61. Brazilian Journal Of Medical And Biological Research - Glycosuria in glomerular diseases histopathology and clinical correlations Tubular dysfunction is not uncommon in glomerular diseases and is frequently http://www.scielo.br/scielo.php?script=sci_arttext&pid=S0100-879X1998000500005

62. Soluble Interleukin-2 Receptor In Patients With Glomerular Diseases -- Chen Et A types of glomerular diseases; 40 healthy volunteers were used as control. All patients with glomerular diseases had levels of serum sIL2R significantly http://pmj.bmjjournals.com/cgi/content/abstract/71/840/617

HOME HELP FEEDBACK SUBSCRIPTIONS ... TABLE OF CONTENTS Author Keyword(s) Vol Page [Advanced] This Article Submit a response Alert me when this article is cited Alert me when eLetters are posted Alert me if a correction is posted Services Email this link to a friend Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal ... Download to citation manager PubMed PubMed Citation Articles by Chen, H. S. Articles by Chen, W. Y. Postgraduate Medical Journal, 1995, Vol 71, 617-622 ARTICLES Soluble interleukin-2 receptor in patients with glomerular diseases HS Chen, MS Wu, TS Yen and WY Chen Department of Internal Medicine, National Taiwan University Hospital, Taipei, Republic of China. In this study, we measured the soluble interleukin-2 receptor (sIL-2R) level to evaluate the cellular immune status in 61 patients with different types of glomerular diseases; 40 healthy volunteers were used as control.

63. Glomerular Diseases - WI, Milwaukee WI glomerular diseases Columbia St. Mary s Community Physicians of Wisconsin, Milwaukee, Ozaukee and Washington County. http://www.columbia-stmarys.org/14553.cfm

Print Search Send to a friend Home �Conditions and Concerns Glomerular diseases Adapted from the National Institutes of Health by HealthGate Editorial Staff Many diseases affect kidney function by attacking the tiny units within the kidney where blood is cleaned. Glomerular diseases include many conditions with a variety of genetic and environmental causes, but they fall into two major categories: Glomerulonephritis (gloh-MAIR-yoo-loh-neh-FRY-tis) describes the inflammation of the membrane tissue in the kidney that serves as a filter, separating wastes and extra fluid from the blood. Glomerulosclerosis (gloh-MAIR-yoo-loh-skleh-ROH-sis) describes the scarring or hardening of the tiny blood vessels within the kidney. Although glomerulonephritis and glomerulosclerosis have different causes, they can both lead to end-stage renal disease (ESRD). What are the kidneys and what do they do? The two kidneys are bean-shaped organs located near the middle of the back, just below the rib cage to the left and right of the spine. Each about the size of a fist, these organs act as sophisticated filters for the body. They process about 400 quarts of blood a day to sift out about 2 quarts of waste products and extra water that eventually leave the body as urine. Blood enters the kidneys through arteries that branch inside the kidneys into tiny clusters of looping blood vessels. Each cluster is called a

64. Glomerular Diseases glomerular diseases is the third most common cause of endstage renal disease (after glomerular diseases Pathogenesis. Pathogenetic mechanisms include http://www.total-health-care.com/illness/glomerular-diseases.htm

Make us your Homepage Bookmark Site Practical First Aid Medical Tests ... Renal Cyst Glomerular diseases Glomerular diseases is the third most common cause of end-stage renal disease (after diabetes and hypertension) in Europe and the USA, accounting for some 10-15% of such patients. Glomerulonephritis is a general term for a group of disorders in which: there is primarily an immunologically mediated injury to glomeruli, although renal interstitial damage is a regular accompaniment the kidneys are involved symmetrically secondary mechanisms of glomerular injury come into play following an initial immune insult (see below) the renal lesion may be part of a generalized disease (e.g. systemic lupus erythematosus, SLE). Glomerular diseases Pathogenesis Pathogenetic mechanisms include: deposition or in situ formation of immune complexes deposition of antiglomerular basement membrane antibody (fewer than 5% of glomerulonephritides) deposition of an immunoglobulin of atypical configuration in glomeruli, as in IgA nephropathy. These pathogenetic mechanisms activate secondary mechanisms that produce glomerular damage.

65. Arch Intern Med -- Modified Serum Albumin In The Pathogenesis Of Glomerular Dise Modified Serum Albumin in the Pathogenesis of glomerular diseases A New Hypothesis. Arch Intern Med. 2002;162356358. http://archinte.ama-assn.org/cgi/content/extract/162/3/356

Select Journal or Resource JAMA Archives of Dermatology Facial Plastic Surgery Family Medicine (1992-2000) General Psychiatry Internal Medicine Neurology Ophthalmology Surgery Student JAMA (1998-2004) JAMA CareerNet For The Media Meetings Peer Review Congress Vol. 162 No. 3, February 11, 2002 Featured Link E-mail Alerts Comments, Opinions, and Brief Case Reports Article Options Full text PDF Send to a Friend Similar articles in this journal Literature Track Add to File Drawer Download to Citation Manager PubMed citation Articles in PubMed by Kozhevnikov AD Contact me when this article is cited Modified Serum Albumin in the Pathogenesis of Glomerular Diseases: A New Hypothesis Arch Intern Med. Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings. In 1984 we first reported that human serum albumin (HSA) in its denatured form was present in a large amount, up to 25% in terms of fully denaturated protein, in the serum of patients with chronic renal failure (CRF) and patients with the nephrotic syndrome (NS).

66. Diagnostic Atlas Of Renal Pathology Normal Growth and Maturation Primary glomerular diseases glomerular diseases THAT CAUSE NEPHROTIC SYNDROME NONIMMUNE COMPLEX Minimal Change Disease and http://www.harcourt-international.com/catalogue/title.cfm?ISBN=1416028714

67. NephCure Helps NIDDK Plan Glomerular Disease Workshop Patients who are impacted by glomerular diseases will be encouraged to participate. Details about the conference will be posted closer to the date of the http://www.nephcure.org/News_niddkworkshop.htm

NephCure Helps NIDDK Plan... Dr. Elias Zerhouni NephCure Helps NIDDK Plan Glomerular Disease Workshop This goal is consistent with the NIH Roadmap to Research initiative developed by NIH Director, Dr. Elias Zerhouni. There are also plans for the workshop to include plenary speakers, discussion on opportunities and challenges in glomerular disease research, and evaluation of what resources are needed to carry out these opportunities and challenges. The workshop will be open to the public and the scientific community. Patients who are impacted by glomerular diseases will be encouraged to participate. Details about the conference will be posted closer to the date of the conference. info@nephcure.org

68. Elsevier.com - Diagnostic Atlas Of Renal Pathology Normal Growth and Maturation Primary glomerular diseases GLOMERULAR Congenital Nephrotic Syndrome of Finnish Type glomerular diseases THAT CAUSE http://www.elsevier.com/wps/product/authors/705094

Home Site map Regional Sites Advanced Product Search ... Diagnostic Atlas of Renal Pathology Book information Product description Audience Author information and services Ordering information Bibliographic and ordering information Book related information Submit your book proposal Other books in same subject area About Elsevier Select your view DIAGNOSTIC ATLAS OF RENAL PATHOLOGY A Companion to Brenner and Rector's The Kidney 7E To order this title, and for more information, go to http://www.elsevierhealth.com/title.cfm?ISBN=1416028714 First Edition By Agnes Fogo , MD, Professor of Pathology, Medicine and Pediatrics, Director, Renal Electron Microscopy laboratory, Vanderbilt University Medical Center, Nashville, TN, USA Michael Kashgarian , MD, Professor of Pathology and Molecular, Cellular, and Developmental Biology; Director of Laboratories; Director, Renal and Electron Microscopy Laboratory, Department of Pathology, Yale University School of Medicine, New Haven, CT Description This new atlas covers all major inflammatory, infectious, pre-neoplastic, and neoplastic diseases of the kidney. Over 600 illustrations help readers to recognize the pathologic features and clinical manifestations of both common and rare renal disorders and to formulate confident and accurate diagnoses. This resource, Diagnostic Atlas of Renal Pathology, is designed as a companion book to, ?The Kidney 7th Edition? by Brenner and Rector. The newest information regarding categorizing and classification of diseases is presented and how this relates to the various morphological lesions illustrated and their clinical significance.

69. Two Different Glomerular Diseases In The Same Patient At An Interval Of 7 Years Family investigation proved negative for glomerular diseases, renal failure, arterial hypertension and diabetes mellitus. In 1991, when he was 25 years old, http://ndt.oxfordjournals.org/cgi/content/full/17/11/2014

JOURNAL HOME HELP FEEDBACK SUBSCRIPTIONS ... TABLE OF CONTENTS QUICK SEARCH: [advanced] Author: Keyword(s): Year: Vol: Page: This Article Extract FREE Full Text (PDF) Alert me when this article is cited ... Alert me if a correction is posted Services Email this article to a friend Similar articles in this journal Similar articles in ISI Web of Science Similar articles in PubMed ... Request Permissions PubMed PubMed Citation Articles by Stefoni, S. Nephrol Dial Transplant (2002) 17: 2014-2016 European Renal Association-European Dialysis and Transplant Association Case Reports Two different glomerular diseases in the same patient at an interval of 7 years Letizia Soverini Paola Preda Gianandrea Pasquinelli Claudio Orsi Rita Prandini Alba Vangelista and Sergio Stefoni Nephrology, Dialysis and Renal Transplantation Unit and Electron Microscopy Unit, Pathology Department, St Orsola University Hospital, Bologna, Italy Keywords: double glomerulopathy; IgA nephritis; membranous glomerulonephritis Introduction Top Introduction Case Discussion References Overlapping of different glomerulopathies has already been observed in renal biopsies from diabetic as well as non-diabetic patients ]. Unlike other reports, the present paper describes a patient

70. Abnormal Podocyte CR-1 Expression In Glomerular Diseases: Association With Glome However, the majority of biopsies from patients with glomerular diseases showed abnormal CR1 expression (59 of 74), most evident in proliferative biopsies http://ndt.oxfordjournals.org/cgi/content/abstract/2/5/304

JOURNAL HOME HELP FEEDBACK SUBSCRIPTIONS ... TABLE OF CONTENTS QUICK SEARCH: [advanced] Author: Keyword(s): Year: Vol: Page: This Article Alert me when this article is cited Alert me if a correction is posted Services Email this article to a friend Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal ... Request Permissions PubMed PubMed Citation Articles by Nolasco, F. E. Articles by Reuben, R. ORIGINAL ARTICLES Abnormal podocyte CR-1 expression in glomerular diseases: association with glomerular cell proliferation and monocyte infiltration FE Nolasco, JS Cameron, B Hartley, RA Coelho, G Hildredth and R Reuben Servico de Nefrologia, Hospital Curry Cabral, Portugal. The expression of CR-1 complement receptors on glomerular epithelial cells, was studied in 77 renal biopsies from patients with (74) or without (3) glomerular diseases, employing an anti-CR-1 monoclonal antibody, and an indirect immunoperoxidase technique. Four patterns of CR-1 expression were recognised: normal (18); generally decreased (6); focal/segmental partial

71. Penn State Faculty Research Expertise Database (FRED) Newer glomerular diseases. Citation, Abt AB. Cohen AH. Newer glomerular diseases. 1996 Nov. Semin Nephrol. 16(6)50110. Faculty Authors, Arthur B. Abt http://fred.hmc.psu.edu/ds/retrieve/fred/publication/9125794

72. ÿþ HTML HEAD META Http - Equiv = Content - Type In the United States , the major glomerular diseases causing end stage renal Patients with glomerular disease may be classified into two groups http://www.acpmedicine.com/abstracts/sam/med1005.htm

Glomerular Diseases Abstract March 2005 Section 10 Nephrology V Glomerular Diseases Raimund H. Pichler, M.D. Assistant Professor of Medicine, Division of Nephrology, University of Washington School of Medicine Stuart J. Shankland, M.D. Associate Professor of Medicine and Director, Nephrology Fellowship Program, Division of Nephrology, University of Washington School of Medicine © 2005 WebMD Inc. All Rights Reserved. WebMD Professional Publishing, P.O. Box 1819, Danbury CT 06813 (800-545-0554 or 203-790-2087)

73. Renal Endothelin-1 And Endothelin Receptor Type B Expression In Glomerular Disea Renal Endothelin1 and Endothelin Receptor Type B Expression in glomerular diseases with Proteinuria. INGO LEHRKE , RÜDIGER WALDHERR , EBERHARD RITZ and http://www.jasn.org/cgi/content/abstract/12/11/2321

HOME HELP FEEDBACK SUBSCRIPTIONS ... TABLE OF CONTENTS QUICK SEARCH: [advanced] Author: Keyword(s): Year: Vol: Page: This Article Full Text Full Text (PDF) Alert me when this article is cited ... Citation Map Services Email this article to a friend Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal ... Cited by other online articles PubMed PubMed Citation Articles by LEHRKE, I. Articles by WAGNER, J. J Am Soc Nephrol 12:2321-2329, 2001 American Society of Nephrology Renal Endothelin-1 and Endothelin Receptor Type B Expression in Glomerular Diseases with Proteinuria INGO LEHRKE EBERHARD RITZ and Department of Nephrology, University of Heidelberg, Germany. Abstract . The endothelin (ET) system has been studied extensively in experimental models of progressive chronic renal disease, but there is limited information regarding the ET system in renal patients. First, the expression of human ET-1, as well as ET receptor type A (ET-R A ) and ET-R B , was studied in 26 renal biopsies from patients with different renal diseases. Gene expression

74. Diagnostic Atlas Of Renal Pathology A Companion To Brenner And Contents. Chapter 1. glomerular diseases Normal Growth and Maturation Primary glomerular diseases glomerular diseases THAT CAUSE NEPHROTIC SYNDROME http://www.intl.elsevierhealth.com/catalogue/titleprint.cfm?ISBN=1416028714

75. Project: Pathophysiology, Immunology And Treatment Of Renal Disease. (www.onderz Leusen, Bosch) Prospective clinical trials in glomerular diseases. INTERNATIONAL Dept. of Nephrology, Mannheim (van der Woude) Abnormalities in HSPG http://www.onderzoekinformatie.nl/en/oi/nod/onderzoek/OND1264315/toon

Login English KNAW Research Information NOD - Dutch Research Database ... Research entire www.onderzoekinformatie.nl site fuzzy match Project: Pathophysiology, immunology and treatment of renal disease. Print View Titel Pathofysiologie, immunologie en behandeling van nieraandoeningen Abstract Period 01/1995 - unknown Related organisations Secretariat: Department of Nephrology (KUN) Related persons Project leader: Prof.dr. J.H.M. Berden Project leader: Dr. F.Th.M. Huysmans Project leader: Prof.dr. R.A.P. Koene Project leader: Dr. W.J.M. Tax Project leader: Prof.dr. J.F.M. Wetzels Researcher: Drs. M.B. Andresdottir Researcher: Dr. J. van den Born Researcher: A.J.W. Branten Researcher: Dr. M.C.J. van Bruggen Researcher: Drs. W.L. Diemont Researcher: Dr. L.B. Hilbrands Researcher: Dr. A.J. Hoitsma Researcher: Dr. F.Th.M. Huysmans Researcher: Dr. R. Licht Researcher: Dr. S.M.A. Peters Researcher: Drs. C.J.I. Raats Researcher: Dr. R.G.L. de S©vaux Researcher: Dr. W.J.M. Tax Researcher: Drs. G. Vervoort Researcher: Prof.dr. J.F.M. Wetzels

76. Antimouse Laminin Antibodies In IgA Nephropathy And Various Glomerular Diseases. from patients with various glomerular diseases (50 cases of IgA nephropathy, between patients with other glomerular diseases and normal controls. http://www.pdg.cnb.uam.es/UniPub/iHOP/gp/6682109.html

Antimouse laminin antibodies in IgA nephropathy and various glomerular diseases.IgG, IgA and IgM class antibodies to mouse laminin and human fibronectin in sera from patients with various glomerular diseases (50 cases of IgA nephropathy enzyme-linked immunosorbent assay method. IgA antimouse laminin antibody titers in sera from IgA nephropathy patients were significantly higher (p less than 0.05) than in controls. There were no statistical differences in IgA antimouse laminin antibody titers between patients with other glomerular diseases and normal controls. IgM antimouse laminin antibody was significantly raised (p less than 0.01) in sera from patients with preeclampsia. The reaction of mouse laminin with the IgA nephropathy and preeclampsia sera on each of the IgA and IgM assay systems was inhibited by the antigen at up to 5 micrograms/ml. However, it was not inhibited by anti-C3d, anti-C1q, anti-J chain and antisecretory component sera or saccharides. The reaction of mouse laminin with an exceptionally high-titer IgA antimouse laminin antibody serum from a normal control on the IgA assay system was clearly inhibited by 1 mM of melibiose, which contains alpha-galactosyl residues.

77. APRIL 18, 2002 Renal Disease, 90 percent of which is caused by glomerular diseases;. Whereas glomerular diseases of the kidney cause 360000 people in the United States http://thomas.loc.gov/cgi-bin/query/z?c107:H. Con. Res. 383:

78. Lund University, Doctoral Dissertation Conclusions During the course of glomerular diseases a maintained low urinary excretion of IgG or IgM indicates a salutary prognosis. http://www.lub.lu.se/cgi-bin/show_diss.pl/med_900.html

Lund University, Doctoral Dissertation Faculty of Medicine Nephrology Postal address of Department Public defence of dissertation Segerfalksalen, Wallenberg Neurocentrum, BMC, Lund, fredag den 26-mars 2004, kl. 09.15 Author Omran Bakoush Title and subtitle Diagnostic and prognostic value of proteinuria in chronic renal diseases Abstract In study IV, we observed that patients with type 2 DN had a higher urinary excretion of high molecular weight proteins (IgG and IgM) than patients with type 1 DN, despite similar degree of albuminuria. This suggests partly different patho-physiological mechanisms in diabetic nephropathy (DN) in type 1 and type 2 diabetes mellitus. Patients with type 2 DN have a better preserved ratio of urinary excretion of IgG2/IgG4 than type 1 DN patients, indicating that the charge selectivity is less impaired in type 2 DN. Finally, old but not young hypertensive rats (study V) develop proteinuria as a result of a dysfunction of the glomerular capillary filter, affecting primarily its size-selectivity. The changes are functionally compatible with the appearance in the glomerular barrier of an increased number of unselective pores. Conclusions: During the course of glomerular diseases a maintained low urinary excretion of IgG or IgM indicates a salutary prognosis. Different patho-physiological mechanisms of albuminuria in type 1 and type 2 diabetes have been found, and hypertension induced proteinuria is primarily a size-selective disorder.

79. Entrez PubMed In the present study, primary glomerular diseases showed the following prevalences Primary glomerular diseases were significantly more prevalent in http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=8

80. Glomerular Diseases - Gastonia, North Carolina glomerular diseases Gastonia, North Carolina - courtesy of CaroMont Health. http://www.caromont.org/14538.cfm

Print Search Send to a friend Be Well ... Cancer InDepth �Conditions and Concerns Glomerular diseases Adapted from the National Institutes of Health by HealthGate Editorial Staff Many diseases affect kidney function by attacking the tiny units within the kidney where blood is cleaned. Glomerular diseases include many conditions with a variety of genetic and environmental causes, but they fall into two major categories: Glomerulonephritis (gloh-MAIR-yoo-loh-neh-FRY-tis) describes the inflammation of the membrane tissue in the kidney that serves as a filter, separating wastes and extra fluid from the blood. Glomerulosclerosis (gloh-MAIR-yoo-loh-skleh-ROH-sis) describes the scarring or hardening of the tiny blood vessels within the kidney. Although glomerulonephritis and glomerulosclerosis have different causes, they can both lead to end-stage renal disease (ESRD). What are the kidneys and what do they do? The two kidneys are bean-shaped organs located near the middle of the back, just below the rib cage to the left and right of the spine. Each about the size of a fist, these organs act as sophisticated filters for the body. They process about 400 quarts of blood a day to sift out about 2 quarts of waste products and extra water that eventually leave the body as urine. Blood enters the kidneys through arteries that branch inside the kidneys into tiny clusters of looping blood vessels. Each cluster is called a

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