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         Iga Nephropathy:     more books (15)
  1. Iga Nephropathy: The 25th Year (Contributions to Nephrology) by Gilbert Faure, M. Kessler, et all 1993-06
  2. IgA Nephropathy - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References by ICON Health Publications, 2004-10-08
  3. Iga Nephropathy: Pathogenesis and Treatment (Contributions to Nephrology) by Yasuhiko Tomino, 1999-02
  4. Iga Nephropathy (Topics in Renal Medicine)
  5. IgA Nephropathy Today (Contributions to Nephrology)
  6. Moderately Proteinuric IgA Nephropathy in the Young (Biomedical and Health Research, V. 44)
  7. Iga Nephropathy: Pathogenesis and Treatment (Contributions to Nephrology) by A. R. Clarkson, 1995-02
  8. IgA Nephropathy Medical Guide by Qontro Medical Guides, 2008-07-09
  9. Steroid helps boost survival in IgA nephropathy.(Nephrology)(Immunoglobulin A): An article from: Internal Medicine News by Nancy Walsh, 2008-12-01
  10. Pulmonary capillaritis in IgA nephropathy.: An article from: Southern Medical Journal by Devanand Anantham, Kenneth P.W. Chan, et all 2007-06-01
  11. Primary IgA Nephropathy: Pathophysiology, Diagnosis, and Clinical Management.: An article from: Nephrology Nursing Journal by Betsy Jane Staples, 2001-04-01
  12. Clinical Features & Long Term Prognosis in Two Forms of Mesangial Glomerulonephritis, Iga Nephropathy & Igm Nephropathy (Acta Universitatis Tamperensis) by Juhani Myllymaki, 2009-11-12
  13. Recent Advances in Iga Nephropathy by Kar-neng Lai, 2009-01-12
  14. Iga Mesangial Nephropathy (Contributions to Nephrology) by G. D'Amico, L. Minetti, 1984-09

81. IgA Nephropathy
Abdalla Rifai, Ph.D. The pathogenesis of iga nephropathy, the most common form of glomerulonephritis in the world, is still largely unknown.
http://www2.lifespan.org/Nephrology/research/patho.html
Pathogenesis of IgA Nephropathy
Abdalla Rifai, Ph.D The pathogenesis of IgA nephropathy, the most common form of glomerulonephritis in the world, is still largely unknown. Utilizing an experimental model of IgA immune complex-mediated nephropathy, our laboratory is focused on elucidating in vivo the cellular and molecular mechanisms that are associated with glomerular IgA immune deposits. Identification of the early molecular events induced by glomerular IgA deposition includes establishing a profile of proinflammatory cytokines, chemokines and growth factors that initiate renal tissue injury. In addition, differential expression of extracellular matrix genes involved in the progressive stages of renal fibrosis is being characterized in a chronic model of this disease. This approach promises to provide an opportunity to develop rational therapies for IgA nephropathy. Home Search News Find a Physician ... Site Map
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82. Immunosuppressive Agents For Treating IgA Nephropathy (Cochrane Review)
Abstract of a systematic review of the effects of health care prepared by the Cochrane Collaboration.
http://www.cochrane.org/cochrane/revabstr/AB003965.htm
From The Cochrane Library, Issue 2, 2005
Immunosuppressive agents for treating IgA nephropathy (Cochrane Review)
Samuels JA, Strippoli GFM, Craig JC, Schena FP, Molony DA ABSTRACT What's new in this issue Search abstracts Browse alphabetical list of titles Browse by Review Group A substantive amendment to this systematic review was last made on 09 June 2003. Cochrane reviews are regularly checked and updated if necessary. Background: IgA nephropathy (IgAN) is a world-wide disease and the cause of end-stage renal failure (ESRF) in 15 to 20% of patients within 10 years and in 30 to 40% of individuals within 20 years from the apparent onset of disease. No specific treatment has yet been established but many approaches have been investigated. Objectives: To assess the benefits and harms of immunosuppressive treatment for IgAN. Search strategy: We searched The Cochrane Renal Group's specialized register (May 2003), Cochrane Central Register of Controlled Trials (CENTRAL) (The Cochrane Library, Issue 3, 2002) MEDLINE (1966 - September 2002), EMBASE (1988 - September 2002) and handsearched reference lists of retrieved articles and conference proceedings. Selection criteria: Randomized controlled trials (RCTs) and quasi-RCTs comparing treatment of IgAN with immunosuppressive agents against placebo, no treatment, other immunosuppressive or non-immunosuppressive agents.

83. IgA Nephropathy (Berger�s Disease) - Washington DC
iga nephropathy (Berger�s disease) Washington Hospital Center is located in Washington DC.
http://www.whcenter.org/14781.cfm
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IgA nephropathy (Berger�s disease)
Definition: IgA nephropathy is a kidney disorder characterized by blood in the urine . It is caused by inflammation of internal kidney structures and the deposit of IgA antibodies in kidney tissue.
Alternative Names: Nephropathy - IgA; Berger's disease
Causes, incidence, and risk factors: IgA nephropathy (Berger's disease) is a form of mesangial proliferative nephritis. Structures in the kidney called glomeruli become inflamed, and a type of antibody called IgA deposits in the kidney. The disorder can appear as acute , rapidly progressive, or chronic glomerulonephritis , or as visible or microscopic blood in the urine. IgA nephropathy usually is discovered after one or more episodes of dark or bloody urine in a person with no other symptoms of kidney disorder. Bloody urine may begin during or soon after a respiratory infection. Acute nephritic syndrome or nephrotic syndrome (groups of symptoms associated with decreased kidney functioning) may result.

84. Moderately Proteinuric IgA Nephropathy In The Young (Edited By: R. Coppo And L.
iga nephropathy (IgAN) is one of the most common glomerular diseases worldwide. Its natural history is consistent with a clinical onset in the first
http://www.iospress.nl/html/1586030590.php
Moderately Proteinuric IgA Nephropathy in the Young Volume Biomedical and Health Research
Edited by: R. Coppo and L. Peruzzi
2000, 134 pp., hardcover
ISBN:
Price:

IgA Nephropathy (IgAN) is one of the most common glomerular diseases world-wide. Its natural history is consistent with a clinical onset in the first decades of life. Clinical indicators of poor prognosis are proteinuria and hypertension. Angiotensin-converting enzyme-inhibitors (ACE-I) are a promising treatment, since Angiotensin II plays a key role in glomerular hypertension and permselectivity and modulates mesangial and tubular cell functions. This Concerned Action was designed to evaluate the effect of ACE-I (benazepril 0.2 mg/Kg) versus placebo in young IgAN patients ( 1g/day 50 ml/min/1.73m2). Activity and chronicity indexes are evaluated on renal biopsies together with the spatial distribution of IgA and C3 deposits by confocal microscopy. Ambulatory blood pressure measurements allow the identification of medium-high pressure levels. Enrolled patients are investigated for factors modulating the disease progression, including genetic predictors (gene polymorphism of HLA, IgA switch region, ACE, AT1 receptor, adducin, complement factors) and immunologic parameters (aberrantly glycosylated and macromolecular IgA in serum and cytokine urinary excretion).”
IOS Press
Nieuwe Hemweg 6B, 1013 BG Amsterdam, The Netherlands

85. IgA Nephropathy (Berger’s Disease)
iga nephropathy (Berger s disease) is a form of mesangial proliferative iga nephropathy progresses slowly. In many cases, it does not progress at all.
http://www.shands.org/health/information/article/000466.htm
Disease Injury Nutrition Poison ... Male urinary system
IgA nephropathy (Berger’s disease)
Definition: A kidney disorder characterized by blood in the urine ; caused by inflammation of the internal kidney structures and deposits of IgA antibodies in the kidney mesangial tissue.
Alternative Names: Nephropathy - IgA; Berger's disease
Causes, incidence, and risk factors: IgA nephropathy (Berger's disease) is a form of mesangial proliferative nephritis. Inflammation of the renal glomeruli occurs, and there are IgA (a type of antibody ) deposits in the kidney. The disorder can appear as acute , rapidly progressive, or chronic glomerulonephritis ; or as visible or microscopic hematuria blood in the urine
Berger's disease usually is discovered after one or more episodes of dark or bloody urine in a person with no other symptoms of kidney disorder. Bloody urine may begin during or soon after a respiratory infection. Acute nephritic syndrome or nephrotic syndrome (groups of symptoms associated with decreased kidney functioning) may result. Risk factors include having a personal or family history of IgA nephropathy or Henoch Schonlein purpura (a form of vasculitis that affects many parts of the body, and may cause a kidney lesion that is identical to the lesion of Berger's disease).

86. IgA Nephropathy (Berger’s Disease)
iga nephropathy is a kidney disorder characterized by blood in the urine. It is caused by inflammation of internal kidney structures and the deposit of IgA
http://www.pennhealth.com/ency/article/000466.htm
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IgA nephropathy (Berger’s disease)
Male urinary system Definition: IgA nephropathy is a kidney disorder characterized by blood in the urine . It is caused by inflammation of internal kidney structures and the deposit of IgA antibodies in kidney tissue. Alternative Names: Nephropathy - IgA; Berger's disease Causes, incidence, and risk factors: IgA nephropathy (Berger's disease) is a form of mesangial proliferative nephritis. Structures in the kidney called glomeruli become inflamed, and a type of antibody called IgA deposits in the kidney. The disorder can appear as acute , rapidly progressive, or chronic glomerulonephritis , or as visible or microscopic blood in the urine. IgA nephropathy usually is discovered after one or more episodes of dark or bloody urine in a person with no other symptoms of kidney disorder. Bloody urine may begin during or soon after a respiratory infection. Acute nephritic syndrome or nephrotic syndrome (groups of symptoms associated with decreased kidney functioning) may result.

87. IgA Nephropathy, The Most Common Cause Of Glomerulonephritis, Is Linked To 6q22&
iga nephropathy (IgAN) is the most common form of glomerulonephritis, Familial iga nephropathy. Evidence of an inherited mechanism of disease.
http://www.nature.com/ng/journal/v26/n3/full/ng1100_354.html
@import "/ng/style.css"; NATURE.COM NEWS@NATURE.COM NATUREJOBS NATUREEVENTS ... Help E-MAIL: PASSWORD: Save password Forgotten password? SEARCH JOURNAL Advanced search Journal home Archive Table of Contents ... For librarians NPG Resources Nature Nature Cell Biology Nature Medicine Nature Reviews Genetics ... Browse all publications Letter Nature Genetics
doi:10.1038/81677
Ali G. Gharavi , Yan Yan , Francesco Scolari , F. Paolo Schena , Giovanni M. Frasca , Gian Marco Ghiggeri , Kerry Cooper , Antonio Amoroso , Battista Fabio Viola , Graziana Battini , Gianluca Caridi , Cristina Canova , Anita Farhi , Vairavan Subramanian , Carol Nelson-Williams , Sue Woodford , Bruce A. Julian , Robert J. Wyatt Howard Hughes Medical Institute, Yale University School of Medicine, New Haven, Connecticut, USA. Department of Genetics, Yale University School of Medicine, New Haven, Connecticut, USA. Department of Medicine, Yale University School of Medicine, New Haven, Connecticut, USA. Department of Medicine, Mount Sinai School of Medicine, New York, New York, USA. Division and Chair of Nephrology, Spedali Civili, University of Brescia, Brescia, Italy.

88. IgA Nephropathy Information Diseases Database
iga nephropathy,Berger s disease, Disease Database Information.
http://www.diseasesdatabase.com/ddb1353.htm
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89. Treating IgA Nephropathy -- DILLON 12 (4): 846 -- Journal Of The American Societ
iga nephropathy may be the most common type of glomerulonephritis, worldwide. The chronic nature of iga nephropathy and the possibility of a good
http://www.jasn.org/cgi/content/full/12/4/846
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This Article Full Text (PDF) Alert me when this article is cited Alert me if a correction is posted ... Citation Map Services Email this article to a friend Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal ... Download to citation manager PubMed PubMed Citation Articles by DILLON, J. J. J Am Soc Nephrol 12:846-847, 2001
American Society of Nephrology
EDITORIAL
Treating IgA Nephropathy
JOHN J. DILLON Section of Nephrology, The University of Chicago, Chicago, Illinois. Correspondence to Dr. John J. Dillon, The University of Chicago, 5841 S. Maryland Avenue, MC-5100, Chicago, IL 60637. Phone: 773-702-5168; Fax: 773-753-1461; E-mail: IgA nephropathy may be the most common type of glomerulonephritis, worldwide. Once thought benign, it causes end-stage renal disease in 15 to 20% of individuals within 10 yr of onset ( ) and in 30 to 35% of individuals within 20 yr of onset. Proteinuria, an elevated

90. Controlled Prospective Trial Of Prednisolone And Cytotoxics In Progressive IgA N
Patients selected for moderately progressive iga nephropathy benefit from treatment with iga nephropathy treatment 25 years on can we halt progression?
http://www.jasn.org/cgi/content/abstract/13/1/142
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American Society of Nephrology
Controlled Prospective Trial of Prednisolone and Cytotoxics in Progressive IgA Nephropathy
Francis W. Ballardie and Ian S. D. Roberts *Department of Nephrology, Royal Infirmary and University, Manchester, United Kingdom; and Department of Cellular Pathology, John Radcliffe Hospital, Oxford, United Kingdom. Correspondence to Dr. Francis Ballardie, Manchester Royal Infirmary, Oxford Road, Manchester M13 9WL, United Kingdom. Phone: 44-161-276-4446; Fax: 44-161-276-4196; E-mail: ABSTRACT. In a single-center, multiple-referral source study

91. Rapidly Progressive IgA Nephropathy Kobayashi Naho, Nishida Masashi, Aoyama Mich
Peerreviewed open access biomedical speciality periodical from India covering various aspects of child health.
http://www.ijppediatricsindia.org/article.asp?issn=0019-5456;year=2005;volume=72

92. IgA Nephropathy - Quest Diagnostics Patient Health Library
iga nephropathy is a kidney disorder occuring during childhood and young adulthood. It usually follows a viral infection of the upper respiratory or
http://www.questdiagnostics.com/kbase/nord/nord587.htm
var hwPrint=1;var hwDocHWID="nord587";var hwDocTitle="IgA Nephropathy";var hwRank="1";var hwSectionHWID="nord587-Header";var hwSource="en-usQ2_05";var hwDocType="Nord";
National Organization for Rare Disorders, Inc.
IgA Nephropathy
Important
It is possible that the main title of the report IgA Nephropathy is not the name you expected. Please check the synonyms listing to find the alternate name(s) and disorder subdivision(s) covered by this report.
Synonyms
  • Berger's Disease Idiopathic Renal Hematuria Mesangial IGA Nephropathy
Disorder Subdivisions
  • None
General Discussion
IgA nephropathy is a chronic kidney disease that usually first appears during adolescence and young adulthood and often progresses to kidney failure. It usually follows a viral infection of the upper respiratory or gastrointestinal tracts. The major symptom is the passing of blood in the urine (hematuria). There may be associated pain in the loin area.
Resources
American Kidney Fund, Inc.
6110 Executive Boulevard
Suite 1010
Rockville, MD 20852

93. Complications Of IgA Nephropathy In A Non-Insulin-Dependent Diabetes Model, The
Complications of iga nephropathy in a NonInsulin-Dependent Diabetes Model, the Akita Mouse. TOSHIYUKI HASEYAMA,1,2 TOSHIKI FUJITA,1 FUJIKO HIRASAWA,1
http://www.sasappa.co.jp/online/search/file/page1.cgi?page=0111980404

94. IgA Nephropathy Foundation
provide vital and the latest information on the diagnosis and treatment of iga nephropathy to patients, their families, and their healthcare providers.
http://www.pslgroup.com/dg/dd246.htm

95. Northwestern Memorial Hospital - Exceptional Care In The Heart Of Chicago
Nephropathy IgA; Berger s disease. Causes, incidence, and risk factors. iga nephropathy (Berger s disease) is a form of mesangial proliferative nephritis
http://www.nmh.org/nmh/adam/adamencyclopedia/HIEArticles/000466.htm
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      Diseases / Conditions Injury Nutrition Poisons ... Male urinary system
      IgA nephropathy (Berger?s disease)
      Definition: IgA nephropathy is a kidney disorder characterized by blood in the urine . It is caused by inflammation of internal kidney structures and the deposit of IgA antibodies in kidney tissue.
      Alternative Names: Nephropathy - IgA; Berger's disease
      Causes, incidence, and risk factors: IgA nephropathy (Berger's disease) is a form of mesangial proliferative nephritis. Structures in the kidney called glomeruli become inflamed, and a type of antibody called IgA deposits in the kidney. The disorder can appear as acute , rapidly progressive, or chronic glomerulonephritis , or as visible or microscopic blood in the urine. IgA nephropathy usually is discovered after one or more episodes of dark or bloody urine in a person with no other symptoms of kidney disorder. Bloody urine may begin during or soon after a respiratory infection. Acute nephritic syndrome or nephrotic syndrome (groups of symptoms associated with decreased kidney functioning) may result.

96. Karger Publishers
The author of this volume has studied iga nephropathy for nearly 25 years, almost as long iga nephropathy, considered to be an immunecomplex-mediated
http://content.karger.com/ProdukteDB/produkte.asp?Aktion=showproducts&ProduktNr=

97. Karger Publishers
It is becoming evident that iga nephropathy is the commonest variety of primary iga nephropathy was initially considered a benign disease with a
http://content.karger.com/ProdukteDB/produkte.asp?Doi=45467

98. ScienceDaily -- Browse Topics: Health/Conditions_and_Diseases/Genitourinary_Diso
Vanderbilt Medical Center An article about iga nephropathy. iga nephropathy - A personal story of Darryl who had this disease. From the beginning, to the
http://www.sciencedaily.com/directory/Health/Conditions_and_Diseases/Genitourina
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99. CIN'2003. Geddes. Natural History Of IgA Nephropathy
iga nephropathy (IgAN) was first described in 1969 1 and is now The incidence of endstage renal disease (ESRD) due to iga nephropathy in USA between
http://www.uninet.edu/cin2003/conf/geddes/geddes.html

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... Voltar ao inicio cin2003 New insights into the natural history of IgA nephropathy in adults. Colin C. Geddes FRCP. (Gla)
Consultant Nephrologist, Western Infirmary, Glasgow, United Kingdom
Introduction IgA nephropathy (IgAN) was first described in 1969 [1] and is now recognised as the commonest primary glomerulopathy. The long-term outcome is highly variable [2]. Knowledge about the natural history of IgAN provides prognostic information, informs decisions about the risks and benefits of therapeutic options, can refine the design of clinical trials and enhances our understanding of the pathophysiology of progressive renal disease. This review will consider questions patients might ask and highlight recently published studies that have added to our knowledge about the natural history of IgAN. Clinical course of IgAN Reports from post mortem examinations and patients with recurrent IgAN in a renal transplant suggest that the hallmark of IgAN, mesangial deposition of IgA immune complexes, may be associated with a period of no clinical manifestation. At least a proportion of patients will subsequently develop microscopic haematuria which may be accompanied by proteinuria and hypertension. It is not known if, given enough time, all patients with mesangial IgA deposition would develop haematuria and proteinuria. Of the patients who develop proteinuria and hypertension a proportion will develop impaired renal function which is usually progressive.

100. HDCN -- SPNSG-NKFT: Basic Nephrology Lecture Series. May, 1998. IgA Nephropathy.
iga nephropathy Symposium supported by educational grants from the National Kidney Clinical Features and Prognostic Indicators of iga nephropathy
http://www.hdcn.com/symp/98iga/98iga.htm
Online Slide/Audio Symposium Basic Nephrology Lecture Series
May, 1998
IgA Nephropathy Symposium supported by educational grants from the National Kidney Foundation of Texas and in cooperation with the IgA Nephropathy Foundation.
IgA Nephropathy From October 28 - November 30, 1999, these talks will be in the FREE ZONE. You need to be a registered user of HDCN. To register for the FREE ZONE of HDCN click here . After November 30, 1999, you will need to have a PAID SUBSCRIPTION to HDCN to access these talks.
The Talks
Clinical Features and Prognostic Indicators of IgA Nephropathy

Ronald J. Hogg, M.D.
J. Charles Jennette, M.D.
Evolution of Treatment for IgA Nephropathy

Bryson Waldo, M.D.
Omega-3 and IgA Nephropathy
James V. Donadio, Jr., M.D. Role of ACE Inhibitors in the Management of IgA Nephropathy Daniel C. Cattran, M.D. Pathologic Features and How They Impact Diagnosis Richard K. Sibley, M.D. Faculty and Guests Daniel C. Cattran, M.D. Professor of Medicine, University of Toronto, Toronto, Ontario, Canada. James V. Donadio, Jr., M.D.

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