Fact Sheet : Graves Disease
In Graves disease, a form of hyperthyroidism, the immune system produces Graves disease was named for the Irish physician Sir Robert Graves,
http://www.lifetimetv.com/shows/strongmed/fact/fact1_21.html

Extractions: Graves' disease occurs in less than one-quarter of 1 percent of the population, and every year about a half a million new cases are diagnosed. It is four to eight times more common in women than men and typically begins between the ages of 20 and 40, although it can occur in children and the elderly. While the exact cause of Graves' disease is not known, a genetic disposition to autoimmune disorders seems to be needed in order to develop the disease. Other factors that contribute to the development of Graves' disease include heredity, one's immune system, age, sex hormones and stress. Many thyroid specialists believe that Graves' disease may have its onset after an external stressor (such as death in the family), a viral infection or pregnancy. Although Graves' disease is not contagious, it has been known to occur coincidentally with husbands and wives. Symptoms Like other thyroid problems, Graves' disease usually begins slowly. Symptoms include fatigue, weight loss, restlessness, rapid heartbeat, muscle weakness (especially involving your upper arms and thighs), heat intolerance, tremors, enlarged thyroid gland, heart palpitations, increased sweating, nervousness and irritability, thinning hair, increased appetite, lighter menstrual periods and more frequent bowel movements. Graves' disease can also affect the eyes, usually an elevation of the upper eyelids, making the eyes appear larger. About 5 percent of patients, however, experience more serious problems in which the tissue behind the eyes becomes inflamed and swollen, making the eyes protrude. A few patients have blurred or double vision.
Hyperthyroidism (Graves Disease) - Lucile Packard Children's Hospital
In newborns, the cause of hyperthyroidism (also called graves disease) is a graves disease in adults is an autoimmune disorder characterized by the
http://www.lpch.org/DiseaseHealthInfo/HealthLibrary/diabetes/graves.html

Extractions: Endocrinologists Hyperthyroidism (Graves Disease) Hyperthyroidism means overactivity of the thyroid gland, resulting in too much thyroid hormone in the bloodstream. The over-secretion of thyroid hormones leads to overactivity of the body's metabolism. In newborns, the most common cause of an overactive thyroid is called neonatal Graves disease, which can be life threatening. However, hyperthyroidism rarely occurs in children and adolescents. In newborns, the cause of hyperthyroidism (also called Graves disease) is a mother who has or has had Graves disease herself. Graves disease in adults is an autoimmune disorder characterized by the production of antibodies that stimulate the thyroid gland. When a pregnant woman has these antibodies, they can cross the placenta and affect the fetus' thyroid gland. Graves disease in pregnant woman can result in stillbirth, miscarriage, or premature birth. The following are the most common symptoms of hyperthyroidism in a newborn. However, each baby may experience symptoms differently. Symptoms may include:
InterPro: IPR002167 Graves Disease Carrier Protein
graves disease carrier protein (GDC) is a protein of as yet uncharacterised IPR002167graves disease carrier protein 3151. ModBase MB_P16260 36-325
http://www.ebi.ac.uk/interpro/DisplayIproEntry?ac=IPR002167
Shedding Of Thyrotropin Receptor Subunit Causes Graves Disease
graves disease came to national attention in the early 1990s when former United States President and First Lady, George and Barbara Bush, developed the
http://www.eurekalert.org/pub_releases/2003-06/joci-sot061003.php

Extractions: Journal of Clinical Investigation Graves disease, the most common cause of goiter and hyperthyroidism in the United States, came to national attention in the early 1990s when former United States President and First Lady, George and Barbara Bush, developed the condition. The etiology of Graves disease is multifactorial, and nongenetic factors are thought to play an important role. Sandra McLachlan and researchers at Cedars-Sinai Medical Center in Los Angeles, California now show that it is an unusual structural feature of the thyrotropin receptor (TSHR) that plays a major role in the development of this disease. Graves disease is the result of the production of autoantibodies to the TSHR located on the surface of thyroid cells. These antibodies bind the TSHR and stimulate it to overproduce thyroid hormones, which results in hyperthyroidism. In the June 16 issue of the Journal of Clinical Investigation, Sandra McLachlan and her team report that studies in a new adenovirus-mediated animal model of Graves disease revealed that cleavage of the TSHR A subunit can induce or amplify the immune response to the TSHR to a greater extent than the intact TSHR molecule. The data, increase our understanding as to why autoantibodies specifically arise to the TSHR, stimulate the thyroid, and result in Graves hyperthyroidism. However, the mechanisms responsible for TSHR cleavage will require further exploration.
TWiki . Mygrid . GravesDisease
GravesStoryBoard Storyboard for the graves disease Demonstrator - A user GD_poster_for_EPSRC_e-Science_meeting.ppt graves disease NFKBIE poster for
http://twiki.mygrid.org.uk/twiki/bin/view/Mygrid/GravesDisease

Extractions: Mygrid Bioinformatics Technologies Main ... Search GravesDiseaseInterview GravesDiseaseScenario - An outline of a user scenario that involves computational approaches to discover genes important in Graves' disease and identify associated SNP's AlansAnnotationPipelineWalkThrough - A walk-through of two different approaches that biologists may take to answer the question, "Tell me everything there is to know about my gene", aka the annotation pipeline of the GravesDiseaseScenario GravesStoryBoard - Storyboard for the Graves Disease Demonstrator - A user perspective AnilWipat - 23 Jan 2003 GravesStoryBoardDetails - trying to nail down precise types, etc. ChrisGreenhalgh - 23 Apr 2003 GDScenarioReqs - visualization and interaction requirements for GD scenario - above with details of proposed implementation, types, etc. GDScenarioNetBeansReqs - requirements for NetBeans workbench distilled from the above. ChrisGreenhalgh - 29 Apr 2003 Attachment Action: Size: Date: Who: Comment: GDworkflows_1.pdf
Postgraduate Medicine: The Many 'faces' Of Graves' Disease
Describes the clinical manifestations and biochemical markers that aid in diagnosis as well as other disorders that may mimic graves'.
http://www.postgradmed.com/issues/1999/10_15_99/felz.htm

Extractions: Michael W. Felz, MD; Peter P. Stein, MD VOL 106 / NO 5 / OCTOBER 15, 1999 / POSTGRADUATE MEDICINE CME learning objectives This page is best viewed with a browser that supports tables Part 1 of this article, which illustrates the clinical "faces" of patients with Graves' disease, was published in the October 1, 1999, issue, page 57. Preview : Deceptive signs and symptoms of Graves' disease, a common thyrotoxic condition, sometimes lead to delayed diagnosis and misdirected management. In this article, Drs Felz and Stein blend clinical experience with a review of the literature to clarify bedside and differential diagnosis, laboratory findings, and selective management options for this multisystem autoimmune syndrome. Part 1 of this article, which illustrates the clinical "faces" of patients with Graves' disease, was published in the October 1 issue, page 57.
Graves' Disease Resource Page - Thyroid Disease Treatment Options, Information,
This graves disease website, authored by a facilitator for the National graves disease Foundation, contains indepth information on important medical
http://home.velocitus.net/deecee/

Extractions: Graves' Disease is a condition of autoimmunity primarily affecting the thyroid gland, and is the most common cause of hyperthyroidism (excess thyroid hormone). In some people the antibodies also affect the eyes, causing them to protrude from their orbits and/or retracting the eyelids, producing a buggy-eyed or "staring" appearance, and this sometimes results in double vision and other problems. Rarelyusually in people who also have obvious eye diseasethe skin over the shins and tops of feet is affected (pretibial myxedema). Even more rarely, the myxedema can occur on other skin areas. Since thyroid imbalances affect the function of every cell and every part of the body (including the brain), Graves' Disease presents with many physical and neuropsychological symptoms, which vary somewhat from one person to another. Depending on treatment choice, it can take many monthsand often several yearsto stabilize thyroid levels, and for the mind and body to return mostly to normal. Several studies have found that a percentage of patients have ongoing problems, even after successful treatment. Following treatment, many patients
Thyroid Eye Disease (Graves' Disease)
People with excess thyroid hormones (graves disease) can develop many eye problems. One characteristic symptom is a protrusion of the eye (exophthalmos),
http://www.steen-hall.com/graves.html

Extractions: The thyroid gland is located in the neck. It manufactures two thyroid hormones that are essential for metabolism and growth. The thyroid takes iodine from the food we eat and uses it to make thyroid hormones. If thyroid hormones are in short supply, a person feels sluggish, has a slow heartbeat, and can even feel depressed. Doctors call this condition hypothyroidism. If a newborn has hypothyroidism, the baby can develop a condition called cretinism. The baby may have slowed development and poor intelligence unless the condition is corrected.

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