41. Neurological Research: Cerebral Edema Leading To Decompressive Craniectomy: An A Full text of the article, cerebral edema leading to decompressive craniectomyAn assessment of the preceding clinical and neuromonitoring trends from http://www.findarticles.com/p/articles/mi_qa3909/is_200307/ai_n9295323

@import url(/css/us/style1.css); @import url(/css/us/searchResult1.css); @import url(/css/us/articles.css); @import url(/css/us/artHome1.css); Advanced Search Home Help IN free articles only all articles this publication Automotive Sports 10,000,000 articles - not found on any other search engine. FindArticles Neurological Research Jul 2003 Content provided in partnership with 10,000,000 articles Not found on any other search engine. Featured Titles for AAACN Viewpoint ABNF Journal, The AIDS Treatment News AMAA Journal ... View all titles in this topic Hot New Articles by Topic Automotive Sports Top Articles Ever by Topic Automotive Sports Cerebral edema leading to decompressive craniectomy: An assessment of the preceding clinical and neuromonitoring trends Neurological Research Jul 2003 by Strege, Rainer J Lang, Erhard W Stark, Andreas M Scheffner, Heike ... Et al Save a personal copy of this article and quickly find it again with Furl.net. It's free! Save it. The aim of this study was to examine the pre-operative clinical and neuromonitoring courses in patients with a decompressive craniectomy to assess and to compare clinical and neuromonitoring signs indicating extensive cerebral edema. We conducted a retrospective analysis of the clinical signs and courses of simultaneous monitoring of intracranial pressure (ICP) and cerebral oxygenation (P^sub ti^O^sub 2^) in 26 consecutive patients who were sedated and treated with a decompressive craniectomy due to extensive cerebral edema after aneurysmal subarachnoid hemorrhage (SAH) (n = 20) or severe head injury (SHI) (n = 6). Pathological monitoring trends always preceded clinical deterioration. In 18 of 26 patients extensive cerebral edema was indicated solely by increasing ICP >20 mmHg or decreasing P^sub ti^O^sub 2^

42. [Brainlife] Cerebral Edema cerebral edema associated with Gliadel wafers Two case studies NeuroOncology,Volume 7, Number 1, Pages 84 89, January 2005. (Review Article/Case Study http://www.brainlife.org/treatment/edema.htm

Brainlife Treatment Cerebral Edema Brain Tumors Broholm H , Rubin I, Kruse A, Braendstrup O, Schmidt K, Skriver EB, Lauritzen M Nitric oxide synthase expression and enzymatic activity in human brain tumors Clin Neuropathol. 2003 Nov-Dec;22(6):273-81. (Laboratory Investigation, Abstract) Glioblastoma Michael A. Badruddoja , Hendrikus G. J. Krouwer, Scott D. Rand, Kelly J. Rebro, Arvind P. Pathak, Kathleen M. Schmainda Antiangiogenic effects of dexamethasone in 9L gliosarcoma assessed by MRI cerebral blood volume maps Neuro-Oncology, Volume 5, Number 4, Pages 235 243 ( October 2003) Animal Study, Abstract) Emil L. Weber and Eric A. Goebel Cerebral edema associated with Gliadel wafers: Two case studies Neuro-Oncology, Volume 7, Number 1, Pages 84 89, January 2005. (Review Article/Case Study, Abstract+) Gliomas Badie B , Schartner JM, Hagar AR, Prabakaran S, Peebles TR, Bartley B, Lapsiwala S, Resnick DK, Vorpahl J Microglia cyclooxygenase-2 activity in experimental gliomas: possible role in cerebral edema formation Clin Cancer Res 2003 Feb;9(2):872-7 (Animal Study, Abstract)

43. Dr. Koop - Acute Mountain Sickness High altitude cerebral edema; Altitude anoxia; Altitude sickness; Mountainsickness; High altitude pulmonary edema. Causes, incidence, and risk factors http://www.drkoop.com/ency/93/000133.html

Home Health Reference Acute mountain sickness Jul 29, 2005 Search: Dr.Koop MEDLINE Diseases Symptoms Procedures Natural Medicine ... Drug Library Inside DrKoop News Archive Animations Health Videos Health Tools ... Newsletters Acute mountain sickness Injury Disease Nutrition Poison ... Prevention Acute mountain sickness Definition: Acute mountain sickness is an illness that can affect mountain climbers, hikers, skiers, or travelers who ascend too rapidly to high altitude (typically above 8,000 feet or 2,400 meters). This is especially for persons who normally reside at or near sea level. Alternative Names: High altitude cerebral edema; Altitude anoxia; Altitude sickness; Mountain sickness; High altitude pulmonary edema Causes, incidence, and risk factors: Reduced atmospheric pressure and a lower concentration of oxygen at high altitude are the causes of this illness. It affects the nervous system, lungs, muscles, and heart. Symptoms can range from mild to life-threatening. In most cases the symptoms are mild, but in severe cases fluid collects in the lungs ( pulmonary edema ) causing extreme shortness of breath and further decreasing oxygenation.

44. Factors Associated With Cerebral Edema In Children With Diabetic Ketoacidosis (D Background cerebral edema (CE) is an uncommon but devastating complication ofDKA in children. The factors associated with this complication are unclear. http://www.aemj.org/cgi/content/abstract/7/5/559

HOME HELP FEEDBACK SUBSCRIPTIONS ... TABLE OF CONTENTS This Article Alert me when this article is cited Alert me if a correction is posted Services Similar articles in this journal Alert me to new issues of the journal Download to citation manager PubMed Articles by Kuppermann, N. Articles by Glaser, N. Academic Emergency Medicine Volume 7, Number 5 559, Society for Academic Emergency Medicine PEDIATRIC EMERGENCIES Factors Associated with Cerebral Edema in Children with Diabetic Ketoacidosis (DKA) Nathan Kuppermann Peter Barnett Ian McCaslin David Nelson Jennifer Trainor Jeffrey Louie Francine Kaufman Kimberly Quayle Mark Roback Richard Malley and Nicole Glaser University of California, Davis, Sacramento A BSTRACT Background: Cerebral edema (CE) is an uncommon but devastating complication of DKA in children. The factors associated with this complication are unclear. Objective: To identify factors associated with CE in children with DKA. Methods: We performed a retrospective case-control study of DKA-related CE in children. We reviewed DKA admissions from 1982-97 at 10 pediatric referral centers.

45. Mild Hypothermia Reduces Cardiac Arrest-induced Cerebral Edema In Rats -- Xiao E cerebral edema secondary to increased microvascular permeability was determinedby brain wetto-dry weight ratio and cerebral spinal fluid pressure (CSFP). http://www.aemj.org/cgi/content/abstract/8/5/511-a

HOME HELP FEEDBACK SUBSCRIPTIONS ... TABLE OF CONTENTS This Article Alert me when this article is cited Alert me if a correction is posted Services Similar articles in this journal Alert me to new issues of the journal Download to citation manager PubMed Articles by Xiao, F. Articles by Conrad, S. A Academic Emergency Medicine Volume 8, Number 5 511, Society for Academic Emergency Medicine Neuronal Injury Mechanism Mild Hypothermia Reduces Cardiac Arrest-induced Cerebral Edema in Rats Feng Xiao Shu Zhang Steven Alexander Jian Huang Donna L Carden Thomas C Arnold and Steven A Conrad Louisiana State University Health Sciences Center: Shreveport, LA A BSTRACT Objectives: survival and neurological outcome after experimental cardiac arrest (CA). However, the mechanism by which hypothermia improves CA-induced brain damage is not clear. The purpose of this study was to test the hypothesis that mild hypothermia attenuates CA-induced increases in cerebral microvascular permeability. Methods: Eighteen male Sprague-Dawley rats (300-400 grams) were randomized into 3 groups. The animals in group 1 (

46. Institute For Healthcare Improvement: Combined Lung Injury, Meningitis And Cereb Combined lung injury, meningitis and cerebral edema How permissive can hypercapniabe? Intensive Care Medicine. 1998;24616–619. http://www.ihi.org/IHI/Topics/CriticalCare/Sepsis/Literature/CombinedLungInjuryM

Log In/Register Contact Us Site Map More Search Options ... About Us Combined lung injury, meningitis and cerebral edema: How permissive can hypercapnia be? Tasker RC, Peters MJ. Combined lung injury, meningitis and cerebral edema: How permissive can hypercapnia be? Intensive Care Medicine. 1998;24:616–619. This case study describes the management of hypercapnia. This item has not yet been rated View article abstract Your Privacy

47. Baylor Neurology Case Of The Month Systemic lupus erythematosus with cerebral edema and leukoencephalopathy The presence of cranial nerve deficits and cerebral edema in this patient also http://www.bcm.edu/neurology/challeng/pat48/summary.html

Patient #48 Summary and Discussion Jennifer Lynch, MD and Dennis R. Mosier, MD, PhD Diagnosis: Systemic lupus erythematosus with cerebral edema and leukoencephalopathy Hospital Course: She returned to the Ben Taub emergency room two days later with tachypnea, and was readmitted to the hospital with left ear pain, dizziness, tachycardia (to 120/min), and progressive hypoxemia. Records of her recent "pneumonia" (not available on the first admission) described considerable pleuritic chest pain, with no causative organisms isolated. Her condition rapidly declined, and she was intubated for mechanical ventilation, with bright red blood evident upon initial suctioning. Bronchoscopy was performed, but did not detect any sites of ongoing bleeding, and no growth of bronchial washings or atypia of cells was found. The patient had been empirically treated with valacyclovir in view of her history of herpes simplex, but no conclusive evidence of viral infection was found. Together with the clinical or historical features of CNS dysfunction, episodic polyarthralgias, anemia of chronic disease

48. Neurosurgery - Abstract: Volume 12(6) June 1983 P 599-605 Cerebral Edema Associa The cerebral edema, as judged by computed tomographic scan, associated withsupratentorial meningiomas was assessed in 55 cases. http://www.neurosurgery-online.com/pt/re/neurosurg/abstract.00006123-198306000-0

LWWOnline LOGIN eALERTS REGISTER ... Articles in Press Cerebral edema associated with... Cerebral edema associated with meningiomas. Neurosurgery. 12(6):599-605, June 1983. Gilbert, J J; Paulseth, J E; Coates, R K; Malott, D Abstract: The cerebral edema, as judged by computed tomographic scan, associated with supratentorial meningiomas was assessed in 55 cases. No relationship to the occurrence or the degree of edema could be established with respect to meningioma location, histological type, tumor vascularity, cellularity, number of mitotic figures, necrosis, calcification, or cortical invasion. The larger the meningioma, the more likely the presence of and the severity of cerebral edema. The edema is a significant factor in the occurrence of clinical signs and symptoms. A biopsy of cerebral cortex and white matter underlying a transitional meningioma in a patient with associated cerebral edema demonstrated perivascular astrocytic end-feet swelling in the cortex and considerable extracellular fluid in the white matter. The ultrastructural appearance is similar to that seen with primary and metastatic brain tumors and with experimental vasogenic cerebral edema. Subscribe to RSS feed utrdc-pt01 Release 4.0

49. Neurosurgery - Abstract: Volume 14(3) March 1984 P 295-301 Cerebral Edema Associ cerebral edema associated with meningiomas possible role of a A retrospectivestudy of cerebral edema in 40 patients with intracranial meningiomas http://www.neurosurgery-online.com/pt/re/neurosurg/abstract.00006123-198403000-0

LWWOnline LOGIN eALERTS REGISTER ... Articles in Press Cerebral edema associated with... Cerebral edema associated with meningiomas: possible role of a secretory-excretory phenomenon. Neurosurgery. 14(3):295-301, March 1984. Philippon, J; Foncin, J F; Grob, R; Srour, A; Poisson, M; Pertuiset, B F Abstract: A retrospective study of cerebral edema in 40 patients with intracranial meningiomas seems to demonstrate that the extent of edema is not related to anatomical factors such as location. Histological type is more significant; cerebral edema is usually observed with meningothelial forms. Furthermore, the study of tumor cells by electron microscopic methods indicates that secretory-excretory activity is closely associated with with the production of peritumoral edema. The ultrastructural aspects of this secreted material are described, but further biochemical studies are necessary to determine its exact nature. The possible role of tumoral steroid receptors in the secretory-excretory phenomenon is discussed. Subscribe to RSS feed utrdc-pt02 Release 4.0

50. Radiology In Ped Emerg Med, Vol 5, Case 6 This is the case in generalized cerebral edema, subdural hematoma, The ventriclesare slitlike due to cerebral edema and acute hemorrhage resulting in http://www.hawaii.edu/medicine/pediatrics/pemxray/v5c06.html

Intracranial Hypertension and Brain Herniation Syndromes Radiology Cases in Pediatric Emergency Medicine Volume 5, Case 6 Loren G. Yamamoto, MD, MPH Kapiolani Medical Center For Women And Children University of Hawaii John A. Burns School of Medicine This is a 5-year old female who is brought to the emergency department at 8:00 a.m. because she was poorly responsive when her mother awoke her in the morning. This prompted her mother to drive her to the E.D. There is a history of headache and vomiting during the evening and night. There is no history of trauma. Exam: VS T36.7 (rectal), P92, R32, BP 137/97. She is minimally responsive. Pupils equal and reactive. There are no signs of external trauma. Within minutes of arrival, she exhibits extensor posturing. She is orally intubated using the rapid sequence induction method with atropine, thiopental, and vecuronium. She is hyperventilated. End-tidal CO2 monitoring is used to keep her pCO2 in the 25 mmHg range. A loading dose of phenytoin is administered. An emergency CT scan is ordered. View CT scan. The image on the left is a high CT cut which should show the sulci and gyri well. Due to increased ICP, the cortex is compressed up against the calvarium losing the distinctness of the sulci and gyri. The space between the cortex and the calvarium is obliterated. The sulci/gyri sign cannot be totally relied upon in some instances. In cases of external hydrocephalus or chronic (or subacute) subdural effusions, fluid collects over the cortex. The fluid space between the cortex and the calvarium appears to be increased and the sulci/gyri may appear prominent. View prominent sulci/gyri.

51. Researchers Find Way To Detect Dangerous Brain Swelling In Diabetic Children Therefore, the diagnosis of cerebral edema needs to be made at the bedside No one ever forgets their cerebral edema cases, whether it s the ones you ve http://www.news-medical.net/?id=4157

52. Researchers Find Way To Detect Dangerous Brain Swelling In The screening method was 96 percent accurate in detecting cerebral edema.In addition, the approach identified four cases of cerebral edema among the 69 http://www.news-medical.net/print_article.asp?id=4157

53. HIGH ALTITUDE CEREBRAL EDEMA OR MOUNTAIN SICKNESS - Travel Medicine Program - Pu Travellers to high altitudes, both mountains climbers and trekkers, should beadvised of the potential hazard of acute mountain sickness. http://www.phac-aspc.gc.ca/tmp-pmv/travel/cerebr_e.html

HIGH ALTITUDE CEREBRAL EDEMA OR MOUNTAIN SICKNESS Travellers to high altitudes, both mountains climbers and trekkers, should be advised of the potential hazard of acute mountain sickness. Tour companies offer itineraries to Nepal where trekkers climb to over 6,000 metres, to Mount Kilimanjaro in East Africa 6,000 metres, to Andean passes in South Africa 4,000 metres, and to Swiss Alps. These trips are often advertised as being quite easy. However, only those who are healthy and well trained should take part in such expeditions. Anyone in doubt, especially with a history of cardiac or respiratory complaint should undergo medical examination and seek medical advice. Even those who are fit run the risk of acute Mountain sickness during rapid ascent to high altitudes if they spend more than half a day above 2,500 metres. A number of studies have shown a correlation between incidence of that disease and altitude: in the Swiss Alps, high altitude sickness have been observed in 9% of climbers at 2,850 meters, 34% at 3,650 metres, and 53% at 4,559 metres. Having normal heart and lung functions based on examination findings (such as an ECG) does not imply a reduced risk. It is often the young and fit who fall ill.

54. Cerebral Edema According to the Monro Kellie Model, an increase in the volume of brain tissueresulting from an increase in its water content (ie cerebral edema) must http://uscneurosurgery.com/infonet/5036/resuscitation new.htm

GH 5036 Cerebral resuscitation Key concepts Tissue water Herniation Increased ICP Monro Kellie Hypothesis Diuresis ... Self-assessment Introduction Neither the brain nor the spinal cord can expand too much without pushing up against the bone that surrounds them. According to the Monro Kellie Hypothesis of intracranial pressure dynamics, intracranial pressure and volume are directly related: as the volume of brain and other tissue inside the skull goes up, since the skull cannot expand, pressure inside it goes up. Intracranial volume can be divided into three "compartments". If one compartment's volume goes up or down the total volume goes down unless one or both of the other compartments increase their volume to compensate. top Pathophysiology Intracranial volume can be divided into three "compartments". If one compartment's volume goes up or down the total volume goes down unless one or both of the other compartments increase their volume to compensate. Edema Edema is excessive water in tissue. Brain edema results from entry into brain tissue of water leaking through an incompetent blood brain barrier Herniation An local increase in brain volume in an area anatomically predisposed to herniation can cause movement of tissue from one to another intracranial compartment (herniation).

55. Cerebral Edema And Brain Swelling A. Vasogenic cerebral edema (most common form of edema). 1. Increased permeabilityof small vessels B. Cytotoxic cerebral edema (cellular brain edema) http://kobiljak.msu.edu/CAI/Pathology/Intra_F/Intra_2.html

II. CEREBRAL EDEMA AND BRAIN SWELLING A. Vasogenic Cerebral Edema (most common form of edema) 1. Increased permeability of small vessels (breakdown of blood-brain barrier) 2. Escape of proteins, fluids into extracellular space, especially of white matter B. Cytotoxic Cerebral Edema (cellular brain edema) 1. Increased permeability of cell membranes 2. Excess fluid accumulates intracellularly; may occur with ischemia or with other conditions such as metabolic poisons or water intoxication. C. Hydrocephalic (Interstitial) Edema 1. Fluid flows from CSF into brain through ventricular lining in cases of hydrocephalus. Previous Section Course Index Section Index Next Section

56. Cerebral Edema In The Himalayas: Too High, Too Fast! Highaltitude cerebral edema is a well-recognized outcome for trekkers withinitial acute mountain sickness who climb too high, too fast.1,2 Symptoms of http://www.wemjournal.org/wmsonline/?request=get-document&issn=1080-6032&volume=

57. The Cerebral Etiology Of High-altitude Cerebral Edema And Acute Mountain Sicknes Wilderness and Environmental Medicine Vol. 10, No. 2, pp. 97–109. The cerebraletiology of highaltitude cerebral edema and acute mountain sickness http://www.wemjournal.org/wmsonline/?request=get-abstract&issn=1080-6032&volume=

58. BioMed Central | Abstract | Cerebral Edema: Hypertonic Saline Solutions Opinion cerebral edema Hypertonic Saline Solutions Anish Bhardwaj MD and JohnA Ulatowski MD PhD Neuroscience Critical Care Division Johns Hopkins http://www.biomedcentral.com/1092-8480/1/179/abstract

home journals A-Z subject areas advanced search ... Issue 3 Viewing options Abstract Full text PDF Associated material: PubMed record Related literature: Articles citing this article on Google Scholar Other articles by authors Bhardwaj A ... Similar articles (PubMed) Tools: E-mail to a friend Download references Opinion Cerebral Edema: Hypertonic Saline Solutions Anish Bhardwaj MD and John A Ulatowski MD PhD Neuroscience Critical Care Division Johns Hopkins University School of Medicine Meyer 8-140, 600 N Wolfe St, Johns Hopkins Hospital, Baltimore, MD, 21287, USA Current Treatment Options in Neurology Published Abstract Our experience, and that of others, suggests that hypertonic saline solution therapy reduces intracranial pressure and lateral displacement of the brain in patients with cerebral edema. This therapy appears most promising in patients who have head trauma or postoperative cerebral edema. Studies comparing hypertonic saline therapy with conventional therapies are limited. Additional randomized studies are needed to determine its safety and optimum duration of benefit and to determine the lesions most likely to be improved. To date, the cost effectiveness of hypertonic saline therapy is unknown. Caution is advised regarding its use until the results of more definitive trials investigating its efficacy and safety are known. Terms and Conditions Privacy statement Information for advertisers Contact us

59. BioMed Central | Full Text | Cerebral Edema: Hypertonic Saline Solutions To obtain access to Current Treatment Options in Neurology through your institutionuse the options below. If you would like information about a personal http://www.biomedcentral.com/1092-8480/1/179

home journals A-Z subject areas advanced search ... my BioMed Central To obtain access to Current Treatment Options in Neurology through your institution use the options below. If you would like information about a personal subscription, please contact subs@phl.cursci.com Click here to view an abstract of this article Click here to login if you are already a subscriber to Current Treatment Options in Neurology Subscribe to Current Treatment Options in Neurology Register for a free online trial Ask your librarian to investigate institutional access Athens users please click here to gain access If you believe you are seeing this page in error, or cannot activate your subscription, e-mail us info@biomedcentral.com Terms and Conditions Privacy statement Information for advertisers ... Contact us

60. IngentaConnect Cerebral Edema Associated With Gliadel Wafers: Two Case Studies cerebral edema associated with Gliadel wafers Two case studies. Authors Emil L.Weber1; Eric A. Goebel. Source NeuroOncology, Volume 7, Number 1, http://www.ingentaconnect.com/content/dup/no/2005/00000007/00000001/art00009

Home About Ingenta Ingenta Labs Help For Publishers Why go online? Why choose IngentaConnect? Why choose CustomConnect? Access and authentication ... Contact us For Researchers About IngentaConnect Search and browse Publications available Accessing articles ... Register For Librarians Why choose IngentaConnect? Why choose IngentaConnect Premium? Activating Subscriptions Purchasing articles ... Browse Publications Cerebral edema associated with Gliadel wafers: Two case studies Authors: Emil L. Weber ; Eric A. Goebel Source: Neuro-Oncology , Volume 7, Number 1, January 2005, pp. 84-89(6) Publisher: Duke University Press View Table of Contents full text options Abstract: While the introduction of carmustine wafers (Gliadel wafers) into the tumor resection cavity has been shown to be a beneficial therapy for malignant glioma, it is recognized that clinically significant cerebral edema is a potential adverse effect. Following are two clinical case reports demonstrating profound cerebral edema associated with implantation of Gliadel wafers. As a result, one of these individuals had premature death. A brief literature review is provided to assist in explaining the mechanisms by which clinically significant cerebral edema may develop. Document Type: Case report DOI: Affiliations: Tri-State Neurosurgical, Inc., Evansville, IN 47710, USA

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