81. Faculty/Kelly positron emission tomography (PET) are being conducted in humans with variousforms of myocardial diseases due to ischemia, hypertension, and diabetes. http://cardiology.wustl.edu/faculty/kelly.html

Daniel P. Kelly, MD Professor of Medicine, Pediatrics, Director, Center for Cardiovascular Research Co-Director, Cardiovascular Division Office location : 810 Clinical Sciences Research Bldg., North Tower Addition Telephone Fax E-mail : dkelly@im.wustl.edu Current Research Interests The Kelly laboratory is interested in regulatory mechanisms related to the control of cardiac energy metabolism in the developing and diseased heart. We have found that a member of the nuclear hormone receptor superfamily, the peroxisome proliferator-activated receptor a (PPAR a ), serves as a key regulator of mitochondrial energy production in the normal postnatal mammalian heart. The activity of PPAR a is reduced in the hypertrophied and failing heart. Conversely, PPAR a activity is abnormally “super-activated” in the diabetic heart. To model these disease states, genetically modified mice with reduced or increased cardiac PPAR a function have been developed. Using the PPAR

82. UCSD Medical Web Store - Product Detail For Principles Of Molecular Cardiology. Inherited myocardial diseases Elizabeth M. McNally Receptorsignaling pathwaysin heart failure Shayela Suvarna, Liza Barki-Harrington, Miwako Suzuki, http://webmedbooks.com/ucsd/content/productdetail.aspx/upc=eff4fe94-60c6-4102-93

You Are Here: Home Medical Books Medicine Cardiology Principles of Molecular C... Sign In My Account Search All Products Medical Books Medical Supplies Faculty Authors for Principles of Molecular Cardiology. By Runge, Marschall S. MD, PhD Click for larger image List Price: Your Price: Savings: Usually ships within 24 hours More product information Product Information Binding: Cloth Specs: 617 pages 4.87 Lbs ISBN: Edition: Publisher: Humana Press Publish Date 04/05 Series: Contemporary Cardiology Description: This text provides a look into the advances in the fields of molecular biology, development and genetics in relation to cardiology. It offers to the reader an introduction to molecular cardiology via a section of introductory overviews. The authors provide chapters on disease-oriented topics written by leaders in the field. These experts have gathered a concise and thorough discussion of the relevant molecular, cellular, genetic and developmental biological processes that underlie and contribute to cardiovascular disease. This book presents a reference that any scientist in the field would want to own as a broad overview of the field. Also a valuable tool to any cardiology fellow would benefit from due to the insights it provides into the common diseases in the field. Table Of Contents: Cardiovascular genetics Christopher Semsarian, Christine Seidman

83. RIKEN NEWS September 2003 Some myocardial diseases have been found to be caused by mutations in the genesencoding troponin. In addition, the group of Dr. Iwao Otsuki, http://www.riken.jp/engn/r-world/info/release/news/2003/sep/

Zeroing in on the regulatory mechanism of muscle contraction - An approach focusing on the three-dimensional structure and movement of actin filaments - Zeroing in on the regulatory mechanism of muscle contraction An approach focusing on the three-dimensional structure and movement of actin filaments RIKEN Harima Institute Structural Biochemistry Laboratory Chief Scientist, Dr. Yuichiro Maeda Great progress has been made in the research of "actin filaments," which play a part in the regulation of muscle contraction. Chief Scientist Yuichiro Maeda and his colleagues in the Structural Biochemistry Laboratory have become the first in the world to solve the three-dimensional structure of troponin. Troponin is one of the proteins that compose actin filaments, and it plays an important role in the regulation of muscle contraction. This landmark achievement had been awaited for 40 years, since the proposal of the calcium ion (Ca ) theory of regulation of muscle contraction and the discovery of troponin, a molecule to which Ca binds. "Our goal is to first elucidate the three-dimensional structure of actin filaments, and then to use the knowledge to fully clarify the regulatory mechanisms of muscle contraction. Now that we have determined troponin's three-dimensional structure, it is possible to get to the heart of the questions concerning troponin's behavior and functions," explained Dr. Maeda. How can structural biology uncover the regulatory mechanisms of muscle contractions? This article will take you to the forefront of cutting-edge research.

84. Week 9 Infectious Endocarditis, NonIschemic myocardial diseases, Cardiac Tumors. Dr.Nandkumar. Pathology. Atherosclerosis, Ischemic Heart Disease. Dr. Nandkumar http://www.med.uiuc.edu/m2/calendar/Week 9.htm

Week 9 Monday October 10 Tuesday October 11 Wednesday October 12 Thursday October 13 Friday October 14 FCP Cardiovascular Disease: FCP Cardiovascular Disease: FCP Cardiovascular Disease: Pharmacology Clinical Use of Inotropic Drugs Dr. Cook READING DAY (Assigned study room: Forum B) Pathology Valvular Heart Diseases, Rheumatic Fever/Rheumatic Heart Disease Dr. Nandkumar Pathology Infectious Endocarditis, Non-Ischemic Myocardial Diseases, Cardiac Tumors Dr. Nandkumar Pathology Atherosclerosis, Ischemic Heart Disease Dr. Nandkumar FCP Cardiovascular Disease: Afternoon Epidemiology 8 Clinical Trials Dr. Gayed Pharmacology Antiarrhythmic Drugs I Dr. Wang Pharmacology Antiarrhythmic Drugs II Dr. Wang Pharmacology Cardiovascular Review Dr. Wang Epidemiology Midterm Exam Review (optional) Dr. Gayed

85. UC Davis Graduate Group In Comparative Pathology MD Kittleson Acquired cardiovascular/myocardial diseases. JE Madigan - Equineinfectious diseases. KG Magdesian - Equine and comparative gastrointestinal http://www.vetmed.ucdavis.edu/pmi/comppath/FACUL.HTM

UC Davis Graduate Group in Comparative Pathology GGCP Faculty There are approximately 100 faculty members in the Graduate Group in Comparative Pathology representing most departments in the Schools of Medicine and Veterinary Medicine. Return to GGCP Home Faculty from the School of Medicine Faculty from the School of Veterinary Medicine Human Anatomy K.L. Erickson Macrophage immunobiology Hematology and Oncology K.S. Lam Application of cominatorial chemistry in basic research, drug development, and cancer proteomics Medical Dermatology S.A. Grando - Immunopathology with blistering dermatoses Internal Medicine C.L. Bowlus Expression of serine protease in cortical thymic epithelial cells and its role in T cell development M.M-J. Chang- Biology of conducting airway epithelium S. Dandekar Molecular pathology, animal models for AIDS J.S. Powell - Gene therapy of genetic disorders T.P. Prindiville - Gastrointestinal immunology D.L. Robbins - Immunobiology of rheumatoid arthritis M. Vajdy -

86. 8th International Congress On Shock Wave Therapy In Vienna 8th International Congress on Shock Wave Therapy in Vienna Remarkable Resultsin Wound Healing and Curing myocardial diseases http://www.prnewswire.co.uk/cgi/news/release?id=147108

87. Heart Attack (myocardial Infarction) By MedicineNet.com Overview on heart attack (myocardial infarction) and heart attack symptoms Coronary heart diseases include heart attacks, sudden unexpected death, http://www.medicinenet.com/heart_attack/article.htm

document.writeln(''); MedicineNet Home Heart Home Page > Heart Attack Search Tips Printer-Friendly Format FREE Newsletters Email to a Friend ... Next Heart Attack (Myocardial Infarction) Revising Medical Author: Dennis Lee, MD Revising Medical Editor: Daniel Kulick, MD Jay Marks, MD What is a heart attack? What causes a heart attack? ... Heart Attack At A Glance What is a heart attack? A heart attack (also known as a myocardial infarction) is the death of heart muscle from the sudden blockage of a coronary artery by a blood clot. Coronary arteries are blood vessels that supply the heart muscle with blood and oxygen. Blockage of a coronary artery deprives the heart muscle of blood and oxygen, causing injury to the heart muscle. Injury to the heart muscle causes chest pain and pressure. If blood flow is not restored within 20 to 40 minutes, irreversible death of the heart muscle will begin to occur. Muscle continues to die for 6-8 hours at which time the heart attack usually is "complete." The dead heart muscle is replaced by scar tissue. Approximately one million Americans suffer a heart attack each year. Four hundred thousand of them die as a result of their heart attack.

88. ClinicalTrials.gov - Information On Clinical Trials And Human Research Studies: Coronary Disease; Diabetes Mellitus; Heart diseases; myocardial Ischemia Heart diseases; myocardial Infarction; myocardial Ischemia http://www.clinicaltrials.gov/ct/screen/BrowseAny?path=/browse/by-sponsor/NIH/NH

89. ClinicalTrials.gov - Information On Clinical Trials And Human Research Studies: Conditions Albinism; Intestinal Disease; Kidney Disease; myocardial Disease; Conditions Cardiovascular diseases; Coronary Disease; myocardial http://www.clinicaltrials.gov/ct/gui/action/FindCondition?ui=D009202&recruiting=

90. Breakthrough In National Diseases: Common Factor Behind Myocardial Infarction, R A common gene variant has been identified as the risk factor behind a number ofcommon diseases by research scientists at Karolinska Institutet and the http://www.sciencedaily.com/releases/2005/05/050506155139.htm

@import "/styles/navbar.css"; @import "/styles/tabStyles.css"; Set home page Bookmark site Add search Latest News ... Email to friend Text Size A A A Front Page ... Advertise With Us Source Karolinska Institutet Date Print this page Email to friend Breakthrough In National Diseases: Common Factor Behind Myocardial Infarction, Rheumatism And MS A common gene variant has been identified as the risk factor behind a number of common diseases by research scientists at Karolinska Institutet and the Centre for Molecular Medicine (CMM), Stockholm, Sweden. Up to a quarter of the population could be affected. Related News Stories (June 30, 2005) Parents whose children are born with a low birth weight run greater risk of dying of cardiovascular diseases. Even the fathers are at greater risk. These findings are published in a new report by ... full story Single Stem Cells From Bone Heal A Broken Heart (February 9, 2005) Myocardial infarction results in irreversible damage to the heart that can cause congestive heart failure. The lasting damage results from the limited ability of the myocardium to regenerate and ... full story (May 19, 1998)

91. Chapter 4: Myocardial Infarction - Cardiovascular Diseases Acute myocardial infarction characteristically presents with an acute onset ofsevere Cholesterol and coronary heart disease screening and treatment. http://hebw.uwcm.ac.uk/cardio/chapter4.html

CARDIOVASCULAR DISEASES Team Leader: Dr David Fone Date of completion: 4: Myocardial infarction This bulletin is a supplement to, not a substitute for, professional skills and experience. Users are advised to consult the supporting evidence for a consideration of all the implications of a recommendation. The Statements The Evidence 4.1 Clinical guidelines Acute myocardial infarction characteristically presents with an acute onset of severe chest pain at rest i . Clinical guidelines and audit standards for the management of acute myocardial infarction are available from the British Cardiac Society i , the American Heart Association ii and European Society of Cardiology iii (Health gain notation - 1 "beneficial" i. de Bono DP, Hopkins A, for a working party of the joint audit committee of the British Cardiac Society and the Royal College of Physicians of London. The management of acute myocardial infarction . British Cardiac Society, 1996. http://www.cardiac.org.uk/ (Type V evidence - expert opinion) ii.

92. Myocardial Disorders Topic - Unified Search Environment diseases of the myocardium. Synonyms and Source Vocabularies myocardial disordersMTH/PN/NOCODE CST/HT/CV/CARD/MYO AOD/DE/0000005386 MTH/MM/U013349 http://www.use.hcn.com.au/portals/shared/subject.`Myocardial disorders`/home.htm

Myocardial disorders Topic Tree Definition: Diseases of the myocardium. Synonyms and Source Vocabularies: Myocardial disorders Myocardial Diseases myocardium disorder heart muscle disorder Heart Diseases Arrhythmia Carcinoid Heart Disease Cardiac Output, Low ... Ventricular Dysfunction Myocardial disorders Cardiomyopathy, Alcoholic Cardiomyopathy, Congestive Hypertrophic Cardiomyopathy Cardiomyopathy, Restrictive ... Myocarditis

93. Breakthrough In National Diseases: Breakthrough in national diseases Common factor behind myocardial infarction,rheumatism and MS. A common gene variant has been identified as the risk http://info.ki.se/article_en.html?ID=3411

94. The 4th World Congress Of Echocardiography And Vascular Ultrasound 15.1515.25, myocardial Performance In Hypertensive Heart Disease. Doppler EchoStudy, Ariesta Ann SOENATRA, MD (Indonesia) http://www.egyptheart.org/echo2000/iscu4m_d2.htm

The 4th World Congress of Echocardiography And Vascular Ultrasound Final SCIENTIFIC PROGRAM Thursday January 20th, 2000 Program Search The Program WEDNESDAY JANUARY Program ... Day three Hall A, B, C PLENARY SESSION-II Chairpersons Mohamed ATTIA (Egypt); Peter HANRATH (Germany); Ryozo OMOTO (Japan) Is Teaching Of Physical Examination Still Justified In The Era Of Echocardiography? Andres JAUSSI, MD (Switzerland) Second Harmonic Imaging: New Standard In Stress Echocardiography Peter HANRATH, MD (Germany) 3 D Echocardiography: Current Status And Future Directions Natesa PANDIAN, MD (USA) Early Noninvasive Recognition Of The Candidate For Sudden Death Mario MARANHAO, MD (Brazil) Intra-operative transesophageal echocardiography: Its newer implications Ryozo OMOTO, MD (Japan) Break Hall A SCIENTIFIC SESSION-8 EMERGING ROLE OF ECHO IN CRITICAL CARE Chairpersons Sherif MOKHTAR (Egypt); Mikel SMITH (USA); Otto KAMP (Netherlands) Plenary Lecture: Echocardiography In Acute Cardiac Emergencies Satish KAUSHIK, MD (India)

95. Myocardial DNA Strand Breaks Are Detected In Biopsy Tissues From Patients With D in mechanisms of progressive loss of functional cardiac units in these myocardialdiseases. Key words dilated cardiomyopathy, myocarditis, TUNEL method http://www.clinicalcardiology.org/briefs/9808briefs/21-591.html

Search this site: AUGUST 1998: Contents Previous Next Myocardial DNA Strand Breaks Are Detected in Biopsy Tissues from Patients with Dilated Cardiomyopathy Yukihiko Seki, M.D., Hisashi Kai, M.D., Ph.D.,* Mamiko Kai, Ph.D.,* Akihiko Muraishi, M.D., Kyo Adachi, M.D., Ph.D., Tsutomu Imaizumi, M.D., Ph.D. Third Department of Internal Medicine and *Institute of Cardiovascular Disease, Kurume University School of Medicine, Kurume, Japan Summary Background: Progressive damage of cardiomyocytes with interstitial and replacement fibrosis accompanied by less inflammatory cell infiltration is observed in patients with dilated cardiomyopathy (DCM), suggesting some other mechanisms rather than necrotic cell death. Hypothesis: The aim of this study was to assess the possible involvement of apoptotic process in the pathogenesis of DCM and myocarditis. Methods: Endomyocardial biopsy was performed in patients with DCM (n = 9), myocarditis (n = 4), or atypical chest pain syndrome (as controls; n = 5). The TUNEL method was used for in situ detection of oligonucleosomal DNA strand breaks. Results: Conclusion: The DNA strand breaks were detected in cardiomyocytes in patients with DCM and mainly in interstitial cells in myocarditis. It is possible that the DNA strand breaks can be involved in mechanisms of progressive loss of functional cardiac units in these myocardial diseases.

96. Cardiovascular Pathology Index Heart, left ventricle, acute myocardial infarction, gross Miscellaneous CardiacDiseases. Peripheral coronary artery with sclerosis, microscopic http://www-medlib.med.utah.edu/WebPath/CVHTML/CVIDX.html

Cardiovascular Pathology Index Return to the organ system pathology menu. Tutorials Go to the tutorial on myocardial infarction. The following images are present: Normal Heart Normal heart, gross Normal aortic valve, gross Normal tricuspid valve, gross Normal coronary artery, microscopic ... Normal myocardium, medium power microscopic Atherosclerotic Cardiovascular Disease Coronary artery with atherosclerotic narrowing, microscopic Coronary artery with recanalized thrombosis, microscopic Coronary artery with calcific atherosclerosis, microscopic Coronary artery atherosclerosis, occlusive, microscopic ... Coronary artery with recent thrombus, cross section, gross Myocardial Infarction Heart, left ventricle, acute myocardial infarction, gross Heart, left ventricle and septum, myocardial infarction, gross Myocardium, contraction band necrosis, microscopic Myocardium, acute myocardial infarction, 1 to 2 days, microscopic ... Heart, coronary artery bypass graft, gross Arterial Dissection Aorta, dissection with tear in arch, gross Heart, dissection with tear through media, low power microscopic Hemopericardium with cardiac tamponade, gross Aorta, dissection, gross ... Arachnodactyly, Marfan's syndrome, gross Infective Endocarditis Aortic valve, infective endocarditis, gross

97. Shock Wave Therapy Shows Remarkable Results In Wound Healing And Shock wave therapy shows remarkable results in wound healing and curing myocardialdiseases. Medical Procedure News. Published Tuesday, 31May-2005 http://www.news-medical.net/print_article.asp?id=10530

98. Atrial Fibrillation mitral stenosis, and mixed mitral valve disease), thyrotoxicosis, myocardialdiseases including those due to infiltrative processes, infection, http://www.americanheart.org/presenter.jhtml?identifier=1596

99. Echocardiography Coronary artery disease obscuring giant cell myocarditisa case report. Giant cell myocarditis is a rare but devastating disease that usually affects http://www2.umdnj.edu/~shindler/giantcel.html

Giant Cell Myocarditis E-chocardiography Journal: Alphabetical List Chronological List Images Home Page ... Echocardiographic Description of Recurrent Idiopathic Giant-Cell Myocarditis in Cardiac Allograft . Circulation. 2003;108:e1 Fiedler's myocarditis. Who Named it. Giant Cell Myocarditis Responding to Immunosuppressive Therapy. Chest. 2000;117:905-907 Idiopathic Giant-Cell Myocarditis - Natural History and Treatment. Abstract NEJM Final Diagnosis. UPMC Pathology case. Shashi Ariyanayagam-Baksh, MBBS. A Rare Cause of Rapidly Evolved Stenosis of the Mitral Valve. Hellenic J Cardiol 2003. Giant Cell Myocarditis pathology slide. Case report with echo image. J Nippon Med Sch 2004. Dilated and Restrictive Cardiomyopathies Print version. The Cleveland Clinic. Dilated and Restrictive Cardiomyopathies. Photos The Cleveland Clinic. Myocarditis eMedicine Wai Hong W. Tang, MD. Myocarditis eMedicine David S. Howes, MD. Autopsy case with heart photos. The Indian Journal of Chest Disease. Giant Cell Arteritis Presenting With Annuloaortic Ectasia. Chest. 2002;121:1365-1367. J Am Soc Echocardiogr. 2004 Oct;17(10):1073-6.

100. Temple University Hospital - Department Of Surgery - About Us The cardiac service treats a spectrum of vascular, coronary artery and myocardialdiseases. Temple is one of the busiest adult heart transplant center in http://www.templehealth.org/surgeryresidency/about/

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