61. Miller Fisher Syndrome In Adult Onset Still's Disease: Case Report And Review Of KEY WORDS miller fisher syndrome, Still s disease, Guillain–Barré syndrome,Neurological manifestations, Adult Still s disease, gangliosides. http://rheumatology.oxfordjournals.org/cgi/content/abstract/41/2/216

JOURNAL HOME HELP FEEDBACK SUBSCRIPTIONS ... TABLE OF CONTENTS QUICK SEARCH: [advanced] Author: Keyword(s): Year: Vol: Page: This Article Full Text Full Text (PDF) Alert me when this article is cited ... Alert me if a correction is posted Services Email this article to a friend Similar articles in this journal Similar articles in ISI Web of Science Similar articles in PubMed ... Download to citation manager Search for citing articles in: ISI Web of Science (1) Request Permissions PubMed PubMed Citation Articles by Desai, S. S. Articles by Deodhar, A. Related Collections Connective tissue disease Rheumatology 2002; 41: 216-222 British Society for Rheumatology Grand Rounds in Rheumatology Miller Fisher syndrome in adult onset Still's disease: case report and review of the literature of other neurological manifestations S. S. Desai E. Allen and A. Deodhar Oregon Health Sciences University, 3181 SW Sam Jackson Park Road, CR 119, Portland, OR 97201, USA Abstract Adult-onset Still's disease (AOSD) is a multi-system inflammatory disorder characterized by high spiking fevers, evanescent salmon-coloured

62. Bioline International Official Site (site Up-dated Regularly) Short Report Recurrent miller fisher syndrome A Case Report Miller fishersyndrome (MFS) is a variant of Guillain-Barre syndrome characterized by http://www.bioline.org.br/abstract?id=ni02102&lang=en

63. Guillain-Barré Syndrome - May 15, 2004 - American Family Physician acute motor sensory axonal neuropathy (AMSAN), miller fisher syndrome, Yuki N, Hattori T. Clinical features and prognosis of miller fisher syndrome. http://www.aafp.org/afp/20040515/2405.html

64. Guillain-Barré Syndrome: Definition And Much More From Answers.com In a different and infrequent variant called miller fisher syndrome, MillerFisher Syndrome, however, is a descending weakness, proceeding in the http://www.answers.com/topic/guillain-barr-syndrome

showHide_TellMeAbout2('false'); Business Entertainment Games Health ... More... On this page: Dictionary Diagnosis Medical WordNet Wikipedia Mentioned In Or search: - The Web - Images - News - Blogs - Shopping Guillain-Barr© Syndrome Dictionary Guil·lain-Bar·r© Syndrome gē-yăn bə-rā n. A temporary inflammation of the nerves, causing pain, weakness, and paralysis in the extremities and often progressing to the chest and face. It typically occurs after recovery from a viral infection or, in rare cases, following immunization for influenza. [After Georges Guillain (1876–1961) and Jean Alexandre Barr© (1880–1967), French neurologists.] Diagnosis Guillain-Barre Syndrome What is Guillain-Barre Syndrome? Guillain-Barre Syndrome is a disease of the nervous system due to damage to the myelin sheath around nerves. The myelin sheath acts as an insulator the same as rubber or plastic around electrical wires. Guillain-Barre Syndrome is the most frequent acquired (as opposed to inherited) nerve disease (neuropathy). In many cases it follows shortly after a virus infection. It is also rarely associated with immunizations, surgery, and childbirth. What causes Guillain-Barre Syndrome?

65. NZORD Support Groups miller fisher syndrome. Both GBS and CIDP have more than one type. There are alsoother, more rare related conditions, both acute and chronic, http://www.nzord.org.nz/internal.asp?CategoryID=100006&ArticleID=100170

66. Journal Of Neuro-Ophthalmology - UserLogin syndrome with ophthalmoplegia and in the MillerFisher syndrome (8), ophthalmoplegia in miller fisher syndrome and Guillain-Barre syndrome clinical http://www.jneuro-ophthalmology.com/pt/re/jneuroophth/fulltext.00041327-20020600

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67. AJNR -- Sign In Page miller fisher syndrome (MFS) is characterized by the clinical triad of ophthalmoplegia,ataxia, Neuropathy in the miller fisher syndrome clinical and http://www.ajnr.org/cgi/content/full/25/4/645

HOME HELP FEEDBACK SUBSCRIPTIONS ... SEARCH QUICK SEARCH: [advanced] Author: Keyword(s): Year: Vol: Page: This item requires a subscription to American Journal of Neuroradiology Online. Full Text MR Imaging Findings of Spinal Posterior Column Involvement in a Case of Miller Fisher... Inoue et al. AJNR Am J Neuroradiol. This Article Abstract Figures Only Full Text (PDF) ... Alert me if a correction is posted Services Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal Download to citation manager PubMed PubMed Citation Articles by Inoue, N. Articles by Ushio, Y. To view this item, select one of the options below: Sign In User Name Sign in without cookies. Can't get past this page? Help with Cookies. Need to Activate? Password Forgot your user name or password? Purchase Short-Term Access Pay per Article - You may access this article (from the computer you are currently using) for 7 days for US$10.00 Pay for Admission - You may access all content in American Journal of Neuroradiology Online (from the computer you are currently using) for 7 days for US$25.00.

68. Entrez PubMed Bickerstaff s brainstem encephalitis, miller fisher syndrome and GuillainBarresyndrome overlap with negative anti-GQ1b antibodies. http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=1

69. Entrez PubMed miller fisher syndromea presenting clinical manifestation of lung cancer in apreviously apparently healthy individual. Csepany T, Boczan J, Magyar MT, http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=1

70. Miller Fisher Syndrome And Haemophilus Influenzae Infection -- Koga Et Al. 57 (4 Objective— To examine the association between miller fisher syndrome (MFS) and Does Haemophilus influenzae Infection Lead to miller fisher syndrome? http://www.neurology.org/cgi/content/abstract/57/4/686

HOME HELP FEEDBACK SUBSCRIPTIONS ... TABLE OF CONTENTS QUICK SEARCH: [advanced] Author: Keyword(s): Year: Vol: Page: This Article Figures Only Full Text Full Text (PDF) ... Citation Map Services Email this article to a friend Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal ... Cited by other online articles PubMed PubMed Citation Articles by Koga, M. Articles by Hirata, K. Related Collections All Infections Post-infectious All Neuromuscular Disease Peripheral neuropathy ... Guillain-Barre syndrome Neurology American Academy of Neurology Articles Miller Fisher syndrome and Haemophilus influenzae infection Michiaki Koga, MD, PhD; Nobuhiro Yuki, MD, PhD; Tadashi Tai, MD, PhD; and Koichi Hirata, MD, PhD From the Department of Neurology (Drs. Koga, Yuki, and Hirata), Dokkyo University School of Medicine, Tochigi; and Department of Tumor Immunology (Dr. Tai), Tokyo Metropolitan Institute of Medical Science, Tokyo, Japan. Address correspondence and reprint requests to Dr. Michiaki Koga, Department of Neurology, Yamaguchi University School of Medicine, Minami-Kogushi 1-1-1, Ube, Yamaguchi 755-8505, Japan; e-mail: Objective: Fisher syndrome (MFS) and antecedent Haemophilus influenzae infection.

71. Combined Pre- And Postsynaptic Action Of IgG Antibodies In Miller Fisher Syndrom Reduction in quantal content by miller fisher syndrome (MFS) serum or purifiedimmunoglobulin G (IgG). Ordinate quantal content relative to the control http://www.neurology.org/cgi/content/figsonly/56/1/67

HOME HELP FEEDBACK SUBSCRIPTIONS ... TABLE OF CONTENTS QUICK SEARCH: [advanced] Author: Keyword(s): Year: Vol: Page: This Article Abstract Full Text Full Text (PDF) ... Citation Map Services Email this article to a friend Similar articles in this journal Alert me to new issues of the journal Download to citation manager PubMed Articles by Buchwald, B. Articles by Toyka, K.V. Related Collections All Immunology All Neuromuscular Disease Guillain-Barre syndrome Combined pre- and postsynaptic action of IgG antibodies in Miller Fisher syndrome B. Buchwald, MD*; J. Bufler, MD*; M. Carpo, MD, PhD; F. Heidenreich, MD; R. Pitz, PhD; J. Dudel, MD; and K.V. Toyka, MD View larger version [in a new window] View larger version [in a new window] m = 0.7. (B) MFS-IgG of Patient 11; quantal content m = 0.2. (C) Wash-out (control solution); quantal content m = 0.5. All data were recorded from the same nerve terminal. The number of records evaluated for each distribution were 1,500 to 3,000, depending on quantal content. The solid line marks the gaussian distribution of single quanta. View larger version [in a new window] Figure 3. Concentration-dependent reduction in peak current amplitude by Miller Fisher syndrome (MFS) immunoglobulin G (IgG) from Patient 5. Amplitudes of single pulses of 1.0 mM acetylcholine to an outside-out patch were plotted versus time. Pulses were applied with a frequency of 0.25 Hz. The horizontal bars indicate duration of IgG application at the respective IgG concentration. Note that the reduction in peak current amplitude comes on faster and is more pronounced with high antibody concentrations.

72. GlycoWord / Glycopathology-A03 miller fisher syndrome is a variant of GuillainBarré syndrome, characterized bythe triad of ophthalmoplegia, ataxia and areflexia. http://www.glycoforum.gr.jp/science/word/glycopathology/GD-A03E.html

Immune-mediated Neuropathy and Antibody against Glycoconjugates Figure legend A hypothesis for the role of anti-GQ1b IgG antibody in the pathogenesis of ophthalmoplegia. Anti-GQ1b IgG antibodies specifically bind to GQ1b antigen that is localized in the paranodal myelin of the cranial nerves innervating ocular muscles to cause ophthalmoplegia. The other nerves, which lack in the dense localization of GQ1b antigen in the paranodal myelin, are not involved. Antibodies that recognize the disialosyl residue of ganglioside GD1b are specifically associated with sensory ataxic neuropathy. Experimental sensory ataxic neuropathy is induced by sensitization of rabbits with GD1b, which is localized in the primary sensory neurons of dorsal root ganglia. Antibodies against several other glycoconjugates, including myelin-associated glycoprotein, GM1, GD1a, GalNAc-GD1a and galactocerebroside, are known to be present in patients with some immune-mediated neuropathies. Some of the antibodies may bind to the glycoconjugate(s) with unique localization and determine the distribution of the damage in the pathogenesis of autoimmune neuropathies. Susumu Kusunoki (Department of Neurology, School of Medicine, University of Tokyo)

73. Directory Of Open Access Journals Title, Recurrent miller fisher syndrome a case report. Abstract,miller fisher syndrome (MFS) is a variant of GuillainBarre syndrome characterized http://www.doaj.org/abstract?id=86400&toc=y

74. NORD - National Organization For Rare Disorders, Inc. Chronic Inflammatory Demyelinating Polyneuropathy (CIDP); MillerFisher Syndrome polyradiculoneuropathy) are recognized miller fisher syndrome, http://www.rarediseases.org/search/rdbdetail_abstract.html?disname=Guillain Barr

75. PNS Meeting Platform Abstracts - Inflammatory Neuropathies miller fisher syndrome (MFS) is associated with antiGT1a/GQlb antibodies in 90%of cases. MFS follows C. jejuni enteritis and structural studies on C. http://pns.ucsd.edu/Plat1.abstracts.html

Back to Program PNS Home Inflammatory Neuropathies Abstracts Platform 1 THE MEMBRANE ATTACK COMPLEX OF COMPLEMENT (C5b-9) RESCUES SCHWANN CELLS (SchC) FROM APOPTOSIS IN EXPERIMENTAL ALLERGIC NEURITIS (EAN). Hafer-Macko C.E. Koski C.L. University of Maryland, Baltimore, Maryland, USA. MEMBRANE ATTACK COMPLEX OF COMPLEMENT (C5b9) INDUCES PROLIFERATION AND RESCUE FROM APOPTOSIS OF SCHWANN CELLS (SchC) Dashiell S.M., Rus H., Koski C.L University of Maryland, Baltimore, Maryland, USA C5b-9 deposition on SchC of GBS patients or EAN rats does not induce cytolysis secondary to complement regulatory protein expression. To determine if sublytic C5b-9 induces SchC mitogenesis and survival in vitro, rat SchC in serum free medium (N2) were stimulated with C5b-9 assembled from purified proteins (C5,6,7,8,9) or b -neuregulin (NRG). C5b-9 increased 3H-thymidine incorporation 10-fold by 24 hrs over N2 alone or N2 with heat inactivated C5b-9. Cell cycle analysis with propidium iodide and FACS showed SchC in N2 were synchronized in G1/G0 at 24 hrs. C5b-9 shifted 50% of SchC into S phase similar to NRG (100 ng/ml). C5b-9 stimulated SchC doubling as assessed by vital dye exclusion. Pertussis toxin and PD098,059, but not a PKA inhibitor, prevented C5b-9 mediated thymidine incorporation suggesting signal transduction was mediated via Gi proteins and ERK kinase. ERK1 activity, increased 2-fold by 20 min. Serum withdrawal and culture in N2 that caused 40-50% of SchC to undergo apoptosis was prevented by NRG. Likewise, C5b-9 inhibited the number of apoptotic cells and reduced TUNEL-positive SchC. C5b-9, like NRG, is a potent trophic factor stimulating SchC mitogenesis and apoptotic rescue and may contribute to peripheral nerve repair following immune-mediated demyelination.

76. Serum Antibodies Against Gangliosides And Campylobacter Jejuni Lipopolysaccharid Seven patients with miller fisher syndrome (MFS), six in the acute phase and onein the recovery phase, were investigated for serum antibodies against http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=175580

77. Guillain-Barre Syndrome- And Miller Fisher Syndrome-Associated Campylobacter Jej GuillainBarré Syndrome- and miller fisher syndrome-Associated Campylobacterjejuni Lipopolysaccharides Induce Anti-GM1 and Anti-GQ1b Antibodies in Rabbits http://iai.asm.org/cgi/content/abstract/69/4/2462

This Article Full Text Full Text (PDF) Alert me when this article is cited ... Alert me if a correction is posted Services Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal Download to citation manager ... Books from ASM Press PubMed PubMed Citation Articles by Ang, C. W. Articles by van Doorn, P. A. Infection and Immunity, April 2001, p. 2462-2469, Vol. 69, No. 4 0019-9567/01/$04.00+0 DOI: 10.1128/IAI.69.4.2462-2469.2001 American Society for Microbiology Campylobacter jejuni Lipopolysaccharides Induce Anti-GM and Anti-GQ Antibodies in Rabbits C. W. Ang, M. A. De Klerk, H. P. Endtz, B. C. Jacobs, J. D. Laman, and P. A. van Doorn Departments of Neurology, Immunology, Erasmus University, and Dijkzigt University Hospital, Rotterdam, The Netherlands Received 23 June 2000/Returned for modification 7 September 2000/Accepted 22 January 2001 Campylobacter jejuni (GBS) and Miller Fisher syndrome (MFS) by molecular mimicry between C. jejuni

78. Food Research Institute Briefings: Campylobacter -- Chronic Effects Campylobacter jejuni lipopolysaccharides in GuillainBarré and Miller Fishersyndrome. in patients with Guillain-Barré or miller fisher syndrome. http://www.wisc.edu/fri/briefs/campy.htm

FRI Briefings Campylobacter — Chronic Effects first published July 1998 prepared by M. Ellin Doyle , Ph.D. Food Research Institute, UW-Madison Foodborne illness is typically short-lived and primarily involves gastrointestinal symptoms. However, certain pathogens cause more life-threatening disease and certain individuals — because of age or immune status — are more likely to experience severe symptoms. More serious effects are often associated with specific pathogens: Clostridium botulinum is often lethal; E. coli O157.H7 has become infamous for causing hemolytic uremic syndrome in children; Listeria can cause miscarriages and toxoplasmosis is an important cause of congenital malformations; septicemia may result from infections with Salmonella and Listeria Foodborne bacteria have also been shown to cause chronic effects (sequelae), including arthritis, ankylosing spondylitis, renal disease, cardiac and neurological disorders, and nutritional and malabsorptive disorders. Some data indicate that 2–3% of the population as a whole develop chronic effects after foodborne illness, but follow-up of some outbreaks of salmonellosis revealed that 15–16% of cases suffered reactive arthritis in the months following their initial illness ( ). Persons positive for the major histocompatibility class antigen B27 have a significantly greater relative risk for developing some reactive diseases. As many as 20% of exposed, genetically susceptible persons may suffer these complications.

79. KoreaMed - Basic Search Brainstem Encephalitis Mimicking miller fisher syndrome. The possiblerelationship of the miller fisher syndrome and brainstem encephalitis are http://www.koreamed.org/SearchBasic.php?RID=106486&DT=1&QY=J Korean Neurol Asso

80. Miller Fisher's Syndrome (www.whonamedit.com) miller fisher s syndrome A disturbance thought to be a variant of the GuillainBarrésyndrome usually characterised by total external ophthalmoplegia, http://www.whonamedit.com/synd.cfm/1508.html

Home List categories Eponyms A-Z Biographies by country ... Contact Whonamedit.com does not give medical advice. This survey of medical eponyms and the persons behind them is meant as a general interest site only. No information found here must under any circumstances be used for medical purposes, diagnostically, therapeutically or otherwise. If you, or anybody close to you, is affected, or believe to be affected, by any condition mentioned here: see a doctor. A recommendation: Hypography is an open community about science and all things related Miller Fisher's syndrome Also known as: Fisher's syndrome Synonyms: Ophthalmoplegia-ataxia-areflexia syndrome, polyradiculoneuritis, type Fisher. Associated persons: Charles Miller Fisher Description: A disturbance thought to be a variant of the Guillain-Barré syndrome usually characterised by total external ophthalmoplegia, ataxia, and loss of tendon reflexes. Early symptoms include fever, headache, and pneumonia. They are followed by facial paralysis, diplopia, external ophthalmoplegia, and paresthesia of the arms and trunk. Reported in male patients between 38 and 65 years of age. Complete recovery is usual.

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