161. Virtual Children's Hospital: CQQA: Anemia Common Questions, Quick Answers on anemia. anemia in children is most oftencaused by not eating enough iron, but can have other causes. http://www.vh.org/pediatric/patient/pediatrics/cqqa/anemia.html

Pediatrics Common Questions, Quick Answers Anemia (Iron Deficiency) Donna D'Alessandro, M.D. Lindsay Huth, B.A. Peer Review Status: Internally Reviewed Creation Date: February 2002 Last Revision Date: April 2002 Common Questions, Quick Answers What is anemia? What causes anemia? Who can be anemic? ... When should I call the doctor? What is anemia? Anemia is having fewer red blood cells than is normal. Anemics do not have enough red blood cells. What causes anemia? The body uses iron to make red blood cells and to build muscle and strong bones. Red blood cells carry oxygen. Anemia in children is most often caused by not eating enough iron, but can have other causes. Heavy menstrual periods can cause anemia if too much iron is lost in the blood. Who can be anemic? Anyone who does not get enough iron can be anemic. It is especially important for infants and teens to get enough iron. Girls who are menstruating need lots of iron. What are the symptoms of anemia? There may be none. Symptoms are hard to notice unless the anemia is severe. Children may feel weak and get tired easily.

162. B+ Home A charitable organization providing assistance to children with Fanconi anemia within the St. Louis and St. Charles Missouri metro areas. http://www.bpositiveinc.com

B Positive! Bone Marrow Drive Schedule UPDATE B Positive, Inc. P.O. Box 533 Chesterfield, Mo. 63006-0533 (314) 503-BPOS (2767) In December, 2000, two St. Louis area siblings, Matthew Pearl, age 4, and Alexandra Pearl, age 6, were diagnosed with Fanconi Anemia . At the same time, they discovered that each child had B+ blood types and that their only hope for finding a cure was successful bone marrow transplant. The odds of any given individual, other than an immediate family member, being a perfect match is about one in a million. Alex received her bone marrow transplant in May, 2001 and is recovering well... however, no match for Matt has been found yet. We need your help! For more about Alex and Matt, visit their web site at www.alexandmatt.com Welcome to the B Positive, Inc. Web Site! For those who do not know exactly what Fanconi Anemia is, it is a genetic, life-threatening, inherited blood disorder that leads to bone marrow failure. It occurs equally in males and females and is found in all ethnic groups.

163. Anemia On Sympatico / MSN Powered By MediResource anemia is a condition where the number of healthy red blood cells (RBCs) in theblood is less than normal. RBCs transport oxygen throughout the body, http://mediresource.sympatico.ca/channel_disease_detail.asp?channel_id=158&menu_

164. Alex And Matt Pearl Personal stories of two children fighting Fanconi anemia. http://www.alexandmatt.com

PHOTO GALLERY UPDATED!! OVER 500 PHOTOS!! Hi, most of you may know our story; we are Alexandra and Matthew Pearl In December 2000 and January 2001 we were both diagnosed with a rare, life threatening blood disease called Fanconi anemia. It causes bone marrow failure; our bodies cannot make the blood we need to survive. Our only hope is a bone marrow transplant. Cardinals Help With Bone Marrow Drive For Eureka Child Read Press Release Alex has found a perfect match and is recovering from her May 31, 2001 transplant in Cincinnati, Ohio. There is no perfect match for Matthew, in any registry throughout the entire world. We need your help, please consider getting added to the National Bone Marrow Registry through the Pearl Million-Donor March. Refer to the ' How You Can Help ' section.

165. Fanconi Anemia Fanconi anemia (FA), first described in 1927 by a Swiss pediatrician Guido Fanconi, Canadian Fanconi anemia Research Fund / La Fondation Canadienne de http://www.fanconicanada.org/sys-tmpl/fanconianemia/

Home 2006 Toronto Fundraiser 2005 National Anemia Patient Conference Fanconi Anemia ... Donate Now FAMILIES Families Welcome Page PHYSICIANS/RESEARCHERS Sitemap Fanconi Anemia Fanconi Anemia What is FA? Fanconi Anemia (FA), first described in 1927 by a Swiss pediatrician Guido Fanconi, is the most common of the inherited anemias that lead to progressive, severe bone marrow failure, also known as aplastic anemia. The effects of the disease are devastating, leaving patients weak, prone to severe bleeding due to insufficient blood clotting and susceptible to infection. FA is a genetic disorder that occurs equally in males and females and is found in all ethnic groups. Though considered primarily a blood disease, it may affect all systems of the body. A Fanconi Anemia patient often, but not always, has other physical defects detectable at the time of birth ranging from minor to serious. Patients are also at an increased risk for developing leukemia and other cancers. Many children do not survive to adulthood. How is FA diagnosed?

166. Adam Day Learn about a boy who passed away after a fight with Fanconi anemia. Find out how to make a donation to the Fanconi anemia Research Fund. http://www.adam.interactiveinc.com/

Posted by Darla on February 28, 1997 at 03:02:11: Soaring with angels Greetings All: We, again would like to thank everyone for their sincere and heartfelt support. We literally could not have come as far as we did without all of you. We regretfully inform you all that Adam passed away at 9:11 PM, Minnesota time, on February 27, 1997. He was peaceful and pain-free when he passed. His family was with him, as was most of the staff from 4A, who have care for him in the entire duration of our stay here. We will happily inform everyone, though that Adam is now soaring with the angels, watching down protectively on all of us. We're sure he is having go-cart races with Mark Muellen and showing his knife collection to Joey Adamson and fishing with John Dumouchel. We will miss him but we know he is in a better place. May God bless all of you and shower you with goodness, health and many miracles. Thank you again. The Day Family Adam's Uncle Bob went to Minnesota during 1/30-2/3 and took some photos. Adam was in great spirits despite the constant interruptions by a wide variety of physicians and assistants. Go to: http://www.geocities.com/Heartland/Hills/2219/

167. Biblioteca De Salud: Anemia Falciforme Translate this page La anemia falciforme es una enfermedad hereditaria de los glóbulos rojos. Los síntomas de la anemia falciforme son causados por una hemoglobina anormal. http://www.nacersano.org/centro/9388_9967.asp

18 de septiembre de 2005 Antes de Quedar Embarazada Durante su Embarazo Complicaciones El Parto ... Las Familias en la NICU Biblioteca de Salud Anemia Falciforme Sickle Cell Disease Association of America 16 S. Calvert Street, Suite 600 Baltimore MD 21202 www.sicklecelldisease.org Sickle Cell Information Center Box 109 Grady Memorial Hospital 80 Jessie Hill Jr. Drive S.E. Atlanta GA 30303 www.scinfo.org National Heart, Lung and Blood Institute Information Center P.O. Box 30105 Bethesda MD 20824-0105 www.nhlbi.nih.gov Referencias 2. National Heart, Lung, and Blood Institute. Sickle Cell Anemia. Consultado 15/4/04, www.nhlbi.nih.gov. Biblioteca de Salud Embarazo Ácido Fólico El Bajo Peso al Nacer El Embarazo Después de los 35 Años El Embarazo en las Adolescentes ... Un Buen Estado Físico para los Dos Infecciones/Enfermedades Durante el Embarazo Anormalidades del Líquido Amniótico Condiciones de la Placenta El VIH y el SIDA en el Embarazo Herpes Genital ... Trastornos Hepáticos durante el Embarazo Pruebas Diagnósticas Amniocentesis Análisis de la Sangre Materna Muestra del Villus Coriónico (CVS) Ultrasonido Alcohol, Drogas y Medicamentos Durante el Embarazo Accutane El Consumo de Alcohol Durante el Embarazo El consumo de drogas ílicitas durante el embarazo El Fumar Durante el Embarazo ... Talidomida Pérdida del Embarazo Aborto Espontáneo Embarazos Ectópicos y Molares Muerte del Feto Muerte del Neonato Defectos de Nacimiento y Condiciones Genéticas Acondroplasia Anemia Falciforme Anomalías Cromosómicas Defectos Cardíacos Congénitos ... Talasemia Para Recién Nacidos

168. Vitamin B12 Deficiency / Pernicious Anemia: Patient Information: Arizona Telemed Therefore vitamin B12 deficiency results in anemia, an RBC deficiency limitingthe amount Vitamin B12 deficiency is usually caused by pernicious anemia, http://www.telemedicine.arizona.edu/patient_info/benign_disorders/disorders/vita

Patient Information Resource: Benign Hematologic (Blood) Disorders A collaborative project of the Arizona Telemedicine Program , the Arizona Health Sciences Library and the Arizona Cancer Center See: Vitamin B Deficiency: Pernicious Anemia Vitamin B deficiency results in anemia, an RBC deficiency limiting the amount of oxygen in the tissues. Vitamin B deficiency is usually caused by pernicious anemia, a condition in which the digestive system is altered and cannot absorb vitamin B systemic lupus erythematosus , myxedema), or drug action. Among the drugs that may inhibit absorption are histamine blockers, such as cimetidine (Tagamet) and ranitidine (Zantac). Multiple symptoms result from malabsorption of vitamin B . Anemia, mentioned above, is associated with weakness, pallor, elevated heart rate, and elevated breathing rate (see Anemia ). In addition, the patient may be especially prone to infection and may bruise or bleed easily (see Neutropenia and Thrombocytopenia ). Other symptoms include sore tongue, numbness, and tingling.

169. ClinicalTrials.gov - Information On Clinical Trials And Human Research Studies: anemia, Refractory, with Excess of Blasts (43 recruiting studies). 14. anemia,Sickle Cell (31 recruiting studies). 15. Antithrombin III Deficiency (1 http://www.clinicaltrials.gov/ct/screen/BrowseAny?path=/browse/by-condition/hier

170. Having Patience When You Are The Patient! A positive survival story. http://www.geocities.com/hotsprings/villa/4803

My name is Shari and I am an Aplastic Anemia Survivor. I am just beginning to build this page, so please be patient! In the mean time, please feel free to e-mail me at MIRACL2100@aol.com Thank you! SOMETHING ABOUT ME!!! I am a 33 year old female in remission with Aplastic Anemia. I was diagnosed in November of 1985. I was treated with over 200 blood transfusions(red cells and platelets), prednisone(steroids), ATG(Anti-Thymocyte Globulin), then, eventually had a splenectomy(removal of the spleen) in September of 1986. I was told I was in remission in the Spring of 1987. I can't ever be told that I'm cured, because, I have never had a bone marrow transplant. A bone marrow transplant is the only known CURE. Unfortunately, a perfect match could not be found for me. When I was diagnosed, my white count didn't exist at all. My hemoglobin was 4.0, and my platelet count was only 500(normal is 150,000-350,000). Therefore, my immune system was "shot". I was open to get all kinds of infections. I was bruised all over, had terrible migraines, and low blood pressure. The doctors even had to stop my menstrual cycle so I wouldn't bleed the little blood that I had left. You may be thinking, well, what's the miracle!! Well, that was all of the negative information. Now, for the positive: I've been in remission for 10 years now; I went on to finish college(I was a junior when I got sick), and, I got married. I didn't know if I'd make it through the Aplastic Anemia, and, not only have I survived the illness, I brought a new life into this world! On March 3, 1995, I delivered a healthy beautiful girl named Alexa. Her middle name is Faythe(for all the faith I had to get through the illness). I had the child that I never thought I could have, due to all of the medications I had taken.

171. Equine Infectious Anemia The equine infectious anemia virus (EIAV) is categorized as a retrovirus itcontains genetic RNA material, which it uses to produce DNA. http://www.aphis.usda.gov/vs/nahps/equine/eia/

UMR (PDF) Fact Sheet (PDF) EIA-1996 (PDF) EIA Static Maps ... Other Equine Facts EIA is a viral disease of members of the horse family. The equine infectious anemia virus (EIAV) is categorized as a retrovirus: it contains genetic RNA material, which it uses to produce DNA. This DNA is then incorporated into the genetic makeup of infected cells. Identified in France in 1843 and first tentatively diagnosed in the United States in 1888, EIA has commanded a great deal of attention over the years. There is no vaccine or treatment for the disease. It is often difficult to differentiate from other fever-producing diseases, including anthrax, influenza, and equine encephalitis.

172. Ilir's Progress With Aplastic Anemia Includes information about aplastic anemia and Ilir's progress with this disease. http://www.geocities.com/ilir_status/

Ilir Kullolli On this page About Ilir Kullolli Aplastic Anemia Blood elements ... Links On this site Pictures of Ilir Discussion List Recent Pictures. New!!! This page was last updated on: July 5, 2001 Ilir Kullolli is an International Student from Albania. He was attending Cerro Coso Community College in Ridgecrest, California when he was diagnosed with Aplastic Anemia. He was hospitalized on November 17, 1999. After getting better he transferred to Wentworth Institute of Technology in Boston, Massachusetts and graduated with a Bachelor of Science in Computer Engineering in December 2003. Now he's doing very well and he seems to be sick-free. Aplastic Anemia: An injury to the bone marrow; specifically the stem cells. Stem cells make red and white cells and platelets. The injury may be genetic, caused by viruses, drugs or other toxins. One known cause is an antibiotic used in Europe called Chloramphenocol. People who get this have a 1 in 30,000 chance of contracting blood disorders such as Aplastic Anemia. Ilir received this drug three years ago in Albania when he had appendicitis. The doctor is skeptical of this as a cause because of the time between it and the onset of the disease. [back to index] Blood elements Hemoglobin: Carries oxygen from the lungs to the body and Carbon Dioxide back to the lungs.

173. MEdIC - Health Explorer - Iron Overload MEdIC Aplastic anemia patients who receive repeated blood cell transfussions may develop iron overload. A look at this condition and who needs treatment. http://medic.med.uth.tmc.edu/ptnt/00001041.htm

Iron Overload Aplastic anemia patients who receive repeated blood cell transfussions may develop iron overload. Iron from transfused red cells builds up in the blood and eventually accumulates in the heart, liver, pancreas, and endocrine organs. This excess iron may eventually damage vital organs and cause complications like liver disease, heart disease, and diabetes mellitus. Normal body iron stores are 3-4 grams. Each unit of transfused red cells contains 200-250 mg of iron. Thus, a patient who receives 2 units of blood each month would accumulate approximately 5-6 g of extra iron in one year. Without treatment to remove excess iron, damage to the heart and other organs occurs in patients who have received as few as 100 units of blood, or 20 grams of excess iron. Visible signs of iron overload, such as bronze or slate grey skin pigmentation, don't usually appear until enough iron has accumulated to cause tissue damage. In the United States, the only way to prevent or treat iron overload is with the iron chelating drug deferoxamine (Desferal). Desferal binds excess body iron and promotes its excretion by the kidneys in urine and via the bile in feces. Desferal is administered by subcutaneous or intravenous infusion by a small portable pump about the size of a Walkman. Typically the patient inserts a subcutaneous needle and wears the pump for 9-12 hours each day, usually at night while sleeping. Severely iron overloaded patients may need a continuous infusion through an indwelling central venous catheter. Several studies have demonstrated that regular chelation therapy with Desferal can remove excess body iron, prevent organ damage, and prolong life.

174. Anemia Cases A case study of a 50 year old woman complaining of anorexia and shortness of breath for the past few weeks. http://medocs.ucdavis.edu/IMD/420A/case/anemias/cases/ACase2/AnemiaCase2.html

Anemia Case Studies Case #2 History: A 50 year old woman complains of anorexia and shortness of breath for the past few weeks. She feels well but has lost 30 pounds over the last 6 months. Ten years ago she was treated for anemia with iron pills which she took for only a short time. On exam there is a slight yellow tinge to her skin and sclera, a smooth red tongue, and difficulty with fine motor coordination. CBC: RBC 1.70 million/ul, Hct. 19.3%, Hbg. 6.5 g/dl, MCV 114 fl, RDW 22, WBC 2,000/ul, Retics: 2.6% (44,200/ul). Peripheral Blood Smears: 100x Smear (250 x 350) 100x Smear (440 x 640) 1000x Smear (250 x 350) 1000x Smear (440 x 640) View All Additional Test Results Order Additional Test Results Questions: 1. Explain why this patient is pancytopenic. Answer 2. List other causes of this type of anemia. Answer 3. What test confirms the diagnosis? Answer

175. Perncious An indepth overview of pernicious anemia and links to other informative sites. http://www.geocities.com/Athens/Acropolis/6338/pernicious.html

DRACULA'S Campaign Against Pernicious Anemia WELCOME TO ANOTHER WACKY MEDICAL WEBSITE created by Cynthia Donlan. If you have any questions or just need someone to talk to who understands what you are going through don't hesitate to E-mail me! I don't bite. Also check out my homepage by clicking on the link below. Finally,At the Medical Website I developed, I have information on various other Autoimmune diseases such as Systemic Lupus Erythematosus, Sjogren's Syndrome, Myasthenia Gravis, Diabetes and many many more. Just click on the back button below!!! GOOD EVENING! Hello I am Count Dracula. I have been known for a long time to have an affinity for blood so I have gone to medical school to become a Hematologist. As part of my training we were taught about the various types of anemia. Those conditions both sickened and frightened me. What is a vampire to do if they lose their food source? I need healthy blood from healthy people so that I don't become deficient. This WebPage is brought to you by Blood Brothers, a fraternal order of Transylvanian Vampire Physicians. We hope you find this informative! Stay Healthy and try to be O+ about life! WHAT IS PERNICOUS ANEMIA?

176. InteliHealth: Pernicious Anemia Details about this disorder, what it is, the symptoms, diagnosis, prevention, treatment, prognosis and when to call a doctor. http://www.intelihealth.com/IH/ihtIH/WSCHN000/9339/20862.html

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177. Anaemia Headlines. DNA Down Under. New section on RNA interference as a tool to blockvirus replication. Anaemia. The erythrocyte as seen by the routine http://www.uq.edu.au/vdu/HDUAnaemiaBackground.htm

Virology The Basics Taxonomy Links ... HOME Headlines DNA Down Under New section on RNA interference as a tool to block virus replication. Anaemia The erythrocyte as seen by the routine haematologist is essentially at the final stage of maturation. The "cell" is anucleate, without mitochondria, lysosomes or endoplasmic reticulum (ER), and relies on the Embden-Meyerhof and Heptose Monophosphate pathways to maintain the cell's shape etc. All cells (except for T cells) are produced in haematopoietic tissue called Bone Marrow . In a child, this is found in the tibia. In an adult, bone marrow is found in the anterior/posterior iliac crest, sternum, ribs, vertebrae and skull. The bone marrow houses the most primitive haematological cells - called Stem cells. These cells are pluripotential, which means they are able to differentiate into a range of haematological cell types. The bone marrow is influenced by Erythropoietin produced mostly in the kidneys, but also the liver.

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