21. Entrez PubMed A case of massive proteinuria following an Amatch living donor transplantationis described. All at http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=7

22. Entrez PubMed Development of nil disease and focal segmental glomerulosclerosis (FGS) insequential renal biopsies http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=6

23. Minimal Change Disease Minimal change nephrotic syndrome; nil disease; Lipoid nephrosis; Idiopathicnephrotic syndrome of childhood. Treatment http://adam.about.com/encyclopedia/000496trt.htm

zJs=10 zJs=11 zJs=12 zJs=13 zc(5,'jsc',zJs,9999999,'') zCMt='a00' About Healthcare Center Healthcare Center Essentials ... Surgeries and Procedures zau(256,152,145,'gob','http://z.about.com/5/ad/go.htm?gs='+gs,''); Allergies Arthritis Asthma Breast Cancer ... Help zau(256,140,140,'el','http://z.about.com/0/ip/417/C.htm','');w(xb+xb+' ');zau(256,140,140,'von','http://z.about.com/0/ip/496/6.htm','');w(xb+xb); Search Healthcare Center ENCYCLOPEDIA INDEX Injury Disease Nutrition Poison ... Z Minimal change disease Overview Symptoms Treatment Prevention Alternative Names: Minimal change nephrotic syndrome; Nil disease; Lipoid nephrosis; Idiopathic nephrotic syndrome of childhood Treatment: Swelling may be treated with diuretics, blood pressure control, and ACE inhibitor medicines. You may receive instructions on how much salt to allow in your diet. Corticosteroids can cure minimal change disease in a vast majority of children. Some patients may require maintenance on steroids to sustain the remission. A minority of children may relapse after they stop taking steroids. If that happens, they usually respond well to a repeat course of the steroids. Adults do not respond to steroids quite as well as children, but a majority still do find steroids effective. The frequency of relapses and steroid dependence may be greater in adults. Frequent relapsers (more than 3 relapses) may need cytotoxic therapy. In most cases, this involves a course of cyclophosphamide. Other medicines that have been used include cyclosporine and chlorambucil.

24. Minimal Change Disease Minimal change nephrotic syndrome; nil disease; Lipoid nephrosis; Idiopathicnephrotic syndrome of childhood. Symptoms. There may be symptoms of nephrotic http://adam.about.com/encyclopedia/000496sym.htm

zJs=10 zJs=11 zJs=12 zJs=13 zc(5,'jsc',zJs,9999999,'') zCMt='a00' About Healthcare Center Healthcare Center Essentials ... Surgeries and Procedures zau(256,152,145,'gob','http://z.about.com/5/ad/go.htm?gs='+gs,''); Allergies Arthritis Asthma Breast Cancer ... Help zau(256,140,140,'el','http://z.about.com/0/ip/417/C.htm','');w(xb+xb+' ');zau(256,140,140,'von','http://z.about.com/0/ip/496/6.htm','');w(xb+xb); Search Healthcare Center ENCYCLOPEDIA INDEX Injury Disease Nutrition Poison ... Z Minimal change disease Overview Symptoms Treatment Prevention Alternative Names: Minimal change nephrotic syndrome; Nil disease; Lipoid nephrosis; Idiopathic nephrotic syndrome of childhood Symptoms: There may be symptoms of nephrotic syndrome: swelling especially around the eyes, feet and ankles, and in the abdomen foamy appearance of the urine weight gain (from fluid retention) poor appetite Signs and tests: A physical examination would not reveal much except swelling. Blood tests and urinalysis reveal results typical of nephrotic syndrome, including high levels of urine protein, low blood albumin levels, and high cholesterol. A kidney biopsy and examination of the tissue with an electron microscope would show the classic changes of minimal change disease. An

25. JN 2000; Vol.13 N°4: 275-281 Out of a total of 172 biopsies, 85 were nil disease, 52 were mild mesangial This was due to a lower number of relapses/year in the nil disease and mild http://www.sin-italy.org/jnonline/vol13n4/275.html

Table of Contents Original Investigation JNEPHROL 2000; 13: 275-281 Clinical significance and long-term evolution of minimal change histopathologic variants and of IGM nephropathy among Egyptians Ahmed F. Donia, Mohamed A. Sobh, Fatma E. Moustafa, Mohamed A. Bakr, Mohamed A. Foda - Urology and Nephrology Center, University of Mansoura - Egypt Keywords: Evolution, IgM nephropathy, Minimal change nephrotic sydrome variants Introduction Minimal-change nephrotic syndrome (MCNS) is defined morphologically on the basis of normal glomeruli by light microscopy, absent or non-specific immunoglobulin deposits by immunofluorescence, and diffuse epithelial cell foot-process effacement by electron microscopy (1). Based on electron microscopic and other findings, new data suggest that the podocyte phenotype may help define MCNS and focal segmental glomerulosclerosis (FSGS) (2). Additionally, podocyte vacuolization and effacement may have a diagnostic and prognostic role in MCNS (3). The absence or presence of podocytes in urine may differentiate MCNS from mesangial proliferation (4). In 1981, an ISKDC report (5) stated that although the glomeruli in MCNS are customarily normal by light microscopy (nil disease), the histopathologic concept of minimal changes does, however, include minor abnormalities. Accordingly, MCNS was divided into five subcategories (variants): (a) Nil disease, (b) Focal glomerular obsolescence, (c) Mild mesangial thickening, (d) Focal tubular changes, and (e) Mild mesangial hypercellularity. The ISKDC report described the histopathologic changes seen in these five subgroups and assessed their clinical significance.

26. SUNY Stony Brook Pathology Department HBP310 Urinary And Male Genital Outline n Lipoid nephrosis (nil disease). n Membranous glomerulonephritis. n Focalsegmental sclerosis. w Several causes. n AIDS. n Intravenous drug abuse http://www.path.sunysb.edu/coursemat/hbp310urinarymg_outline.htm

URINARY AND MALE GENITAL OUTLINE Male Genitourinary System w Dual function w Congenital abnormalities common w Infertility n Cryptorchidism Infection w Urethritis n Chlamydia n Gonorrhea w Ulcers and condyloma n Herpes n Syphilis w Hematocoele w Torsion w Varicocoele Tumors w Germ cell origin w Parallel to ovarian tumors w Cryptorchidism w Seminoma most common w Self diagnosis Prostate w Hyperplasia n Hormonal n Obstructive w Symptoms w Cancer n Most common, 3rd in fatality n Age related n Cause unknown n Spread to bone Bladder w Cancer n Environmental w Infection n Instrumentation n Colonization n Reflux n Flora Renal Syndromes w Acute renal failure n Oliguria n Anuria w Nephritic w Nephrotic w Isolated Hematuria w Tubular Defects (metabolic) Kidney w Infection n Acute w Ascending n Own flora n Reflux w Hematogenous n Chronic n Stones Tumors w w Adenocarcinoma w w Horseshoe kidney w Dysplasia w Autosomal Dominant Adult Polycystic Kidney Disease (ADPK) n Cysts lead to renal failure n Hypertension n Berry aneurysm w Other cysts Nephritic and Nephrotic Syndrome w Nephrotic n Loss of protein n Decreased serum albumin n Edema w Nephritic n Oliguria n Azotemia (Urea, Creatinine)

27. Sports & Minimal Change Disease - Health And Medical Information Produced By Doc Also known as minimal change nephrotic syndrome; nil disease; lipoid nephrosis;and idiopathic nephrotic syndrome of childhood. http://www.medicinenet.com/script/main/art.asp?articlekey=24127

28. Case Based Pediatrics Chapter Minimal change disease or nil disease accounts for 8085% of cases of primaryidiopathic nephrotic syndrome in childhood. 2. b and d. http://www.hawaii.edu/medicine/pediatrics/pedtext/s13c02.html

Case Based Pediatrics For Medical Students and Residents Department of Pediatrics, University of Hawaii John A. Burns School of Medicine Chapter XIII.2. Nephrotic Syndrome Paul J. Eakin, MD September 2002 Return to Table of Contents A previously well 5 year old male presents to your office with the chief complaint of facial puffiness. His mother noticed this a few days ago and it seems to be worsening. He has no other symptoms, but about two weeks ago had "a bad cold." Exam: VS T 37, HR 90, RR 20, BP 92/55. He is alert and cooperative with the examination. His face shows moderate periorbital edema. His eyes are non-injected, his conjunctiva are not edematous and his throat is not red. His heart is regular without murmurs. Heart sounds are normal. His lung exam shows good aeration, with no crackles or rhonchi. Abdomen is soft, non-tender, non-distended and without masses or shifting dullness. No hepatosplenomegaly. He has normal male genitalia with no scrotal edema. The dorsal surfaces of his hands and feet have mild pitting edema. He has brisk capillary refill and 2+ pulses. No rashes are noted. Urinalysis shows 4+ protein, and a specific gravity of 1.030. His chemistry panel is remarkable for protein of 2 g/dL, serum albumin of 1.4 g/dL and cholesterol of 350 mg/dL. BUN and creatinine are normal.

29. Disease, Medication, Symptom Etc Database Index : N Diseases Database nil disease see Minimal change glomerulonephritis Nilutamide Nimesulide Nimodipine Nimorazole Nimustine Ninth cranial nerve palsy see 9th cranial http://www.diseasesdatabase.com/disease_index_n.asp

Diseases Database Index Sponsors Contact ... Previous Page Disease, medication, symptom etc database index : N Search Nabilone Nabothian gland cyst Naboth's follicles see Nabothian gland cyst Nabumetone N-acetylglutamate synthetase deficiency N-acetyl-L-cysteine see Acetylcysteine NADH-dependent methemoglobin reductase deficiency NADH-diaphorase deficiency see NADH-dependent methemoglobin reductase deficiency Nadifloxacin Nadolol NADPH-dependent methemoglobin reductase deficiency NADPH-reductase deficiency see NADPH-dependent methemoglobin reductase deficiency Nadroparin Nadroparine see Nadroparin Naegeli-Franceschetti-Jadassohn syndrome Naegleria Naegleria fowleri see Naegleria Naegleria fowlerii see Naegleria Naegleriasis see Naegleria Nafamostat Nafarelin Nafcillin Nafidimide see Amonafide Nafronyl see Naftidrofuryl Nafronyloxalate see Naftidrofuryl Naftidrofuryl Naftifine Nager acrofacial dysostosis Nager syndrome see Nager acrofacial dysostosis Nail abnormality Nail biting see Onychophagia Nail corrugation see Ridged nails Nail discoloration Nail disorders see Nail abnormality Nail fungal infection see Tinea unguium Nail overgrowth see Onychauxis Nail pitting Nail ridging see Ridged nails Nail signs see Nail abnormality Nail thickness increased see Pachyonychia Nail-patella syndrome see Hereditary onycho-osteodysplasia Naito-Oyanagi disease see Dentatorubropallidoluysian degeneration Nalbuphine Nalidixic acid Nalmefene ... Nance-Horan syndrome Nance-Insley syndrome see Otospondylomegaepiphyseal dysplasia Nance-Sweeney chondrodysplasia see Otospondylomegaepiphyseal dysplasia Nandrolone Nanocephalic dwarf

30. Turtles-own [is_sick? Days Quaran] ; Days = No Of Days Turtles 315 478 Number of Sick Time Number 0.0 100.0 0.0 100.0 true true PENS Number 1.0 0 16777216 false BUTTON 54 110 121 143 nil disease NIL 1 T OBSERVER http://web.cz3.nus.edu.sg/~chenk/gem2503_3/project/Group5/GEM - Project - (final

turtles-own [is_sick? days Quaran] ; days => no of days turtles left to recover ; is_sick? =>is the turtle sick ; Quaran => no of days left to be free from quarantine to setup ca ;clear all setup-quarantine-area crt percent * screen-size-x * screen-size-y ;create no. of turtles with a percent w.r.t screen area ask turtles [ set xcor (random (screen-edge-x * 1.5) ) - (screen-edge-x / 1.1 ) set ycor (random (screen-edge-x * 1.5) ) - (screen-edge-x / 1.1 ) set Quaran ;default Quaran = ifelse who < p-mask and is_sick? = false and pcolor = 10) ; only if the above 3 citeria is true, turtles catch disease ; 1) probability of getting sick 2) not sick 3)on a patch of a mask person [ if mask = true [ set shape "circle" ] set color yellow set is_sick? true set days 4 + random 3 ;new transmitted disease last for 4 to 7 days ] end to update setup-quarantine-area ask turtles [ if is_sick? = true [ if mask = true [ set shape "circle" ] if pcolor != 20 [ ifelse shape = "circle" [stamp 10] [stamp black] ] set days days - 1 ;decrease the number of days left to recovery ] if quaran > [set quaran quaran - 1] ;decrease the number of days to be quarantine ] end to disease ;create random disease ask turtles [ if who

31. Minimal Change Disease Medical Reference - MedicineOnline.com Alternative names Minimal change nephrotic syndrome; nil disease; Lipoid nephrosis;Idiopathic nephrotic syndrome of childhood http://www.medicineonline.com/reference/Diseases_and_Conditions/Immune_Disorders

Medical References Health Topics Medical Dictionary Drug Information Index ... Providers Login Search for Containing Exact Match The web References [ DISEASES AND CONDITIONS ] Minimal change disease Diseases and Conditions Immune Disorders > Minimal change disease Minimal change disease Sections >> Illustrations Alternative names Definition Causes, incidence, and risk factors ... Prevention Illustrations Male urinary system Alternative names Minimal change nephrotic syndrome; Nil disease; Lipoid nephrosis; Idiopathic nephrotic syndrome of childhood Definition Minimal change disease is a kidney disorder. Each kidney is made of more than a million units called nephrons. Each nephron has a tuft of blood vessels called a glomerulus, which is surrounded by a Bowman''s Capsule. Together, these structures filter blood and form urine. Minimal change disease is one cause of nephrotic syndrome . It is named "minimal change" because the nephrons appear totally normal under a light microscope. The condition is only seen using an electron microscope, which reveals changes in the Bowman''s Capsule in the nephrons. Urine tests also show high protein levels. Causes, incidence, and risk factors

32. Virtual Hospital: University Of Iowa Family Practice Handbook, Fourth Edition: N The exception is minimalchange disease or nil disease. Minimal-change diseasehas the best prognosis, with 90% of children and 50% of adults responding http://www.vh.org/adult/provider/familymedicine/FPHandbook/Chapter08/15-8.html

University of Iowa Family Practice Handbook, Fourth Edition, Chapter 8 Nephrology and Urology: Proteinuria, Nephrotic syndrome, and Nephritic Urine Sudha Rajavel, MD and Rosanna Yuk-Kuen Kao, MD, PhD Department of Family Medicine University of Iowa Hospitals and Clinics and College of Medicine Peer Review Status: Externally Peer Reviewed by Mosby Nephrotic Urine versus Nephritic Urine . The nephrotic urine contains a large amount of protein but does not contain elements indicating active inflammation such as WBCs and RBC casts. In contrast, the nephritic urine is suggestive of acute renal inflammation and will contain protein, RBC casts, blood by dipstick, and WBCs. The differential diagnosis can be narrowed based on whether a nephritic or nephrotic urine is present. Nephrotic Syndrome. General . Nephrotic syndrome is not a disease but rather the renal manifestation of multiple underlying causes. The primary disease may be renal in origin, such as minimal-change disease, or may be a systemic illness with renal manifestations, such as diabetes mellitus with nephropathy. Nephrotic syndrome may be related to glomerulonephritis and other causes of nephritis. Once active renal disease (nephritis) is no longer ongoing (that is, burned out), the patient may end up with nephrotic syndrome and a nephrotic urine. Definition Presentation Anorexia, malaise, edema, anasarca, or pleural effusions.

33. Renal Unit - Case 7 Answers -  Pathology 6000 Laboratory - Department Of Pathol Since minimal change disease (nil disease, lipoid nephrosis) is the most commoncause of nephrotic syndrome in children, treatment is often tried without http://www.uchsc.edu/pathology/6000/a-renc7.htm

RENAL UNIT CASE 7 Answers: 1. Nephrotic syndrome. Poor appetite - possible intestinal edema, ascites. Tired and listless - possibly relative hypotension due to fluid shift. Puffy eyes, edema and nocturia-edema collects in dependent areas (high venous pressure) during the day, in areas of loose connective tissue about the eyes when head is lower at night. Recumbent posture allows some mobilization of fluid from legs at night and therefore nocturia. Apparent weight gain is fluid. Review lecture notes on proteinuria and nephrotic syndrome. 2. Clinical information which can help resolve the differential diagnosis of nephrotic syndrome and identify secondary forms: A) History: a. System review to detect involvement of other organ systems. b. Drug history, exposure to environmental toxins and allergens. c. Family history of diabetes or renal disease. d. History of chronic inflammatory disease that could predispose to amyloidosis.* B) Physical examination: a. Masses which might be neoplasms.*

34. RETINA - UserLogin 1 The terms minimal change disease and nil disease reflect the histopathologicobservations on renal biopsy of either normal findings or mild mesangial http://www.retinajournal.com/pt/re/retina/fulltext.00006982-200406000-00019.htm

LWWOnline LOGIN eALERTS REGISTER ... Archive You are attempting to access protected content. To access this content please login using an established account or create/activate an account. If you have already created/ activated an online account, please login below: User Name: Password: Note: passwords are CASE SENSITIVE If you are a new user or guest visiting an LWWonline site for the first time, please complete the new account setup process to view or purchase content. Forgot your password? Subscribe Renewal Information Subscribe to RSS feed utrdc-pt02 Release 4.0

35. Nephrotic Syndrome : Information And Treatment Minimal Change Glomerulopathy/ nil disease/ Lipoid nephrosis/ Idiopathic NephroticSyndrome of childhood A glomerular disorder characterized clinically by http://www.nephroticsyndrome.com/relatedcon.htm

about us contact us Dr Shah testimonials ... press Search other diseases Chalazion Child Asthma Eczema Fissure-in-ano Hepatitis C Hair Falling Irritable Bowel Syndrome Homoeopathy Information Lichen Planus Psoriasis Tonsils Trigeminal Neuralgia Urticaria Ulcerative Colitis Vitiligo What is it Symptoms Causes Pathology ... Flash Demonstration Download While diagnosing NS, the clinician has to closely examine other similar conditions, as shown below. It may be noted that this makes a suggested reading only for those who want to go into the detail of understanding Nephrotic Syndrome. Nephrotic Syndrome is the outcome of ample number of diseases that damage the filtering units of the kidneys in a certain way that allows them to leak protein into the urine. Hence there are array of diseases, which eventually end up in Nephrotic Syndrome. Some of the diseases that cause Nephrotic Syndrome, such as glomerulonephritis, affect only the kidney. Other diseases that cause Nephrotic Syndrome, such as diabetes and lupus, affect other parts of the body as well. It should be also noted that in vast majority of cases exact cause couldn’t be pinpointed. A. Local causes (primarily affecting kidneys)

36. Part I - The Question Paper -Tuesday 16 May 1995 D A 13 n/a 1 p/t n/a nil 2 nil disease 4 n/a n/a 5 nil nil 1 Enviro nil nil n/a EHOs nil Dental nil Mental nil Sexual n/a D A nil disease nil Enviro nil http://notes.nt.gov.au/lant/hansard/HANSARD7.NSF/0/d0a448b1998c429c6925645f000e9

37. Surgery Of Modern Warfare change, the nil disease,. wherein the binding substance leaves the blood, drawingwith it the body1s water;. and without the binder, http://www.surgeryofmodernwarfare.com/archives/july2103/july2103.html

Surgery of Modern Warfare A poem about a pathologist and a cadaver poem by Surgery's very first surgeon-contributor, Noah Raizman The Pathologist Discusses His Life The foot processes fuse and efface themselves, despite this being a disease of minimal change, the nil disease, wherein the binding substance leaves the blood, drawing with it the body1s water; and without the binder, pressure forces the water out, so little left in the blood, we hold it back, puff ourselves out, a disobedient child holding his breath and refusing to move we classify this entity as a single point in our cartography of suffering, this disease of minimal change, the patients of stasis, young and bedridden they are the worst kind, but some are like me, that is to say old and sclerosed, a lifetime of deposition, microscopically obliterated but if instructed to look closer, with the advantage of stains and metals the movement is ultrastructural which is perhaps true of everything but what can be said of that visible only to electrons? Only this: we see it through their starry, charged eyes.

38. Bacterial Fruit Blotch Of Melons (watermelon, Rockmelon And Honeydew) A certificate of nil disease detection gives a degree of surety that the seed isclean. Rockmelon and honeydew seed is currently screened for disease http://www.dpi.qld.gov.au/horticulture/11630.html

Access keys Skip to primary navigation Skip to secondary navigation Skip to content ... Safe, sustainable systems Bacterial fruit blotch of melons (Watermelon, rockmelon and honeydew) R G O’Brien and Christine Horlock, Agency for Food and Fibre Sciences, Horticulture . Figure 1. Bacterial fruit blotch of watermelon In Queensland, the fruit blotch disease of watermelon has occurred sporadically over the last 25 years. We have regarded it as a minor disease but recently fruit blotch has shown that, given suitable conditions, it can cause severe damage. Since mid 1995, blotch has been noticed in the Ayr, Bowen, Chinchilla and Emerald districts. The severity of the disease is greatly modified by weather conditions and outbreaks are often associated with periods of wet, windy weather. Hail damage can greatly increase disease severity. In 1996, a second strain which also infects rockmelon and honey dew was identified and these crops must now be considered at risk from this disease. Figure 2. Rockmelon leaf showing angular bacterial blotch lesions Symptoms (Links to more symptom images at end) Seedlings The disease can be seed borne. Water-soaked areas appear on cotyledons, these collapse after a few days to become pale-brown coloured, dry areas. If the disease progresses, the water-soaked areas enlarge, affecting the stem which may collapse causing seedling death. There can be a rapid spread of the disease in crowded production areas.

39. Lipoid Nephrosis minimal change disease; foot process disease; nil disease; minimal change nephropathy;minimal change glomerulopathy. ICD9-CM 581.3 lipoid nephrosis http://www.5mcc.com/Assets/SUMMARY/TP0540.html

Lipoid nephrosis DESCRIPTION: Nephrosis characterized by edema, albuminuria, changes in lipids and proteins in the blood, accumulation of globules of cholesterol esters in the tubular epithelium of the kidney. CAUSES: loss of negative charge in glomerular capillary wall Synonyms: minimal change disease foot process disease nil disease minimal change nephropathy minimal change glomerulopathy ICD-9-CM: 581.3 lipoid nephrosis Author(s): Mark R. Dambro, MD

40. Minimal Change Nephrotic Syndrome In Children: Deaths During The First 5 To 15 Y although mesangial abnormalities and tubular atrophy were associated withhigher mortality than nil disease or focal glomerular obsolescence. http://pediatrics.aappublications.org/cgi/content/abstract/73/4/497

QUICK SEARCH: [advanced] Author: Keyword(s): Year: Vol: Page: This Article P Rs: Submit a response Alert me when this article is cited Alert me when P ... Alert me if a correction is posted Services E-mail this article to a friend Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal ... Cited by other online articles PubMed PubMed Citation Minimal change nephrotic syndrome in children: deaths during the first 5 to 15 years' observation. Report of the International Study of Kidney Disease in Children Of 521 children with a previously untreated nephrotic syndrome, as defined by proteinuria greater than or equal to 40 mg/h/m2 and serum albumin less than or equal to 2.5 g/dL, entering the International Study of Kidney Disease in Children between January 1967 and April 1976, 389 showed minimal changes on renal biopsy. Of these, seven boys and three girls died, all before July 1972. Infection was the cause of death in six patients. One child died of dural sinus thrombosis, one died as a result of

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