Clinical Neuropharmacology - UserLogin this disease, although gabapentin was found capable of improving cerebellar and other cerebellar diseases associated with a predominant or selective http://www.clinicalneuropharm.com/pt/re/clnneupharm/fulltext.00002826-200309000-
Section I - Data Used In College And University Catalogs. Course 31, Washburn, FA Lecture 4 Asymmetrical CNS diseases; cerebellar diseases. 32,Washburn, FA Lecture 5 cerebellar diseases cont.; spinal cord diseases http://notes.cvm.okstate.edu/cvm/syllabus.nsf/0/b672f8b5c9dd1b5e86256d33004c98b0
Extractions: Health You are here: Home News and Events News Articles Study Finds Psychiatric Disorders are Common in People with Cerebellar Degeneration For release: Wednesday, September 25, 2002 Overview A new study shows that most patients with movement disorders caused by damage to the cerebellum also have psychiatric symptoms. The study suggests that patients with cerebellar diseases may benefit from screening and treatment of psychiatric symptoms. Get Web page suited for printing Email this to a friend or colleague For decades, researchers have known that the cerebellum, a fist-sized structure at the back of the brain, helps to coordinate the body and to fine-tune movements. A growing body of research conducted during the last decade supports the notion that the cerebellum also plays a role in cognition, emotion, and other non-motor symptoms. Now, a new study shows that most patients with movement disorders caused by damage to the cerebellum also have psychiatric symptoms. The study suggests that patients with cerebellar diseases may benefit from screening and treatment of psychiatric symptoms.
The Role Of The Cerebellum In Cognition And Behavior A Selective Psychopathology in Patients With Degenerative cerebellar diseases A Comparison to CerebellarSubcortical Circuits and Mania in Cerebellar Disease http://neuro.psychiatryonline.org/cgi/content/abstract/12/2/193
Center For Functional And Molecular Imaging Deuschl G, Toro C. Zeffiro T, Massaquoi S, Hallett M. (1996) Adaptation motorlearning of arm movements in patients with cerebellar diseases. http://cfmi.georgetown.edu/members/thomas_zeffiro.php
HDLighthouse.org psychiatric morbidity of patients with degenerative cerebellar diseases. cerebellar diseases, compared with 21 patients with Huntington s disease http://hdlighthouse.org/abouthd/statistics/updates/0033psyco.shtml
Extractions: HD Statistics HD Lighthouse Editors Comment: The following small study suggests that HD patients have more mental problems than those with other brain conditions. The study also suggests that a surprisingly large percentage of neurologically healthy folks, including the researchers, have the same problems. Jerry METHOD: The study included a series of 31 patients with degenerative cerebellar diseases, compared with 21 patients with Huntington's disease and 29 neurologically healthy comparison subjects. Comprehensive psychiatric evaluations, including the Structured Clinical Interview for DSM-IV and psychopathology rating scales, were administered.
Department Of Neuroscience-Faculty the cerebellum coordinates movement, and how its malfunction in disease causes also to provide potential therapeutic targets in cerebellar diseases. http://neuroscience.aecom.yu.edu/faculty/primary_faculty_pages/khodakhah.html
Extractions: We are fortunate that the circuitry of the cerebellum is well understood. Purkinje cells are the sole output of the computational circuitry of the cerebellar cortex. These spontaneously active cells encode the timing signals required for motor coordination in their rate and pattern of activity. In the laboratory our efforts are focused in two areas: 1) understanding the mechanisms that give rise to, and regulate, the intrinsic spontaneous activity of Purkinje cells and 2) elucidating the role of synaptic transmission is shaping the output of Purkinje cells.
Biomedicine - Huntington Psychopathology in patients with degenerative cerebellar diseases a comparisonto Huntington s disease. The American journal of psychiatry. http://www.bioteach.ubc.ca/Biomedicine/Huntington/
Extractions: Graphics:Jen Philpot Huntington disease (HD) is a rare, progressive and fatal autosomal dominant neurodegenerative disorder, typically of adult onset. In 1872, Dr. George Huntington (1850-1916), a family doctor in the U.S., published the first paper on the disease that he called "Huntington's chorea" . A chorea is an abnormal, involuntary movement. The name comes from the Greek word chorea, which means dance. In 1983, HD was the first gene mapped to a chromosomal locus using an anonymous marker technique . After a ten-year struggle, during which fundamental strategies of positional cloning were developed, the causative mutation, a CAG trinucleotide repeat expansion, was identified . Since then, HD research and clinical practice have exerted a strong influence on the approach to phenotype-genotype relationships, the practice of genetic (especially presymptomatic) testing, and the understanding of the pathogenesis of neurodegeneration.
R&D: Medicine - Neurology system) in various cerebellar diseases Investigation of the influence oftactile Horak, FB, Diener, HC Cerebellar control of postural scaling and http://www.uni-essen.de/fet/fue/eng/fb14/neurolo.htm
Extractions: Quantitative determination and therapy of incipient arteriosclerotic alterations in the human carotid arteries using 3-D reconstructions of images of the carotid artery Histological morphology of sonographic patterns in duplex sonography Dynamic and static models of brain glucose metabolism using FDG and PET. Animal experiments on the acute inflammatory meningeal reaction at the blood/brain barrier and on meningeosis carcinomatosa Investigations of natural and pathological autoimmunity in neuromuscular diseases using the techniques of analytical and preparative biochemistry Neuroimmunological questions which are being primarily investigated using the model of murine toxoplasma encephalitis and listeriosis.
Textbook Of Descriptive Medicine - Kichlu And Bose XIV Demeylinating disease 864-865 XV - Heredetary cerebellar diseases 865-867 Ch. X - Nervous System - Section II - Diseases of voluntary muscles http://www.wholehealthnow.com/books/descriptive-medicine.html
The Annals -- Sign In Page cerebellar diseases such as cancer, ischemic attack, or alcoholism. We report2 new cases of cerebellar ataxia secondary to local anesthesia carried out http://www.theannals.com/cgi/content/full/38/5/828
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Directory Of Open Access Journals Key words, Adolescent; Adult; Atrophy; cerebellar diseases; pathology; Cerebellum;pathology; Cerebral Cortex; pathology; Female; Human; Magnetic Resonance http://www.doaj.org/abstract?id=86390&toc=y
Cerebellar Abiotrophy The clinical signs are suggestive of cerebellar disease, particularly if theyare seen in a breed in which abiotrophy is known to occur. http://www.upei.ca/~cidd/Diseases/nervous system disorders/cerebellar abiotrophy
Extractions: related terms: cerebellar and extrapyramidal nuclear abiotrophy, x-linked cerebellar ataxia What is cerebellar abiotrophy? The cerebellum is the part of the brain that regulates the control and coordination of movement. In this condition, cells in the cerebellum mature normally before birth, but then deteriorate prematurely causing clinical signs associated with poor coordination and lack of balance. The Purkinje cells in the cerebellum are primarily involved; cells in other areas of the brain may also be affected. How is cerebellar abiotrophy inherited? An autosomal recessive mode of inheritance has been confirmed or is strongly suspected for the abiotrophies listed below, with the exception of x-linked cerebellar ataxia in the English pointer, which has an x-linked mode of inheritance. What breeds are affected by cerebellar abiotrophy Neonatal cerebellar abiotrophy (very rare) - Affected cells start to degenerate before birth, so that signs of cerebellar dysfunction are present at birth or when the pup first walks. Beagle, samoyed
Extractions: (advertisement) Home Specialties Resource Centers CME ... Patient Education Articles Images CME Advanced Search Consumer Health Link to this site Back to: eMedicine Specialties Neurology Neuro-oncology Last Updated: March 16, 2005 Rate this Article Email to a Colleague Synonyms and related keywords: paraneoplastic neurological syndrome, PCD, breast cancer, ovarian cancer, uterine cancer, lung cancer, occult gynecologic cancers, malignancy AUTHOR INFORMATION Section 1 of 10 Author Information Introduction Clinical Differentials ... Bibliography Author: Abbas Mehdi, MD , Director MDA Center of Central California, Neurology, California Neurological Center, Inc Coauthor(s): David Y Ko, MD , Laboratory Director, Associate Professor, Department of Neurology, University of Southern California Medical Center Abbas Mehdi, MD, is a member of the following medical societies: American Academy of Neurology Editor(s): Frederick M Vincent, Sr, MD , Professor of Clinical Medicine, Psychiatry, and Neurology, Dept of Medicine, Dept of Psychiatry, Dept of Neurology, Michigan State University, College of Human Medicine,College of Osteopat;
The Cerebellum And Its Disorders - Cambridge University Press Infectious diseases radiology and treatment of cerebellar abscesses Jeffrey Other infectious diseases MarioUbaldo Manto; 17. cerebellar disorders in http://www.cambridge.org/aus/catalogue/catalogue.asp?isbn=0521771560
The Pathological Protein information about prion diseases and announcement of a new book. there is nosign of a onesidedness of the cerebellar disease. http://www.thepathologicalprotein.com/_wsn/page4.html
Extractions: Josef Gerstmann described members of the "H" family suffering from an inherited disease now called Gerstmann-Sträussler-Scheinker syndrome, or GSS (Scheinkers name wasnt attached to the disease at first). The disease proved to be a transmissible spongiform encephalopathy in experiments reported in 1981 by D. Carleton Gajduseks team at the National Institutes of Health. In 1989, Stanley Prusiner and Karen Hsaio showed that GSS resulted from a mutation in the prion protein gene. The change in the genetic sequence is a point mutation: a single nucleotide was changed, resulting in an altered amino acid in the prion protein. Several different point mutations are now known to produce GSS. Gerstmann described an unusual reflex action of one of his GSS patients. Turning her head one way, either by herself or by the force of a physicians hands, would cause her arms to swing to the opposite side, and vice versa. Below is a translation of that 1928 case report. (The original appeared as: "Über ein noch nicht beschriebenes Reflexphänomen bei einer Erkrankung des zerebellaren Systems," in Wiener Medizinische Wochenschrift, Volume 78, pages 906908, 1928.)
