61. Penn State Faculty Research Expertise Database (FRED) , A hemostatic disorder characterized by a poor anticoagulant response to activated protein C (APC).......activated protein c resistance. http://fred.hmc.psu.edu/ds/retrieve/fred/meshdescriptor/D020016

62. Obstetrical & Gynecological Survey - UserLogin Changes of Resistance to Activated Protein C in the Course of Pregnancy and Obstetric Implications of activated protein c resistance and Factor V Leiden http://www.obgynsurvey.com/pt/re/obgynsurv/fulltext.00006254-199901000-00003.htm

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63. Case - Activated Protein C Resistance In The Absence Of Factor V Leiden Mutation Two patients with deep vein thrombosis following treatment with thalidomide are presented. These cas. http://www.docguide.com/news/content.nsf/cases/41F4099DDEB81E5185256F87007275C0

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64. Abstract The relation of activated protein c resistance with ABO blood groups. Abstract number P0942. Ozcan* M., Kasapoglu† A., Topcuoglu* P., Arslan* O. http://www.blackwellpublishing.com/isth2003/abstract.asp?id=9116

65. LU:research - Lund University Institutional Archive Title, Reactions to awareness of activated protein c resistance carriership a descriptive study of 270 women. Fulltext, Available as PDF http://lu-research.lub.lu.se/php/gateway.php?who=lr&method=getfile&file=archive/

66. LU:research - Lund University Institutional Archive Title, The discovery of activated protein c resistance. Fulltext, Not available in this archive. Author, Dahlbäck, B http://lu-research.lub.lu.se/php/gateway.php?who=lr&method=getfile&file=archive/

67. Journal Of Pediatric Gastroenterology And Nutrition - Fulltext: Volume 26(2) Feb activated protein c resistance in Pediatric Inflammatory Bowel Disease Conclusions activated protein c resistance does not seem to play a major role in http://www.jpgn.org/pt/re/jpgn/fulltext.00005176-199802000-00009.htm

68. Protein C - Enpsychlopedia activated protein c resistance is the inability of protein C to cleave factors V and/or VIII. This may be hereditary or acquired. http://psychcentral.com/psypsych/Protein_C

home resource directory disorders quizzes ... support forums Protein C Protein C is a major physiological anticoagulant . It is a vitamin K -dependant serine protease enzyme EC http://us.expasy.org/cgi-bin/nicezyme.pl?3.4.21.69 ) that is activated by thrombin into activated protein C (APC). The activated form (with protein S as a cofactor ) degrades Factor Va and Factor VIIIa Contents showTocToggle("show","hide") 1 Genetics 2 Role in disease 3 Pharmacology 4 External link ... edit Genetics The PROC gene is located on the second chromosome edit Role in disease Protein C deficiency is a rare genetic disorder that predisposes to venous thrombosis and habitual abortion . If homozygous , this presents with a form of disseminated intravascular coagulation in newborns termed purpura fulminans ; it is treated by replacing the defective protein C. Activated protein C resistance is the inability of protein C to cleave factors V and/or VIII. This may be hereditary or acquired. The best known and most common hereditary form is Factor V Leiden . Acquired forms occur in the presence of elevated Factor VIII concentrations. Warfarin necrosis is acquired protein C deficiency due to treatment with the vitamin K inhibitor anticoagulant warfarin . In initial stages of action, inhibition of protein C may be stronger than inhibition of the vitamin K-dependant coagulation factors (II, VII, IX and X), leading to paradoxical activation of coagulation and

69. Thrombosis And Haemostasis activated protein c resistance (FVQ506) and Pregnancy Activated protein C (APC) resistance, due to a point mutation in the factor V gene (FVQ506), http://www.schattauer.de/zs/thromb/artikel.asp?path=/zs/thromb/1999/4/&nummer=th

70. Portal Toolkit Invalid Site URL activated protein c resistance Associated with Maternal Floor Infarction Factor V Leiden with activated protein c resistance is found in up to 5% of the http://ppv.ovid.com/pt/re/ajrp/fulltext.00001833-200219050-00008.htm

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71. PHS 398 (Rev. 5/01), Biographical Sketch Format Page activated protein c resistance in ischemic stroke not due to factor V arginine506 glutamine mutation. Stroke 271163-6, 1996. 15. Macko RF, Ameriso SF, http://www.bme.ogi.edu/~trant/bme507/AndrasGruber.htm

Principal Investigator/Program Director (Last, First, Middle): Gruber, Andras BIOGRAPHICAL SKETCH Provide the following information for the key personnel in the order listed on Form Page 2. Follow this format for each person. DO NOT EXCEED FOUR PAGES. NAME Andras Gruber , M.D. POSITION TITLE Associate Professor of Biomedical Engineering EDUCATION/ TRAINING Begin with baccalaureate or other initial professional education, such as nursing, and include postdoctoral training.) INSTITUTION AND LOCATION DEGREE (if applicable) YEAR(s) FIELD OF STUDY Semmelweis Medical Univ., Budapest , HU MD Medicine Postgraduate Medical School Budapest , HU Board Cert. Cert. Internal Medicine The Scripps Research Institute, La Jolla CA Postdoc biochem A. Positions and Honors Positions and Employment Medical residency, and staff physician, Internal Medicine, Postgraduate Med. School Budapest , HU Staff physician , Internal Medicine, Szonyi T. County Hospital Vac , HU Postdoctoral Research Fellow The Scripps Research Institute, La Jolla CA Research Associate

72. Entrez PubMed Resistance to the anticoagulant action of activated protein C, APC resistance, is a highly prevalent risk factor for venous thrombosis among individuals of http://ghr.nlm.nih.gov/gene=f5/show/PubMed

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73. MSRGSNet/Genetic Drift/Venous Thrombosis And The Factor V (Leiden) Mutation The mutation causes resistance to activated protein C (APC), and this induces Previous finding of activated protein c resistance by laboratory analysis http://www.mostgene.org/gd/gdvol14b.htm

Previous Section This Issue- Table of Contents Next Section Vol. 14: Spring, 1997 Molecular Genetic Testing in Mainstream Medicine Venous Thrombosis and the Factor V (Leiden) Mutation Introduction Who should be tested? Deep vein thrombosis and pulmonary embolism Known genetic causes ... Illustrative case report Introduction Who should be tested? Indications include: Venous thrombosis or pulmonary embolism Transient ischemic attacks or premature stroke Peripheral vascular disease, particularly lower extremity occlusive disease History of a thrombotic event Family history of thrombosis or known factor V mutation in a relative Prior to major surgery, pregnancy, postpartum, oral contraceptive use or estrogen therapy if there is a personal or family history of thrombosis. Previous finding of activated protein C resistance by laboratory analysis Deep vein thrombosis and pulmonary embolism Venous thrombosis and pulmonary embolism pose a serious health problem. In this country half a million people are hospitalized each year and 50,000-100,000 deaths occur due to venous thrombosis which is also a leading cause of maternal death. The incidence of symptomatic venous thrombosis cases is approximately 1 in 1000 people per year. Venous thrombosis is a multifactorial condition caused by a combination of genetic, aquired or environmental influences. Natural anticoagulant systems (the protein C system and antithrombin III) are in place to keep coagulation in check. Excess clotting occurs when there is a disturbance in one of the coagulation inhibitor mechanisms or in natural lysis of clots.

74. Acquired Activated Protein C Resistance Is Associated With Lupus Anticoagulants Acquired activated protein c resistance (APCR) has been hypothesized as a possible mechanism by which antiphospholipid antibodies (APLAs) cause thrombotic http://www.bloodjournal.org/cgi/content/abstract/97/4/844

QUICK SEARCH: [advanced] Author: Keyword(s): Year: Vol: Page: This Article Full Text Full Text (PDF) Alert me when this article is cited ... Citation Map Services Email this article to a friend Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal ... Rights and Permissions PubMed PubMed Citation Articles by Male, C. Articles by Andrew, M. E. Related Collections Plenary Papers Blood, 15 February 2001, Vol. 97, No. 4, pp. 844-849 PLENARY PAPER Acquired activated protein C resistance is associated with lupus anticoagulants and thrombotic events in pediatric patients with systemic lupus erythematosus Christoph Male Lesley Mitchell James Julian Patricia Vegh Penny Joshua Margaret Adams Michelle David and Maureen E. Andrew Acquired activated protein C resistance (APCR) has been hypothesized as a possible mechanism by which antiphospholipid antibodies (APLAs) cause thrombotic events (TEs). However, available evidence for an association of acquired APCR with APLAs is limited. More importantly, an association of acquired APCR with TEs has not

75. Hyperprothrombinemia May Result In Acquired Activated Protein C Resistance -- Tr Activated protein C (APC) resistance1 due to the factor V (FV) gene mutation Rodeghiero F, Tosetto A. activated protein c resistance and factor V Leiden http://www.bloodjournal.org/cgi/content/full/96/9/3295

QUICK SEARCH: [advanced] Author: Keyword(s): Year: Vol: Page: This Article Full Text (PDF) Alert me when this article is cited Alert me if a correction is posted ... Citation Map Services Email this article to a friend Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal ... Rights and Permissions PubMed PubMed Citation Articles by Tripodi, A. Articles by Mannucci, P. Blood, 1 November 2000, Vol. 96, No. 9, pp. 3295-3296 CORRESPONDENCE To the editor: Hyperprothrombinemia may result in acquired activated protein C resistance Activated protein C (APC) resistance due to the factor V (FV) gene mutation G1691A (single-letter nucleotide codes) and the prothrombin gene mutation G20210A are the most common genetic disorders associated with venous thrombosis. APC resistance not due to the FV gene mutation G1691A, and the hyperprothrombinemia secondary to the prothrombin gene mutation G20210A, are also independent risk factors for venous thrombosis. Although it has been surmised that high levels of prothrombin in plasma may result in an increased rate of thrombin generation or increased thrombin potential

76. Haema/Áßìá activated protein c resistance in the absence of factor V Leiden mutation is associated with an increased risk of thrombotic complications in patients http://www.mednet.gr/eae/haema/h81-18.htm

HAEMA (ÁÉÌÁ) The Journal of the Hellenic Society of Haematology Journal's Home Page Volume 8, Issue 1 Volume 7, Supplement 1 Volume 7, Issue 4 ... Volume 1, Issue 1 4 Papadiamantopoulou str. 115 28 Athens GREECE Tel.-FAX: +30210 7211806 E-mail: eae@hol.gr Activated protein C resistance in the absence of factor V Leiden mutation is associated with an increased risk of thrombotic complications in patients with multiple myeloma receiving first-line thalidomide-dexamethasone therapy Abderrahman Abdelkefi , Neila Ben Romdhane , Mohamed Younes , Sami Guermazi Centre National de Greffe de Moelle Osseuse, Tunis Institut Pasteur de Tunis, Tunisia Haema 2005; 8(1):118-120 Abstract. Key words: thrombophilia, myeloma, thalidomide, deep vein thrombosis, activated protein C resistance. Correspondence: Abderrahman Abdelkefi, MD, Centre National de Greffe de Moelle Osseuse. Rue Jebel Lakhdar, Bab Saadoun 1006, Tunis, Tunisia, Tel.:00216.71775929, Fax:00216.71565428, e-mail: aabdelkefi@yahoo.fr

77. Juvenile Temporal Arteritis And Activated Protein C Resistance -- Granel Et Al. activated protein c resistance due to a factor V mutation associated with familial ischemic stroke. J Neurosurg Sci 1997;41373–8.Medline http://ard.bmjjournals.com/cgi/content/full/63/2/215

HOME HELP FEEDBACK SUBSCRIPTIONS ... TABLE OF CONTENTS Author Keyword(s) Vol Page [Advanced] This Article Extract Full Text (PDF) Submit a response ... Alert me if a correction is posted Services Email this link to a friend Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal ... Download to citation manager PubMed PubMed Citation Articles by Granel, B Articles by Weiller, P-J Related Collections Other Rheumatology Annals of the Rheumatic Diseases LETTER Juvenile temporal arteritis and activated protein C resistance B Granel J Serratrice N Ene P E Morange P Disdier and P-J Weiller Correspondence to: Dr P Disdier; Accepted 12 May 2003 Keywords: giant cell arteritis; juvenile temporal arteritis; vasculitis; protein C resistance Juvenile temporal arteritis (JTA) is a rare benign vascular lesion limited to the branches of the external carotid artery, first recognised by Lie et al Histological features of JTA include vasculitis (without giant cells and granulomas), lymphocytes

78. Juvenile Temporal Arteritis And Activated Protein C Resistance -- Granel Et Al. Juvenile temporal arteritis and activated protein c resistance. B Granel1 , J Serratrice1 , N Ene1 , PE Morange2 , P Disdier1 and PJ Weiller1 http://ard.bmjjournals.com/cgi/content/extract/63/2/215

HOME HELP FEEDBACK SUBSCRIPTIONS ... TABLE OF CONTENTS Author Keyword(s) Vol Page [Advanced] This Article Full Text Full Text (PDF) Submit a response ... Alert me if a correction is posted Services Email this link to a friend Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal ... Download to citation manager PubMed PubMed Citation Articles by Granel, B Articles by Weiller, P-J Related Collections Other Rheumatology Annals of the Rheumatic Diseases LETTER Juvenile temporal arteritis and activated protein C resistance B Granel J Serratrice N Ene P E Morange P Disdier and P-J Weiller Correspondence to: Dr P Disdier; patrick.disdier@ap-hm.fr Accepted 12 May 2003 Keywords: giant cell arteritis; juvenile temporal arteritis; vasculitis; protein C resistance The first 150 words of the full text of this article appear below. Juvenile temporal arteritis (JTA) is a rare benign vascular lesion limited to the branches of the external carotid artery

79. Activated Protein C Resistance And Factor V Leiden Mutation Are Independent Risk Background Resistance to activated protein C due to the factor V R506Q (Leiden) mutation is the activated protein c resistance Is a Risk Factor for VTE http://www.annals.org/cgi/content/abstract/130/8/643

Search Annals: Advanced search Home Current Issue Past Issues ... ACP Online Article Table of Contents Full Text of this article PDF of this article (PDFs free after 6 months) Figures/Tables List Related articles in Annals Services Send comment/rapid response letter Notify a friend about this article Alert me when this article is cited Add to Personal Archive ... ACP Search PubMed Articles in PubMed by Author: Rodeghiero, F. Tosetto, A. Related Articles in PubMed PubMed Citation ... PubMed ARTICLE Activated Protein C Resistance and Factor V Leiden Mutation Are Independent Risk Factors for Venous Thromboembolism Francesco Rodeghiero, MD and Alberto Tosetto, MD Background: Resistance to activated protein C due to the factor V R506Q (Leiden) mutation is the most common clotting abnormality in patients with venous thromboembolism. Objective: To evaluate the risk for venous thromboembolism associated with the factor V Leiden mutation or with resistance to activated protein C in the general population. Design: Cross-sectional survey. Setting: General community of Vicenza, Italy.

80. Activated Protein C Resistance In Patients With Central Retinal Vein Occlusion - The blood samples were analysed for activated protein c resistance with We have recently shown that activated protein c resistance was increased http://bjo.bmjjournals.com/cgi/content/full/81/10/832

HOME HELP FEEDBACK SUBSCRIPTIONS ... TABLE OF CONTENTS Author Keyword(s) Vol Page [Advanced] This Article Abstract Full Text (PDF) Submit a response ... Citation Map Services Email this link to a friend Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal ... Cited by other online articles PubMed PubMed Citation Articles by Larsson, J Articles by Bauer, B Related Collections Related Article Br J Ophthalmol 832-834 ( October ) Activated protein C resistance in patients with central retinal vein occlusion J Larsson, a A Sellman, b B Bauer a a Department of Ophthalmology, Lund University Hospital, Sweden, b Medical Center Hospital of Helsingborg, Sweden Correspondence to: Dr J Larsson, Department of Ophthalmology, Lund University Hospital, S-221 85 Lund, Sweden. Accepted for publication 26 June 1997 Abstract Top Abstract Introduction Patients and methods Results Discussion References AIM/BACKGROUND A new defect in the anticoagulant system has recently been discovered activated protein C resistance. The frequency of this

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