Neurology -- Sign In Page Lateral pontine and extrapontine myelinolysis associated with Extrapontinemyelinolysis with involvement of the hippocampus in three children with http://www.neurology.org/cgi/content/full/64/3/574
Extractions: This Article Figures Only Full Text (PDF) Correspondence: ... Alert me if a correction is posted Services Email this article to a friend Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal ... Download to citation manager PubMed PubMed Citation Articles by van der Helm-van Mil, A. H.M. Articles by Harinck, H. I.J. Related Collections All Imaging
Neurosurgery - October 1998, Volume 43, Issue 4 extrapontine myelinolysis and Behavioral Change after Transsphenoidal PituitarySurgery Case Report. Masahiko Tosaka, MD; Hideaki Kohga, MD http://www.neurosurgery-online.com/pt/re/neurosurg/toc.00006123-199810000-00000.
Extractions: Abstract References (17) View full size inline images Transplantation Volume 61(4) 27 February 1996 pp 658-661 Fryer, Jonathan P. ; Fortier, Marielle V. ; Metrakos, Peter ; Verran, Deborah J. ; Asfar, Sami K. ; Pelz, David M. ; Wall, William J. ; Grant, David R. ; Ghent, Cameron N. Multiorgan Transplant Service and Department of Radiology, University Hospital, University of Western Ontario, London, Ontario, Canada Multiorgan Transplant Service. Department of Radiology. Address correspondence to Dr. Jonathan P. Fryer, Department of Surgery, Northwestern University, 303 East Superior, Suite 532, Chicago, IL 60611. Received 7 June 1995. Accepted 5 September 1995. Article Outline Figures/Tables In a recent series of 44 liver transplants we identified both extrapontine myelinolysis (EPM)-characteristic of cyclocosporine neurotoxicity-and central pontine myelinolysis (CPM) in 5 recipients posttransplant. An additional 2 recipients had EPM only posttransplant. MRIs performed in 4 asymptomatic recipients were normal. Large perioperative shifts in serum sodium, hypomagnesemia, and high cyclosporine levels may play a role in the development of these lesions, although the evidence from this study is inconclusive. In addition to supportive care, dilantin was started in patients who had seizures; aggressive magnesium replacement was initiated for hypomagnesemia, and cyclosporine levels were reduced in all patients. All patients demonstrated a slow steady recovery and all but 2 are home at the time of writing. CPM may be more prevalent than previously appreciated following liver transplantation, although its prognosis may not be as dismal.
Extractions: In a recent series of 44 liver transplants we identified both extrapontine myelinolysis (EPM)-characteristic of cyclocosporine neurotoxicity-and central pontine myelinolysis (CPM) in 5 recipients posttransplant. An additional 2 recipients had EPM only posttransplant. MRIs performed in 4 asymptomatic recipients were normal. Large perioperative shifts in serum sodium, hypomagnesemia, and high cyclosporine levels may play a role in the development of these lesions, although the evidence from this study is inconclusive. In addition to supportive care, dilantin was started in patients who had seizures; aggressive magnesium replacement was initiated for hypomagnesemia, and cyclosporine levels were reduced in all patients. All patients demonstrated a slow steady recovery and all but 2 are home at the time of writing. CPM may be more prevalent than previously appreciated following liver transplantation, although its prognosis may not be as dismal.
Extractions: HOME HELP FEEDBACK SUBSCRIPTIONS ... TABLE OF CONTENTS This Article Full Text (PDF) Submit a related Letter to the Editor Alert me when this article is cited ... Citation Map Services Email this article to a friend Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal ... Download to citation manager PubMed PubMed Citation Articles by Maghnie, M. Mohamad Maghnie Eugenio Genovese Stefan Lundin Federico Bonetti and Departments of Pediatrics (M.M., F.B., M.A.), Radiology (E.G.), University of Pavia, IRCCS Policlinico S. Matteo, Pavia, Italy; Department of Clinical Pharmacology (S.L.), Lund University Hospital, Lund, Sweden Address all correspondence and requests for reprints to: Mohamad Maghnie, Department of Pediatrics, University of Pavia, IRCCS Policlinico S.Matteo, 27100 Pavia. Italy. E-mail
Clinical Neuropharmacology - UserLogin CPM) or, rarely, other cerebral regions (extrapontine myelinolysis; EPM) (1) . Medlej R. Tamraz J. extrapontine myelinolysis treatment with TRH. http://www.clinicalneuropharm.com/pt/re/clnneupharm/fulltext.00002826-200003000-
Current Opinion In Neurology - UserLogin Abbreviations CT computed tomography EPM extrapontine myelinolysis CPM central Clinical features of central pontine and extrapontine myelinolysis TOP http://www.co-neurology.com/pt/re/coneuro/fulltext.00019052-200012000-00014.htm
World J Gastroenterol None of the patients showed extrapontine myelinolysis. central pontine andextrapontine myelinolysis. Acta Neurobiol Exp 2001; 61 141144 http://www.wjgnet.com/1007-9327/10/2540.asp
Extractions: MEDLINE Resumen. Summary Introduction and clinical case. We present a case of extra-pontine myelinolysis caused by acute hypernatraemia in which a complex picture of late onset extrapyramidal features, choreodystonia and parkinsonism developed. Repeated physical examinations, neurophysiological and neuroimaging studies using magnetic resonance all indicated an extra-pontine site of the lesions, which symmetrically affected the striate and to a lesser extent both thalami. We review the relevant literature available and analyze the cases described as having abnormal movements associated with a myelinolytic syndrome. Conclusions.
Current Opinion In Critical Care - UserLogin Osmotic demyelination central pontine myelinolysis and extrapontine myelinolysisTOP. Hyponatremia is a common complication of patients with liver disease http://www.co-criticalcare.com/pt/re/cocritcare/fulltext.00075198-199904000-0000
Instituto Nacional De Salud Translate this page Extrapontine osmotic myelinolysis is a rare nervous system complication. Central pontine and extrapontine myelinolysis the osmotic demyelination http://www.ins.gov.co/biomedica/ver_articulo.php?id_articulo=41
UCSF - Neuroradiology Interesting Case: December, 2001 patchy and asymmetric lesions); extrapontine myelinolysis (requires anappropriate clinical context; often associated with central pontine myelinolysis) http://www.neurorad.ucsf.edu/previouscases/12012001.html
Extractions: 167 cases, over 1000 figures at $139.00. Six months prior to presentation, this 13 year old girl developed fatigue and weight loss. She then presented with a generalized seizure and lapsed into coma for three days. Upon awakening, she was disoriented and inappropriate. An MR scan was obtained. Fig A Fig B Fig C Fig D Reversibility of T2-prolonging lesions has been documented, suggesting that they represent areas of edema and possibly microinfarction.
[Dysphagia] CPM extrapontine myelinolysis symmetrically affects thalamocapsular regions, neostriatum,and other territories. Myelinolysis usually results from the rapid http://list.dysphagia.com/dysphagia/2002-July/msg00370.html
Extractions: This Article Abstract Figures Only Full Text (PDF) ... Citation Map Services Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal Download to citation manager PubMed PubMed Citation Articles by Ruzek, K. A. Articles by Miller, G. M. American Journal of Neuroradiology 25:210-213, February 2004
Extractions: This Article Alert me when this article is cited Alert me if a correction is posted Services Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal Download to citation manager ... Cited by other online articles PubMed PubMed Citation Articles by Ho, V. B. Articles by Geyer, C. A. VB Ho, CR Fitz, CC Yoder and CA Geyer Department of Radiology, Walter Reed Army Medical Center, Washington, DC 20307-5001. Osmotic myelinolysis is a distinctive clinical syndrome with characteristic MR features in the central pons (central pontine myelinolysis) and in other locations (extrapontine myelinolysis). We describe the resolving MR features in an adolescent who has experienced complete neurologic recovery. Regions of involvement manifested increased T2 signal intensity. The extrapontine involvement was noted to resolve earlier with interim-increased T1-weighted signal. The mechanism for the variable
Accessing Article Menger H, Jorg J. Outcome of central pontine and extrapontine myelinolysis.J Neurol 1999; 246 700705. Article PubMed ChemPort http://www.nature.com/bmt/journal/vaop/ncurrent/full/1705115a.html
Extractions: Most users gain access to full text articles through a site license. This is available to institutional customers only. For further information visit the Librarian Gateway. Full-text articles are also available through a number of other options: I want to purchase this article Select this option to view this article immediately and access it for seven days. I am already a personal subscriber to Bone Marrow Transplantation Personal subscribers to Bone Marrow Transplantation can view this article. To do this you need to associate your subscription with your registration via the My Account page. If you already have an active online subscription, log in via the login box in the top right-hand corner of this page.
Log In Problems Central pontine and extrapontine myelinolysis associated with type 2 diabeticpatient with hypokalemia. Diabetes Res Clin Pract. 2005;687580. http://www.medscape.com/viewarticle/505496_print
Log In Problems in the splenium were first described in Italian red wine drinkers but havebeen documented in cases of malnutrition and extrapontine myelinolysis. http://www.medscape.com/viewarticle/505496_3
