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         Pcc Deficiency:     more detail
  1. Optimization of cultural and nutritional conditions for accumulation of poly-@b-hydroxybutyrate in Synechocystis sp. PCC 6803 [An article from: Bioresource Technology] by B. Panda, P. Jain, et all 2006-07-01

81. Microarray Analysis Of The Genome-Wide Response To Iron Deficiency And Iron Reco
Microarray Analysis of the GenomeWide Response to Iron deficiency and IronReconstitution in the pcc 6803 Plant Physiol. Singh et al. 132 (4) 1825.
http://www.plantphysiol.org/cgi/content/full/132/4/1825/DC1
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This Article Abstract Full Text Services Alert me to new issues of the journal Microarray Analysis of the Genome-Wide Response to Iron Deficiency and Iron Reconstitution in the Cyanobacterium Synechocystis sp. PCC 6803
Plant Physiol. Singh et al. 132 (4): 1825.
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HELP FEEDBACK ... ASPB Publications PLANT PHYSIOLOGY THE PLANT CELL

82. Prenatal And Genetic Mg Deficiency
PRENATAL AND GENETIC MAGNESIUM deficiency IN CARDIOMYOPATHY; POSSIBLE VITAMINAND TRACE MG Bianchetti, E. Girardin, pcc Sizonenko and L. Paunier.
http://www.mgwater.com/genetic.shtml
THE MAGNESIUM WEB SITE
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Paul Mason, Editor

83. Molecular Characterization Of IdiA And Adjacent Genes In The Cyanobacteria Synec
pcc 6301 and pcc 7942 grown under Fe deficiency. The intensity of this transcriptwas considerably reduced in Fesufficient culture.
http://mic.sgmjournals.org/cgi/content/abstract/145/6/1473
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ARTICLES
Molecular characterization of idiA and adjacent genes in the cyanobacteria Synechococcus sp. strains PCC 6301 and PCC 7942
KP Michel, F Kruger, A Puhler and EK Pistorius
Biologie VIII: Zellphysiologie, Universitat Bielefeld, 33501 Bielefeld, Germany IdiA (iron-deficiency-induced protein A) is a protein expressed at highly elevated levels in Synechococcus sp. strains PCC 6301 and PCC 7942 under Fe- or Mn-limiting growth conditions. Besides being similar to two bacterial Fe-binding proteins, SfuA and FbpA, IdiA shows similarity to two

84. IdiA, A 34 KDa Protein In The Cyanobacteria Synechococcus Sp. Strains PCC 6301 A
In the cyanobacteria Synechococcus pcc 6301 and pcc 7942 a protein with an deficiency Induction of the idiA Gene of Synechococcus elongatus pcc 7942
http://mic.sgmjournals.org/cgi/content/abstract/142/9/2635
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ARTICLES
IdiA, a 34 kDa protein in the cyanobacteria Synechococcus sp. strains PCC 6301 and PCC 7942, is required for growth under iron and manganese limitations
KP Michel, HH Thole and EK Pistorius
Biologie VIII: Zellphysiologie, Universitat Bielefeld, Germany. In the cyanobacteria Synechococcus PCC 6301 and PCC 7942 a protein with an apparent molecular mass of about 34 kDa (called IdiA for iron- deficiency-induced protein A) accumulates under iron and managanese limitation. IdiA from Synechococcus PCC 6301 was partially sequenced

85. What Is The PCC
Admission with Stipulation Students who complete the pcc but have one low required to enroll in a course that fulfills their deficiency each semester,
http://www.nku.edu/~aarc/restdefpcc.htm
What does it mean to be restricted or stipulated?
When a student applies to NKU, the Office of Admissions will review the student's high school record and ACTE/SAT/COMPASS scores. The University has specific score cutoffs in the areas of English, Math and Reading which determine a student's admission status - Regular, Restricted or Stipulated. Students are identified as having a low ACTE score if any of the English, Math, and/or Reading subscores are less than an 18. Students with SAT scores rather than ACT scores for admission purposes must meet the following criteria: "low SAT score" in Verbal section is less than 480; "low SAT score" in Math section is less than 440. Equivalent COMPASS scores are used for students who take that test. In addition, students' records are examined to determine if they have met the Pre-College Curriculum, which is a set of specified courses completed in a high school program, as follows:
  • English - 4 units (English I, II, III, IV) Mathematics - 3 units (Algebra I, Algebra II, and Geometry) Science - 2 units (Biology I; Chemistry I or Physics I)

86. The Molecular Basis Of Human 3-methylcrotonyl-CoA Carboxylase Deficiency -- Baum
a patient with isolated PC deficiency, and a patient with isolated pcc We harvested the cells after 72 hours and measured MCC and pcc activity
http://www.jci.org/cgi/content/full/107/4/495
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Article
The molecular basis of human 3-methylcrotonyl-CoA carboxylase deficiency
Matthias R. Baumgartner Shlomo Almashanu Terttu Suormala Cassandra Obie Robert N. Cole Seymour Packman E. Regula Baumgartner and David Valle McKusick-Nathans Institute of Genetic Medicine, Johns Hopkins University, Baltimore, Maryland, USA
Howard Hughes Medical Institute, and
Department of Biological Chemistry, Johns Hopkins University, Baltimore, Maryland, USA
Department of Pediatrics, University of California, San Francisco, California, USA Address correspondence to: David Valle, 802 PCTB, Johns Hopkins University, 725 N. Wolfe Street, Baltimore, Maryland 21205, USA. Phone: (410) 955-4260; Fax: (410) 955-7397; E-mail: Received for publication December 6, 2000

87. Hemophilia Pediatric Oncall
Hemophilia B results from deficiency or abnormality of factor IX. pcc iswidely used to treat bleeding in patients with inhibitors to factor VIII.
http://www.pediatriconcall.com/fordoctor/DiseasesandCondition/hemophilia.asp
DOCTOR CORNER Search GO Home Back Ask Doctor Post Query Education Image Gallery Teaching Files Question of the day Specialist Answers Latest Updates Conference Journal Search Original Articles Clinical Trials Search For Pediatricians Hospitals Special Schools Drug Index ... Sign Out
HEMOPHILIA
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Dr Ira Shah
M.D, DCH(Gold Medalist), FCPS, DNB

Q: What is hemophilia?
A: Hemophilia is an inherited disorder in which there is inability to form an effective clot and hence causes prolonged bleeding in a patient. It is seen exclusively in males. A person with hemophilia does not bleed faster, only longer. Q: How common is hemophilia?
A:
Hemophilia affects approximately 1 in 5,000 live male births. Q: How is hemophilia inherited ?
A: There are two types of hemophilia - Hemophilia A and Hemophilia B. Hemophilia A results from deficiency or abnormality of factor VIII. Hemophilia B results from deficiency or abnormality of factor IX. Both are inherited as

88. Hemophilia Of The Sunshine State
Aquired Immune deficiency Syndrome (AIDS)a disease caused by a virus that attacks Prothrombin Complex Concentrate (pcc)- plasma-derived product that
http://www.hemophilia.com/consumers/glossary/
Glossary [A] [B] [C] [D] ... [V-Z]
A Acetaminophen (Tylenol) - a substance used to reduce pain or fever.
Acute -developing quickly with severe symptoms but not lasting a long time.
Aquired Immune Deficiency Syndrome (AIDS) -a disease caused by a virus that attacks the body's immune system.
AHF -Antihemophilic factor.
Albumin -a blood protein that is purified and virally inactivated. Used in monoclonal and some recombinant factor products for stabilization purposes.
ALT -alanine aminotransferase; a liver protein that is released into the blood when the liver is damaged. Increased levels may indicate hepatitis infection.
AST -aspartate aminotransferase; a liver protein that is released into the blood when the liver is damaged.

89. Visualization Of Chromatin Events During DNA Excision Repair In XP Cells: Defici
in XP cells deficiency in localized but not generalized chromatin events Since the regions of localized decondensation in the G1 pcc have been
http://carcin.oxfordjournals.org/cgi/content/abstract/7/12/1975
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ARTICLES
Visualization of chromatin events during DNA excision repair in XP cells: deficiency in localized but not generalized chromatin events
WN Hittelman
Cells derived from patients with xeroderma pigmentosum (XP) are known to be compromised in excision repair after u.v. irradiation, but the exact site of the molecular defect in the repair pathway is not known. The purpose of this study was to examine the ability of XP cells to alter their chromatin in preparation for excision repair. Treatment of quiescent normal human fibroblasts with u.v. light results in two types of chromatin changes that

90. Iron-Deficiency Anemia
Anemia is a reduction in the number of red blood cells (RBCs); in the amount ofhemoglobin in the blood (hemoglobin is the ironcontaining pigment of the
http://www.pccnaturalmarkets.com/health/Concern/Iron_Deficiency.htm

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... Printer-friendly version Iron-Deficiency Anemia
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Natural ways to
beat iron-deficiency anemia
Iron deficiency, whether it is severe enough to lead to anemia or not, can have many non-nutritional causes or may simply be caused by a lack of dietary iron. Symptoms of iron deficiency without anemia may include:
  • Fatigue Mood changes Decreased cognitive function
    Find the cause Iron deficiency can have many non-nutritional causes, including some serious diseases, so work with your healthcare provider to investigate why you are low in iron Take iron as directed Know your iron level To avoid possible problems related to iron overload, have your blood tested regularly for both high and low iron while you are taking iron supplements Drinking coffee or tea with iron supplements inhibits iron absorption Get your vitamin C
These recommendations are not comprehensive and are not intended to replace the advice of your doctor or pharmacist. Continue reading the full iron-deficiency anemia article for more in-depth, fully-referenced information on medicines, vitamins, herbs, and dietary and lifestyle changes that may be helpful.
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About iron-deficiency anemia
Anemia is a reduction in the number of red blood cells (RBCs); in the amount of hemoglobin in the blood (hemoglobin is the iron-containing pigment of the red blood cells that carry oxygen from the lungs to the tissues); and in another related index called hematocrit (the volume of RBCs after they have been spun in a centrifuge). All three values are measured on a complete blood count, also referred to as a CBC. Iron-deficiency anemia can be distinguished from most other forms of anemia by the fact that it causes RBCs to be abnormally small and pale, an observation easily appreciated by viewing a blood sample through a microscope.

91. Vitamin B12 Deficiency
An abnormally low level of vitamin B12 (cobalamin) is a factor in many disorders.
http://www.pccnaturalmarkets.com/health/Concern/Vitamin_B12_Deficiency.htm

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Also indexed as: Pernicious Anemia
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An abnormally low level of vitamin B12 (cobalamin) is a factor in many disorders. True PA is probably an autoimmune disease. The immune system destroys cells in the stomach that secrete intrinsic factor. Many people with PA have both chronic inflammation of the stomach lining, called atrophic gastritis, and antibodies that fight their intrinsic factor-secreting cells. The term pernicious anemia is sometimes used colloquially to refer to any anemia caused by vitamin B12 deficiency. Vitamin B12 deficiency can be due to malabsorption of dietary B12 despite normal levels of intrinsic factor. For example, celiac disease and may cause B12 malabsorption, which can lead to anemia. Less common causes of B12 deficiency include gastrointestinal surgery, pancreatic disease , intestinal parasites , and certain drugs.

92. Publications
pcc 6803 wild type and a histidine kinase Slr1759free mutant. for irondeficiency induction of the idiA gene of Synechococcus elongatus pcc 7942.
http://www.uni-bielefeld.de/biologie/Zellphysiologie/team/pistorius/publications

Aktuelles
Überblick Fakultäten / Einrichtungen Forschung ... Kontakt  Sie befinden sich hier: Universität Bielefeld Startseite der Fakultät für Biologie Molekulare Zellphysiologie die Gruppe ... Previous Co- workers
Nodop A, Suzuki I, Barsch A, Niehaus K, Michel KP, Pistorius EK (2005) Comparative analyses of Synechocystis sp. PCC 6803 wild type and a histidine kinase Slr1759-free mutant.
Arch. Microbiol. Submitted Neumann K, Stephan DP, Ziegler K, Hühns M, Broer I, Lockau W, Pistorius EK (2005) Production of cyanophycin, a suitable source for the biodegradable polymer polyaspartate, in transgenic plants.
Plant Biotechnology J. 3: 249-258 Neufeld S, Zinchenko V, Stephan DP, Bader KP, Pistorius EK (2004) On the functional significance of the polypeptide PsbY for photosynthetic water oxidation in the cyanobacterium Synechocystis sp. strain PCC 6803.
Mol. Gen. Genomics 271: 485-467 Michel KP, Pistorius EK (2004) Adaptation of the photosynthetic electron transport chain in cyanobacteria to iron deficiency: The function of IdiA and IsiA
Physiologica Plantarum, 120: 36-50

93. Prof. Dr. Elfriede K. Pistorius
cyanobacteria Characterization and functional analysis of an L-amino acid
http://www.uni-bielefeld.de/biologie/Zellphysiologie/team/pistorius/

Aktuelles
Überblick Fakultäten / Einrichtungen Forschung ... Kontakt  Sie befinden sich hier: Universität Bielefeld Startseite der Fakultät für Biologie Molekulare Zellphysiologie die Gruppe ... Previous Co- workers
Biologie VIII: Molekulare Zellphysiologie
Fakultät für Biologie
Universität Bielefeld
Postfach 10 01 31
D-33501 Bielefeld
Germany
e.pistorius@uni-bielefeld.de
Ph.D. Degree from Purdue University, West-Lafayette, Indiana, U.S.A.
Biochemistry Department: Ph.D. Supervisor: Prof. Dr. Bernard Axelrod
Thesis: Studies on isoenzymes of soybean lipoxygenase
Research assistent at Max-Planck-Institut, Forschungsstelle Vennesland (Prof. Dr. Birgit Vennesland), Berlin Habilitation at Technische Universität Berlin: Fachbereich für Physikalische und Angewandte Chemie. Venia Legendi: Biochemie Research assistent: Biologie VIII: Zellphysiologie (Prof. Dr. Georg H. Schmid), Fakultät für Biologie, Universität Bielefeld Venia Legendi: Biochemie der Pflanzen Außerplanmäßige Professorin at Fakultät für Biologie Supervisor of the research group: Molecular biology of cyanobacteria integrated into Biologie VIII: Zellphysiologie (Prof. Dr. Georg H. Schmid) and since 2002: Molekulare Zellphysiologie (Prof. Dr. Dorothee Staiger)

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