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Cerebellar Diseases:     more books (29)

Cerebellar Disorders: A Practical Approach to Diagnosis and Management Handbook of Cerebellar Diseases (Neurological Disease and Therapy) Cerebellar Infarct Midline Tumors Minimally Invasive Endoscopic Neurosurgery (Advances in Neurosurgery) by Bernhard Ludwig Bauer, M. Brock, 1994-04 Parenchymatous Atrophy of the Cerebellum: A Contribution to the Symptomatology of Intrinsic Cerebellar Disease by La Salle Archambault, 1918-01-01 Comparison of patients with Parkinson's disease or cerebellar lesions in the production of periodic movements involving event-based or emergent timing [An article from: Brain and Cognition] by R.M.C. Spencer, R.B. Ivry, 2005-06-01 Cerebellar functions (Nervous and mental disease monograph series) by André Thomas, 1912 The localisation of cerebral disease: Being the Gulstonian lectures of the Royal College of Physicians for 1878 by David Ferrier, 1879 Cerebellar abscess secondary to ear disease: Illustrated by a case successfully treated by operation : with remarks on diagnosis, and with a table of published cases by Theodore D Acland, 1896 Cerebellar abscess secondary to ear disease, by Charles Alfred Ballance, 1896 Cerebellar Functions. Nervous and Mental Disease Monograph Series No. 12 by Andre Thomas] Andre-Thomas [ie, 1912-01-01 Malignant pleural mesothelioma with scalp, cerebellar, and finger metastases: a rare case.(Disease/Disorder overview): An article from: Southern Medical Journal by Asiye Kanbay, Kivilcim Ipek Oguzulgen, et all 2007-01-01 CURRENT Diagnosis & Treatment in Neurology (LANGE CURRENT Series) by John Brust, 2006-09-14 Speech motor programming in hypokinetic and ataxic dysarthria [An article from: Brain and Language] by K.A. Spencer, M.A. Rogers, Hemangiomas of cerebellum and retina (Lindau's disease): With the report of a case by Harvey Cushing, 1928

lists with details

81. Physical Diagnosis Table Of Physical Findings
    cerebellar Disease. Roth s Spots, FUNDUSCOPIC Retinal hemorrhages with pale orwhite centers. cerebellar Disease. Tophi, Nodules of urate deposits,  
    http://www.geocities.com/doctor_uae/pdx_table_of_findings.htm
    
    Extractions: Return to Physical Diagnosis Download a copy of this study guide Finding Description, Comments Associated Disease(s) Adie's Pupil Similar to Argyll Robertson Pupil, except that accommodation is also impaired. May also see impaired deep tendon reflexes. Adie's Syndrome Angioid Streaks FUNDUSCOPIC: Pigmented lines radiating outward from the optic disc Pseudoxanthoma Elasticum Angiokeratomas Purplish, red papules, on lower abdomen, groin, or scrotum. Fabry's Disease : Hereditary Glycolipid Lipidosis. Arcus Senilis Gray band of opacity around the cornea., a normal finding with aging. Argyll Robertson Pupils No pupillary light reflex, but accommodation is intact. Neurosyphilis Argyll Robertson Pupil (1) Weak or absent direct pupillary reflex, (2) Retained ability to accommodate for near vision, (3) Failed pupillary dilation after atropine administration. Tabes Dorsalis (Neurosyphilis) Arteriovenous (AV) Nicking FUNDUSCOPIC: Arteriolar narrowing and compression of veins, where the arteries cross the veins. Due to sclerotic changes in both arteries and veins.

82. Neurobiology Of Disease Journal Entry 9
    disease maps to the same 3cM interval as the spinal cerebellar ataxia 3 This interval also contains the spinal cerebellar ataxia 3 (SCA3) gene,  
    http://www.neuro.wustl.edu/journal/jrnl9.htm
    
    Extractions: The gene for Machado-Joseph disease maps to the same 3-cM interval as the spinal cerebellar ataxia 3 gene on chromosome 14q G. Stevanin: , P. S. Sousa: , G. Cancel: , A. Durr: , 0. Dubourg: , G. A. Nicholson: , J. Weissenbach: , E. Jardim: , Y. Agid: , E. Cassa: , A. Brice: )INSERM U289, Hopital de la Salpetriere, Paris, France, )Departamento de Neurologia, Hospital das Clinicas, Universidade de Sao Paulo, Ribeirao Preto, Sao Paulo, Brasil, )Department of Medicine, Molecular Genetics and Medicine Laboratory, University of Sydney, Concord Hospital, New South Wales, Australia and )Genethon, Evry, France Correspondence: Dr Alexis Brice, INSERM U289, Hopital de la Salpetriere, 47 Bd de I'Hopital, 75651 Parix Cedex 13, France. Fax: 00/33/l/44.24.36.58. Machado-Joseph disease (MJD) is an autosomal dominant neurodegenerative disorder in families of Portuguese-Azorean ancestry. The gene responsible for MJD has been assigned to a 29-cM interval on chromosome 14q. A large Brazilian family with MJD was genotyped with six new microsatellite markers spanning 19 cM on chromosome 14q. Linkage analysis and haplotype reconstruction reduced the MJD candidate region to a 3-cM interval between markers D14S280 and D14S81, permitting positional cloning. This interval also contains the spinal cerebellar ataxia 3 (SCA3) gene, responsible for a genetic subtype of the type I autosomal dominant cerebellar ataxias, clinically related to MJD. This result supports the hypothesis that abnormalities in the same gene may be responsible for both disorders. The minor clinical differences between the two diseases may result from allelic heterogeneity.

83. Cognitive Impairments In Cerebellar Degeneration A Comparison
    While patients with cerebellar disease have exhibited a variety of cognitive andaffective impairments,10 their disordered executive functioning is  
    http://neuro.psychiatryonline.org/cgi/content/full/16/2/176

84. Ataxia, Marie's
    Hereditary cerebellar Ataxia; Nonne s Syndrome; PierreMarie s Disease This disease entry is based upon medical information available through the  
    http://webcenter.health.webmd.netscape.com/hw/brain_nervous_system/nord403.asp
    
    Extractions: Marie's Ataxia is an inherited disorder of impaired muscle coordination usually beginning during young adulthood or middle age. This hereditary form of ataxia is characterized by unsteady walking. Nerve degeneration and muscle atrophy in the legs, head and neck area and arms may occur. Cases that begin later in life may be mild with symptoms that can often be treated successfully.

85. Sociedade Brasileira De Geriatria E Gerontologia
    It differs from primary cerebellar disease by the slowness of movement and absence of Other features of cerebellar disease include an impaired checking  
    http://www.sbgg.org.br/profissional/artigos/neurological3.htm
    
    Extractions: Familiarity with the common causes of abnormal gait in the elderly is important in helping the clinician to focus the neurological examination.4,9-13 The most frequent etiologies in 120 elderly outpatients referred to a neurologist for undiagnosed gait disorder6 and in another series of 50 patients admitted to a neurology service with gait impairment14 are shown in Table 1. Up to one-third of patients had a potentially treatable cause for the gait disorder in these series.14,15

86. Excitotoxic Aspects Of Lithium NeurotoxicityExcitotoxic Aspects Of Lithium Neuro
    cerebellar disease may not be prominent during the most acute Evidence offocal cerebellar disease secondary to lithium has been documented with  
    http://psycprints.ecs.soton.ac.uk/perl/local/psyc/makedoc?id=524&type=html

87. West EXCITOTOXIC ASPECTS OF LITHIUM NEUROTOXICITY The Article
    Use of a cerebellar disease model (produced by neurotoxins like harmaline) Evidence of focal cerebellar disease secondary to lithium has been documented  
    http://psycprints.ecs.soton.ac.uk/archive/00000524/02/psyc.96.7.32.lithium-toxic

88. Hospital Tour
    cerebellar Disease Cause unknown. Spinal Cord Disease Cause unknown. Parkinson s Disease, Multiple Sclerosis and cerebellar Disease  
    http://www.kznhealth.gov.za/medicine/referral.htm
    
    Extractions: Hospital Tour Home Site Map Clinic / Unit Referral Criteria Endocrine Clinic Lipid Clinic Neurology clinic Ward D1 - Cardiac ... Ward H2 - Infectious Unit Endocrine Clinic Any patient with an endocrine disorder e.g. Pituitary problem, adrenal gland problem, thyroid glad problem, bone abnormalities or growth abnormalities. The patient must be referred with a detailed referral letter, all necessary blood results and other special investigations e.g. X-rays attached. An appropriate appointment date will then be given. Lipid Clinic Cholesterol more than 7 and/or triglyceride more than 3, not responding to 10 mg daily of atorvastatin. The patient must be referred with a detailed referral letter, all necessary blood results including a recent lipogram attached. An appropriate appointment date will then be given. Neurology Clinic Epilepsy - Poorly controlled/Intractable - Newly diagnosed requiring investigation.

89. Login To BioOne
    diseases affecting the cerebellum often result in ataxia and intention tremors . Several disease processes involving the avian cerebellum have been  
    http://www.bioone.org/bioone/?request=get-document&issn=1082-6742&volume=017&iss

90. Department Of Medicine – Alfred Hospital
    movement recordings and tapping regularity in cerebellar disease. project would investigate the reliability of these measures in cerebellar disease,  
    http://www.med.monash.edu.au/medicine/alfred/research/deptmed.html
    
    Extractions: Location: Dept of Medicine, Monash Medical School, Alfred Hospital Description: Cardiac transplantation is currently the therapy of choice for end-stage heart disease resistant to other medical or surgical therapy. The main cause of graft and patient loss after the first post transplant year is cardiac allograft vasculopathy. This project is part of an extensive prospective longitudinal study of endomyocardial biopsies from patients with cardiac allografts. The aim of the study is to analyse specific features of biopsies during the first year post transplantation, to determine their value as predictive factors for the subsequent development of cardiac allograft vasculopathy. The factors to be examined include fibroblast growth factor-2, fibroblast growth factor -9, plasminogen activator inhibitor-1, vascular endothelial growth factor and genes of the renin-angiotensin and endothelin systems.

91. TOLUENE AND OPTIC ATROPHY MEDLINE Rinsho Shinkeigaku 1992 Apr
    cerebellar disease is usually associated with toluene exposure; in the more severecases there is often radiological evidence of irreversible cerebellar  
    http://www.webshells.com/medsrch/toluatrf.txt
    
    Extractions: ==TOLUENE AND OPTIC ATROPHY== MEDLINE Rinsho Shinkeigaku 1992 Apr;32(4):421-425 [A case of chronic toluene intoxication with atrophy of cerebrum, cerebellum and brainstem on CT and MRI]. [Article in Japanese] Fujita K, Koga Y, Takemasa K, Koike H, Akai J Department of Neuropsychiatry, School of Medicine, Kyorin University. A 28-year-old man developed neurological disorders consisting of cerebellar symptoms and dementia over a period of 8 years of inhalation of toluene. He also developed bilateral optic atrophy. CT scan and MRI revealed atrophy of cerebrum, cerebellum and brainstem. The severity of neurological symptoms corresponded with the findings of CT and MRI. Furthermore, MRI (T2) showed reduced signal intensity in bilateral thalamus. This patient was also admitted to another hospital 2.5 years ago. Compared with the previous findings, present examinations showed significant progress of the atrophy of cerebrum and brainstem. It was suggested from the results of ABR that the disturbance of the brainstem was aggravated through this period. Acta Neurol Scand 1985 Nov;72(5):512-517 Nervous system effects of long-term occupational exposure to toluene. Juntunen J, Matikainen E, Antti-Poika M, Suoranta H, Valle M Forty-three male rotogravure printers with long-term toluene exposure and 31 age- and sex-matched offset printers without toluene exposure were examined in detail. Clinical, neurophysiological, neuropsychological and neuroradiological examinations and assessment of autonomic functions did not reveal any statistically significant differences between the groups. The results suggest that occupational long-term exposure to toluene under these circumstances does not have clinically significant adverse effects on the nervous system. Exposure to toluene seemed to be associated with heavy drinking. Arq Neuropsiquiatr 1994 Mar;52(1):90-92 Cerebellar atrophy related to chronic exposure to toluene. Case report. Damasceno BP, de Capitani EM Faculdade de Ciencias Medicas (FCM), Universidade Estadual de Campinas (UNICAMP), SP, Brasil. A 31-year-old woman presented slowly progressing ataxia and neurasthenic symptoms after 14-year occupational exposure to low concentration toluene vapour. Examination disclosed only cerebellar signs. Cognitive functions were normal except moderate visuo-spatial and constructive deficit. CT imaging showed severe pancerebellar atrophy without pathological signs in other brain structures. Two years after she was removed from workplace, CT imaging and ataxia showed no worsening, while visuo-constructive function improved. The authors warn against possible neurotoxic risk associated with this kind of exposure. Hum Toxicol 1989 Jul;8(4):293-300 Chronic neurological toxicity associated with exposure to volatile substances. Lolin Y Department of Chemical Pathology, National Hospitals for Nervous Diseases, London, UK. 1. The main neurological disorders associated with chronic VSA are peripheral neuropathy, cerebellar disease, chronic encephalopathy and dementia. Apart from peripheral neuropathy, the clinical features are non-specific, evidence for solvent-related toxicity is in most cases circumstantial and there is no clear dose/response relationship. 2. Peripheral neuropathy is mainly associated with n-hexane and methyl n-butyl ketone. 3. Cerebellar disease is usually associated with toluene exposure; in the more severe cases there is often radiological evidence of irreversible cerebellar atrophy. 4. Chronic encephalopathy and dementia are the most serious consequence of solvent exposure, particularly to toluene in abusers and to mixed solvents in industrial workers. Postmortem studies in some abusers have shown generalized axonal degeneration, demyelination and brain atrophy. 5. Further studies on low level solvent exposure are needed as little is known about the neurological consequences of mild VSA, especially as regards individual susceptibility and possible interactions between solvents and other toxins such as ethanol. Ann Neurol 1988 Jun;23(6):611-614 Toluene abuse causes diffuse central nervous system white matter changes. Rosenberg NL, Kleinschmidt-DeMasters BK, Davis KA, Dreisbach JN, Hormes JT, Filley CM Department of Neurology, University of Colorado Health Sciences Center, Denver. We describe the findings of magnetic resonance imaging (MRI) of the brain in 6 chronic toluene vapor abusers and the neuropathological findings in 1 abuser not studied by MRI. MRI in 6 chronic toluene abusers revealed the following abnormalities: (1) diffuse cerebral, cerebellar, and brainstem atrophy; (2) loss of differentiation between the gray and white matter throughout the central nervous system; and (3) increased periventricular white matter signal intensity on T2-weighted images. Another chronic toluene abuser (MRI not performed) died as a result of acute toluene overdose. The brain displayed diffuse, ill-defined myelin pallor, maximal in cerebellar, periventricular, and deep cerebral white matter. Neurons were preserved throughout, axonal swelling or beading was not seen, gliosis was minimal, and occasional, scant perivascular macrophage collections were seen. Taken in concert, these findings suggest that the pathological and MRI abnormalities are due to either increased water content of the white matter or subtle toluene-induced metabolic changes in myelin. Neurology 1986 May;36(5):698-702 Neurologic sequelae of chronic solvent vapor abuse. Hormes JT, Filley CM, Rosenberg NL Neurologic abnormalities were seen in 13 of 20 patients with a history of chronic solvent vapor (primarily toluene) abuse for 2 or more years. The patients were evaluated after an abstinence period of at least 4 weeks, to avoid neurologic effects of acute intoxication. Neurologic signs included cognitive (60%), pyramidal (50%), cerebellar (45%), and brainstem/cranial nerve (25%) findings. Eight of nine CTs revealed diffuse atrophy of cerebral hemispheres, cerebellum, and brainstem. BAERs were abnormal in three of four patients, and EEG abnormalities were seen in three of seven patients. Chronic exposure to solvent vapor may cause persistent neurologic impairment. Neurology 1983 Oct;33(10):1337-1340 Multifocal central nervous system damage caused by toluene abuse. Lazar RB, Ho SU, Melen O, Daghestani AN Four toluene abusers had evidence of severe multifocal central nervous system damage. Impairment of cognitive, cerebellar, brainstem, auditory, and pyramidal tract function, as well as CT evidence of cerebral cortical, cerebellar, and brainstem atrophy, have been noted. In addition, we found opsoclonus, ocular flutter, and ocular dysmetria. All three patients tested had abnormal brainstem auditory evoked potentials, indicative of brainstem dysfunction. The patient with opsoclonus had CT evidence of brainstem, cerebellar, and cerebral cortical atrophy. J Adolesc Health Care 1982 Aug;3(1):37-39 Solvent abuse associated cortical atrophy. Schikler KN, Seitz K, Rice JF, Strader T Eleven of 42 toluene inhalers were evaluated with computed tomography scans because of neurologic abnormalities. Six of the 11 were found to have cerebral cortical atrophy. In addition, two of the six had cerebellar atrophy. All six had been exposed to toluene for at least 10 years. This study suggests that atrophy of the central nervous system may occur in chronic toluene abusers. Int Arch Occup Environ Health 1980;46(3):219-231 Neurological picture of organic solvent poisoning in industry. A retrospective clinical study of 37 patients. Juntunen J, Hupli V, Hernberg S, Luisto M A retrospective evaluation of neurological, neuroradiological, neurophysiological, psychological, and laboratory findings on 37 patients, remitted to the Institute of Occupational Health, Helsinki because of suspected poisoning due to organic solvents, was performed. Patient selection was made on the basis of performed pneumoencephalography (PEG). Most of the patients had been exposed to a mixture of solvents (19 cases). Carbon disulphide exposure had occurred in six cases, trichloroethylene in five cases, and the rest of the patients had been exposed to styrene (one case), thinner (two cases), toluene (1 case), methanol (1 case), and carbon tetrachloride (two cases). Clinical neurological findings comprised slight psycho-organic alteration, cerebellar dysfunction, and peripheral neuropathy. The PEG showed changes suggesting brain atrophy in 63% of the patients. Slight asymmetric central atrophy and localized cortical atrophy were the most frequent findings. The main electroencephalographic finding was slight diffuse slow-wave. Electroneuromyography showed slight changes suggesting peripheral neuropathy in 23 of the 28 patients examined. Psychological alterations were seen in all patients: personality changes and psychomotor disturbances were the most common findings. Because individual constitutional differences existed, no clear-cut exposure-effect relationship could be established. Thus, neurological evaluation of all those exposed to neurotoxic agents who present symptoms, regardless of the degree of current exposure, is important.

92. Crossed Cerebral - Cerebellar Diaschisis : MRI Evaluation. Chakravarty A Neurol
    However, inspite of marked cerebellar volume loss in one hemisphere, none of thecases showed any clinical evidence of cerebellar disease.  
    http://www.neurologyindia.com/article.asp?issn=0028-3886;year=2002;volume=50;iss

93. Cerebello-Olivary And Lateral (Accessory) Cuneate Degeneration In A Juvenile Ame
    Key words American Miniature horses; cerebellar disease; equid; neurodegeneration.There are numerous reports in the veterinary literature of  
    http://www.vetpathology.org/cgi/content/full/37/3/271
    
    Extractions: HOME HELP FEEDBACK SUBSCRIPTIONS ... TABLE OF CONTENTS This Article Abstract Full Text (PDF) Alert me when this article is cited ... Alert me if a correction is posted Services Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal Download to citation manager PubMed PubMed Citation Articles by Fox, J. Articles by Scarratt, W. Vet Pathol BRIEF COMMUNICATIONS AND CASE REPORTS J. Fox R. Duncan P. Friday B. Klein and W. Scarratt Abstract A 12-month-old American Miniature horse colt was presented to the Virginia Tech Veterinary Teaching Hospital with a 7-month history of progressive ataxia. Physical examination revealed a head intention tremor, base-wide stance, and ataxia. Necropsy findings were confined to the brain. There were bilateral areas of liquefactive necrosis and cavitation corresponding to the dorsal accessory olivary and lateral (accessory) cuneate nuclei.

94. Typical Features Of Cerebellar Ataxic Gait -- Stolze Et Al. 73 (3): 310 -- Journ
    Keywords cerebellar disease; gait ataxia; movement analysis Twelve patientswith cerebellar disease (CD) of various causes with a clear cut cerebellar  
    http://jnnp.bmjjournals.com/cgi/content/full/73/3/310
    
    Extractions: Vol Page [Advanced] This Article Abstract Full Text (PDF) Submit a response ... Citation Map Services Email this link to a friend Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal ... Cited by other online articles PubMed PubMed Citation Articles by Stolze, H Articles by Deuschl, G Related Collections Other Neurology Journal of Neurology Neurosurgery and Psychiatry

95. Transmission Of Creutzfeldt-Jakob Disease Via A Corneal Transplant -- Heckmann E
    EEG, CCT, and MRT disclosed a cerebellar disease a definite aetiology could Truncal ataxia, cerebellar dysarthria, bilateral dysmetria on finger to  
    http://jnnp.bmjjournals.com/cgi/content/full/63/3/388
    
    Extractions: Vol Page [Advanced] This Article Abstract Full Text (PDF) Submit a response ... Citation Map Services Email this link to a friend Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal ... Cited by other online articles PubMed PubMed Citation Articles by Heckmann, J G J Neurol Neurosurg Psychiatry 388-390 ( September ) J G Heckmann, a C J G Lang, a F Petruch, b A Druschky, a C Erb, c P Brown, d a a Department of Neurology, University of Erlangen Nuremberg, Germany, b Department of Neurology, Bezirkskrankenhaus Ansbach, Germany, c d National Institutes of Health, Laboratory of Central Nervous System Studies, Bethesda, Maryland, USA

96. Clinical Trial: Gluten-Free Diet In Patients With Gluten Sensitivity And Cerebel
    Recognizing gluten sensitivity in patients with cerebellar ataxia would beimportant for two Condition. Celiac Disease cerebellar Ataxia Healthy  
    http://www.clinicaltrials.gov/ct/gui/show/NCT00006492
    
    Extractions: Home Search Browse Resources ... About Gluten-Free Diet in Patients with Gluten Sensitivity and Cerebellar Ataxia This study has been completed. Sponsored by: National Institute of Neurological Disorders and Stroke (NINDS) Information provided by: National Institutes of Health Clinical Center (CC) ClinicalTrials.gov Identifier: Purpose This study will screen patients with cerebellar ataxia to check for antibodies that indicate allergy to gluten (wheat protein) and will study the effect of a gluten-free diet in patients with these antibodies. Patients with cerebellar ataxia have problems with coordination, resulting in "clumsiness" and unsteadiness of posture and walking. There are many known causes of cerebellar ataxia, but in many patients the cause is unknown and there are no available treatments. Cerebellar ataxia has been recognized as a complication of celiac disease, a syndrome characterized by sensitivity to gluten. Recognizing gluten sensitivity in patients with cerebellar ataxia would be important for two reasons: it would be one of the rare causes of the disease that are potentially treatable, and it would identify patients at risk for developing gastrointestinal cancers, particularly intestinal lymphoma. Patients with cerebellar ataxia of known or unknown cause and normal healthy volunteers of any age are eligible for this study.

97. J. Cogn. Neurosci. -- Sign In Page
    The impaired execution of movements in cerebellar disease may influence the results It is well known that basal ganglia disease, compared to cerebellar  
    http://jocn.mitpress.org/cgi/content/full/14/3/493
    
    Extractions: This Article Abstract Full Text (PDF) Alert me when this article is cited ... Alert me if a correction is posted Services Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal Download to citation manager PubMed PubMed Citation Articles by Witt, K. Articles by Deuschl, G. To view this item, select one of the options below: Sign In User Name Sign in without cookies.

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