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         Atherosclerosis:     more books (100)
  1. Atherosclerosis XI
  2. Current Developments in Atherosclerosis Research
  3. Tobacco Smoking and Atherosclerosis (Advances in Experimental Medicine and Biology)
  4. Oophorectomy Increases Risk for Atherosclerosis.(Brief Article): An article from: Family Practice News by Doug Brunk, 2001-02-01
  5. Coenzyme Contents of Arterial Tissue (Monographs on atherosclerosis) by John Esben Kirk, 1974-01-01
  6. Bile Acids (Monographs on atherosclerosis) by William T. Beher, 1976-01-01
  7. Metabolic syndrome predicts subclinical atherosclerosis in adults.(Across Specialties): An article from: Clinical Psychiatry News by Bruce Jancin, 2005-07-01
  8. Vitamin Contents of Arterial Tissue (Monographs on atherosclerosis) by John Esben Kirk, 1973-01-01
  9. Smooth muscle cells in atherosclerosis (Monographs on atherosclerosis) by Jack C Geer, 1972
  10. Diet and Drugs in Atherosclerosis: European Atherosclerosis Group Meeting, Lugano, Switzerland by Noseda, 1981-09
  11. Inflammatory and Infectious Basis of Atherosclerosis (Progress in Inflammation Research)
  12. Dietary Proteins and Atherosclerosis by G. Debry, 2003-11-24
  13. Hypercholesterolemia: Clinical and Therapeutic Implications (Atherosclerosis Reviews) (Vol 18) by Joseph Stokes, 1988-06
  14. Atherosclerosis: Is It Reversible?

121. Atherosclerosis
atherosclerosis, also called hardening or blockage of the arteries, In atherosclerosis, a fatty substance called plaque builds up in the walls of
http://www.pfizer.ca/english/your health/heart health/atherosclerosis/default.as

122. Intima Media Thickness And Atherosclerosis
Intima Media Thickness and atherosclerosis. To begin the lecture, click the STARTbutton above. If you are the first time visitor, you might want to know
http://www.pitt.edu/~super1/lecture/lec3281/
Lists of Lectures Front Page
Intima Media Thickness and Atherosclerosis
To begin the lecture, click the START button above. If you are the first time visitor, you might want to know [ How to navigate within and outside the lecture This is a beta version. Your comments to this version would be highly appreciated as well. Submit Your comments

123. Pharmaceutical Research And Manufacturers Of America
atherosclerosis. Complete a new search atherosclerosis GlaxoSmithKlineResearch Triangle Park, NC. Phase I. 677116 (LpPLA2 inhibitor) none / none
http://www.phrma.org/newmedicines/newmedsdb/drugs.cfm?indicationcode=Atheroscler

124. Cholesterol
Cholesterol is also strongly implicated in the development of atherosclerosis fatty the subject may be at increased risk of developing atherosclerosis.
http://users.rcn.com/jkimball.ma.ultranet/BiologyPages/C/Cholesterol.html
Cholesterol
Cholesterol is small molecule, one of the steroids . It is essential to life. It has also been responsible for 17 Nobel Prizes, countless pages of reports in scientific journals and the popular press, and mounting anxiety on the part of health-conscious people. Why? The human body contains about 100 g of cholesterol. Most of this is incorporated in the membranes from which cells are constructed and is an indispensable component of them. The insulating layers of myelin wound around neurons are especially rich in cholesterol. In far smaller quantities, but no less important, cholesterol is starting ingredient for the synthesis of the steroid hormones: Cholesterol is also the precursor from which the body synthesizes vitamin D One of the major uses of cholesterol is the synthesis of bile acids . These are synthesized in the liver from cholesterol and are secreted in the bile There is also an unceasing transport of cholesterol in the blood between the liver and all the other tissues. Most of this cholesterol travels complexed with fatty acids and protein in the form of low density lipoproteins LDL s). Cells that need cholesterol trap and ingest LDLs by

125. Clinical Trial: Atherosclerosis Prevention In Pediatric Lupus Erythematosus (APP
atherosclerosis Prevention in Pediatric Lupus Erythematosus (APPLE). This studyis currently recruiting patients. Verified by National Institute of
http://www.clinicaltrials.gov/ct/gui/show/NCT00065806
Home Search Browse Resources ... About Atherosclerosis Prevention in Pediatric Lupus Erythematosus (APPLE) This study is currently recruiting patients.
Verified by National Institute of Arthritis and Musculoskeletal and Skin Diseases (NIAMS) March 2005 Sponsored by: National Institute of Arthritis and Musculoskeletal and Skin Diseases (NIAMS) Information provided by: National Institute of Arthritis and Musculoskeletal and Skin Diseases (NIAMS) ClinicalTrials.gov Identifier: Purpose High cholesterol levels are common in people with Systemic Lupus Erythematosus (SLE). Atorvastatin is a drug that reduces cholesterol levels. This study will test whether atorvastatin can reduce cholesterol levels in children with SLE. Condition Intervention Phase Lupus Erythematosus, Systemic
Drug: Atorvastatin
Phase III

MedlinePlus
related topics: Lupus
Study Type: Interventional
Study Design: Treatment, Randomized, Double-Blind, Placebo Control, Factorial Assignment, Efficacy Study Official Title: A Multicenter, Randomized, Double-Blind, Placebo-Controlled Study to Test the Safety and Efficacy of Lipitor (Atorvastatin) in Reducing the Progression of Carotid IMT in Early Childhood SLE Further Study Details:
Expected Total Enrollment: 280 Study start: September 2003; Expected completion: April 2009

126. What's Hot: Atherosclerosis And Dyslipidemia
atherosclerosis is a type of arteriosclerosis, and is characterized by deposits of atherosclerosis is a slow, progressive disease, and if you have been
http://www.diabetic-lifestyle.com/articles/apr99_whats_1.htm
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what's hot
april 99 Diabetic-Lifestyle What's Hot informs and stimulates with monthly in-depth articles on diabetic health topics. Diabetic-Lifestyle offers recipes, menus, medical updates, entertaining, travel - practical information to enhance life while managing diabetes on a daily basis. - Home Atherosclerosis and Dyslipidemia We continue our articles on cardiovascular disease with this article on another aspect of the problem that has great significance for diabetics. Arteriosclerosis is the general term for thickening and hardening of the arteries. This occurs normally as we age. Atherosclerosis is a type of arteriosclerosis, and is characterized by deposits of fatty substances, cholesterol, cellular waste products, calcium, and fibrin in the inner lining of the arteries. This buildup is called plaque. Plaque can partially or totally block the flow of blood through an artery. When plaque occurs two things can happen: hemorrhaging into the plaque and/or formation of a clot or thrombus on the plaque's surface. If either of these occurs and blocks the entire artery, a heart attack or stroke may result. Atherosclerosis is a slow, progressive disease, and if you have been reading research on the subject, you know that the process has been found to begin in children and adolescents. It affects large and medium sized arteries, but the type of artery and the progressive time table appear to vary with the individual.

127. University Of Chicago Hospitals: Atherosclerosis
Detailed information on atherosclerosis, including causes, disease progression,symptoms, diagnostic, and treatment information.
http://www.uchospitals.edu/online-library/content=P00197
@import "/css/style.css"; @import "/css/gs.css"; Home About the Hospitals Donate or Volunteer Contact Us ... Cardiac Conditions and Diseases
Atherosclerosis
What is atherosclerosis?
Atherosclerosis is a type of arteriosclerosis caused by a build-up of plaque in the inner lining of an artery. (Arteriosclerosis is a general term for thickening or hardening of the arteries.) Plaque is made up of deposits of fatty substances, cholesterol, cellular waste products, calcium, and fibrin, and can develop in medium or large arteries. The artery wall becomes thickened and looses its elasticity. Atherosclerosis is a slow, progressive disease that may start as early as childhood. However, the disease has the potential to progress rapidly.
What causes atherosclerosis?
It is unknown exactly how atherosclerosis begins or what causes it. Some scientists think that certain risk factors may be associated with atherosclerosis, including:
  • elevated cholesterol and triglyceride levels high blood pressure smoking diabetes mellitus (type 1 diabetes) obesity physical inactivity
How does the disease progress?

128. Stanford Hospital And Clinics - Stanford University Medical Center - Atheroscler
atherosclerosis is a type of arteriosclerosis caused by a buildup of plaque in the Specific treatment for atherosclerosis will be determined by your
http://www.stanfordhospital.com/healthLib/atoz/cardiac/athero.html
Print-friendly View Cardiac Surgeons
Cardiologists

Heart and Lung Transplant Specialists

Interventional Cardiologists
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Vascular Surgeons
Cardiovascular Diseases
Atherosclerosis
What is atherosclerosis?

Atherosclerosis is a type of arteriosclerosis caused by a build-up of plaque in the inner lining of an artery. (Arteriosclerosis is a general term for thickening or hardening of the arteries.) Plaque is made up of deposits of fatty substances, cholesterol, cellular waste products, calcium, and fibrin, and can develop in medium or large arteries. The artery wall becomes thickened and looses its elasticity. Atherosclerosis is a slow, progressive disease that may start as early as childhood. However, the disease has the potential to progress rapidly.
What causes atherosclerosis?
It is unknown exactly how atherosclerosis begins or what causes it. Some scientists think that certain risk factors may be associated with atherosclerosis, including the following:
  • elevated cholesterol and triglyceride levels high blood pressure smoking type 1 diabetes obesity physical inactivity
How does the disease progress?

129. Cardiovascular Diabetology | Full Text | How Hyperglycemia Promotes Atherosclero
How hyperglycemia promotes atherosclerosis molecular mechanisms atherosclerosisaccounts for virtually 80% of all deaths among diabetic patients.
http://www.cardiab.com/content/1/1/1
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Review How hyperglycemia promotes atherosclerosis: molecular mechanisms Doron Aronson and Elliot J Rayfield Cardiology Division, Rambam Medical Center, 31096 Haifa, Israel Clinical Professor of Medicine Mount Sinai School of Medicine, New York, NY 10029, USA USA Visiting Physician, Rockefeller University Hospital, New York, NY 10021, USA Cardiovascular Diabetology The electronic version of this article is the complete one and can be found online at: http://www.cardiab.com/content/1/1/1 Received Accepted Published Outline Abstract Abstract Advanced glycosylation end products Protein kinase c Oxidative stress ... References Outline Abstract Advanced glycosylation end products Protein kinase c Oxidative stress ... References Both type I and type II diabetes are powerful and independent risk factors for coronary artery disease (CAD), stroke, and peripheral arterial disease ]. Atherosclerosis accounts for virtually 80% of all deaths among North American diabetic patients, compared with one third of all deaths in the general North American population

130. Annual Reviews - Error
Table 2 Mendelian disorders relevant to atherosclerosis1 Genetics ofatherosclerosis. In Textbook of Cardiovascular Medicine, 2nd Edition, ed. E Topol.
http://arjournals.annualreviews.org/doi/full/10.1146/annurev.genom.5.061903.1759
An Error Occurred Setting Your User Cookie A cookie is a small amount of information that a web site copies onto your hard drive. Annual Reviews Online uses cookies to improve performance by remembering that you are logged in when you go from page to page. If the cookie cannot be set correctly, then Annual Reviews cannot determine whether you are logged in and a new session will be created for each page you visit. This slows the system down. Therefore, you must accept the Annual Reviews cookie to use the system. What Gets Stored in a Cookie? Annual Reviews Online only stores a session ID in the cookie, no other information is captured. In general, only the information that you provide, or the choices you make while visiting a web site, can be stored in a cookie. For example, the site cannot determine your email name unless you choose to type it. Allowing a web site to create a cookie does not give that or any other site access to the rest of your computer, and only the site that created the cookie can read it. Please read our for more information about data collected on this site.

131. Gene Clusters Predict Atherosclerosis Severity, Susceptibility
Duke University Medical Center researchers have identified specific clusters ofgenes within human aortas that appear to predict with great specificity
http://www.eurekalert.org/pub_releases/2004-09/dumc-gcp092804.php
Public release date: 28-Sep-2004
E-mail Article

Contact: Richard Merritt
Merri006@mc.duke.edu

Duke University Medical Center
Gene clusters predict atherosclerosis severity, susceptibility
DURHAM, N.C. Duke University Medical Center researchers have identified specific clusters of genes within human aortas that appear to predict with great specificity which patients may be at highest risk for developing atherosclerosis, as well as the severity of the disease. For the researchers, this is an important first of many steps toward developing highly individualized approaches to identifying and treating atherosclerosis that are tailored to and informed by a patient's unique genetic make-up. Atherosclerosis is a disorder marked by the thickening and clogging of blood vessels, which over time can deprive the heart of necessary oxygen and nutrients. While factors such as diet, smoking, cholesterol levels and inactivity are important in the development of atherosclerosis, the researchers said that heredity plays a crucial role in how the body responds to these environmental factors. "Instead of trying to find a specific gene that might be implicated in the development atherosclerosis, we took the novel approach of trying identify clusters of genes that may help us better understand the progression of the disease," said Duke cardiologist David Seo, M.D. The results of the Duke research are scheduled to appear in the October 2004 issue of the journal Arteriorsclerosis, Thrombosis and Vascular Biology and are published early on-line at

132. FOODnetBASE: Food Sciences Online
Dietary Proteins and atherosclerosis provides a thorough review of the role ofproteins in the development of atherosclerosis. The author reviews early
http://www.foodnetbase.com/ejournals/books/book_summary/summary.asp?id=1236

133. DHEA For Weight Loss: Miracle Drug Or Unproven Experiment?
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DHEA for weight loss: miracle drug or unproven experiment?
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