61. NOVA Online | Neanderthals On Trial | Tracing Ancestry With MtDNA That is, all people alive today can trace some of their genetic heritage through loops of genetic code known as mitochondrial DNA, or mtDNA for short. http://www.pbs.org/wgbh/nova/neanderthals/mtdna.html

window.self.name = "mtd_parent"; var gaJsHost = (("https:" == document.location.protocol) ? "https://ssl." : "http://www."); document.write(unescape("%3Cscript src='" + gaJsHost + "google-analytics.com/ga.js' type='text/javascript'%3E%3C/script%3E")); Scientists explore humankind's past with mtDNA. Tracing Ancestry with MtDNA By Rick Groleau In 1987, three scientists announced in the journal Nature that they had found a common ancestor to us all, a woman who lived in Africa 200,000 years ago. She was given the name "Eve," which was great for capturing attention, though somewhat misleading, as the name at once brought to mind the biblical Eve, and with it the mistaken notion that the ancestor was the first of our species the woman from whom all humankind descended. The "Eve" in question was actually the most recent common ancestor through matrilineal descent of all humans living today. That is, all people alive today can trace some of their genetic heritage through their mothers back to this one woman. The scientists hypothesized this ancient woman's existence by looking within the cells of living people and analyzing short loops of genetic code known as mitochondrial DNA, or mtDNA for short. In recent years, scientists have used mtDNA to trace the evolution and migration of human species, including when the common ancestor to modern humans and Neanderthals lived though there has been considerable debate over the validity and value of the findings. In reproduction, the nuclear DNA of one parent mixes with the nuclear DNA of the other. MtDNA, on the other hand, passes on from mother to offspring unaltered.

62. ANNALS ONLINE -- Sign In Page mitochondrial GENETIC DEFECT LEADS TO NUCLEAR GENOMIC INSTABILITY . mitochondrial Genetic Polymorphisms and Pancreatic Cancer Risk Cancer Epidemiol. http://www.annalsnyas.org/cgi/content/full/1019/1/260

Search Advanced Search This item requires a subscription to Annals of the New York Academy of Sciences Online. Strategies for Engineered Negligible Senescence: Why Genuine Control of Aging May Be Foreseeable Volume 1019 published June 2004 Ann. N.Y. Acad. Sci. doi: 10.1196/annals.1297.043 New York Academy of Sciences description purchase this volume Mitochondrial Dysfunction Is a Common Phenotype in Aging and Cancer KESHAV K. SINGH To view this item, select one of the options below: Sign In Academy Members Log in at the Academy site Librarians Activate your Institutional Subscription Sign Up Individuals Join the Academy Institutions Subscribe for Full Access Purchase Short-Term Access Pay per Article - You may access this article (from the computer you are currently using) for 1 day for US$15.00 Pay for Admission - You may access all content in Annals of the New York Academy of Sciences Online (from the computer you are currently using) for 24 hours for US$30.00. Regain Access - You can regain access to a recent Pay per Article or Pay for Admission purchase if your access period has not yet expired.

63. 561 570..572 Your browser may not have a PDF reader available. Google recommends visiting our text version of this document. http://www.sciencemag.org/cgi/reprint/312/5773/570.pdf

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