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AJR -- Sign In Page Clin Radiol1993; 47137 138Medline; Moriwaka F, Tashiro K, Maruo Y, Nomura M,Hamada K, Kashiwaba T. MR imaging of pontine and extrapontine myelinolysis. http://www.ajronline.org/cgi/content/full/182/3/809
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Extractions: Clinical bottom line (level 4) Mortality was higher in patients with acute onset hyponatraemia or symptomatic hyponatraemia. The commonest causes of hyponatraemia were congestive heart failure, iatrogenic causes (fluid overload or diuretics) and SIADH. Arieff et al: Medicine 1976; 55 (2): 121-129 Outcomes studied: mortality with chronic symptomatic hyponatraemia mortality with asymptomatic hyponatraemia cause: congestive heart failure cause: SIADH cause: post-surgical (overtransfusion with 5% dextrose) fluid overload cause: dieuretic use usually due to diuretic use cause: chronic renal failure cause: acute renal failure
Extractions: This Article Alert me when this article is cited Alert me if a correction is posted Services Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal Download to citation manager ... Cited by other online articles PubMed PubMed Citation Articles by Atkin, S. Articles by Mathew, B European Journal of Endocrinology, Vol 135, Issue 2, 245-247 A female aged 53 years was found to have a suprasellar lesion, which was shown to be a Rathke's cyst after removal by transsphenoidal surgery. She presented 16 days postoperatively, and following two grand mal seizures was found to be profoundly hyponatraemic (sodium 101 nmol/l). She was initially thought to have the syndrome of inappropriate antidiuretic hormone and was treated accordingly, but central venous pressure measurement revealed the hypovolaemia of cerebral salt wasting syndrome. The patient subsequently developed severe neurological sequelae after the correction of her hyponatraemia, following the development of extrapontine myelinolysis. Cerebral salt wasting syndrome is a rare cause of hyponatraemia following pituitary transsphenoidal surgery, which may mimic the syndrome of inappropriate antidiuretic hormone secretion. This case emphasizes the poor prognosis that may result from the rapid correction of profound hyponatraemia.
Posters P13 Central extrapontine myelinolysis due to polydipsia causing basal ganglia extrapontine myelinolysis is a rare complication but may occur without http://laeknabladid.is/2002/fylgirit/14/nc/nr/130/
Extractions: Methods: 944 patients were admitted at the Department of Neurology, Bispebjerg Hospital with the diagnoses TIA (n=190), ischemic stroke (n=634) or hemorrhagic stroke (n=120). The patients` mean levels of s-hcy were compared in the three diagnoses, and in patients with recurrent stroke versus patients without recurrent stroke during the following year. Due to log normal distribution, data was analysed after log 10 transformation. A multivariate logistic regression model was set up after univariate analysis including variables at a significance level of 0.1. Results: Patients with an ischemic event (TIA or ischemic stroke) (n=824) had a mean s-hcy of 11.99 mmol/l (CI 95%: 11.64-12.39), patients with hemorrhagic stroke had a mean s-hcy of 10.52 mmol/l (CI 95%: 9.73-11.38). The difference between the two groups was significant, p=0.003. After one year we could document recurrent stroke in 44 cases, these patients had mean s-hcy levels at the primary admission of 14.29 mmol/l (CI 95%: 11.51-17.70) whereas patients without recurrent stroke had mean levels of s-hcy of 11.69 mmol/l (CI 95%: 11.53-12.27), p=0.005. S-hcy reached significance in multivariate logistic regression model.
The Neurologist - UserLogin Myelinolysis typically affects the pons but various extrapontine brain areas can be Salvesen R. extrapontine myelinolysis after surgical removal of a http://www.theneurologist.org/pt/re/neurologist/fulltext.00127893-200311000-0000
[Dysphagia] Central Pontine Myelinosis case that central pontine myelinolysis (CPM) and extrapontine myelipolysis (EPM)appeared Central pontine myelinolysis clinical and MRI correlates. http://list.dysphagia.com/dysphagia/2002-July/msg00357.html
OMIM ENTRY 277900 liver transplantation who developed postoperative central pontine and extrapontinemyelinolysis and then went on to develop new extrapyramidal symptoms http://www.eng.uiowa.edu/~tscheetz/sequence-analysis/examples/OMIM/omim_wilson.h
Extractions: ATPase, Cu(2+)-TRANSPORTING, BETA POLYPEPTIDE, INCLUDED; ATP7B, INCLUDED Gene Map Locus: 13q14.3-q21.1 Note: pressing the symbol will find the citations in MEDLINE whose text most closely matches the text of the preceding OMIM paragraph, using the Entrez MEDLINE neighboring function. In Wilson disease, the basal ganglia and liver undergo changes that express themselves in neurologic manifestations and signs of cirrhosis, respectively. A disturbance in copper metabolism is somehow involved in the mechanism. Low ceruloplasmin ( ) is found in the serum. Shokeir and Shreffler (1969) advanced the hypothesis that ceruloplasmin functions in enzymatic transfer of copper to copper-containing enzymes such as cytochrome oxidase. Supporting the hypothesis was the finding of markedly reduced levels of activity of cytochrome oxidase in Wilson disease and moderate reductions in heterozygotes. The Kayser-Fleischer ring is a deep copper-colored ring at the periphery of the cornea which is frequently found in Wilson disease and is thought to represent copper deposits.
Abstracts Of Papers Presented At The 97th Research Meeting Of He was also found to have hyponatraemia and MR Imaging revealed extrapontinemyelinolysis. Subsequently he had repeated episodes of autonomic dysfunction in http://bhj.org/journal/2003_4502_april/abstractsfrommrcbh_392.htm
Extractions: The Electrodiagnostic laboratory of Bombay Hospital is one of the pioneering centres in India, well equipped with pentium based state of art electromyograph machines. The department has a good infrastructure and a very good reputation to its credit. The continuous quality management is thus required to cater the quality services.
