61. PubMed Medline Query A clinical study of a large inbred kindred with pure familial spastic paraplegia . families with the pure form of hereditary spastic spinal paralysis. http://assoc.wanadoo.fr/asl.spastic/fichierslies/medline.htm

Docs Per Page: Entrez Date limit: 30 Days 60 Days 90 Days 180 Days 1 Year 2 Years 5 Years 10 Years No Limit citations 1-50 displayed (out of 144 found), page 1 of 3 Citation report Abstract report MEDLINE report ASN.1 report Related articles protein links nucleotide links structure links genome links for the articles selected (default all). documents on this page through Loansome Doc El-Shanti HE, et al. [See Related Articles] A clinical study of a large inbred kindred with pure familial spastic paraplegia. Brain Dev. 1999 Oct;21(7):478-82. PMID: 10522526; UI: 99450361. Cruz Martinez A, et al. [See Related Articles] Central motor conduction in hereditary motor and sensory neuropathy and hereditary spastic paraplegia. Electromyogr Clin Neurophysiol. 1999 Sep;39(6):331-5. PMID: 10499202; UI: 99428990. Hazan J, et al. [See Related Articles] A fine integrated map of the SPG4 locus excludes an expanded CAG repeat in chromosome 2p-linked autosomal dominant spastic paraplegia. Genomics. 1999 Sep 15;60(3):309-19.

62. Article : FAHR Disease - A Rare Neurodegenerative Disorder ; Author : ; Co-Autho Fahr Disease or familial idiopathic basal ganglia calcification is loss ofmotor accomplishments, symmetrical spastic paralysis, athetosis, http://www.ijri.org/articles/current_issue/Neuroradiology 383-384.htm

Neuroradiology FAHR Disease - A Rare Neurodegenerative Disorder R Malik, VK Pandya, d naik Ind J Radiol Imag 2004 14:4:383-384 Keywords: Fahr Disease, basal ganglia, ataxia, athetosis, idiopathic, calcification INTRODUCTION :- Fahr Disease is a rare degenerative neurological disorder characterized by the presence of abnormal calcium deposition and associated cell loss in the areas of brain that control movement, including basal ganglia and cerebral cortex. The condition was first described by Fahr in 1930. According to reports in medical literature, Fahr Disease is often familial. It is believed to have autosomal dominant inheritance but a few cases have been reported to have autosomal recessive inheritance and even some sporadic cases have been reported in literature. The association between the abnormal phenotypes and abnormal genes remain unclear despite the recent mapping to chromosome 14q of a susceptible locus for Fahr Disease. Fig. 1 Fig. 2 CASE REPORT :- DISCUSSION :- Fahr Disease or familial idiopathic basal ganglia calcification is characterized by bilateral basal ganglia clacification .The most common site of calcification is the globus pallidus . However additional areas of calcification are putamen, caudate nucleus, internal capsule, dentate nucleus, thalamus, cerebellum and cerebral white matter. The calcium deposits occur in the extracellular and extravascular space often surrounding the capillaries. It is not clear whether the calcification in Fahr Disease is a metastatic deposition, secondary to local disruption of blood brain barrier, or is due to disorder of neuronal calcium metabolism.

63. A Gene Encoding A Putative GTPase Regulator Is Mutated In Familial Amyotrophic L ALS2 is characterized by a progressive spasticity of limb and facial muscles onset of hereditary ascending spastic paralysis with bulbar involvement. http://www.nature.com/ng/journal/v29/n2/full/ng1001-166.html

@import "/ng/style.css"; NATURE.COM NEWS@NATURE.COM NATUREJOBS NATUREEVENTS ... Help E-MAIL: PASSWORD: Save password Forgotten password? SEARCH JOURNAL Advanced search Journal home Archive Table of Contents ... For librarians NPG Resources Nature Nature Cell Biology Nature Medicine Nature Reviews Genetics ... Browse all publications Article Nature Genetics doi:10.1038/ng1001-166 There is an Erratum (November 2001) associated with this Article. There is an Erratum (November 2001) associated with this Article. A gene encoding a putative GTPase regulator is mutated in familial amyotrophic lateral sclerosis 2 Shinji Hadano , Collette K. Hand , Hitoshi Osuga , Yoshiko Yanagisawa , Asako Otomo , Rebecca S. Devon , Natsuki Miyamoto , Junko Showguchi-Miyata , Yoshinori Okada , Roshni Singaraja , Denise A. Figlewicz , Thomas Kwiatkowski , Betsy A. Hosler , Tally Sagie , Jennifer Skaug , Jamal Nasir , Robert H. Brown Jr , Stephen W. Scherer , Guy A. Rouleau , Michael R. Hayden NeuroGenes, International Cooperative Research Project, Japan Science and Technology Corporation, Tokai University School of Medicine, Isehara, Kanagawa 259-1193, Japan. Department of Molecular Neuroscience, The Institute of Medical Sciences, Tokai University, Isehara, Kanagawa, Japan.

64. ADN Hyperkalemic Periodic paralysis, 1055 1 6 3. Neoplasms, Astrocytomer AdrenocorticalCancer familial spastic Paraplegia Amyotrophic Lateral Sclerosis, 32 http://www.medicine.mcgill.ca/neurogen/cell_dnae2.htm

Introduction Stored DNA / Cell Lineages Accessibility Blood Sampling Shipping of Blood Samples Reception of Samples Withdrawal of DNA or Cell Lineages from the Bank INTRODUCTION Table 1 ). However, the use of the Bank's material remains subject to specific agreements between involved parties (consent of participants and approval of projects by an ethics committee) which could limit the accessibility of some of the material to specific uses. The Bank also allows medical doctors and researchers the possibility of storing DNA and making cell lines without having to invest in the necessary infrastructure. STORED DNA / CELL LINEAGES Table 1 . Diseases for which cell lines already exist and/or for which DNA is stored. The number of specimens includes affected persons and non-affected family members of affected individuals. Category Disease Number of specimens Audiology Hereditary Orthophonic Problems Cardiology Cardiomyopathy Coronary Artery Disease Cerebellar Various Ataxias Acadian Ataxia Charlevoix-Saguenay Ataxia Friederich Ataxia Periodic Familial Ataxia Spastic Ataxia Spino-cerebellar Ataxia Mental Deficiencies Autism Mental Retardation Rett Syndrome Demyelating Diseases Adrenoleucodystrophy Monoclonal Gammopathy Multiple Sclerosis Endocrine Pseudohypothyroidism

65. Mobility Impairments And Neurological Conditions Hereditary spastic Paraplegia / familial spastic Paraparesis paralysis (seealso MS, Spinal Injury and other conditions leading to paralysis) http://www.netreach.net/~abrejcha/mobility.htm

This is part of the Brejcha Personal and Disability Resource Site at http://www.netreach.net/~abrejcha, and after reading this page you can click here to go back to Organizations and Resources . But for now, Welcome to my: INTERNET DISABILITY RESOURCES May 7, 2005 TO START National Institute of Neurological Disorders and Stroke (NINDS) at http://www.ninds.nih.gov is the Federal Government's leading supporter of biomedical research on nervous system disorders, including MS. Amputee: http://www.amputee-center.com offers a range of resources, links and information. Amyotrophic Lateral Sclerosis (ALS or Lou Gherig's): The ALS Survival Guide at http://www.lougehrigsdisease.net is an excellent and resource-rich site. Another great site is the Beat ALS page of Doug's Home Page at http://home.earthlink.net/~jakesan/beatals.html . The PALS Webring at http://www.users.bigpond.com/paulfran/JoinPALS.htm has a bunch of info and links on this condition. Preoject ALS at http://www.projectals.org is a key player in working to get stem cell research going for ALS and other conditions (as shown in the recent CBS television movie "Jenifer" about the founding of the organization) Ataxia: The National Ataxia Foundation at http://www.ataxia.org

66. Full Listing BOWEL DEFECTS AND ABNORMALITIES, BRACHIAL PLEXUS paralysis, BRAIN INJURED, familial HYPERLIPIDAEMIA, familial spastic PARAPLEGIA, FAMILY, http://www.doctor.gp/help/full_listing.htm

DIRECTORY OF BRITISH SELF HELP GROUPS AND SUPPORT ORGANISATIONS by Steve and Julie Garrill Home

67. 1. Easy One. Which Affected Several Members Of The English Royal are investigating for A. familial Alzheimer s B. familial parkinsonism (*) C.familial prion What virus is the cause of tropical spastic paralysis? http://www.pathguy.com/lectures/cns0102.txt

68. VI. DISEASES OF THE NERVOUS SYSTEM AND SENSE ORGANS INFLAMMATORY 342.0 Flaccid hemiplegia 342.1 spastic hemiplegia 342.9 Unspecified 343 disease 359.3 familial periodic paralysis Hypokalemic familial periodic http://www.nber.org/mortality/1995/docs/ch06.txt

69. Gene Discovered For A Dementing Brain Disease the affected person progressively develops severe dementia and spastic paralysis . The NYU group named the disease familial British Dementia. http://hdlighthouse.org/see/aggregates/addeposits.htm

Amyloid deposits are chacteristic of HD. Alzheimer pathology is found in about two-thirds of HD patients. Jerry 06/24/99 NYU Medical Center, 24-Jun-99 Amyloid is a general term used to describe proteins that form so-called beta-sheets, which fold in a particular way to form deposits in the brain. A number of brain disorders associated with dementia are linked to amyloid, including Alzheimer's, the most common form of senile dementia. While they all share a common fingerprint, namely the deposition of amyloid and neuronal loss, the nature and distribution of the amyloid protein within the brain differs with each disease for unknown reasons. A single alteration in the DNA alphabet occurring in a newly discovered human gene causes an unusual form of hereditary dementia characterized by amyloid deposits in the brain, one of the hallmarks of Alzheimer's disease, according to a new study by New York University School of Medicine scientists. The discovery solves a longstanding medical mystery involving descendants of a British woman who died in 1883 and may help lead to a better understanding of Alzheimer's and other dementias. The NYU School of Medicine researchers found the genetic alteration, or mutation, in a gene they dubbed "BRI." The unusual mutation, which occurs at a "stop codon," produces an abnormal protein fragment that forms plaques in the brain of affected family members, according to the new study in June 24 issue of Nature. The researchers developed a blood test to detect the mutation.

70. SPF - HSP General Information HSP is also known as Hereditary spastic Paraparesis, familial spastic Paraplegia Hereditary CharcotDisease, spastic Spinal paralysis, Diplegia Spinalis http://www.sp-foundation.org/hsp.htm

General information Reviewed by John K. Fink, M.D. , SPF Medical Advisor Index: What is HSP? What is (Apparently Sporadic) Spastic Paraplegia? What are the symptoms? How severe will my symptoms get? ... Where can I get more information? What is HSP? HSP is a term for a group of rare, inherited neurologic disorders along the motor neuron disease continuum. Their primary symptom is progressive spasticity (stiffness) and weakness of the leg and hip muscles. There are at least twenty types of HSP and the genetic causes are known for eleven. The disorder is estimated to affect some 20,000 people in the U.S. There are many different names used for HSP. The most common are Hereditary Spastic Paraplegia (or Paraparesis), Familial Spastic Paraparesis (or Paraplegia) and Strümpell-Lorrain Disease. Others are Spastic Paraplegia, Hereditary Charcot-Disease, Spastic Spinal Paralysis, Diplegia Spinalis Progressiva, French Settlement Disease, Troyer syndrome and Silver syndrome.

71. MedlinePlus Medical Encyclopedia: Hyperkalemic Periodic Paralysis Unlike other forms of periodic paralysis (such as familial periodic paralysisand thyrotoxic Weakness is flaccid (loose) rather than spastic (tight). http://www.nlm.nih.gov/medlineplus/ency/article/000316.htm

@import url(/medlineplus/images/advanced.css); Skip navigation Medical Encyclopedia Other encyclopedia topics: A-Ag Ah-Ap Aq-Az B-Bk ... Z Hyperkalemic periodic paralysis Contents of this page: Illustrations Alternative names Definition Causes, incidence, and risk factors ... Prevention Illustrations Muscular atrophy Alternative names Return to top Paramyotonia congenita; Periodic paralysis - hyperkalemic Definition Return to top Hyperkalemic periodic paralysis is associated with elevated levels of potassium in the bloodstream. The disorder causes occasional episodes of muscle weakness Causes, incidence, and risk factors Return to top The cause of hyperkalemic periodic paralysis is believed to be a genetic muscle disease. In most cases it is inherited directly; in other cases, it occurs randomly in a family group. The disorder involves attacks of muscle weakness or paralysis , alternating with periods of normal muscle function. Attacks usually begin in early childhood. Multiple daily attacks are not uncommon. Attacks typically last only 1 to 2 hours, but can sometimes last as long as a day. They tend to occur while resting after exercise or exertion. Attacks may also be triggerd by cold expsoure or eating small amounts of potassium. Unlike other forms of periodic paralysis (such as familial periodic paralysis and thyrotoxic periodic paralysis ), hyperkalemic periodic paralysis is not associated with low potassium in the bloodstream (

72. MedlinePlus Medical Encyclopedia: Thyrotoxic Periodic Paralysis There is a similar disorder, hypokalemic periodic paralysis (familial periodic Weakness is constant rather than spastic (spasmodic) and is greater in http://www.nlm.nih.gov/medlineplus/ency/article/000319.htm

@import url(/medlineplus/images/advanced.css); Skip navigation Medical Encyclopedia Other encyclopedia topics: A-Ag Ah-Ap Aq-Az B-Bk ... Z Thyrotoxic periodic paralysis Contents of this page: Illustrations Alternative names Definition Causes, incidence, and risk factors ... Prevention Illustrations Thyroid gland Alternative names Return to top Periodic paralysis - thyrotoxic Definition Return to top Thyrotoxic periodic paralysis a syndrome characterized by intermittent episodes of muscle weakness that occurs in people with high levels of thyroid hormone ( thyrotoxicosis , hyperthyroidism). Causes, incidence, and risk factors Return to top Thyrotoxic periodic paralysis is a rare condition that occurs only in people with thyrotoxicosis (high thyroid hormone levels). It is seen most commonly in Asian men. There is a similar disorder, hypokalemic periodic paralysis (familial periodic paralysis), that is an inherited condition and is not associated with high thyroid levels. Thyrotoxic periodic paralysis involves attacks of muscle weakness or paralysis alternating with periods of normal muscle function. Attacks usually begin after symptoms of hyperthyroidism have developed. The frequency of attacks varies from daily to yearly. Episodes of muscle weakness may last for a few hours or may persist for several days.

73. Hereditary "pure" Spastic Paraplegia: A Clinical And Genetic Study Of 22 Familie Infantile ascending hereditary spastic paralysis (IAHSP) Clinical features in Quality Assessment of Whole Genome Mapping Data in the Refined familial http://jnnp.bmjjournals.com/cgi/content/abstract/44/10/871

HOME HELP FEEDBACK SUBSCRIPTIONS ... TABLE OF CONTENTS Author Keyword(s) Vol Page [Advanced] This Article Submit a response Alert me when this article is cited Alert me when eLetters are posted Alert me if a correction is posted ... Citation Map Services Email this link to a friend Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal ... Cited by other online articles PubMed PubMed Citation Articles by Harding, A. E. Journal of Neurology, Neurosurgery, and Psychiatry, 1981, Vol 44, 871-883 PAPERS Hereditary "pure" spastic paraplegia: a clinical and genetic study of 22 families AE Harding In 22 families with the "pure" form of hereditary spastic paraplegia inheritance was autosomal dominant in 19 and autosomal recessive in three. Examination of intrafamilial correlation of age of onset in the dominant cases suggested that the disorder is genetically heterogeneous. Two forms

74. ATSDR - Motor Neuron Disease/Amyotrophic Lateral Sclerosis: Preliminary Review O A lower motor neuron condition that primarily results in paralysis of muscles in the familial spastic papaparesis (FSP). familial HTLVI myelopathy http://www.atsdr.cdc.gov/NEWS/alsreport.html

Search Index Home Glossary ... Contact Us MOTOR NEURON DISEASE/AMYOTROPHIC LATERAL SCLEROSIS: PRELIMINARY REVIEW OF ENVIRONMENTAL RISK FACTORS AND MORTALITY IN BEXAR COUNTY, TEXAS March 5, 2002 Prepared by: Curtis W. Noonan, Ph.D. Lakeisha Sykes, M.P.H. Roberta Hilsdon Health Investigations Branch Agency for Toxic Substances and Disease Registry Division of Health Studies Introduction General Overview of ALS Environmental Risk Factors for MND/ALS Heavy Metals ... Table 3. SMRs for MND in Bexar County, Texas using Texas and the United States as standard populations. Introduction Several former workers from Kelly Air Force Base (AFB) and community members residing near the base have expressed concern about amyotrophic lateral sclerosis (ALS) and the possible association with environmental contaminants. Representatives from the South Texas Chapter of the ALS Association, the Air Force Institute for Environment, Safety and Occupational Risk Analysis (AFIERA), the San Antonio Metropolitan Health District (SAMHD), and the Texas Department of Health are conducting an evaluation of ALS cases with previous connections to Kelly AFB. AFIERA also is supporting a mortality study of former Kelly AFB civilian workers that will evaluate ALS among other endpoints. To complement these efforts, the Agency for Toxic Substances and Disease Registry has prepared the following report. This report begins with a general overview of ALS, including its clinical and epidemiological characteristics. This description is followed by a review of the available literature on suspected environmental risk factors associated with the disease. Finally, this report includes a presentation of the ALS mortality experience for Bexar County in recent years. It is expected that this report will provide a useful context within which community members concerned about ALS and potential associations with Kelly Air Force Base may begin to seek answers.

75. Screening British CADASIL Families For Mutations In The NOTCH3 Gene -- DE LANGE A form of familial presenile dementia with spastic paralysis. Brain 1940;63237261.6. Sourander P, Walinder J. Hereditary multi-infarct dementia. http://jmg.bmjjournals.com/cgi/content/full/37/3/224

HOME HELP FEEDBACK SUBSCRIPTIONS ... TABLE OF CONTENTS Author Keyword(s) Vol Page [Advanced] This Article Extract Full Text (PDF) Submit a response ... Citation Map Services Email this link to a friend Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal ... Cited by other online articles PubMed PubMed Citation Articles by DE LANGE, R P J Articles by CLAIR, D M S. Related Collections Genetics J Med Genet 224-225 ( March ) Letters to the editor Screening British CADASIL families for mutations in the gene E DITOR CADASIL (Cerebral Autosomal Dominant Arteriopathy with Subcortical Infarcts and Leucoencephalopathy) is a hereditary form of multi-infarct vascular dementia. Clinical symptoms often present in middle adult life (30-50 years of age) and include recurrent subcortical ischaemic strokes, migraine with or without aura, major psychiatric symptoms, and dementia. Magnetic resonance

76. Science In Motion: Common Molecular Pathological Themes Emerge In The Hereditary Lesions to the tract result in spastic paralysis, a disabling clinical picture in Genetic localisation of a new locus for recessive familial spastic http://jmg.bmjjournals.com/cgi/content/full/40/2/81

HOME HELP FEEDBACK SUBSCRIPTIONS ... TABLE OF CONTENTS Author Keyword(s) Vol Page [Advanced] This Article Abstract Full Text (PDF) Submit a response ... Citation Map Services Email this link to a friend Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal ... Cited by other online articles PubMed PubMed Citation Articles by Reid, E Related Collections Genetics Journal of Medical Genetics BMJ Publishing Group REVIEW ARTICLE Science in motion: common molecular pathological themes emerge in the hereditary spastic paraplegias E Reid Correspondence to: ABSTRACT The hereditary spastic paraplegias are a group of neurodegenerative conditions that all share the principal clinical feature of progressive lower limb spastic paralysis, caused by either failure of development or progressive degeneration of the corticospinal tract. The conditions are characterised by extreme genetic heterogeneity

77. Spastic Paresis - A Collection Of References using dilute procaine indicate that spastic paralysis in goats is caused by More recently, the condition was reported in two familial related Pygmy http://kinne.net/paresis.htm

Spastic Paresis A collection of resources for you and your veterinarian Maxine Kinne From: American College of Veterinary Internal Medicine 7th Annual Veterinary Medicine Forum, May 1989 Spastic Paresis in Pygmy Goats J. Baker, D. Ciszewski, C. Lowrie, T. Mullaney College of Veterinary Medicine, Michigan State University, East Lansing, MI Spastic paresis is reported for the first time in goats from North America. It was concluded that spastic paresis in goats shares many similarities with the disease in cattle. The possibility of inheritance is suggested by 2 of the goats being related. Results of surgical desafferentation indicate that the defect is in the myotatic reflex (stretch reflex). Results from the epidural block using dilute procaine indicate that spastic paralysis in goats is caused by a relative over-stimulation (or lack of inhibition) of the gamma efferent motor neurons. From: Goat Medicine (1994, P. 170)

78. Systemic Amyloid Deposits In Familial British Dementia -- Ghiso Et Al. 276 (47): (1) as familial presenile dementia with spastic paralysis. The pedigree has beenfollowed and expanded since then (2), and common ancestors have been http://www.jbc.org/cgi/content/full/276/47/43909

HOME HELP FEEDBACK SUBSCRIPTIONS ... TABLE OF CONTENTS QUICK SEARCH: [advanced] Author: Keyword(s): Year: Vol: Page: Originally published In Press as doi:10.1074/jbc.M105956200 on September 13, 2001 J. Biol. Chem., Vol. 276, Issue 47, 43909-43914, November 23, 2001 This Article Abstract Full Text (PDF) All Versions of this Article: most recent Purchase Article View Shopping Cart Alert me when this article is cited Alert me if a correction is posted ... Citation Map Services Email this article to a friend Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal ... Download to citation manager PubMed PubMed Citation Articles by Ghiso, J. A. Articles by Frangione, B. Systemic Amyloid Deposits in Familial British Dementia Jorge A. Ghiso Janice Holton Leticia Miravalle Miguel Calero Tammaryn Lashley Ruben Vidal Henry Houlden Nicholas Wood Thomas A. Neubert Agueda Rostagno Gordon Plant , and Blas Frangione From the Departments of Pathology

79. Clinical Study: 96-N-0089, Characteristics Of Idiopathic Familial Voice Disorder familial vocal fold paralysis can be a lifethreatening disorder that can cause Condition(s) Laryngeal Disease spastic Dysphonia Voice Disorder http://clinicalstudies.info.nih.gov/detail/A_1996-N-0089.html

Protocol Number: 96-N-0089 Title: Characteristics of Idiopathic Familial Voice Disorders Number: 96-N-0089 Summary: The purpose the study is to determine the genetic causes of specific voice disorders that run in families. Researchers are particularly interested in two conditions; 1. Spasmodic dysphonia 2. Vocal fold paralysis Familial vocal fold paralysis can be a life-threatening disorder that can cause difficulty with vocal fold movement for breathing and voice and sometimes for swallowing. Studies are ongoing at the NIH to better understand the pathophysiology and to relate it to the genetic pattern of inheritance. Families are being recruited to participate in these studies and are being provided with further information on the disorder and genetic counseling if desired. Physician referral is requested for affected members of families with vocal fold paralysis of an unknown cause occurring over at least 2 generations. All travel, lodging, examination and counseling costs are covered for both affected and unaffected members of a family. Examinations include: voice, laryngeal, neurological, electrodiagnostic testing, genetic counseling, and radiological studies. Sponsoring Institute: National Institute of Neurological Disorders and Stroke (NINDS) Recruitment Detail Type: Active Accrual Of New Subjects Gender: Referral Letter Required: Yes Population Exclusion(s): None Eligibility Criteria: INCLUSION CRITERIA: Symptoms present during speech and not apparent at rest

80. Common Chronic Disease Patterns In Arabian Gulf, Saudi Arabia & Yemen - Analysis Hereditary familial diseases of nervous system. Herditary ataxia, 3, paralysis agitans, 21, 42. Cerebral spastic infantile paralysis, 18, 30 http://www.bhj.org/books/arabs/vi.htm

Analysis of 3000 Arab Patients Admitted To Jaslok Hospital Total Arabs Indians Encephalitis, myelitis and encephalomyelitis Meningitis Late effects of intracranial abcesss or pyogenic infection Herditary ataxia Hereditary diseases of the striato- pallidal system Hereditary neuromuscular disorders Other diseases of C N S Other cerebral paralysis Paralysis agitans Cerebral spastic infantile paralysis Epilepsy Migraine Other disease of brain Other diseases of spinal cord Motor neurone disease Diseases of nerves and peripherial ganglia Other diseases of of peripherial nerves except autonomic Facial paralysis Trigeminal neuralgia Sciatica Diseases of peripherial autonomic nervous system Inflammatory diseases of the eye Choroiditis Other inflammtion of uveal tract Blepharitis Hordeolum Keratitis Iritis Inflammation of lacrimal glands and ducts Other inflammatory diseases of eye Cataract Detachment of retina Blindness Glaucoma Corneal opacity Strabismus Pterygium Refractive errors Otitis media without mention of mastoiditis Other deafness Mastoiditis without mention of otitis media

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