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         Anemia:     more books (100)
  1. Anemia in Women: Self-Help and Treatment by M.D. Joan Gomez, 2002-10-14
  2. Understanding Anemia (Understanding Sickness & Health Series) by M.D.Ed Uthman, 1998-03-01
  3. The Iron Disorders Institute Guide to Anemia by Cheryl Garrison, 2009-06-01
  4. In the Blood: Sickle Cell Anemia and the Politics of Race (Critical Histories) by Melbourne Tapper, 1998-01-01
  5. Dying in the City of the Blues: Sickle Cell Anemia and the Politics of Race and Health by Keith Wailoo, 2001-03-26
  6. Menace In My Blood: My Affliction With Sickle-Cell Anemia by Ola Tamedu, 2006-01-24
  7. Endless Love by Elizabeth A. Ryan, 2007-09-07
  8. Immune Hemolytic Anemias by Lawrence D. Petz, George Garratty, 2003-12-16
  9. Heavy Menstrual Flow and Anemia: Self Help Book by Susan M. Lark, 1996-03-01
  10. Anemias and Other Red Cell Disorders by Kenneth Bridges, Howard A. Pearson, 2007-12-13
  11. Blood And Circulatory Disorders Sourcebook: Basic Consumer Health Information About The Blood And Circulatory System And Related Disorders, Such as Anemia ... Diseases, Cancer o (Health Reference Series) by Amy L. Sutton, 2005-06-01
  12. Sickle Cell Anemia - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References by ICON Health Publications, 2004-03-12
  13. Turning Blood Red: The Fight for Life in Cooley's Anemia by Arthur Bank, 2008-11-28
  14. Hope and Destiny: A Patient's and Parent's Guide to Sickle Cell Anemia by Allan F. Platt Jr. PA-C, Alan Sacerdote MD, 2006-04-01

161. B+ Home
A charitable organization providing assistance to children with Fanconi anemia within the St. Louis and St. Charles Missouri metro areas.
B Positive! Bone Marrow Drive
UPDATE B Positive, Inc.
P.O. Box 533
Chesterfield, Mo. 63006-0533
(314) 503-BPOS (2767)
In December, 2000, two St. Louis area siblings, Matthew Pearl, age 4, and Alexandra Pearl, age 6, were diagnosed with Fanconi Anemia . At the same time, they discovered that each child had B+ blood types and that their only hope for finding a cure was successful bone marrow transplant. The odds of any given individual, other than an immediate family member, being a perfect match is about one in a million.
Alex received her bone marrow transplant in May, 2001 and is recovering well... however, no match for Matt has
been found yet. We need your help!
For more about Alex and Matt, visit their web site at Welcome to the B Positive, Inc. Web Site! For those who do not know exactly what Fanconi Anemia is, it is a genetic, life-threatening, inherited blood disorder that leads to bone marrow failure. It occurs equally in males and females and is found in all ethnic groups.

162. Virtual Children's Hospital: CQQA: Anemia
Common Questions, Quick Answers on anemia. anemia in children is most oftencaused by not eating enough iron, but can have other causes.
Pediatrics Common Questions, Quick Answers
Anemia (Iron Deficiency)
Donna D'Alessandro, M.D.
Lindsay Huth, B.A.
Peer Review Status: Internally Reviewed
Creation Date: February 2002
Last Revision Date: April 2002 Common Questions, Quick Answers What is anemia?
  • Anemia is having fewer red blood cells than is normal. Anemics do not have enough red blood cells.
What causes anemia?
  • The body uses iron to make red blood cells and to build muscle and strong bones. Red blood cells carry oxygen. Anemia in children is most often caused by not eating enough iron, but can have other causes. Heavy menstrual periods can cause anemia if too much iron is lost in the blood.
Who can be anemic?
  • Anyone who does not get enough iron can be anemic. It is especially important for infants and teens to get enough iron. Girls who are menstruating need lots of iron.
What are the symptoms of anemia?
  • There may be none. Symptoms are hard to notice unless the anemia is severe. Children may feel weak and get tired easily.

163. Alex And Matt Pearl
Personal stories of two children fighting Fanconi anemia.
PHOTO GALLERY UPDATED!! OVER 500 PHOTOS!! Hi, most of you may know our story; we are Alexandra and Matthew Pearl In December 2000 and January 2001 we were both diagnosed with a rare, life threatening blood disease called Fanconi anemia. It causes bone marrow failure; our bodies cannot make the blood we need to survive. Our only hope is a bone marrow transplant. Cardinals Help With Bone
Marrow Drive For Eureka Child
Read Press Release Alex has found a perfect match and is recovering from her May 31, 2001 transplant in Cincinnati, Ohio. There is no perfect match for Matthew, in any registry throughout the entire world. We need your help, please consider getting added to the National Bone Marrow Registry through the Pearl Million-Donor March. Refer to the ' How You Can Help ' section.

164. Pernicious Anemia
Pernicious anemia is caused by an inability to absorb vitamin B12 (cobalamin)that is Lack of vitamin B12 produces megaloblastic anemia, nerve damage,
Home Educational Topics
Pernicious Anemia
Pernicious anemia is caused by an inability to absorb vitamin B12 (cobalamin) that is naturally found in certain foods. These foods are all of animal origin and include meat, milk and dairy products, and eggs. Vitamin B12 is not found in plants. Although bacteria in the large intestine produce vitamin B12, it is not absorbed into the blood stream from this site. Most people need only 2 micrograms of vitamin B12 daily but the average diet provides about 5 to 30 micrograms a day. When it is ingested, vitamin B12 needs to be chemically linked to a substance called intrinsic factor, which is produced in the stomach. In the animation on the right , vitamin B12 is shown in pink and the intrinsic factor is shown in blue. They combine in the stomach and pass into the small intestine where the intrinsic factor helps the vitamin B12 get absorbed into the circulation. Through the circulation, the vitamin is transported to the liver where it is stored, being released back into the circulation as needed. Without intrinsic factor, vitamin B12 is not absorbed and pernicious anemia occurs. Other causes of vitamin B12 deficiency produce the same symptoms as pernicious anemia. Diseases of the small intestine that cause malabsorption may cause vitamin B12 deficiency. Vegans (individuals who consume absolutely no foods of animal origin) are at risk of vitamin B12 deficiency due insufficiency of this vitamin in their diet. But it takes a long time to deplete all the vitamin B12 that is stored in the liver. Even if the absorption of vitamin B12 is suddenly cut off (for example, as in gastrectomy, surgical removal of the stomach), there is enough vitamin B12 stored in the liver to last for one to five years.

165. Adam Day
Learn about a boy who passed away after a fight with Fanconi anemia. Find out how to make a donation to the Fanconi anemia Research Fund.
Posted by Darla on February 28, 1997 at 03:02:11:
Soaring with angels Greetings All:
We, again would like to thank everyone for their sincere and heartfelt support. We literally could not have come as far as we did without all of you. We regretfully inform you all that Adam passed away at 9:11 PM, Minnesota time, on February 27, 1997. He was peaceful and pain-free when he passed. His family was with him, as was most of the staff from 4A, who have care for him in the entire duration of our stay here. We will happily inform everyone, though that Adam is now soaring with the angels, watching down protectively on all of us. We're sure he is having go-cart races with Mark Muellen and showing his knife collection to Joey Adamson and fishing with John Dumouchel. We will miss him but we know he is in a better place. May God bless all of you and shower you with goodness, health and many miracles. Thank you again. The Day Family Adam's Uncle Bob went to Minnesota during 1/30-2/3 and took some photos. Adam was in great spirits despite the constant interruptions by a wide variety of physicians and assistants. Go to:

166. Anemia On Sympatico / MSN Powered By MediResource
anemia is a condition where the number of healthy red blood cells (RBCs) in theblood is less than normal. RBCs transport oxygen throughout the body,

167. - Information On Clinical Trials And Human Research Studies:
anemia, Refractory, with Excess of Blasts (183 recruiting studies). 14. anemia,Sickle Cell (31 recruiting studies). 15. Antithrombin III Deficiency (1

168. Fanconi Anemia
Fanconi anemia (FA), first described in 1927 by a Swiss pediatrician Guido Fanconi, Canadian Fanconi anemia Research Fund / La Fondation Canadienne de


2006 Toronto Fundraiser

2005 National Anemia Patient Conference

Fanconi Anemia
Donate Now

Families Welcome Page

Fanconi Anemia
Fanconi Anemia
What is FA?
Fanconi Anemia (FA), first described in 1927 by a Swiss pediatrician Guido Fanconi, is the most common of the inherited anemias that lead to progressive, severe bone marrow failure, also known as aplastic anemia. The effects of the disease are devastating, leaving patients weak, prone to severe bleeding due to insufficient blood clotting and susceptible to infection. FA is a genetic disorder that occurs equally in males and females and is found in all ethnic groups. Though considered primarily a blood disease, it may affect all systems of the body. A Fanconi Anemia patient often, but not always, has other physical defects detectable at the time of birth ranging from minor to serious. Patients are also at an increased risk for developing leukemia and other cancers. Many children do not survive to adulthood. How is FA diagnosed?

169. Having Patience When You Are The Patient!
A positive survival story.
My name is Shari and I am an Aplastic Anemia Survivor. I am just beginning to build this page, so please be patient! In the mean time, please feel free to e-mail me at Thank you! SOMETHING ABOUT ME!!! I am a 33 year old female in remission with Aplastic Anemia. I was diagnosed in November of 1985. I was treated with over 200 blood transfusions(red cells and platelets), prednisone(steroids), ATG(Anti-Thymocyte Globulin), then, eventually had a splenectomy(removal of the spleen) in September of 1986. I was told I was in remission in the Spring of 1987. I can't ever be told that I'm cured, because, I have never had a bone marrow transplant. A bone marrow transplant is the only known CURE. Unfortunately, a perfect match could not be found for me. When I was diagnosed, my white count didn't exist at all. My hemoglobin was 4.0, and my platelet count was only 500(normal is 150,000-350,000). Therefore, my immune system was "shot". I was open to get all kinds of infections. I was bruised all over, had terrible migraines, and low blood pressure. The doctors even had to stop my menstrual cycle so I wouldn't bleed the little blood that I had left. You may be thinking, well, what's the miracle!! Well, that was all of the negative information. Now, for the positive: I've been in remission for 10 years now; I went on to finish college(I was a junior when I got sick), and, I got married. I didn't know if I'd make it through the Aplastic Anemia, and, not only have I survived the illness, I brought a new life into this world! On March 3, 1995, I delivered a healthy beautiful girl named Alexa. Her middle name is Faythe(for all the faith I had to get through the illness). I had the child that I never thought I could have, due to all of the medications I had taken.

170. Vitamin B12 Deficiency / Pernicious Anemia: Patient Information: Arizona Telemed
Therefore vitamin B12 deficiency results in anemia, an RBC deficiency limitingthe amount Vitamin B12 deficiency is usually caused by pernicious anemia,
Patient Information Resource:
Benign Hematologic (Blood) Disorders A collaborative project of the Arizona Telemedicine Program , the Arizona Health Sciences Library and the Arizona Cancer Center See: Vitamin B Deficiency: Pernicious Anemia Vitamin B deficiency results in anemia, an RBC deficiency limiting the amount of oxygen in the tissues. Vitamin B deficiency is usually caused by pernicious anemia, a condition in which the digestive system is altered and cannot absorb vitamin B systemic lupus erythematosus , myxedema), or drug action. Among the drugs that may inhibit absorption are histamine blockers, such as cimetidine (Tagamet) and ranitidine (Zantac). Multiple symptoms result from malabsorption of vitamin B . Anemia, mentioned above, is associated with weakness, pallor, elevated heart rate, and elevated breathing rate (see Anemia ). In addition, the patient may be especially prone to infection and may bruise or bleed easily (see Neutropenia and Thrombocytopenia ). Other symptoms include sore tongue, numbness, and tingling.

171. Ilir's Progress With Aplastic Anemia
Includes information about aplastic anemia and Ilir's progress with this disease.
Ilir Kullolli On this page About Ilir Kullolli
Aplastic Anemia

Blood elements
On this site Pictures of Ilir
Discussion List

Recent Pictures. New!!!
This page was last updated on: July 5, 2001 Ilir Kullolli is an International Student from Albania. He was attending Cerro Coso Community College in Ridgecrest, California when he was diagnosed with Aplastic Anemia. He was hospitalized on November 17, 1999. After getting better he transferred to Wentworth Institute of Technology in Boston, Massachusetts and graduated with a Bachelor of Science in Computer Engineering in December 2003. Now he's doing very well and he seems to be sick-free. Aplastic Anemia: An injury to the bone marrow; specifically the stem cells. Stem cells make red and white cells and platelets. The injury may be genetic, caused by viruses, drugs or other toxins. One known cause is an antibiotic used in Europe called Chloramphenocol. People who get this have a 1 in 30,000 chance of contracting blood disorders such as Aplastic Anemia. Ilir received this drug three years ago in Albania when he had appendicitis. The doctor is skeptical of this as a cause because of the time between it and the onset of the disease. [back to index] Blood elements Hemoglobin: Carries oxygen from the lungs to the body and Carbon Dioxide back to the lungs.

172. MEdIC - Health Explorer - Iron Overload
MEdIC Aplastic anemia patients who receive repeated blood cell transfussions may develop iron overload. A look at this condition and who needs treatment.
Iron Overload Aplastic anemia patients who receive repeated blood cell transfussions may develop iron overload. Iron from transfused red cells builds up in the blood and eventually accumulates in the heart, liver, pancreas, and endocrine organs. This excess iron may eventually damage vital organs and cause complications like liver disease, heart disease, and diabetes mellitus. Normal body iron stores are 3-4 grams. Each unit of transfused red cells contains 200-250 mg of iron. Thus, a patient who receives 2 units of blood each month would accumulate approximately 5-6 g of extra iron in one year. Without treatment to remove excess iron, damage to the heart and other organs occurs in patients who have received as few as 100 units of blood, or 20 grams of excess iron. Visible signs of iron overload, such as bronze or slate grey skin pigmentation, don't usually appear until enough iron has accumulated to cause tissue damage. In the United States, the only way to prevent or treat iron overload is with the iron chelating drug deferoxamine (Desferal). Desferal binds excess body iron and promotes its excretion by the kidneys in urine and via the bile in feces. Desferal is administered by subcutaneous or intravenous infusion by a small portable pump about the size of a Walkman. Typically the patient inserts a subcutaneous needle and wears the pump for 9-12 hours each day, usually at night while sleeping. Severely iron overloaded patients may need a continuous infusion through an indwelling central venous catheter. Several studies have demonstrated that regular chelation therapy with Desferal can remove excess body iron, prevent organ damage, and prolong life.

173. Anemia Cases
A case study of a 50 year old woman complaining of anorexia and shortness of breath for the past few weeks.
Anemia Case Studies
Case #2
A 50 year old woman complains of anorexia and shortness of breath for the past few weeks. She feels well but has lost 30 pounds over the last 6 months. Ten years ago she was treated for anemia with iron pills which she took for only a short time. On exam there is a slight yellow tinge to her skin and sclera, a smooth red tongue, and difficulty with fine motor coordination. CBC: RBC 1.70 million/ul, Hct. 19.3%, Hbg. 6.5 g/dl, MCV 114 fl, RDW 22, WBC 2,000/ul, Retics: 2.6% (44,200/ul).
Peripheral Blood Smears:
100x Smear (250 x 350) 100x Smear (440 x 640) 1000x Smear (250 x 350) 1000x Smear (440 x 640)
View All Additional Test Results
Order Additional Test Results
1. Explain why this patient is pancytopenic. Answer
2. List other causes of this type of anemia. Answer
3. What test confirms the diagnosis? Answer

174. Perncious
An indepth overview of pernicious anemia and links to other informative sites.
DRACULA'S Campaign Against Pernicious Anemia WELCOME TO ANOTHER WACKY MEDICAL WEBSITE created by Cynthia Donlan. If you have any questions or just need someone to talk to who understands what you are going through don't hesitate to E-mail me! I don't bite. Also check out my homepage by clicking on the link below. Finally,At the Medical Website I developed, I have information on various other Autoimmune diseases such as Systemic Lupus Erythematosus, Sjogren's Syndrome, Myasthenia Gravis, Diabetes and many many more. Just click on the back button below!!! GOOD EVENING! Hello I am Count Dracula. I have been known for a long time to have an affinity for blood so I have gone to medical school to become a Hematologist. As part of my training we were taught about the various types of anemia. Those conditions both sickened and frightened me. What is a vampire to do if they lose their food source? I need healthy blood from healthy people so that I don't become deficient. This WebPage is brought to you by Blood Brothers, a fraternal order of Transylvanian Vampire Physicians. We hope you find this informative! Stay Healthy and try to be O+ about life! WHAT IS PERNICOUS ANEMIA?

175. InteliHealth: Pernicious Anemia
Details about this disorder, what it is, the symptoms, diagnosis, prevention, treatment, prognosis and when to call a doctor.
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Vitamin B12 Deficiency
  • What Is It?
  • 176. OHSU Division Of Hematology & Medical Oncology
    Details about many blood products and the risks of transfusion.
    Contact Information:
    3181 SW Sam Jackson Park Rd
    Portland, OR 97239-3098
    Phone: (503) 494-6594
    Email: Find a Doctor FAQ Sheet Construction Updates
    Healthcare Professionals
    General Information
    Upcoming Events
    ASH 2004
    Faculty Spotlight
    Craig Nichols, M.D.
    Division Chief
    "The disease of cancer will be banished from life by calm, unhurrying, persistent men and women, working with every shiver of feeling controlled and suppressed in hospitals and laboratories, and the motive that will conquer cancer will not be pity or horror; it will be curiosity to know how and why."
    H.G. Wells, 1901

    177. Anaemia
    Headlines. DNA Down Under. New section on RNA interference as a tool to blockvirus replication. Anaemia. The erythrocyte as seen by the routine
    The Basics


    DNA Down Under
    New section on RNA interference as a tool to block virus replication. Anaemia The erythrocyte as seen by the routine haematologist is essentially at the final stage of maturation. The "cell" is anucleate, without mitochondria, lysosomes or endoplasmic reticulum (ER), and relies on the Embden-Meyerhof and Heptose Monophosphate pathways to maintain the cell's shape etc. All cells (except for T cells) are produced in haematopoietic tissue called Bone Marrow . In a child, this is found in the tibia. In an adult, bone marrow is found in the anterior/posterior iliac crest, sternum, ribs, vertebrae and skull. The bone marrow houses the most primitive haematological cells - called Stem cells. These cells are pluripotential, which means they are able to differentiate into a range of haematological cell types. The bone marrow is influenced by Erythropoietin produced mostly in the kidneys, but also the liver.

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