41. Immune Disorders Message Board bergers disease Ivan 211912 2/19/00 (1). Re bergers disease - beth 193605 5/25/00 (0). Idopathic Thrombocytopenia Purpura - Sherri 202238 2/13/00 http://www.healthboards.com/immune-disorders/

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43. AllRefer Health - IgA Nephropathy (Berger's Disease) (Berger's Disease, Nephropa IgA Nephropathy (Berger s disease) (Berger s disease, Nephropathy IgA) information center covers causes, prevention, symptoms, diagnosis, treatment, http://health.allrefer.com/health/iga-nephropathy-bergers-disease-info.html

AllRefer Channels :: Yellow Pages Reference Health Home ... Contact Us Quick Jump ADD/ADHD Allergies Alzheimer's Disease Arthritis Asthma Back Pain Breast Cancer Cancer Colon Cancer Depression Diabetes Gallbladder Disease Heart Attack Hepatitis High Cholesterol HIV/AIDS Hypertension Lung Cancer Menopause Migraines/Headaches Osteoporosis Pneumonia Prostate Cancer SARS Stroke Urinary Tract Infection 1600+ More Conditions Alternative Medicine Health News Symptoms Guide Special Topics ... Medical Encyclopedia Web health.allrefer.com You are here : AllRefer.com Health IgA Nephropathy (Berger's Disease) IgA Nephropathy (Berger's Disease) Definition Treatment Expectations or Prognosis Complications ... Go To Main Page Alternate Names : Berger's Disease, Nephropathy - IgA Definition IgA nephropathy is a kidney disorder characterized by blood in the urine ; caused by inflammation of the internal kidney structures and deposits of IgA antibodies in the kidney mesangial tissue. Male Urinary System IgA nephropathy (Berger's disease) is a form of mesangial proliferative nephritis. Inflammation of the renal glomeruli occurs, and there are IgA (a type of

44. AllRefer Health - IgA Nephropathy (Berger's Disease): Treatment (Berger's Diseas IgA Nephropathy (Berger s disease) (Berger s disease, Nephropathy IgA) information center covers Treatment. http://health.allrefer.com/health/iga-nephropathy-bergers-disease-treatment.html

AllRefer Channels :: Yellow Pages Reference Health Home ... Contact Us Quick Jump ADD/ADHD Allergies Alzheimer's Disease Arthritis Asthma Back Pain Breast Cancer Cancer Colon Cancer Depression Diabetes Gallbladder Disease Heart Attack Hepatitis High Cholesterol HIV/AIDS Hypertension Lung Cancer Menopause Migraines/Headaches Osteoporosis Pneumonia Prostate Cancer SARS Stroke Urinary Tract Infection 1600+ More Conditions Alternative Medicine Health News Symptoms Guide Special Topics ... Medical Encyclopedia Web health.allrefer.com You are here : AllRefer.com Health IgA Nephropathy (Berger's Disease) : Treatment of IgA Nephropathy (Berger's Disease) IgA Nephropathy (Berger's Disease) Definition Treatment Expectations or Prognosis Complications Support Groups Calling Your Health Care Provider ... Go To Main Page Alternate Names : Berger's Disease, Nephropathy - IgA Treatment The goal of treatment is to relieve symptoms and prevent or delay of chronic renal failure Antihypertensive medications and diuretics may be given to control high blood pressure and edema . Control of blood pressure is the most important measure to delay renal damage. Sodium (salt) and fluids may be restricted to control

45. Bergers Glomerulonephritis Of The Kidney bergers glomerulonephritis of Kidney From the Virtual Pathology Museum, Department of •Berger s disease is a mild form of IgA glomerulonephritis. http://pathweb.uchc.edu/eAtlas/GU/1542.htm

Search Frames Search No frames PathWeb Home Feed Back ... About Bergers Glomerulonephritis Click on Image to Enlarge it Bergers Glomerulonephritis The mesangial or stalk regions of the glomerular tuft show granular fluorescence for IgA. Bergers glomerulonephritis is an IgA mediated immune complex disorder. Direct immunofluorescence shows granular mesangial fluorescence for IgA and C3. (Description By:H. Yamase, M.D. ) (Image Contrib. by: UCHC ) Bergers Glomerulonephritis Etiology Probably infectious, especially mucosal infections in respiratory or gastrointestinal tracts. Pathogenesis The disease is defined by the presence of IgA immune complexes in the glomerular tufts. Presumably mucosal derived IgA combines with antigens to form circulating immune complexes that deposit in the glomerular mesangium., Epidemiology This disease is probably the most common form of glomerulonephritis world wide. The disease tends to be mild and relatively asymptomatic so many cases are undiagnosed.

46. Untitled Document The doctor said I had a disease called IgA Nephropathy or bergers disease. They said the immediate choice I needed to make was whether to receive http://www.transweb.org/people/recips/lifepost/athletics/us_games/98/people/athp

Luke W. Onken Age: Transplant: Kidney from cadaver donor May 4, 1991 Hometown: Davenport, Iowa Events: 5000 meter, 1500 meter 1500 M race walk, softball throw, three-on-three basketball. My story: My message to the world: Transplants work! Transplants change lives. Transplants save lives. Please consider organ donation. Talk to your family about organ donation. You can be a hero just by signing a donor card. Return to People Index Last modified: 11 May 2000

47. Healthopedia.com - Berger's Disease (IgA Nephropathy, Berger's Nephropathy) Berger s disease is generally believed to be an autoimmune disorder that results in kidney In Berger s disease, the filtering parts of the kidney, http://www.healthopedia.com/bergers-disease/

Set Homepage Email Page Favorites Home Health Centers Medical Encyclopedia Medical Symptoms ... Special Topics Related Resources Health Centers Cardiovascular (Circulatory System) Allergic Reaction to a Medication Autoimmune Disorders Blood in the Urine ... Kidney Transplant Other Topics Sodium You are here : Healthopedia.com Medical Encyclopedia Diseases and Conditions Berger's Disease Category : Health Centers Cardiovascular (Circulatory System) Berger's Disease Alternate Names : IgA Nephropathy, Berger's Nephropathy Pictures and Images Attribution Berger's disease is generally believed to be an autoimmune disorder that results in kidney damage and may lead to kidney failure. An autoimmune disorder is a condition in which the person produces antibodies to his or her own tissues. What is going on in the body? The job of the kidneys is to make urine by filtering the blood. In Berger's disease, the filtering parts of the kidney, called the glomeruli, become damaged. The damage is due to deposits of proteins called antibodies. Antibodies are normally made by the immune system to fight infections. For some unknown reason, people with Berger's disease have either increased production or reduced clearance of IgA antibodies. The higher level of antibodies clogs up the filtering system of the kidney. What are the causes and risks of the disease?

48. Healthopedia.com - Berger's Disease: Treatment & Monitoring (IgA Nephropathy, Be What are the treatments for the disease? Treatment of Berger s disease usually focuses on controlling high blood pressure and reducing protein loss in the http://www.healthopedia.com/bergers-disease/treatment.html

Set Homepage Email Page Favorites Home Health Centers Medical Encyclopedia Medical Symptoms ... Special Topics Related Resources Health Centers Cardiovascular (Circulatory System) Allergic Reaction to a Medication Autoimmune Disorders Blood in the Urine ... Kidney Transplant Other Topics Sodium You are here : Healthopedia.com Medical Encyclopedia Diseases and Conditions Berger's Disease Category : Health Centers Cardiovascular (Circulatory System) Berger's Disease Alternate Names : IgA Nephropathy, Berger's Nephropathy Berger's Disease Pictures and Images Attribution What are the treatments for the disease? Treatment of Berger's disease usually focuses on controlling high blood pressure and reducing protein loss in the urine. This is usually done with one of the blood pressure medications called ACE inhibitors. Examples include enalapril and captopril. Fish oil and corticosteroids such as prednisone may help slow down the kidney damage in some cases. Conditions that aggravate kidney failure, such as infections and obstructions, should be treated promptly. Dietary modifications are important as kidney failure progresses. Individuals should follow the diet prescribed by the healthcare provider, which generally focuses on limiting sodium , protein, and fluid.

49. Laboratory Of Gabriele Bergers - UCSF Department Of Neurosurgery bergers Laboratory. Principal Investigator Gabriele bergers PhD in distinctive preclinical trials targeted at different stages of disease progression. http://neurosurgery.medschool.ucsf.edu/neurosurgery_research/btrc/bergers_lab.ht

Referrals Clinical Trials Department Newsletter Additional Links ... Bergers Laboratory Bergers Laboratory Principal Investigator: Gabriele Bergers PhD Assistant Professor of Neurological Surgery Principal Investigator, Brain Tumor Research Center Current Research Projects Hypoxia and Neovascularization: Cause or Consequence of Glioblastoma Multiforme Progression? Principal Investigator : Gabriele Bergers PhD Co-Investigator : Randall Johnson PhD, University of California San Diego Identification of tumor-endothelial specific factors in low-grade astrocytomas and glioblastoma multiforme Principal Investigator : Gabriele Bergers PhD Co-Investigator : Mitchel Berger MD Endogenous mouse models of gliogenesis Principal Investigator : Gabriele Bergers PhD Co-Investigator : William A. Weiss MD, PhD Translations from preclinical to clinical studies have always posed a challenge to extrapolate animal data to the clinical setting. Specifically, subcutaneous human tumor xenograft models, which are most commonly used for glioblastoma studies, have been criticized because of the immunosuppressed and artificial environment in which established high-passage tumor cell lines grow. For these reasons, spontaneous tumor models have gained greater acceptance because in such models, normal cells transform to malignancy within their normal microenvironments, thus better recapitulating the human situation. Our goal is to develop such an endogenous mouse model of GM. First we will establish mice that lack p53 in astrocytes (GFAP-Cre/p53flox), a criterion that has been shown to be one of the earliest events in gliogenesis. Indeed astrocytes, deficient in p53, start proliferating but do not transform. We will then induce subsequential expression of different oncogenes and growth factors (e.g., Ha-ras, K-ras, v-erB, VEGF) in these mice utilizing either a tet-inducible system or the avian RCAS virus system to reiterate a multistep pathway of gliogenesis.

50. Gabriele Bergers - UCSF Department Of Neurosurgery Gabriele bergers PhD, Assistant Professor of Neurological Surgery at UCSF. preclinical trials at three distinct stages of disease progression aimed at http://neurosurgery.medschool.ucsf.edu/faculty_staff/department_faculty/bergers.

Referrals Clinical Trials Department Newsletter Additional Links ... Gabriele Bergers PhD Gabriele Bergers PhD Assistant Professor of Neurological Surgery Principal Investigator, Brain Tumor Research Center UCSF Neurological Surgery faculty since 2001 Read about Dr. Bergers's current research Dr. Bergers studies the functional importance of angiogenesis in tumor development and progression. She has utilized a transgenic mouse model of pancreatic islet cell carcinogenesis to elucidate mechanisms and impact of angiogenesis during tumor formation. She identified that the angiogenic factor VEGF is critical for the angiogenic switch and is regulated posttranscriptionally, via proteolytic release from the extracellular matrix by the metalloproteinase MMP-9 that is expressed by inflammatory cells. This work was published in Nature Cell Biology in 2000. She furthermore translated the studies into experimental therapeutic approaches and designed preclinical trials at three distinct stages of disease progression aimed at preventing the angiogenic switch in premalignant lesions, intervening in the rapid expansion of small tumors, or inducing the regression of large end-stage cancers. Dr. Bergers was the first to publish that angiogenesis inhibitors have different efficacies depending on the stage of carcinogenesis being targeted (Science 1999). She is currently investigating the underlying mechanisms that lead to stage-specific regulation of angiogenesis. Dr. Bergers' laboratory has expanded its studies to elucidate crucial factors that are implicated in the formation of new blood vessels during gliogenesis and to investigate the functional importance of these factors for the development and progression of glioblastomas utilizing mouse models of glioblastoma. In 2002, Dr. Bergers received the Kimmel Scholar Award, a substantial financial award given to selected promising scientists engaged in cancer research who are at an early stage of their career.

51. ESynopsis Of Bergers Glomerulonephritis Of The Kidney eSynopsis of bergers glomerulonephritis of Kidney From the Electonic Synopsis of Cotran RS, Kumar V, Robbins SL Robbins Pathologic Basis of disease. http://esynopsis.uchc.edu/S258.htm

Prev GU Menu PathWeb Home Feed Back ... Next Bergers Glomerulonephritis Etiology Probably infectious, especially mucosal infections in respiratory or gastrointestinal tracts. Pathogenesis The disease is defined by the presence of IgA immune complexes in the glomerular tufts. Presumably mucosal derived IgA combines with antigens to form circulating immune complexes that deposit in the glomerular mesangium., Epidemiology This disease is probably the most common form of glomerulonephritis world wide. The disease tends to be mild and relatively asymptomatic so many cases are undiagnosed. Some cases seem to be related to mucosal infections such as respiratory infections and gastrointestinal disorders such as celiac disease and hepatic diseases. Immunogenetics, DR4, ?HLA Bw35. General Gross Description The kidneys in Berger's glomerulonephritis show no gross abnormalities that reflect the pathologic process. General Microscopic Description Berger's disease is a mild form of IgA glomerulonephritis. The glomeruli show modest increase in mesangial cellularity.

52. Web Focus - Cell Biology Of Disease - Nature Cell Biology Journal home Focuses Cell biology of disease Articles G bergers, R Brekken, G McMahon, TH Vu, T Itoh, K Tamaki, K Tanzawa, P Thorpe, S Itohara, http://www.nature.com/ncb/webfocus/disease/articles2000.html

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53. EMedicine - Hyperkeratosis Lenticularis Perstans (Flegel Disease) : Article By D Flegel disease likely has both a familial and nonfamilial variant, since several reports Clin Exp Dermatol 1987; 12 231. van de Staak WJ, bergers AM, http://www.emedicine.com/derm/topic183.htm

(advertisement) Home Specialties Resource Centers CME ... Patient Education Articles Images CME Advanced Search Consumer Health Link to this site Back to: eMedicine Specialties Dermatology Papulosquamous Diseases Hyperkeratosis Lenticularis Perstans (Flegel Disease) Last Updated: August 24, 2005 Rate this Article Email to a Colleague Synonyms and related keywords: Flegel's disease, HLP, Odland bodies, keratosis, keratotic papules AUTHOR INFORMATION Section 1 of 11 Author Information Introduction Clinical Differentials ... Bibliography Author: Daniel Roling, MD , Staff Physician, Department of Dermatology, University of Pennsylvania Hospital Coauthor(s): Jacqueline M Junkins-Hopkins, MD , Assistant Professor, Department of Dermatology, University of Pennsylvania School of Medicine Daniel Roling, MD, is a member of the following medical societies: Pennsylvania Medical Society Editor(s): Julia R Nunley, MD , Program Director, Associate Professor, Department of Dermatology, Medical College of Virginia; Richard Vinson, MD

54. Urologic And Male Genital Diseases AntiGlomerular Basement Membrane disease - Balanitis - Bladder diseases Pathol. notes about bergers Glomerulonephritis - Univ of Connecticut/Pathol. http://www.mic.ki.se/Diseases/C12.html

search search staff sitemap ABOUT KAROLINSKA INSTITUTET ... print this page Diseases and Disorders Links pertaining to Urologic and Male Genital Diseases Alert! Patients and laypersons looking for guidance among the target sources of this collection of links are strongly advised to review the information retrieved with their professional health care provider. Start Page Contents: Anti-Glomerular Basement Membrane Disease Balanitis Bladder Diseases Bladder Exstrophy ... Vesico-Ureteral Reflux Urologic and Male Genital Diseases Nephrology and Urology - The Family Practice Handbook, University of Iowa (US) Catheterization Basics - NursingCEU.com About the Foley Catheter - eMedicine/Consumer Health Fournier Gangrene About Fournier Gangrene [Santora and Rukstalis] - eMedicine Genital Diseases, Male Male Genital System Pathology [images; EC Klatt] Sex Organ Homologies - Loyola Univ. (US) Sexually Transmitted Diseases, Bacterial [links] Ed's Lecture Notes on Diseases of the Male Reproductive System [E Friedlander] - (US) [GT Falk] - (UK) The Circumcision Debate - Dr. George

55. Journal Of Neuroimaging -- Sign In Page diseasemodifying therapies in multiple sclerosis an update on recent and ongoing trials Kalkers NF, Barkhof F, bergers E, van Schijndel R, Polman CH. http://jon.sagepub.com/cgi/content/refs/14/3_suppl/54S

JOURNAL HOME HELP CONTACT US SUBSCRIPTIONS ... SEARCH QUICK SEARCH: [advanced] Author: Keyword(s): Year: Vol: Page: This item requires a subscription to Journal of Neuroimaging Online. Impact of Disease-Modifying Therapies on Brain and Spinal Cord Atrophy in Multiple... Rudick J Neuroimaging. This Article Abstract Full Text (PDF) References ... Alert me if a correction is posted Services Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal Download to citation manager ... Reprints and Permissions PubMed PubMed Citation Articles by Rudick, R. A. To view this item, select one of the options below: Sign In User Name Sign in without cookies. Can't get past this page? Help with Cookies. Need to Activate? Password Forgot your user name or password? Purchase Short-Term Access Pay per Article - You may access this article (from the computer you are currently using) for 1 day for US$25.00 Pay for Admission - You may access all content in Journal of Neuroimaging Online (from the computer you are currently using) for 24 hours for US$75.00.

56. The Journal Of Nervous And Mental Disease - UserLogin The Journal of Nervous and Mental disease Volume 188(4) April 2000 pp 242243 Buntinx F, Kester A, bergers J, Knottnerus A (1996) Is depression in http://www.jonmd.com/pt/re/jnmd/fulltext.00005053-200004000-00010.htm

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57. Cancer Chemotherapy - Disease Therapies Protocol Metabolic bone disease induced by prostate cancer rationale for the use of Hanahan D, bergers G, Bergsland E. Less is more, regularly metronomic http://www.lef.org/protocols/prtcl-024refs.html

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58. The Goldhirsh Foundation: Grants Portfolio Project Summaries 2003 Gabriele bergers, Ph.D. University of California San Francisco (San Francisco, CA) Our goal is to closely model the human astrocytoma disease(s). http://www.goldhirshfoundation.org/project_summaries_2004.htm

Foundation Leadership Scientific Advisory Board Grant Recipients Application Information ... Home 2004 Project Summaries Elizabeth Maher, M.D., Ph.D. Charles L. Sawyers, M.D. Arturo Alvarez-Buylla, Ph.D. Gabriele Bergers, Ph.D. ... Terry A. Van Dyke, Ph.D. Recipients of Three-year Awards Elizabeth Maher, M.D., Ph.D. Dana-Farber Cancer Institute (Boston, MA) Identifying the Genetic Switches Driving Glioma Initiation and Progression Glioblastoma (GBM) represents one of the most aggressive and deadly cancers for which there has been little improvement in treatment or basic understanding of disease over the past 25 years of clinical trials and basic science research. Although in its most common form GBM is of highest grade at the time of diagnosis, the ability to study the progression from low-grade astrocytoma to GBM provides a valuable opportunity to identify the key genetic switches governing this transition. Such knowledge of progression genetics would provide both a means of identifying those low-grade patients at increased risk for progression as well as offer novel and more directed drug development opportunities in advanced gliomas including primary and secondary GBM. The support of the Goldhirsh Foundation over the past year has led to the identification of several novel chromosomal regions of gain and loss that are associated with low grade astrocytomas and other regions associated with the transition to secondary GBM. The development of a higher resolution platform for genetic analysis over the past year coupled with the recent implementation of novel bioinformatic tools has been a major advance in our ability to perform these studies and provides even higher confidence that the genes identified will prove to be clinically relevant. The proposed expanded study is designed to ultimately identify those critical genetic elements governing disease progression, grade-specific biology, and therapeutic response.

59. Shepherds Of Good Hope - Les Bergers De L'Espoir Click to go to Les bergers de l`Espoir En Français Social withdrawal, apathy, and fatigue are a constant reminder to me of my disease. http://shepherdsofgoodhope.com/ma2.html

DIVERSITY : Multiplicity of Differences Jennifer J. Holtom from Hope Chronicles, our Spring 2005 Newsletter "EQUALLY LOVED" The great prophet's message of all major religions' could be simplified and understood to be "take care of one another". Our Community at the Shepherds continues to strive not to distinguish between our roles: - client, resident, staff, volunteer, board member, manager, etc., but, to see all as created equal. Understanding each of our roles as better, different or more powerful, only informs us that a lifetime of learning will never be enough to understand the complexities of existence and creation. Events that occur and why they do, can often not be explained or understood, although, science certainly attempts to understand and inform us. Our Creator has created a universe beyond our capacity at this time to understand completely. The message repeatedly heard during the centuries, "if we are hearing", is that there are no differences. We are all of equal value to our Creator. " We are the light!" created equal in our Creator's eyes.

60. Claw Diseases - WSAVA 2002 Carlotti (DN) Claw disease in dogs and cats. European Journal of Small Animal Epidermolyse bulleuse jonctionnelle familiale chez les chiots bergers de http://www.vin.com/proceedings/Proceedings.plx?CID=WSAVA2002&PID=2543

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