Geometry.Net - the online learning center
Home  - Health_Conditions - Myocardial Infarction
e99.com Bookstore
  
Images 
Newsgroups
Page 6     101-115 of 115    Back | 1  | 2  | 3  | 4  | 5  | 6 
A  B  C  D  E  F  G  H  I  J  K  L  M  N  O  P  Q  R  S  T  U  V  W  X  Y  Z  

         Myocardial Infarction:     more books (100)
  1. Thrombolysis and Adjunctive Therapy for Acute Myocardial Infarction (Fundamental and Clinical Cardiology)
  2. Myocardial infarction and cardiac death (Medicinal chemistry)
  3. Efficacy of Myocardial Infarction Therapy
  4. Acute myocardial infarction (Current cardiovascular topics)
  5. Echocardiogram Profiles After Acute Myocardial Infarction by Zeev Vlodaver, 1985-10-24
  6. Acute Myocardial Infarction: Emerging Concepts of Pathogenesis and Treatment
  7. Secondary Prevention in Coronary Artery Disease and Myocardial Infarction (Developments in Cardiovascular Medicine)
  8. Electrocardiography of Acute Myocardial Ischemia and Infarction by Samuel Sclarovsky, 1999-03-24
  9. Myocardial Infarction: Measurement and Intervention (Developments in Cardiovascular Medicine)
  10. Primary Angioplasty in Acute Myocardial Infarction
  11. Pathophysiology of Severe Ischemic Myocardial Injury (Developments in Cardiovascular Medicine)
  12. Purines and Myocardial Protection
  13. Improvement of Myocardial Perfusion: Thrombolysis, angioplasty, bypass surgery (Developments in Cardiovascular Medicine)
  14. Myocardial Contrast Two Dimensional Echocardiography (Developments in Cardiovascular Medicine)

101. Old Myocardial Infarction And Other Forms Of Chronic Ischemic Heart Disease
US Death Rates for Twelve Age groups from Old myocardial infarction and other forms of chronic ischemic heart disease.
http://www.disastercenter.com/cdc/aoldmyoc.html
Death Rates for Twelve Age groups from
Old myocardial infarction and other forms of chronic ischemic heart disease -412,414
Year / Age All ages1 Under 1 year2 1-4 years 5-14 years 15-24 years 25-34 years 35-44 years 45-54 years 55-64 years 65-74 years 75-84 years 85 years and over Age adjusted rate3 Rates on an annual basis per 100,000 All causes
Diseases of heart (390-398,402,404-429)

Rheumatic fever and rheumatic heart disease (390-398)

Hypertensive heart disease -402
... The Disaster Center

* Figure does not meet standards of reliability or precision, see Technical notes.
- Data not available.
... Category not applicable.
1 Figures for age not stated included in "All ages" but not distributed among age groups. 2 Death rates for "Under 1 year" (based on population estimates) differ from Infant mortality rates (based on live births); see Technical notes. 3 For method of computation, see Technical notes. From Table 7. Death rates and age-adjusted death rates for the 15 leading causes of death and selected components in United States, 1979, 1995, and 1996
[Rates on an annual basis per 100,000 population in specified group; age-adjusted rates per 100,000 U.S. standard population;

102. Nikon MicroscopyU: Human Pathology Digital Image Gallery - Myocardial Infarction
Acute myocardial infarction is the medical term for the event commonly referred to as a heart attack.
http://www.microscopyu.com/galleries/pathology/acutemyocardialinfarction.html
MicroscopyU Site Map Small World Contest Interactive Tutorials Basic Concepts ... MicroscopyU Home Human Pathology Digital Image Gallery
Myocardial Infarction (Acute)
Acute myocardial infarction is the medical term for the event commonly referred to as a heart attack. One of the leading causes of morbidity and mortality in America and most other industrialized nations, myocardial infarction involves death to tissues of the heart due to a blockage-related inability of sufficient oxygen to reach the organ. Since 90 to 95 percent of individuals in the United States that experience myocardial infarction and are admitted to a hospital survive, it is important to know the warning signs of the condition so that medical help can be obtained in time. Approximately 300,000 Americans die from heart attacks each year before they reach a hospital. Though symptoms of a heart attack vary, they frequently include chest pain or pressure that may radiate into the jaw, shoulders, arms, or back, shortness of breath, nausea, vomiting, lightheadedness, sweating, and anxiety. A significant number of individuals only experience very mild symptoms that may go unnoticed, in which case the myocardial infarction is sometimes referred to as a silent heart attack. Myocardial infarction is commonly related to the gradual accumulation of cholesterol plaques in the arterial walls known as atherosclerosis . Atherosclerosis is characterized by a narrowing of the lumenal space within arteries and hardening of arterial walls. When atherosclerosis occurs in coronary arteries, there is a risk that a plaque may rupture and a blood clot will form over top of it. If this clot becomes so large that it blocks the blood flow through the artery, acute myocardial infarction occurs. The precise reason that cholesterol plaques rupture and form blood clots is not well understood, but there are a number of factors that can contribute to the event. Some of the best-known contributing factors include high blood pressure, adrenaline levels, and LDL cholesterol, as well as smoking and diabetes mellitus.

103. Nikon MicroscopyU: Human Pathology Digital Image Gallery - Myocardial Infarction
myocardial infarction is the necrosis of a section of heart muscle tissue, most commonly from coronary occlusion.
http://www.microscopyu.com/galleries/pathology/myocardialinfarctionold.html
MicroscopyU Site Map Small World Contest Interactive Tutorials Basic Concepts ... MicroscopyU Home Human Pathology Digital Image Gallery
Myocardial Infarction (Old)
Myocardial infarction is the necrosis of a section of heart muscle tissue, most commonly from coronary occlusion. Often referred to as a heart attack, myocardial infarction occurs suddenly, but is usually the result of long-term conditions. Typically the coronary arteries are thickened with cholesterol plaques over the course of many years, increasingly narrowing the lumenal space. The rupture of a plaque can result in the formation of a blood clot, which causes complete occlusion of the arterial lumen if it grows too large, blocking the flow of blood and the oxygen it contains to the heart. Scientist have not yet determined exactly what causes plaques to rupture, but a number of risk factors for myocardial infarction have been identified, including diabetes, smoking, inactivity, being overweight, high LDL and low HDL cholesterol levels, high blood pressure, and increased adrenaline. Being able to recognize the signs that someone is experiencing a myocardial infarction is important for making sure appropriate care is provided in a timely manner. Patients have a very high survival rate if they are admitted to a hospital, but hundreds of thousands of Americans die each year from heart attacks before the ever reach a medical center. The most common symptoms of myocardial infarction include chest pain or pressure that may radiate into the jaw, shoulders, arms, or back, shortness of breath, nausea, vomiting, low blood pressure, quickened pulse, lightheadedness, sweating, and anxiety. The severity of such indicators can vary significantly depending upon the extent of heart tissue affected. Some individuals may experience such mild symptoms that they do not realize that anything serious is wrong, resulting in what is often called a silent heart attack.

104. Nat' Academies Press, Acute Myocardial Infarction: Setting Priorities For Effect
The Knowledge Base for Clinical Issues in Acute myocardial infarction, 924 (skim). Factors Important for the Selection of Key Patient Management Issues and
http://www.nap.edu/openbook/0309043808/html/
Read more than 3,000 books online FREE! More than 900 PDFs now available for sale HOME ABOUT NAP CONTACT NAP HELP ... ORDERING INFO Items in cart [0] TRY OUR SPECIAL DISCOVERY ENGINE Questions? Call 888-624-8373 Acute Myocardial Infarction: Setting Priorities for Effectiveness Research (1990)
Institute of Medicine ( IOM
Find More Like

This Book
Research ...
Dashboard
NEW!
BUY This Book

CHAPTER SELECTOR:
Openbook Linked Table of Contents Front Matter, pp. i-viii Summary, pp. 1-4 Introduction, pp. 5-8 The Knowledge Base for Clinical Issues in Acute Myocardial I..., pp. 9-24 Factors Important for the Selection of Key Patient Managemen..., pp. 25-28 Key Patient Management Topics for Effectiveness Research in ..., pp. 29-42 Conclusions, pp. 43-44 Bibliography, pp. 45-48 Appendix A: Background and Conduct of the Workshop, pp. 49-62 GO TO PAGE:
Table of Contents SEARCH THIS BOOK:
Front Matter
i-viii Summary skim Introduction skim ... skim The Open Book page image presentation framework is not designed to replace printed books. Rather, it is a free, browsable, nonproprietary, fully and deeply searchable version of the publication which we can inexpensively and quickly produce to make the material available worldwide. For most effective printing, use the "printable PDF page" link available on each OpenBook page's tool block. The 300 x 150 dpi PDF linked to it is printable on your local printer.

105. PharmGKB: Myocardial Infarction
Alternate Names, Infarct, Myocardial; Infarction, Myocardial; Infarctions, Myocardial; Infarcts, Myocardial; Myocardial Infarct; myocardial infarctions;
http://www.pharmgkb.org/do/serve?objId=PA445019&objCls=Disease

106. Myocardial Infarction Risk Assessment From Thrombocyte Activation Antigens
CLASSIF1 Multiparameter Data Classification from Flow Cytometrically determined Thrombocyte Activation Antigens.
http://www.biochem.mpg.de/valet/thrombo1.html
Cell Biochemistry Martinsried
Risk Assessment for Myocardial Infarction
from Thrombocyte Activation Antigens
G.Valet
1. Introduction: General risk indicators like overweight, smoking, high blood pressure, humoral indicators like altered lipid fractions in the peripheral blood, as well as cellular indicators like monocyte/makrophage lipid receptors indicate the risk for myocardial infarction on a statistical but not at an individual person level. - The appearence of thrombocyte activation antigens like or thrombospondin on the thrombocyte surface membrane is the consequence of accelerated blood flow through arteriosclerotically narrowed coronary arteries. Myocardial infarction is ultimately caused by thrombocyte aggregates obstructing such arteries. It seems of interest to determine thrombocyte activation antigen patterns for individual patient risk assessment of myocardial infarction. A blood test would be substantially easier , with lower risk and less costly than coronary angiography from a cathether.

107. Disease Category Listing (463): Heart Attack (Myocardial Infarction)
CenterWatch Listing of Clinical Research Trials for Heart Attack (myocardial infarction)
http://www.centerwatch.com/patient/studies/cat463.html
Clinical Trials: Heart Attack (Myocardial Infarction)
Arizona
Tucson; ACRC / Arizona Clinical Research Center, Inc.
Could you be at risk for a stroke? This is a 3½-year clinical research study to assess the safety and efficacy of an investigational (not approved by the FDA) medication in subjects with normal or low cholesterol levels who could be at risk for a heart attack or stroke.
California
Los Angeles; UCLA Medical Center
Adult Volunteers Wanted For Cardiac MRI Research
Florida
Aventura; Clinical Research Institute of South Florida
Heart Attack and Stroke prevention Ft. Lauderdale; Holy Cross Hospital
ID# 03-05: Cardiac - Evaluation of Cardiac Drug in Patient with Implanted Cardioverter Defibrillator Ft. Lauderdale; Holy Cross Hospital
ID# 02-14: Pacemaker and New Technology Information - Information Validity Ft. Lauderdale; Holy Cross Hospital
ID# 03-26: Myocardial Infarction - Heart Attack Hollywood; South Broward Cardiology Consultants
Comparison trial for the treatment of patients who have experienced Myocardial Infarction (heart attack) Tampa; Clinical Research of West Florida, Inc.

108. FINAMI - Finnish Myocardial Infarction Register
Finnish myocardial infarction Register FINAMI. Introduction Publications Contact information flagfi.gif (117 bytes)
http://www.ktl.fi/eteo/finami/indexen.html
Finnish Myocardial Infarction Register FINAMI
The new FINAMI address is www.ktl.fi/finami
©National Public Health Institute 2002
Last updated 6.9.2005 mt

109. Lack Of Association Between First Myocardial Infarction And Past Use Of Erythrom
Characteristics of casepatients with myocardial infarction and controls The risk of myocardial infarction associated with antihypertensive drug
http://www.cdc.gov/ncidod/eid/vol5no2/jackson.htm

Suggested Citation

Dispatches
Lack of Association between First Myocardial Infarction and Past Use of Erythromycin, Tetracycline, or Doxycycline
Lisa A. Jackson, Nicholas L. Smith, Susan R. Heckbert, J. Thomas Grayston, David S. Siscovick, and Bruce M. Psaty
University of Washington, Seattle, Washington, USA To evaluate the association of prior treatment with antibiotics active against Chlamydia pneumoniae with the risk for incident myocardial infarction, we conducted a population-based case-control study. We found that use of erythromycin, tetracycline, or doxycycline during the previous 5 years was not associated with risk for first myocardial infarction. These results suggest little or no association between the use of these antibiotics and the risk for first myocardial infarction in the primary prevention setting. Chlamydia pneumoniae has been associated with atherosclerotic cardiovascular disease in seroepidemiologic studies, by detection of the organism in atherosclerotic plaque, and in animal model studies ( ). Two small clinical trials to assess the effect of treatment with antibiotics active against

110. IM Quiz: Acute Inferior Myocardial Infarction
This patient is having an acute inferior wall myocardial infarction. Given the patients early presentation and current EKG he is a candidate for
http://meded.ucsd.edu/isp/1994/im-quiz/infmi.htm
Acute Inferior Myocardial Infarction
This patient is having an acute inferior wall myocardial infarction. Given the patients early presentation and current EKG he is a candidate for thrombolytic therapy with TPA. He should be assessed for risk factors and given TPA. Remember to check a right sided EKG to look for posterior (RV) extension of this infarct. This is apparently described as occurring in as many as 50% of inferior myocardial infarctions and in a range of 14-84% (depending on study criteria) of all left ventricular infarctions (1). The clinical triad of RV infarcts consists of :
  • hypotension
  • elevated jugular venous pressure
  • clear lung fields Although quite specific, this triad has a sensitivity of about 25% (2). These signs may not be entirely present if the patient is volume depleted. Patients with RV infarction are volume dependent and giving them nitroglycerin sublingual can cause them to become profoundly hypotensive. This can iatrogenically extend the area of infarction. It is ALWAYS a good rule to get an EKG before doing any interventions with the exception of administering an aspirin. And ALWAYS get a right sided EKG in inferior infarction patterns. For a good review on RV infarction see:
  • References:
  • Cohn JN, Guiha NH, Broder MI, Limas CJ. Right ventricular infarction: clinical and hemodynamic features. Am J Cardiol 1974; 33:209-14.
  • 111. Acute Myocardial Infarction--Quality Improvement In Hospitals—OMPRO, A Hea
    Acute myocardial infarction. Quality indicators Related links Contacts Acute myocardial infarction Surgical Infection Prevention Heart Failure
    http://www.ompro.org/hospital-care/ami.html
    @import url(/style_sheets/newprof.css); @import url(/style_sheets/hospital1.css); Hospital
    Quality Improvement OMPRO Home page Skip the navigation links
    Acute Myocardial Infarction
    Quality indicators
    Related links

    Contacts
    AMI Tools and resources
    Overview
    Cardiovascular disease is the leading cause of morbidity and mortality in the United States. Cardiovascular disease is responsible for almost 50% of all deaths, with persons age 65 and older accounting for about 84% of cardiovascular deaths.
    • Each year, approximately 1.5 million Americans experience an acute myocardial infarction (AMI); almost one-third of those who are older than 65 die during the acute phase of an AMI. Survivors have a chance of related illness and death from two to nine times as high as that of the general population. AMI remains a leading cause for admissions of older adults, accounting for approximately 12 hospitalizations among every 1,000 Medicare beneficiaries. The associated costs were more than $3.6 billion in 1996 or about $9,780 per discharge (excluding payments for services to patients covered by supplemental insurance through managed care organizations). The number of persons who experience an AMI is growing because AMI is more common with increasing age, and the percentage of the population older than 65 is rising. With that increase comes an increase in the number of persons who incur the secondary effects of AMI, including heart failure, recurrent myocardial infarction, stroke, and sudden death.

    112. Laser Thrombolysis In Acute Myocardial Infarction
    Patients with acute myocardial infarction with acute ST elevation by EKG in at least two leads, ongoing ischemia, contraindications or failure of
    http://omlc.ogi.edu/pubs/abs/gregory92.html
    Laser Thrombolysis in acute Myocardial Infarction-Initial Clinical Results
    Gregory KW, Buckley LA, and Block PC, Laser Thrombolysis in acute Myocardial Infarction-Initial Clinical Results Lasers in Surgery and Medicine , (Suppl.) 4:17, 1992.

    113. Coronary Thrombosis (heart Attack)
    the blood supply to that area of the heart muscle will stop. This is known as a coronary thrombosis, a myocardial infarction or heart attack.
    http://www.netdoctor.co.uk/diseases/facts/coronarythrombosis.htm
    Search: All NetDoctor Diseases Medicines NetDoctor.co.uk Home News and features News News archive Newsletter Features Encyclopaedia Diseases Examinations Medicines Premium services SMS services StayQuit thediet Health centres ADHD Allergy and asthma Children's health Depression ... All health centres Discussion and support Discussion forums Support groups Services Ask the doctor Find a hospital Search Medline Test yourself Information About NetDoctor Commercial opportunities NetDoctor.com Coronary thrombosis (heart attack) Reviewed by Dr Neal Uren , consultant cardiologist and Dr Patrick Davey , cardiologist
    What is a coronary thrombosis?
    The heart is surrounded by three major coronary arteries that supply it with blood and oxygen. If a blood clot develops in one of these arteries, the blood supply to that area of the heart muscle will stop. This is known as a coronary thrombosis, a myocardial infarction or heart attack.
    Most commonly a coronary thrombosis will cause severe chest pains behind the sternum (breast bone), often radiating towards the left arm. The area of muscle to which there is insufficient supply stops working properly if the blood clot is not dissolved quickly, eg with thrombosis dissolving (thrombolytic) medication.
    Why does coronary thrombosis occur?

    114. Myocardial Ischemia, Injury And Infarction
    Insufficient blood supply to the myocardium can result in myocardial ischemia, injury or infarction, or all three.
    http://www.americanheart.org/presenter.jhtml?identifier=251

    115. Myocardial Infraction
    infarction means necrosis of myocardial tissue. This gives a permanent damage, although EKG changes can become normal after a longer period of time.
    http://www.technion.ac.il/~eilamp/mi.html
    Myocardial Infarction
    Links Glossary Quizzes Site Map ... Feedback Introduction Infarction of the myocard is accompanied by pain, increased enzyme values and EKG signs. If two of the three typical manifestations are seen it is assumed that an infarction has taken place.
    Dependent on the severity and duration of the oxygen shortage a more or less severe disturbance can happen in the electrical processes in the heart.
    We can distinct the following:
    • Ischemia Lesion Infarction
    Ischemia and Lesion are respectively the less and most worse form of oxygen shortage, but are usually reversible. Infarction means necrosis of myocardial tissue. This gives a permanent damage, although EKG changes can become normal after a longer period of time. Ischemia
    According to theory a negative T-wave indicates ischemia of the total heart wall or in the exterior wall layers of the ventricular muscle, and a high positive T-wave indicates ischemia of the interior wall layers. These changes take place in the first minute of occlusion of the coronary artery and is followed shortly with damage of the myocard. This is the reason why this phase is rarely registered electrocardiographically. Lesion (myocardial injury)
    A lesion causes a displacement of the ST-segment. An ST-depression (descending ST-segment) indicates a lesion of the subendocardial layers and an ST-elevation (ascending ST-segment) a lesion of the total wall. An ST-elevation is also caused by an inflammation of the subepicardial (exterior) layers of the heart muscle, for example pericarditis.

    A  B  C  D  E  F  G  H  I  J  K  L  M  N  O  P  Q  R  S  T  U  V  W  X  Y  Z  

    Page 6     101-115 of 115    Back | 1  | 2  | 3  | 4  | 5  | 6 

    free hit counter