HerbChina2000.com - Herbal Remedies - Ptosis Of Eyelid neurological disease, including myasthenia gravis, chronic progressive externalophthalmoplegia, Horner s syndrome, third cranial nerve weakness, etc.. http://www.herbchina2000.com/therapies/XPD.shtml
Extractions: Help Center View Basket Checkout Ptosis of eyelid DESCRIPTION Ptosis is also called "drooping eyelid." It is caused by weakness of the muscle responsible for raising the eyelid, damage to the nerves which control those muscles, or laxity of the skin of the upper eyelids. Drooping eyelid can be caused by the normal aging process, a congenital abnormality (present before birth), or the result of an injury or disease. Ptosis is of two types, namely congenital ptosis and acquired ptosis. Congenital ptosis is more common than acquired ptosis. Ptosis may involve one or both the eyes and ranges from mild to severe. Congenital Ptosis - Present at birth, the reason for congenital ptosis is the poor development of the eyelid lifting muscle called the levator muscle. A child may have only ptosis or may have other associated eye abnormalities like eye movement disorders, refractive disorders and neurological disorders. Acquired Ptosis - Acquired ptosis may occur at any time after birth, but more commonly in old age. The usual cause for this is weakening of the eyelid lifting muscle, the levator. Weakening of the levator muscle and the resultant droop of upper eyelid may occur due to factors like old age, injury, surgery, muscular disease, and neurological disease, including myasthenia gravis, chronic progressive external ophthalmoplegia, Horner's syndrome, third cranial nerve weakness, etc..
Extractions: This Article Full Text FREE Full Text (PDF) Alert me when this article is cited ... Alert me if a correction is posted Services Email this article to a friend Similar articles in this journal Similar articles in ISI Web of Science Similar articles in PubMed ... Cited by other online articles Search for citing articles in: doi: 10.1093/brain/awg101 Simone Bosbach Cornelia Kornblum and Michael Wagner Department of Psychiatry and Department of Neurology, University of Bonn, Germany Correspondence to: PD Dr Michael Wagner, Neuropsychology Laboratory, Department of Psychiatry, University Hospital of Bonn, Sigmund-Freud-Str. 25, 53105 Bonn, Germany E-mail: *These two authors contributed equally to this work Although neuropsychological deficits have been reported in mitochondrial cytopathies, patients with chronic progressive external ophthalmoplegia
