61. Diffuse Plane Normolipemic Xanthomatosis Associated With Takayasu's Disease And associated with Takayasu s disease and hyperhomocysteinemia A case report with hyperhomocysteinemia and DPNX is the only such report in literature. http://www.ijdvl.com/article.asp?issn=0378-6323;year=2004;volume=70;issue=4;spag

62. ACLS -- Sign In Page Research on familial hyperhomocysteinemia first suggested that elevated circulating levels of homocysteine (Hcy) were linked to atherosclerotic vascular http://www.annclinlabsci.org/cgi/content/full/34/2/175

HOME HELP FEEDBACK SUBSCRIPTIONS ... SEARCH QUICK SEARCH: [advanced] Author: Keyword(s): Year: Vol: Page: This item requires a subscription to Annals of Clinical and Laboratory Science Online. Full Text Effect of Experimental Hyperhomocysteinemia on Cardiac Structure and Function in... Walker et al. Ann Clin Lab Sci. This Article Abstract Full Text (PDF) Alert me when this article is cited ... Alert me if a correction is posted Services Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal Download to citation manager PubMed PubMed Citation Articles by Walker, E. Articles by Wright, G. L. To view this item, select one of the options below: Sign In User Name Sign in without cookies. Can't get past this page? Help with Cookies. Need to Activate? Password Forgot your user name or password? Purchase Short-Term Access Pay per Article - You may access this article (from the computer you are currently using) for 7 days for US$7.00 Pay for Admission - You may access all content in Annals of Clinical and Laboratory Science Online (from the computer you are currently using) for 7 days for US$25.00.

63. HNRCA : Hypercholesterolemia And Hyperhomocysteinemia Associated With Hypothyroi hyperhomocysteinemia and hypercholesterolemia associated with hypothyroidism in the third US National Health and Nutrition Examination Survey. http://hnrc.tufts.edu/publications/detail.php?id=108461

64. HNRCA : Hyperhomocysteinemia Associated With Poor Recall In An Nationally Repres hyperhomocysteinemia associated with poor recall in the third National Health and Nutrition Examination Survey. Am J Clin Nutr 2001;7392733. http://hnrc.tufts.edu/publications/detail.php?id=115256

65. Dr. Rose's Peripheral Brain--CAD RISK FACTORS hyperhomocysteinemia. Severely increased levels in homocystinuria associated with accelerated atherosclerosis; Moderately increased concentrations of http://faculty.washington.edu/momus/PB/cadriskf.htm

CAD RISK FACTORS n.b. For purposes of guiding management of dyslipidemias, NCEP specifies a small list of risk factors as countingSee under " Dyslipidemias " for details. The NCEP recommends counting CAD risk factors among those in the "major" category below, though sedentary lifestyle and dyslipidemias in general aren't counted; HDL < 35 counts as a risk factor, though; Also, HDL > 59 counts as a "negative" risk factor, i.e. if it's there, you subtract 1 from the count of risk factors MAJOR (per JAMA 269:3015, 1993): Smoking Hypertension Dyslipidemias (high LDL, low HDL, high TC/HDL or LDL/HDL ratio) Diabetes Mellitus Unknown whether glycemic control affects risk Raises risk more for women than for men (NEJM 332:1758, 1995-rvw) Sedentary lifestyle OTHERS: High HDL (> 60mg/dl) is a "negative" risk factor per NCEP (see under " dyslipidemias ") Obesity Traditionally considered a weak but significant independent factor; may contribute secondarily through effects on glucose metabolism, BP, etc. 115,000 women enrolled in Nurses' Health study 30-55yo and free of CV disease or Ca at enrollment followed for 16y. In multivariate analysis (adjusted for age, tobacco consumption, menopausal status, use of OCP's and postmenopausal HRT, and parental h/o MI before age 60), the following results were obtained

66. Methylenetetrahydrofolate Reductase Polymorphism (C677T), Hyperhomocysteinemia, hyperhomocysteinemia is associated with ischemic cardiovascular disease Therefore, ICD and VTE may cause hyperhomocysteinemia, rather than vice versa. http://www.bloodjournal.org/cgi/content/abstract/104/10/3046

QUICK SEARCH: [advanced] Author: Keyword(s): Year: Vol: Page: Blood, 15 November 2004, Vol. 104, No. 10, pp. 3046-3051. Prepublished online as a Blood First Edition Paper on June 29, 2004; DOI 10.1182/blood-2004-03-0897. This Article Full Text Full Text (PDF) All Versions of this Article: most recent Alert me when this article is cited Alert me if a correction is posted Citation Map Services Email this article to a friend Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal ... Rights and Permissions PubMed PubMed Citation Articles by Frederiksen, J. Articles by Nordestgaard, B. G. Related Collections Clinical Observations, Interventions, and Therapeutic Trials Hemostasis, Thrombosis, and Vascular Biology Related Letter in Blood Online CLINICAL OBSERVATIONS, INTERVENTIONS, AND THERAPEUTIC TRIALS Methylenetetrahydrofolate reductase polymorphism (C677T), hyperhomocysteinemia, and risk of ischemic cardiovascular disease and venous thromboembolism: prospective and case-control studies from the Copenhagen City Heart Study Jeppe Frederiksen Klaus Juul Peer Grande Gorm B. Jensen

67. Blood -- Sign In Page approach to establish if hyperhomocysteinemia is a cause or simply an effect of the disease. Indeed, hyperhomocysteinemia is not a monogenic condition, http://www.bloodjournal.org/cgi/content/full/105/8/3382-a

QUICK SEARCH: [advanced] Author: Keyword(s): Year: Vol: Page: This item requires a subscription to Blood Online. Full Text Hyperhomocysteinemia: cause or effect of disease? Marcucci et al. Blood. To view this item, select one of the options below: Sign In User Name Sign in without cookies. Can't get past this page? Help with Cookies. Need to Activate? Password Forgot your user name or password? Purchase Short-Term Access Pay per Article - You may access this article (from the computer you are currently using) for 1 day for US$25.00 Regain Access - You can regain access to a recent Pay per Article purchase if your access period has not yet expired. Sign Up Subscribe to the Journal - Subscribe to the print and/or online journal. Access for Patients Access for Patients - Patients desiring immediate access should contact the journal. This Article Full Text (PDF) Alert me when this article is cited Alert me if a correction is posted Services Email this article to a friend Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal ... Rights and Permissions PubMed PubMed Citation Articles by Marcucci, R.

68. HONselect Pulmonary Embolism in a Patient With Pernicious Anemia and Our report highlights the thrombotic risk of hyperhomocysteinemia secondary to We believe that the hyperhomocysteinemia associated with the PA may have http://129.195.254.70/cgi-bin/HONselect?browse C18.452.648.066.480

69. Pulmonary Embolism In A Patient With Pernicious Anemia And Hyperhomocysteinemia Our report highlights the thrombotic risk of hyperhomocysteinemia secondary to PA in a Key Words cobalamin • hyperhomocysteinemia • pernicious anemia http://www.chestjournal.org/cgi/content/abstract/122/4/1487

HOME HELP FEEDBACK SUBSCRIPTIONS ... TABLE OF CONTENTS QUICK SEARCH: [advanced] Author: Keyword(s): Year: Vol: Page: This Article Full Text Full Text (PDF) Submit a response ... Alert me if a correction is posted Services Email this article to a friend Similar articles in this journal Similar articles in ISI Web of Science Similar articles in PubMed ... Cited by other online articles PubMed PubMed Citation Articles by Caldera, A. Articles by Eiger, G. Chest. American College of Chest Physicians Pulmonary Embolism in a Patient With Pernicious Anemia and Hyperhomocysteinemia Angel Caldera, MD Jorge Mora, MD Morris Kotler, MD and Glenn Eiger, MD From the Department of Medicine (Drs. Caldera and Mora), and the Divisions of Cardiology (Dr. Kotler) and Pulmonary and Critical Care Medicine (Dr. Eiger), Albert Einstein Medical Center, Philadelphia, PA. Correspondence to: Angel Caldera, MD, Department of Medicine, Albert Einstein Medical Center, 5401 Old York Rd, Philadelphia, PA 19141; e-mail: We report the case of a 60-year-old woman with a history of ataxia who sought evaluation after a syncopal episode. A diagnostic

70. Hyperhomocysteinemia, Low Folate And Vitamin B12 Concentrations, And Methylene T hyperhomocysteinemia, Low Folate and Vitamin B12 Concentrations, and Methylene The estimated risk of CVT associated with hyperhomocysteinemia, http://stroke.ahajournals.org/cgi/content/abstract/35/8/1790

Published online before print June 10, 2004, doi:10.1161/01.STR.0000132570.24618.78 This Article Full Text Full Text (PDF) All Versions of this Article: most recent 01.STR.0000132570.24618.78v1 Alert me when this article is cited Alert me if a correction is posted Citation Map Services Email this article to a friend Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal ... Request Permissions PubMed PubMed Citation Articles by Cantu, C. Articles by Barinagarrementeria, F. Related Collections Clinical genetics Thrombophilia Thrombosis risk factors Cerebral Venous Thrombosis Stroke. Original Contributions Hyperhomocysteinemia, Low Folate and Vitamin B Concentrations, and Methylene Tetrahydrofolate Reductase Mutation in Cerebral Venous Thrombosis Carlos Cantu, MD, MSc Elisa Alonso, MD Aurelio Jara, BSc Irma Garcia, BSc Fernando Barinagarrementeria, MD Elevated plasma levels of homocysteine are associated with an increased risk of deep-vein thrombosis. association among homocysteine, folate and vitamin B

71. Revista De Nutrição - Translate this page The treatment of hyperhomocysteinemia consists on supplementation of the vitamins This study reviews the hyperhomocysteinemia etiology on chronic renal http://www.scielo.br/scielo.php?pid=S1415-52732005000200008&script=sci_arttext&t

72. Www.nlm.nih.gov/cgi/mesh/2K/MB_cgi?term=Hyperhomoc Acute methionine loadinduced hyperhomocysteinemia enhances To determine the true thrombogenicity of moderate hyperhomocysteinemia and better We conclude that moderate hyperhomocysteinemia plays a role in the http://www.nlm.nih.gov/cgi/mesh/2K/MB_cgi?term=Hyperhomocysteinemia

73. Entrez PubMed hyperhomocysteinemia, defined as an elevated concentration of hyperhomocysteinemia was defined as a fasting homocysteine concentration and/or an http://www.jcircadianrhythms.com/pubmed/9665374

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74. Methylenetetrahydrofolate Reductase (MTHFR) Thermolabile Variant hyperhomocysteinemia, C677T and A1298C Mutations; MTHFR In patients with hyperhomocysteinemia, followup testing for the MTHFR mutation might be http://www.labcorp.com/datasets/labcorp/html/chapter/mono/mg003500.htm

Methylenetetrahydrofolate Reductase (MTHFR) Thermolabile Variant, DNA Analysis Number CPT Related Information Factor II (Prothrombin), DNA Analysis Factor V Leiden Mutation Analysis Thrombotic Risk Profile, DNA Synonyms Hyperhomocysteinemia, C677T and A1298C Mutations; MTHFR Specimen Whole blood, buccal swab (The buccal swab collection kit contains instructions for the use of a buccal swab.) Volume 7 mL whole blood (EDTA) or buccal swab kit Minimum Volume 3 mL whole blood (EDTA) or two buccal swabs Container Lavender-stopper (EDTA) or yellow-stopper (ACD) tube, LabCorp buccal swab kit Storage Instructions Maintain specimen at room temperature or refrigerate. Causes for Rejection Frozen specimen; hemolyzed; quantity not sufficient for analysis; improper container; one buccal swab Use Follow-up evaluation in individuals with hyperhomocysteinemia; evaluation of patients with venous thrombosis Limitations This assay detects only the C677T and A1298C mutations in the MTHFR gene. The diagnosis of hyperhomocysteinemia can not rely on DNA testing alone but should take into consideration clinical findings and other studies, such as serum homocysteine levels. Methodology Polymerase chain reaction (PCR) and restriction enzyme analysis Additional Information Hyperhomocysteinemia has been found in women who have experienced two or more early pregnancy losses, placental infarction and fetal growth retardation, but MTHFR mutation as a cause for early pregnancy loss is still controversial. Homozygosity for C677T has been shown to have a 2 to 3-fold increased risk for neural tube defects (NTDs) such as anencephaly and spina bifida, and compound heterozygosity for C677T and A1298C may also be a risk factor for NTDs. Dietary folic acid supplementation before the fourth week of gestation is well documented in reducing the recurrence risk for open neural tube defects by approximately 75%. It may act by normalizing homocysteine levels.

75. Folate And Cobalamin Deficiencies And Hyperhomocysteinemia In Bangladesh -- Gamb Results The prevalence of hyperhomocysteinemia (men 11.4 µmol/L; women 10.4 µmol/L) was markedly (P 0.0001) greater among men (63%; ± SD 15.3 ± 9.5 http://www.ajcn.org/cgi/content/abstract/81/6/1372

HOME HELP FEEDBACK SUBSCRIPTIONS ... TABLE OF CONTENTS QUICK SEARCH: [advanced] Author: Keyword(s): Year: Vol: Page: This Article Full Text Full Text (PDF) Purchase Article ... Citation Map Services Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal Download to citation manager ... Request Permissions PubMed PubMed Citation Articles by Gamble, M. V Articles by Graziano, J. H American Journal of Clinical Nutrition, Vol. 81, No. 6, 1372-1377, June 2005 American Society for Clinical Nutrition ORIGINAL RESEARCH COMMUNICATION Folate and cobalamin deficiencies and hyperhomocysteinemia in Bangladesh Mary V Gamble Habibul Ahsan Xinhua Liu Pam Factor-Litvak Vesna Ilievski Vesna Slavkovich Faruque Parvez and Joseph H Graziano From the Departments of Environmental Health Sciences (MVG, VI, VS, FP, and JHG), Epidemiology (HA and PF-L), and Biostatistics (XL), Mailman School of Public Health, and the Department of Pharmacology, College of Physicians and Surgeons (JHG), Columbia University, New York, NY, and the New York State Psychiatric Institute, New York, NY (XL) Background: Indian Asian men residing in the United Kingdom have a higher prevalence of hyperhomocysteinemia than do their European counterparts. This has been largely attributed to dietary

76. High Prevalence Of Hyperhomocysteinemia Related To Folate Deficiency And The 677 Background Moderate hyperhomocysteinemia is a risk for neural tube defect Conclusion A high prevalence of hyperhomocysteinemia in coastal West Africa http://www.ajcn.org/cgi/content/abstract/79/4/619

HOME HELP FEEDBACK SUBSCRIPTIONS ... TABLE OF CONTENTS QUICK SEARCH: [advanced] Author: Keyword(s): Year: Vol: Page: This Article Full Text Full Text (PDF) Purchase Article ... Citation Map Services Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal Download to citation manager ... Request Permissions PubMed PubMed Citation Articles by Amouzou, E. K American Journal of Clinical Nutrition, Vol. 79, No. 4, 619-624, April 2004 American Society for Clinical Nutrition ORIGINAL RESEARCH COMMUNICATION High prevalence of hyperhomocysteinemia related to folate deficiency and the 677C T mutation of the gene encoding methylenetetrahydrofolate reductase in coastal West Africa Emile K Amouzou Charles E Adjalla Christian Villaume Ambaliou Sanni and Background: Moderate hyperhomocysteinemia is a risk for neural tube defect and neurodegenerative and vascular diseases and has nutritional, metabolic, and genetic determinants. Its prevalence in sub-Saharan Africa remains unknown. Objective: Our goal was to evaluate the prevalence of hyperhomocysteinemia and the influence of nutritional, metabolic, and genetic determinants

77. Hyperhomocysteinemia Induces Hepatic Cholesterol Biosynthesis And Lipid Accumula hyperhomocysteinemia is an independent risk factor for cardiovascular disorders. hyperhomocysteinemia was induced in SpragueDawley rats by feeding a http://ajpendo.physiology.org/cgi/content/abstract/288/5/E1002

HOME HELP FEEDBACK SUBSCRIPTIONS ... TABLE OF CONTENTS QUICK SEARCH: [advanced] Author: Keyword(s): Year: Vol: Page: Am J Physiol Endocrinol Metab 288: E1002-E1010, 2005. First published January 11, 2005; doi:10.1152/ajpendo.00518.2004 This Article Full Text Full Text (PDF) All Versions of this Article: most recent Alert me when this article is cited Alert me if a correction is posted Citation Map Services Email this article to a friend Similar articles in this journal Similar articles in ISI Web of Science Similar articles in PubMed ... Download to citation manager PubMed PubMed Citation Articles by Woo, C. W. H. Articles by O, K. Hyperhomocysteinemia induces hepatic cholesterol biosynthesis and lipid accumulation via activation of transcription factors Connie W. H. Woo, Yaw L. Siow, Grant N. Pierce, Patrick C. Choy, Gerald Y. Minuk, David Mymin, and Karmin O Departments of Physiology and Animal Science, National Centre for Agri-Food Research in Medicine, St. Boniface Hospital Research Centre, Centre for Research and Treatment of Atherosclerosis, Faculty of Medicine, and

78. Chlamydia Pneumoniae Seropositivity And Hyperhomocysteinemia Are Linked In Patie Concurrently, epidemiologic evidence suggests that hyperhomocysteinemia is Both C. pneumoniae infection and hyperhomocysteinemia have been assumed to http://www.clinchem.org/cgi/content/full/47/7/1304

HOME HELP FEEDBACK SUBSCRIPTIONS ... TABLE OF CONTENTS QUICK SEARCH: [advanced] Author: Keyword(s): Year: Vol: Page: Clinical Chemistry This Article Extract Full Text (PDF) Submit a response ... Alert me if a correction is posted Services Email this article to a friend Similar articles in this journal Similar articles in ISI Web of Science Similar articles in PubMed ... Cited by other online articles Search for citing articles in: ISI Web of Science (11) PubMed PubMed Citation Articles by Wiesli, P. Articles by Schulthess, G. Related Collections Lipids, Lipoproteins, and Cardiovascular Risk Factors Clinical Chemistry. Technical Briefs Chlamydia pneumoniae Seropositivity and Hyperhomocysteinemia Are Linked in Patients with Atherosclerosis Peter Wiesli Friedrich E. Maly Alfredo Meniconi Wolfgang Czerwenka Ulrich Hoffmann Wilhelm Vetter and Georg Schulthess a Department of Internal Medicine, Medical Policlinic, Institute of Clinical Chemistry, and Department of Angiology, University Hospital, CH-8091 Zurich, Switzerland; a address correspondence to this author at: Department of Internal Medicine, University Hospital, CH-8091 Zurich, Switzerland; fax 0041-1-255 45 67, e-mail

79. Selective Screening For Hyperhomocysteinemia In Pediatric Patients -- Vilaseca E hyperhomocysteinemia is a common condition with genetic and environmental Testing for hyperhomocysteinemia in children may be useful for investigation http://www.clinchem.org/cgi/content/extract/44/3/662

HOME HELP FEEDBACK SUBSCRIPTIONS ... TABLE OF CONTENTS QUICK SEARCH: [advanced] Author: Keyword(s): Year: Vol: Page: Clinical Chemistry This Article Full Text Full Text (PDF) Submit a response ... Citation Map Services Email this article to a friend Similar articles in this journal Similar articles in ISI Web of Science Similar articles in PubMed ... Cited by other online articles Search for citing articles in: ISI Web of Science (14) PubMed PubMed Citation Articles by Vilaseca, M. A. Articles by Camacho, J.-A. Related Collections Laboratory Management Pediatric Clinical Chemistry Nutrition Evidence Based Laboratory Medicine and Test Utilization ... Endocrinology and Metabolism Clinical Chemistry. Technical Briefs Selective Screening for Hyperhomocysteinemia in Pediatric Patients ,a Dolores Moyano Rafael Artuch Imma Ferrer Esther Cardo Jaume Campistol Carles Pavia , and Neurol., i a author for correspondence: fax 34-3-2803626 Hyperhomocysteinemia is a common condition with genetic and environmental causes (1), and associated, especially in its

80. Diabetes Forum : Complication_Causes Of  Hyperhomocyst(e)inemia Mellitus Complication Homocysteine AVD Mellitus Free Radicals Nerphropathological changes Oxidation Cause of hyperhomocysteinemia http://www.diabetesforum.net/eng_comp_causehyper .htm

Basics Info Centre Case Studies Myths and Facts ... Site Map Special features select one News Letter Write your own articles Ask questions to doctor Forum Introduction Elevations in homocyst(e)ine are typically caused either by genetic defects in the enzymes involve in homocysteine metabolism or by nutritional deficiencies in vitamin co-factors. Thus, reduction in activity of MS, MTHFR, or CbS, or decreased availability of their co-factors, vitamin B12, folic acid or vitamin B6 respectively, can cause hyperhomocyst(e)inemia. Defects of betaine homocysteine methylene transferase have not been described in literature. Genetic Nutritional Miscellaneous Homocyst(e)ine levels are higher in men than in women, both fasting and post methionine loading, and usually higher in postmenopausal rather than premenopausal women. Homocyst(e)ine also rises with age even when allowance is made for age-related increase in creatinine and decreasing vitamin levels. The rise may be related to age induced reduction in the function of MS or CbS. Other conditions that may be associated with hyperhomocyst(e)inemia are hypothyroidism, impaired kidney function, systemic lupus erythematosus and certain medications, e.g., nicotinic acid, nitrous oxide exposure, theophylline, methotrexate, and L-dopa.

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